ライフサイエンスコーパス: cardiac cycle

1 stole (5.1 mm +/- 1.8) (32% variation during cardiac cycle).

2 during atrial contraction (40% variation per cardiac cycle).

3 rief muscle contractions ( approximately 4-7 cardiac cycles).

4 ponents (PCs) representing breathing and the cardiac cycle.

5 equally incremented time points through the cardiac cycle.

6 n be imposed during a specified phase of the cardiac cycle.

7 the period of minimal cardiac motion in the cardiac cycle.

8 hase) or last third (diastolic phase) of the cardiac cycle.

9 ons of influx vs. efflux via NCX during each cardiac cycle.

10 d in the plane of acquisition throughout the cardiac cycle.

11 wall during the predetermined phases of the cardiac cycle.

12 es in length and force that occur during the cardiac cycle.

13 culation eddies oscillate in size during the cardiac cycle.

14 cardial isopotential mapping during a single cardiac cycle.

15 values are quantified, for all frames in the cardiac cycle.

16 ty of 30 miles per hour and was timed to the cardiac cycle.

17 haracterizing contractile changes during the cardiac cycle.

18 uced by impacts at any other time during the cardiac cycle.

19 delivered during the vulnerable phase of the cardiac cycle.

20 appearance of the myocardium throughout the cardiac cycle.

21 ver the entire epicardial surface during the cardiac cycle.

22 delivered during the vulnerable phase of the cardiac cycle.

23 8 versus 3.6 +/- 0.7 cm2, P = NS) during the cardiac cycle.

24 netium-99m sestamibi, using eight frames per cardiac cycle.

25 essment of phasic events associated with the cardiac cycle.

26 changes in flow over different phases of the cardiac cycle.

27 were monitored during balloon deflation and cardiac cycle.

28 ), with dynamics severalfold slower than the cardiac cycle.

29 cast short-lived LLSTs in human blood during cardiac cycle.

30 perties of the aortic annulus throughout the cardiac cycle.

31 RVOT diameters (RVOTD max and min) during a cardiac cycle.

32 was reconstructed in 10% increments over the cardiac cycle.

33 fails to demonstrate similar modulations by cardiac cycle.

34 trocardiogram at specific time points in the cardiac cycle.

35 re exposed to shear/fluid forces during each cardiac cycle.

36 ges of luminal Ca(2+) that occur through the cardiac cycle.

37 arteriolar endothelium throughout the entire cardiac cycle.

38 and does not occur on the time scale of the cardiac cycle.

39 direction observed during each phase of the cardiac cycle.

40 unaffected by calcium during the course of a cardiac cycle.

41 The TA was traced during a cardiac cycle.

42 data at &75% of the R-R interval during the cardiac cycle.

43 n with sympathetic nerve activity and/or the cardiac cycle.

44 the channel during the resting phase of the cardiac cycle.

45 red with one respiratory cycle performed per cardiac cycle.

46 ergy FLASH frames destroyed bubbles every 15 cardiac cycles.

47 -per-second (FPS) capable of resolving mouse cardiac cycles.

48 ingle image from data acquired over multiple cardiac cycles.

49 ary views only with one frame every multiple cardiac cycles.

50 he transmission rate was once every multiple cardiac cycles.

51 Measurements were averaged over 10 cardiac cycles.

52 table biotic/abiotic interface during normal cardiac cycles.

53 observed contracting during one or multiple cardiac cycles.

54 orm from skin displacement images during the cardiac cycle.).

55 assessments were obtained in three different cardiac cycles (0.92-0.98), rather than in one (0.79-0.9

56 pattern in cardiogram waveform over a single cardiac cycle (~200 ms) of a mouse, which has not been o

57 ges with acquisition window that covered two cardiac cycles (acquisition time, 10-12 min; temporal re

58 and sustain vasodilatation for several more cardiac cycles after contraction.

59 of each marker every 16.7 ms throughout the cardiac cycle, allowing calculation of three-dimensional

60 ration to simulate in vivo conditions of the cardiac cycle and found that I-O forces decrease with in

61 of EPSPs and spike potentials locked to the cardiac cycle and occurring at the end of diastole.

62 e WT-Parv sequesters Ca(2+) too early in the cardiac cycle and prematurely truncates sarcomere shorte

63 vely synchronized, enabling compensation for cardiac cycle and respiratory phase.

64 Flow velocity was measured over the whole cardiac cycle and the wave-free period.

65 o estimate all cardiac time intervals from 1 cardiac cycle and thereby obtain the myocardial performa

66 12 min; temporal resolution, 60 msec) or one cardiac cycle and time-of-flight (TOF) MR angiographic i

67 area of each burst were determined for each cardiac cycle and were placed into 3 mmHg intervals of d

68 ted length is maintained through consecutive cardiac cycles and that hysteresis is minimal.

69 ndividual characteristic ("core") pattern of cardiac cycles and then tracks the changes in the patter

70 e (i.e. number of sympathetic bursts per 100 cardiac cycles) and the area of each burst were determin

71 motion-artifact-free imaging throughout the cardiac cycle, and a fluorescent membrane staining proto

72 tically track the endocardium throughout the cardiac cycle, and show myocardial perfusion defects whe

73 ally induced vasodilatation peaks within 1-2 cardiac cycles, and thus is dissociated from the tempora

74 ating or reducing the problems caused by the cardiac cycle are discussed, including electrocardiogram

75 d-point of the diagram (T50) at which 50% of cardiac cycles are associated with bursts, was inversely

76 mensional blood-flow patterns throughout the cardiac cycle at the widest diameter of the Fontan pathw

77 However, the need to acquire multiple cardiac cycles at varying loads limits its applicability

78 Local blood flow and changes over the cardiac cycle before and after application of atropine w

79 ker coordinates were obtained throughout the cardiac cycle before and during each intervention.

80 The change in MAA throughout the cardiac cycle before ischemia was 17+/-4% in control ani

81 lumes were thus reconstructed throughout the cardiac cycle by combining transverse cardiac image sect

82 fy the role of isovolumic intervals during a cardiac cycle by in vivo visualization of left ventricul

83 ity in muscle nerves are phase-locked to the cardiac cycle by the sinoaortic baroreflexes.

84 g into account valve plane motion during the cardiac cycle by using Fourier-guided, dual-ROI analysis

85 be reliably estimated in humans from single cardiac cycles by a new method that has a potential for

86 ere acquired from three independent complete cardiac cycles by using a 15L8-MHz US transducer.

87 nce (Y: 26 +/- 8 vs. A: 50 +/- 7 bursts (100 cardiac cycles (CC))(-1) , P </= 0.01) were greater in t

88 es) analysis showed that 7 of 14 neurons had cardiac-cycle-correlated rhythms.

89 baseline buffer perfusion, maximal dP/dt per cardiac cycle decreased on average by 30.4%, 40.9%, and

90 es and the plots of dV/dt values through the cardiac cycle demonstrated more gradual developments of

91 had a significant reduction in MRI-assessed cardiac cycle-dependent change in aortic area and disten

92 lder patients with isolated DHF have reduced cardiac cycle-dependent changes in proximal thoracic aor

93 The goal of this study was to determine if cardiac cycle-dependent changes in proximal thoracic aor

94 We hypothesized that a reduction in cardiac cycle-dependent changes in thoracic aortic area

95 ant changes in mean thickness throughout the cardiac cycle, despite the inherent unsteadiness of the

96 aging at pulsing intervals (PIs) of <1 to 30 cardiac cycles during an intravenous infusion of microbu

97 Cardiac cycle dynamics reflect underlying physiological

98 This flow occurred during most of the cardiac cycle, except for 19.6 +/- 2.3% of the cycle whe

99 al regularities induced a phase shift of the cardiac cycle exclusively in the MCS group.

100 racterize beat-by-beat changes in FVC for 15 cardiac cycles following each MSNA burst and a peak resp

101 terize changes in left and right ventricular cardiac cycles following induction of experimental, stre

102 imate the optimal duration and timing in the cardiac cycle for coronary MR angiography.

103 f local myocardial count variations over the cardiac cycle for each patient, and then unpaired t test

104 were gated to the vulnerable portion of the cardiac cycle for the induction of VF.

105 ional imaging of the heart tube over various cardiac cycles for the measurement of cardiac structural

106 erves) was intact, rhythms correlated to the cardiac cycle frequency were found in 20/34 (59 %) of un

107 ion of LV endocardial surface throughout the cardiac cycle, from which global and regional LV volume

108 f the mouse heart and for the animal's rapid cardiac cycle has proven to be a formidable challenge, r

109 cardial perfusion images obtained from whole cardiac cycles have lower myocardial intensity and great

110 d permits analysis of events during a single cardiac cycle; however, at present, RT3-D imaging has po

111 at 14 time points evenly spaced through the cardiac cycle in 10 volunteers and eight patients with C

112 istent through all twelve time points of the cardiac cycle in all five experimental groups and agreed

113 tricle from 3D MR-tagged images for the full cardiac cycle in dogs with cardiac failure and a left bu

114 In vivo DT-CMR was acquired throughout the cardiac cycle in healthy swine, followed by in situ and

115 b) to measure the ASA and VSA throughout the cardiac cycle in healthy volunteers using cardiovascular

116 on mitral valve (MV) deformation during the cardiac cycle in man.

117 r distances remained constant throughout the cardiac cycle in normal hearts, during ischemia, and aft

118 changes in the left ventricle throughout the cardiac cycle in normal Wistar Kyoto rats (WKY) and also

119 ed to measure arterial distension during the cardiac cycle in the brachial arteries of 361 children f

120 trasound transmission rate was one frame per cardiac cycle; in secondary and tertiary views, the tran

121 eformations during the various phases of the cardiac cycle, including aortoventricular and sinotubula

122 The cardiac cycle influences the blood flow velocity profile

123 ses are interrupted (delivered only once per cardiac cycle) instead of conventional 25- to 30-Hz fram

124 We increased information by dividing the cardiac cycle into fewer bins, excluding cycles without

125 The cardiac cycle is composed of an anterograde beat in alte

126 NCX function during the cardiac cycle is determined by intracellular [Ca] and [N

127 The average blood velocity throughout the cardiac cycle is strongly correlated with pulmonary pres

128 resolved SL MR angiographic imaging over two cardiac cycles is a reliable clinical tool for cerebral

129 ements included blood pressure, body height, cardiac cycle length, carotid to femoral artery pulse wa

130 chords, which remain taut during the entire cardiac cycle, limit the motion of the anterior mitral l

131 A digital echocardiogram (often several one cardiac cycle loops) can be stored at one site and forwa

132 When the information for the cardiac cycle modulations and other consciousness-relate

133 rgely to the immediate vasodilatation (first cardiac cycle) observed in response to a brief, single c

134 diminishes minimally or not at all during 3 cardiac cycles of the washout phase); and TMP grade 3 in

135 shout (blush is minimally persistent after 3 cardiac cycles of washout).

136 be the effects of ectopy and slow changes in cardiac cycles on the disturbances in the pattern.

137 ith time-resolved SL MR angiography over one cardiac cycle (P < .001) and TOF MR angiography (P < .00

138 y wedge pressure at any point throughout the cardiac cycle (P=0.290).

139 Fifty per cent of IC neurons exhibited cardiac cycle periodicity, with activity occurring prima

140 eart rate (HR) and HR variability (HRV), and cardiac cycle phase shifts triggered by the processing o

141 AVA) measured during the ejection phase of a cardiac cycle predicts the rate of hemodynamic progressi

142 ly applying this test to activity during the cardiac cycle proved ineffective because subjects-by-tre

143 resolved SL MR angiographic imaging over two cardiac cycles provided a median diagnostic confidence i

144 during tachycardia (mean trend, +15 ms/96.8 cardiac cycles; r(2)=0.92).

145 find that synchronizing imaging scans to the cardiac cycle reduces motion artifacts, significantly im

146 tor neurones in the RVL, identified by their cardiac cycle-related probability of discharge, by the d

147 ow is shifted into the latter portion of the cardiac cycle, resulting in apparent prolongation of sys

148 data obtained at multiple time points in the cardiac cycle show that the glycocalyx remains hydrodyna

149 filament and undergo a powerstroke once the cardiac cycle starts.

150 n, and protein synthesis is regulated by the cardiac cycle, suggesting that mechanotransduction at th

151 ith time-resolved SL MR angiography over two cardiac cycles than with time-resolved SL MR angiography

152 uron showed hyperpolarizations locked to the cardiac cycle that started during late diastole and ende

153 During the cardiac cycle, the angle alpha between mitral annulus an

154 During the cardiac cycle, the release of Ca(2+) from the sarcoplasm

155 During the cardiac cycle, the value of [Na(+)]sm is constrained wit

156 d relaxation (expressed as percentage of the cardiac cycle to adjust for differences in heart rate) s

157 cing Na+) within the time course of a single cardiac cycle to avoid slow secondary effects.

158 somatosensory stimuli is altered across the cardiac cycle to evoke differential changes in bodily st

159 tes are subjected to shear stress during the cardiac cycle under physiological or pathological condit

160 atients showed minor abnormalities of the RV cardiac cycle unrelated to QRSd.

161 nd the stimulation mechanisms throughout the cardiac cycle using the pinwheel protocol, and the resul

162 13 ms), with typically 12 time frames in the cardiac cycle, using a short echo time (5 ms) multislice

163 One or more S-L cardiac cycles, usually the result of a ventricular bige

164 The rest period for coronary arteries in the cardiac cycle varies substantially from patient to patie

165 Change in blood velocity over the cardiac cycle was determined to be approximately 27%.

166 Pulsatility over the cardiac cycle was evaluated by sending a series of pulse

167 at systole and stretch variation during one cardiac cycle was greater in symptomatic group than thos

168 The cardiac cycle was not sensitive to the processing of loc

169 hese data, epicardial deformation during the cardiac cycle was quantified by computing finite strain

170 Annular contraction during the cardiac cycle was similar in the 2 groups (xenograft 9.2

171 Time intervals of the RV cardiac cycle were measured from Doppler recordings.

172 their variation during balloon deflation and cardiac cycles were measured to describe the 3D strut, c

173 (25 to 130 microm), in focus throughout the cardiac cycle, were recorded after infusion of acridine

174 le-shape depth) were measured throughout the cardiac cycle with dedicated quantification software.

175 assess isovolumic PEC and PFR phases of the cardiac cycle with high-frame-rate cine MR images, and P

176 l arteries changed systematically during the cardiac cycle, with the flattest profile occurring durin