ライフサイエンスコーパス: cardiac index

1 eGFR had more women, nonsmokers, and a lower cardiac index.

2 ed with mean pulmonary arterial pressure and cardiac index.

3 with higher right atrial pressure and lower cardiac index.

4 d gas exchange but at the expense of a lower cardiac index.

5 d systemic vascular resistance, and elevated cardiac index.

6 duced systemic vasodilation and elevated the cardiac index.

7 CHF patients and are inversely dependent on cardiac index.

8 pulmonary arterial occlusion pressures, and cardiac index.

9 nses leading to a significant improvement in cardiac index.

10 se pressure, but only angiotensin II reduced cardiac index.

11 idence of hypertension, diabetes and a lower cardiac index.

12 ompared with controls, which correlated with cardiac index.

13 ndex >/=35 mL/m(2) (P<0.01), despite similar cardiac index.

14 0.016) compared with individuals with normal cardiac index.

15 e and negatively with 6-minute walk test and cardiac index.

16 significant association between BMI and both cardiac index (0.003 L/min/m(2); 95% CI, -0.008-0.014; p

17 stance (114 m; 95% CI, 67, 160; p = 0.0002), cardiac index (0.3 L x min(-1) x m(-2); 95% CI, 0.1, 0.4

18 59 m; 95% CI, 29, 89; n = 12, p = 0.001) and cardiac index (0.3; 95% CI, 0.1, 0.4; p = 0.008).

19 gnificantly different from placebo (P=0.10), cardiac index (0.4 L.min(-1).m(-2); 95% confidence inter

20 rease in heart rate (+33 vs. +48 beats/min), cardiac index (+0.6 vs. +1.5 l/m(2)), systolic blood pre

21 TTM33 was associated with decreased cardiac index (-0.4 L/min per m(2); P(group) <0.0001), d

22 In the study cohort (age 57 +/- 14 years, cardiac index 1.9 +/- 0.6 l/min/m(2), left ventricular e

23 /second versus 92.8 +/- 22.7 cm/second), and cardiac index (1.06 +/- 0.30 ml/minute/g versus 0.67 +/-

24 s [WU] vs. 2.1 +/- 1.1 WU; p = 0.017), lower cardiac index (1.6 +/- 0.4 l/min/m(2) vs. 2.7 +/- 0.7 l/

25 s/min vs. 5.9 +/- 11.5 beats/min, p = 0.04), cardiac index (1.6 +/- 1.0 l/min/m(2) vs. 0.8 +/- 1.1 l/

26 e patients had dilated ventricles with a low cardiac index (1.9 +/- 0.6 L/min/m) and high pulmonary a

27 ciated with greater RV systolic dysfunction (cardiac index, 1.9 vs. 2.7 L/min/m2; RV % area change, 2

28 spite similar levels of cardiac dysfunction (cardiac index 2.2 and 2.1 liters/minute/m(2), respective

29 tion: 37.6+/-12.8% versus 29.0+/-9%: P<0.05; cardiac index 2.7+/-0.9 versus 2.2+/-0.4 L.min(-1).m(-2)

30 ressive, systolic function mainly preserved (cardiac index 2.8+/-0.6 [1.9-3.9] L/min per m(2)), and d

31 6 [8.3] vs 12.9 [8.3] Wood units), and lower cardiac index (2.11 [0.69] vs 2.51 [0.92] L/min per m(2)

32 Hg vs. 45.7 +/- 9.4 mm Hg, respectively) and cardiac index (2.3 +/- 0.8 l/min/m(2) vs. 2.2 +/- 0.8 l/

33 , 42 +/- 6 mm; ejection fraction, 65 +/- 8%; cardiac index, 2.6 +/- 0.8 L/min per m(2)), patients had

34 - 8 mm Hg vs. 20 +/- 7 mm Hg, p = 0.02, Fick cardiac index: 2.2 l/min/m(2) [interquartile range: 1.87

35 Compared to placebo, levosimendan increased cardiac index (22%), stroke volume index (15%), and hear

36 ed (mean arterial pressure 70 [65-77] mm Hg, cardiac index 3.3 [2.7-4.0] L/min.m, and SvO2 68.3 [62.8

37 e, prone positioning significantly increased cardiac index (3.0 [2.3-3.5] to 3.6 [3.2-4.4] L/min/m(2)

38 nts with microvascular dysfunction had lower cardiac index (3.1+/-0.7 versus 3.5+/-0.7 L/min per m(2)

39 335 dyne . s . cm(-5) , P < 0.05) and higher cardiac index (3.3 +/- 0.9 versus 2.8 +/- 0.4 L/min/m(2)

40 ssure (median, 11 vs 15 cm H(2)O; P < .001), cardiac index (3.4 vs 3.8 L/min/m(2); P = .001), and glo

41 0.07 vs. 0.29 +/- 0.05, p < .05) and higher cardiac index (4.8 +/- 0.4 vs. 3.4 +/- 0.2 L.min-1.m-2,

42 Hepa Wash also enhanced cardiac index (4.94+/-0.32 vs. 3.36+/-0.25 l/min/m2, p=0

43 sitivity, 68% specificity), and by change in cardiac index (69% sensitivity, 59% specificity), change

44 2] vs 33 mm Hg [95% CI, 30-36], p < 0.0001), cardiac index (76 mL/min/kg [95% CI, 63-91] vs 47 mL/min

45 lung water >/= 10% was predicted by baseline cardiac index (77% sensitivity, 98% specificity) and pul

46 The increased cardiac index accounted for the increased cerebral and p

47 ure variation and the change in stroke index/cardiac index after a fluid challenge.

48 me variation and the change in stroke volume/cardiac index after a fluid or positive end-expiratory p

49 Left ventricular (LV) dysfunction with a low cardiac index after successful CPR contributes to early

50 < 0.05) were Crawford type 2, acuity, SFDN, cardiac index after unclamping, mean arterial pressure d

51 ment, significantly greater increases in the cardiac index and decreases in PCWP were observed with b

52 HES improved liver microcirculation, cardiac index and DO(2)-I, but significantly increased I

53 With the exception of cardiac index and indexed systemic vascular resistance,

54 Microsphere-determined cardiac index and left ventricular myocardial blood flow

55 ours of reperfusion and resulted in elevated cardiac index and local cerebral blood flow compared wit

56 diac energy-dependent workload with improved cardiac index and lower vascular resistance, 2) upgraded

57 (1))apelin-13 (30 to 300 nmol/min) increased cardiac index and lowered mean arterial pressure and per

58 trahydrobiopterin prevented the decreases in cardiac index and mean arterial pressure.

59 jury, there is a high prior probability that cardiac index and mixed venous oxygen saturation are nor

60 vascular resistance index, and PVRI, whereas cardiac index and mixed venous oxygen saturation remaine

61 lar tone and oxygen extraction, whereas both cardiac index and oxygen delivery decreased for patients

62 ase in vascular tone and a reduction in both cardiac index and oxygen delivery.

63 ic left ventricular dysfunction and improved cardiac index and pulmonary and systemic vascular resist

64 pressure, and secondary end points comprised cardiac index and pulmonary arterial pressure at rest an

65 Baseline cardiac index and pulmonary blood volume index were high

66 ed with higher pulmonary pressures and lower cardiac index and pulmonary capacitance (all P<0.05).

67 tion improved cardiac performance, improving cardiac index and pulmonary capillary wedge pressure, bu

68 The cardiac index and pulmonary vascular resistance did not

69 ry arterial pressure, right atrial pressure, cardiac index and pulmonary vascular resistance.

70 Cardiac index and QRS duration were improved by BDOO com

71 Hemodynamic variables such as cardiac index and right atrial pressure have consistentl

72 Cardiac index and stroke volume index were lower in the

73 cular volume indexes or cardiac performance (cardiac index and stroke volume index).

74 is also associated with an acute decrease in cardiac index and systemic O(2) delivery.

75 total Hb, mean pulmonary arterial pressure, cardiac index and systemic oxygen delivery, increases in

76 s pressure and change in stroke volume index/cardiac index and the percentage of fluid responders.

77 disease were excluded, the relation between cardiac index and total brain volume remained (P=0.02).

78 Pulse contour analysis-derived cardiac index and velocity-time integral were measured d

79 worse systolic (lower EF, stroke volume, and cardiac index) and diastolic (shorter deceleration time

80 by increases of pulmonary arterial pressure, cardiac index, and blood oxygen extraction above baselin

81 ressure, pulmonary capillary wedge pressure, cardiac index, and estimated glomerular filtration rates

82 l/min) renal function, despite similar PCWP, cardiac index, and LV ejection fraction.

83 class and improvement in exercise tolerance, cardiac index, and mean pulmonary artery pressure.

84 ved measurements, mean right atrial pressure cardiac index, and mixed venous oxygen saturations.

85 unctional class, mean right atrial pressure, cardiac index, and mixed venous oxygen saturations.

86 Heart rate, mean arterial pressure, cardiac index, and MPAP were not affected by sitaxsentan

87 ially decreased coronary perfusion pressure, cardiac index, and myocardial blood flow.

88 e systemic vascular resistance index (SVRI), cardiac index, and myocardial performance at a targeted

89 300 mg: +33 m, p < 0.01); functional class, cardiac index, and pulmonary vascular resistance also im

90 moderate-severe tricuspid regurgitation, low cardiac index, and raised right atrial pressure were ass

91 ial effusion, pulmonary vascular resistance, cardiac index, and right atrial pressure may be used to

92 ificantly augmented systolic blood pressure, cardiac index, and stroke volume index in this pediatric

93 Systolic blood pressure, cardiac index, and stroke volume index were significantl

94 temperature, arterial pressure, heart rate, cardiac index, and stroke volume index) and metabolic da

95 lent improvement in systolic blood pressure, cardiac index, and stroke volume index, when the ITD alo

96 ors including pulmonary vascular resistance, cardiac index, and vasoreactivity.

97 rger left ventricular volumes, more impaired cardiac indexes, and the presence of cardiac resynchroni

98 evated intracardiac filling pressures, lower cardiac index, anemia, hypoalbuminemia, hyperbilirubinem

99 tment groups did not significantly differ in cardiac index, arrhythmias, peak lactate, inotropic scor

100 Blood samples for cardiac index (arterial and venous) and tissue Doppler (

101 in, with nonpulsatile pump support, a normal cardiac index as well as reinstitution of the Frank-Star

102 Median cardiac index at 6 months was significantly lower than b

103 Median cardiac index at beginning of the treatment was 5.05 L/m

104 subjects and controls, with lower VO(2) and cardiac index at peak, and more severe dyspnea and fatig

105 The cardiac index averaged 0.73 +/- 0.19 mL/min/g and the st

106 ltiple logistic regression analysis baseline cardiac index, baseline pulmonary blood volume index, th

107 is associated with increased SVRI and lower cardiac index because of lower heart rate with unaffecte

108 No differences were observed for cardiac index before and after the operation; at the arr

109 ascular resistance and increased heart rate, cardiac index, blood urea nitrogen (BUN) level, creatini

110 ased heart rate, mean arterial pressure, and cardiac index but decreased tissue perfusion indicated b

111 EOV was associated with slightly lower mean cardiac index but other invasive hemodynamic variables w

112 olume expansion did not significantly change cardiac index, but the oxygen delivery decreased due to

113 Nitric oxide inhalation increased the cardiac index by 24 +/- 11% and the stroke volume index

114 DITPA increased cardiac index (by 18%) and decreased systemic vascular r

115 cardiac magnetic resonance imaging-assessed cardiac index (cardiac output divided by body surface ar

116 riables were related to cardiac MRI-assessed cardiac index (cardiac output/body surface area).

117 s, the Simplified Acute Physiology Score II, cardiac index, cardiac power index, and continuous hemod

118 output divided by body weight was defined as cardiac index; cardiac output divided by heart rate yiel

119 y pressure, in view of widening the range of cardiac index:central venous pressure measurements and i

120 The slope of the multipoint cardiac index:central venous pressure relationship incre

121 pulmonary blood volume index, the change in cardiac index, change in pulmonary blood volume index, a

122 volume index were higher, whereas change in cardiac index, change in pulmonary blood volume index, a

123 in WSES were not correlated with changes in cardiac index, changes in WSED correlated significantly

124 pulmonary capillary wedge pressure and lower cardiac index (CI) but not Paco2 or Pao2.

125 It is widely believed that a reduced cardiac index (CI) is a significant contributor to renal

126 Cardiac index (CI) was significantly increased in both t

127 ulmonary capillary wedge pressure (PCWP) and cardiac index (CI), and by changes in those measures aft

128 QRS duration, cardiac index (CI), and RV dP/dt were measured in 4 diff

129 Shortening fraction, cardiac index (CI), end-systolic wall stress (ESWS), and

130 n fraction (LVEF) and LV-Tei correlated with cardiac index (CI; P<0.001), and LV Tei was most frequen

131 PAP >/=35 mm Hg or mPAP >/=25 mm Hg with low cardiac index [CI <2.0 l/min/m(2)]; severe PH-COPD, seve

132 iac output was indexed to body surface area (cardiac index [CI]) for all analyses.

133 diography) and indexed to body surface area (cardiac index [CI]).

134 e, total peripheral vascular resistance, and cardiac index, compared with effects in sham rats.

135 e in 20 of 24 patients with normalization of cardiac index (complete response [CR]) in 3 of 24, parti

136 Low cardiac index correlated with cool extremities, high cen

137 Whereas cardiac index decreased (4.0 [3.5; 5.3] vs 3.1 [2.6; 3.9

138 increased 4 +/- 1 mm Hg (n = 27, p < 0.01), cardiac index decreased 0.4 +/- 0.1 L/min/m2 (n = 27, p

139 Simultaneously, cardiac index decreased from 3.47 +/- 0.86 L/min/m2 to 3

140 In the remaining patients, cardiac index did not change despite a significant decre

141 olume, left ventricular end-systolic volume, cardiac index, dP/dt max, -dP/dt min, and left ventricul

142 Left ventricular diastolic pressure, cardiac index, dP/dt40 and negative dP/dt were more opti

143 ed postresuscitation myocardial dysfunction (cardiac index, dP/dt40, -dP/dt) was observed with propra

144 up (left ventricular end-diastolic pressure, cardiac index, +dP/dt, -dP/dt, and time constant of expo

145 In Fontan patients, sildenafil improved cardiac index during exercise with a decrease in total p

146 duced left ventricular ejection fraction and cardiac index, elevated pulmonary capillary wedge pressu

147 When coupled with low cardiac index, even mild elevations in PVRI identify pat

148 .5+/-4.9 to 19.8+/-7.0 mm Hg (P<0.0001), and cardiac index (excluding augmented aortic flow) increase

149 ed a passive leg raising-induced increase in cardiac index greater than or equal to 10% with a sensit

150 ed a passive leg raising-induced increase in cardiac index greater than or equal to 10% with a sensit

151 by a passive leg raising-induced increase in cardiac index greater than or equal to 10%.

152 end-expiratory occlusion-induced increase in cardiac index greater than or equal to 5% detected a pas

153 end-expiratory occlusion-induced increase in cardiac index greater than or equal to 6% detected a pas

154 30%), and those with normal PVRI and normal cardiac index (group 2, n=182, 70%).

155 en to identify fluid responders (increase in cardiac index > 15%).

156 Fluid increased cardiac index >/= 15% (44% +/- 39%) in 30 "responders."

157 Volume expansion increased cardiac index >/= 15% in 49% of patients ("volume-respon

158 rognostic features at follow-up, including a cardiac index >/=2.5 L.min(-1).m(-2), 6-minute walk dist

159 mics with right atrial pressure <8 mm Hg and cardiac index >2.5 mg/kg/min(2).

160 tion with right atrial pressure <8 mm Hg and cardiac index >2.5 to 3.0 l/min/m(2).

161 mL/100 mL per minute in those with preserved cardiac index (>/=2.5 L/min per m(2); P=0.003).

162 ded (n=184), individuals with clinically low cardiac index had a higher relative risk of both dementi

163 rdiac index, individuals with clinically low cardiac index had a higher relative risk of dementia (HR

164 01 for both), whereas patients with a higher cardiac index had better survival overall (HR, 0.384; 95

165 sion (dose, 128+/-96 microg per minute), the cardiac index had increased further, to 2.52+/-0.55 lite

166 o a mean of 103+/-67 microg per minute), the cardiac index had increased to 2.22+/-0.44 liters per mi

167 rocirculatory parameters (arterial pressure, cardiac index, heart rate, and pulse pressure variations

168 Cardiac index, heart rate, systemic oxygenation, plasma

169 After implantation, the cardiac index improved from median 0.52 (interquartile r

170 time and stroke volume, cardiac output, and cardiac index in all groups.

171 be a more appropriate treatment target than cardiac index in PAH.

172 nd-expiratory pressure significantly reduced cardiac index in passive leg raising responders (-27% [i

173 Higher cardiac index in the adenosine, lidocaine, and Mg/adenos

174 th concurrent increase of cardiac output and cardiac index in the overall population (p < 0.001).

175 terval and stroke volume, cardiac output, or cardiac index in the overall population.

176 rterial blood flow (corrected for changes in cardiac index) in response to left lung hypoxic challeng

177 mpared with preoperative values, at 1 month, cardiac index increased (1.7 to 2.6 L/min/m(2)) and ther

178 Po2 improved (48 to 60 mm Hg, P=0.0004), and cardiac index increased (4.3 to 5.4 L/min per m2, P=0.00

179 Conversely, cardiac index increased (from 2.6 L.min(-1).m(-2) [IQR,

180 % and 15% groups (p < .05 vs. baseline), and cardiac index increased 130% (p < .05 vs. baseline) at 1

181 Within 48 hours postoperatively, the cardiac index increased 43%, pulmonary capillary wedge p

182 The average cardiac index increased 70.6% by 48 hours after implant,

183 The average cardiac index increased 89.5%, and pulmonary capillary w

184 Cardiac index increased and left ventricular end-diastol

185 However, with treatment cardiac index increased at rest (P=0.006) and peak exerc

186 Cardiac index increased from 1.7 to 2.6 l/min/m(2) witho

187 Each 1-SD unit decrease in cardiac index increased the relative risk of both dement

188 Before sildenafil, cardiac index increased throughout exercise (4.0+/-0.9,

189 ehicle (distilled water), caffeine decreased cardiac index, increased systemic vascular resistance, r

190 Compared with individuals with normal cardiac index, individuals with clinically low cardiac i

191 ssure = 5 cm H2O, we measured the changes in cardiac index induced by end-expiratory occlusion and a

192 We also tested whether the changes in cardiac index induced by passive leg-raising and by an e

193 he changes in pulse contour analysis-derived cardiac index induced by passive leg-raising and end-exp

194 ints were 6-hour and peak cTnT, ECG changes, cardiac index, inotrope and vasoconstrictor use, renal d

195 nine model of severe septic shock with a low cardiac index, intra-aortic balloon counterpulsation pro

196 al and clinical research suggests that lower cardiac index is associated with abnormal brain aging, i

197 Lower cardiac index is associated with an increased risk for t

198 Forearm blood flow, cardiac index, left ventricular dimensions, and mean art

199 Postresuscitation cardiac index, left ventricular end-diastolic pressure,

200 Myocardial function, including cardiac index, left ventricular, dP/dt max (dP/dt max),

201 decreasing cardiac function, even at normal cardiac index levels, is associated with accelerated bra

202 Cardiac index, local cerebral blood flow, and hemodynami

203 a score, edema, positive fluid balance, high cardiac index, low PaO2/FIO2 ratio, and high levels of c

204 h ADHF admitted between 2000 and 2005 with a cardiac index < or =2 l/min/m(2) for intensive medical t

205 In a double-blind fashion, 292 patients (cardiac index < or =2.5 l/min per m(2) and pulmonary cap

206 were adults with ejection fraction </= 25%, cardiac index </= 2.2 l/min/m(2) without inotropes or we

207 gen/fraction of inspired oxygen ratio </=55, cardiac index </=2.2, or ventricular tachycardia or fibr

208 Patients with cardiac index <2.2 L x min(-1) x m(-2) by catheterizatio

209 negative predictive value (93% and 86%) for cardiac index <2.5 and mixed venous oxygen saturation <6

210 s: those with high PVRI (>2 WU.m(2)) and low cardiac index <2.5 L min(-1) m(-2) (group 1, n=70, 30%),

211 significantly lower in patients with reduced cardiac index (<2.5 L/min per m(2)): 94+/-30 mL/100 mL p

212 rtic-valve area, <or=1 cm2), and a depressed cardiac index (<or=2.2 liters per minute per square mete

213 Hemodynamics, including cardiac index, LV dP/dt40, LV negative dP/dt, and LV dia

214 in ejection fraction, fractional shortening, cardiac index, LV dP/dt40, LV negative dP/dt, and LV dia

215 l stress (LVESWS), LVESWS-index, and maximal cardiac index (MCI; a measure of cardiac output at peak

216 eratively, all patients had severely reduced cardiac index (mean, 2.1 L/min/m2).

217 We noninvasively monitored cardiac index, mean arterial blood pressure, heart rate,

218 were associated with pronounced increases in cardiac index, mean arterial pressure, and heart rate an

219 s, renal failure, prolonged ventilation, low cardiac index, myocardial infarction, and stroke.

220 ed ejection fraction, 3D sphericity indices, cardiac index, normalized systolic volume, normalized LV

221 me expansion (500 mL of saline), we measured cardiac index, o2- and Co2-derived variables and lactate

222 ontrast to the dopamine-mediated increase in cardiac index observed at normothermia, high-dose dopami

223 ormal values; the latter were described as a cardiac index of >4.5 L x min(-1) x m(-2), pulmonary art

224 aseline New York Heart Association class IV, cardiac index of 1.7 L/min per m(2), pulmonary capillary

225 % were on at least two inotropes with a mean cardiac index of 1.8 L/min/m2.

226 i 13.0 (+/- 6.7) Wood Units/m(2) and reduced cardiac index of 2.21 (+/- 0.5) L/min/m(2) were recruite

227 the beginning of the treatment with a median cardiac index of 4.2 L/min/m(2) (range, 2.9-5.2; P < .00

228 n/kg, right atrial area of less than 18 cm2, cardiac index of greater than 2.5 L/min/m2, and absent o

229 In the goal-directed therapy arm, a cardiac index of greater than 3 L/min/m was targeted wit

230 recruitment, prone positioning increased the cardiac index only in patients with preload reserve, emp

231 es in systemic hemodynamic variables such as cardiac index or mean arterial pressure.

232 irculation are not useful for predicting low cardiac index or mixed venous oxygen saturation.

233 Sildenafil increased cardiac index (P<0.0001) and stroke volume index (P=0.00

234 sistance, and increased resting and exercise cardiac index (P<0.05 for all) without altering mean art

235 uration gradient (P<0.05) and inversely with cardiac index (P<0.05) for both CHF patients and control

236 th LGSAS had reduced stroke volume index and cardiac index (P=0.003 for both).

237 ulmonary wedge pressures (P=0.002) and lower cardiac indexes (P<0.0001).

238 o (n=101) had no significant effect on donor cardiac index (pooled mean difference, 0.15 L/min/m(2);

239 Cardiac index progressively increased in the device (2.0

240 Cardiac index, pulmonary artery pressure, and pulmonary

241 obtained by transpulmonary dilution such as cardiac index, pulmonary blood volume index, and extrava

242 ted the regression line between the pairs of cardiac index (pulse contour analysis) and central venou

243 The cardiac index, pulse oximetry, transcutaneous oxygen ten

244 ravascular lung water correlated to baseline cardiac index (r = 0.17; p = .001), baseline pulmonary b

245 zosentan caused a dose-dependent increase in cardiac index ranging from 24.4% to 49.9% versus 3.0% wi

246 ed HHT, severe liver involvement, and a high cardiac index related to HHT.

247 Despite the reduction in heart rate, cardiac index remained unchanged under mild therapeutic

248 terin blunted the increase in heart rate and cardiac index seen in the control group without affectin

249 usly altered and dopamine failed to increase cardiac index since stroke index was reduced with increm

250 Cardiac MRI measurements included cardiac index, stroke volume index, global and regional

251 Specifically, arterial pressure, cardiac index, stroke volume index, pH, and creatinine w

252 , cardiac power index (CPI), cardiac output, cardiac index, stroke volume, left ventricular work, lef

253 Changes in cardiac index, systemic and pulmonary vascular resistanc

254 and secondary end points included mean SVRI, cardiac index, systolic function, and lactate levels.

255 olumes (P=0.04) than participants in the top cardiac index tertile (values >2.92).

256 ons revealed that participants in the bottom cardiac index tertile (values <2.54) and middle cardiac

257 diac index tertile (values <2.54) and middle cardiac index tertile (values between 2.54 and 2.92) had

258 levations in PVRI, and when coupled with low cardiac index, this would identify patients at increased

259 Passive leg raising increased cardiac index to a larger extent after (8% +/- 4%) than

260 tory pressure, passive leg raising increased cardiac index to a larger extent than at positive end-ex

261 Increasing cardiac index to achieve an arbitrarily predefined eleva

262 in a more favorable index of forward output (cardiac index) to mechanical energy (pressure-volume are

263 Cardiac index, total peripheral resistance, and blood vo

264 We measured the response of cardiac index (transpulmonary thermodilution) to fluid a

265 mia, high-dose dopamine at 25 degrees C left cardiac index unchanged despite a concomitant increase i

266 GIK significantly increased cardiac index versus placebo (P=0.037).

267 s pressure and change in stroke volume index/cardiac index was 0.18 (95% CI, 0.1-0.25), being 0.28 (9

268 The cardiac index was 1.0 L min(-1) m(-2) (95% CI 0.6-1.4, p

269 ular ejection fraction was 16% (10% to 30%), cardiac index was 1.3 L/min/m (0.7-2.2 ) and systemic re

270 Cardiac index was 1.39 +/- 0.43 L . min(-)(1) . m(-)(2),

271 -37 and 39+/-23 mm Hg, respectively; and the cardiac index was 1.60+/-0.35 liters per minute per squa

272 AP) was 44 +/- 10 mm Hg (range 26-73 mm Hg), cardiac index was 3.5 +/- 0.9 L/min/m(2) , and pulmonary

273 ents with septic acute kidney injury, median cardiac index was 3.5 L/min/m2 (range 1.6-8.7), and medi

274 ry wedge pressure was 5.9 +/- 4.6 mm Hg, and cardiac index was 3.6 +/- 0.6 L . min(-)(1) . m(-)(2).

275 The combination of high PVRI and low cardiac index was an independent risk factor for Fontan

276 The cardiac index was depressed below 3.0 L/min/m2 in 65 of

277 -5 mm Hg; P<0.001), whereas no difference in cardiac index was found between groups.

278 shorter for BDOO compared with CDOO, and the cardiac index was higher with BDOO compared with CDOO.

279 Cardiac index was normal.

280 In multivariable-adjusted models, cardiac index was positively related to total brain volu

281 After surgery, the cardiac index was significantly higher in the OPCABG gro

282 The mean preoperative cardiac index was similar in the 2 surgical groups (2.9+

283 left ventricular end-diastolic pressure, and cardiac index, was significantly improved in pentazocine

284 pressure variation, and the change in stroke/cardiac index were 0.78, 0.72, and 0.72, respectively.

285 ar resistance, capillary wedge pressure, and cardiac index were also obtained at baseline in all pati

286 Cardiac output and cardiac index were increased significantly in 79% patien

287 fraction, left ventricular stroke index and cardiac index were initially (at H0 or H6 according to l

288 icular pressure, dP/dt40, negative dP/dt and cardiac index were measured for an interval of 240 min a

289 sure variation, stroke volume variation, and cardiac index were recorded at tidal volume 6 mL/kg pred

290 Peripheral blood pressure and cardiac index were recorded non-invasively.

291 e 16 (4.2) and 22 (5.3) mm Hg (p = .02), and cardiac indexes were 4.6 (2.8) and 2.2 (0.6) L/min/m (p

292 Cardiac indexes were significantly higher with normotens

293 poreal flows (approximately = 9% of baseline cardiac index) were required in zoniporide and control g

294 avenous (Pyr(1))apelin-13 infusion increased cardiac index, whereas reducing mean arterial pressure a

295 statistically significant change was in the cardiac index which fell slightly but significantly in t

296 n-13 infusion caused a sustained increase in cardiac index with increased left ventricular ejection f

297 The mean increase in cardiac index with the KCV OFF to ON was 0.53 L/min/m(2)

298 2.96, p = 0.307; Q = 44.88, I(2) = 95.54%), cardiac index (WMD: 0.05 L/min/m(2), 95%CI: -0.05, 0.15,

299 Age, baseline cardiac index, World Health Organization functional clas

300 esized that cardiac function, as measured by cardiac index, would be associated with preclinical brai