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1 enerator could represent a paradigm shift in cardiac pacing.
2 are common or controversial indications for cardiac pacing.
3 ), contributes significantly to neuronal and cardiac pacing.
4 ted by hyperpolarization, is a key player in cardiac pacing.
5 rome causes syncope, and symptoms respond to cardiac pacing.
6 cardiomyopathy despite early institution of cardiac pacing.
7 be transferred to the capital for temporary cardiac pacing.
8 , metabolic vasodilation was assessed during cardiac pacing.
9 ber pacemakers for most patients who require cardiac pacing.
10 ker replacement or a complication related to cardiac pacing.
11 atural history of heart failure due to rapid cardiac pacing.
12 s not necessarily imply a permanent need for cardiac pacing.
13 pacemakers and the termination of long-term cardiac pacing.
14 valuation of the methods and indications for cardiac pacing.
18 th combined therapy consisting of continuous cardiac pacing and maximally tolerated beta-blocker ther
20 Sympathetic nerve activity decreased with cardiac pacing and the decline was greater with AV than
22 dications for ICD therapy, no indication for cardiac pacing, and an LVEF of 40% or less, dual-chamber
23 only contribute to a better understanding of cardiac pacing but also may advance current efforts that
28 ble, and expanding clinical use of permanent cardiac pacing in a number of these conditions or syndro
29 e benefits and appropriate uses of permanent cardiac pacing in a variety of pathophysiologic states o
33 ease and enabling beat-to-beat adaptation of cardiac pacing in response to physiological feedback.
36 ts with syncope of unknown origin, selecting cardiac pacing in those with a positive ATP test leads t
42 electrode position for long-term transvenous cardiac pacing is in the apex of the right ventricle.
44 ment of atrioventricular block, dual-chamber cardiac pacing is thought to confer a clinical benefit a
46 ormally functioning hearts and stimulated by cardiac pacing; it thus functioned as an ideal therapeut
48 nts with predominantly AF and secondary SND, cardiac pacing may be the mainstay of therapy for patien
49 icular outflow gradient at rest suggest that cardiac pacing may result in symptomatic and hemodynamic
50 han does ventricular pacing, indicating that cardiac pacing mode may influence sympathetic outflow si
52 oventricular nodal disease, applications for cardiac pacing now include treatment of tachyarrhythmias
53 t pacemaker is increasing, but the effect of cardiac pacing on long-term survival and functional vari
54 by partial reduction of coronary flow, rapid cardiac pacing, or brief ischemia-reperfusion of a remot
55 esuscitation, defibrillation, cardioversion, cardiac pacing, or treatments targeted at the underlying
59 he aim of the study was to determine whether cardiac pacing reduces falls in older adults with cardio
66 less cardiac pacing represents the future of cardiac pacing systems, similar to the transition that o
67 ore amiodarone recipients required temporary cardiac pacing than did recipients of lidocaine or place
70 on Syncope of Uncertain Etiology (ISSUE-3), cardiac pacing was effective in reducing recurrence of s
72 dial diameter response to Ach, adenosine and cardiac pacing were measured in 32 patients with coronar
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