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1 ility of (18)F-florbetaben PET in diagnosing cardiac amyloidosis.
2 ent determinant of myocardial dysfunction in cardiac amyloidosis.
3 ients with symptomatic heart failure from AL cardiac amyloidosis.
4 with worse survival among patients with ATTR cardiac amyloidosis.
5 andard for diagnosis and characterization of cardiac amyloidosis.
6 9m PYP cardiac imaging for detection of ATTR cardiac amyloidosis.
7 ty in patients with amyloid light-chain (AL) cardiac amyloidosis.
8 ility to overcome poor prognosis of advanced cardiac amyloidosis.
9 ned reliably by PSIR and represents advanced cardiac amyloidosis.
10 protinin was found to be useful in detecting cardiac amyloidosis.
11 e curve 0.992, P<0.0001 for identifying ATTR cardiac amyloidosis.
12 endent validation set comprising 41 cases of cardiac amyloidosis.
13 cy of intracardiac thrombosis was present in cardiac amyloidosis.
14 cement CMR was performed in 30 patients with cardiac amyloidosis.
15 n models that predict the log-odds of having cardiac amyloidosis.
16 dial biopsy because of clinical suspicion of cardiac amyloidosis.
17 out the prognostic value of CV-IB in primary cardiac amyloidosis.
18 There is no muscle blood vessel or cardiac amyloidosis.
19 as both sporadic inclusion body myositis and cardiac amyloidosis.
20 be a useful predictor of clinical outcome in cardiac amyloidosis.
21 edictor of clinical outcome in patients with cardiac amyloidosis.
22 myocardial performance in the assessment of cardiac amyloidosis.
24 TTR cardiac amyloidosis and 50 with non-ATTR cardiac amyloidosis [34 with AL amyloidosis and 16 with
25 A total of 116 autopsy or explanted cases of cardiac amyloidosis (55 AL and 61 other type) were ident
29 P imaging of 171 participants (121 with ATTR cardiac amyloidosis and 50 with non-ATTR cardiac amyloid
30 ambda II germ-line genes was associated with cardiac amyloidosis and affected survival adversely.
31 in which 229 participants were evaluated for cardiac amyloidosis and also underwent Tc 99m PYP cardia
32 78 whites over 60 years of age with isolated cardiac amyloidosis and from two control groups (228 cas
34 ardiac imaging as a diagnostic tool for ATTR cardiac amyloidosis and its association with survival in
35 P cardiac imaging distinguishes AL from ATTR cardiac amyloidosis and may be a simple, widely availabl
36 espective of genotype) from patients with AL cardiac amyloidosis and patients with nonamyloid heart f
38 approaches to the diagnosis and treatment of cardiac amyloidosis are discussed, with particular refer
39 d 216 patients with histologically confirmed cardiac amyloidosis at a single center with electrocardi
41 hese conditions, but wild-type transthyretin cardiac amyloidosis (ATTRwt) is increasingly being diagn
43 is a reference standard for the diagnosis of cardiac amyloidosis, but its potential for stratifying r
44 ) cardiac imaging noninvasively detects ATTR cardiac amyloidosis, but the accuracy of this technique
45 myloid deposition in the heart, diagnosis of cardiac amyloidosis (CA) based on these conventional tec
47 meters has been suggested for distinguishing cardiac amyloidosis (CA) from other causes of myocardial
48 unoglobulin amyloid light-chain (AL)-related cardiac amyloidosis (CA) has a worse prognosis than eith
50 atic approach to the evaluation of suspected cardiac amyloidosis can impact the prognosis of patients
52 Clinical clues to promote recognition of cardiac amyloidosis, cardiac sarcoidosis, and cardiac he
53 which tailored interventions are available: cardiac amyloidosis, cardiac sarcoidosis, and cardiac he
56 ptal thickness is >1.98 cm, the diagnosis of cardiac amyloidosis could be made with a sensitivity of
59 l presentation, diagnosis, and management of cardiac amyloidosis, focusing on recent important develo
61 ognosis and treatment of the 2 main types of cardiac amyloidosis, immunoglobulin light chain (AL) and
62 magnetic resonance findings were of definite cardiac amyloidosis in 2, but could be explained solely
63 ging have resulted in greater recognition of cardiac amyloidosis in everyday clinical practice, but t
64 erformed at 3 academic specialty centers for cardiac amyloidosis in the United States in which 229 pa
66 ty for differentiation of patients with ATTR cardiac amyloidosis (irrespective of genotype) from pati
74 etin cardiac amyloidosis (also known as ATTR cardiac amyloidosis) is an increasingly recognized cause
79 e analyzed from 1217 patients with suspected cardiac amyloidosis referred for evaluation in specialis
80 ession analyses among participants with ATTR cardiac amyloidosis showed that an H/CL ratio of 1.6 or
82 r whom there is a high clinical suspicion of cardiac amyloidosis, Tc 99m PYP may be of diagnostic and
84 s a major cause of cardiac dysfunction in AL cardiac amyloidosis, we have previously shown that amylo
85 ture and function and features suggestive of cardiac amyloidosis were assessed in participants who un
86 le method for identifying subjects with ATTR cardiac amyloidosis, which should be studied in a larger
88 ivity and 92% specificity for detecting ATTR cardiac amyloidosis with an area under the curve of 0.96
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