ライフサイエンスコーパス: cardiac rupture

コーパス検索結果 (１語後でソート)

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1 prevented the collagen deposition and caused cardiac rupture.

2 05, 3.16) were independently associated with cardiac rupture.

3 by a paradoxic increase in early deaths from cardiac rupture.

4 ocker use were independently associated with cardiac rupture.

5 7 days (2.9%); 28 recurrent MIs (26.6%); 13 cardiac ruptures (12.4%); 4 pump failures (3.8%); 2 othe

6 promotes ischemic injury, increases risk of cardiac rupture, accentuates post-MI remodeling and left

7 Recurrent MI or cardiac rupture accounts for a high proportion of sudden

8 lay normal cardiac function but are prone to cardiac rupture after acute myocardial infarction.

9 se deficiency prevented aldosterone-enhanced cardiac rupture after myocardial infarction.

10 hic characteristics of patients experiencing cardiac rupture after thrombolytic and adjunctive antico

11 m by oxidative activation of CaMKII, causing cardiac rupture and increased mortality in mice after my

12 =0.04), and postmortem examination confirmed cardiac rupture as the cause of most of the deaths.

13 had a catastrophic prognosis, with frequent cardiac rupture, as the result of markedly reduced colla

14 CONCLUSIONS Cardiac rupture following thrombolytic therapy tends to

15 ming and prevalence as a cause of death from cardiac rupture in patients with acute myocardial infarc

16 t ventricular (LV) pseudoaneurysms form when cardiac rupture is contained by adherent pericardium or

17 Cardiac rupture is preceded by left ventricular dilation

18 BACKGROUND Cardiac rupture is responsible for nearly 15% of all in-

19 showed compromised survival, higher rates of cardiac rupture, more severe left ventricular dilation,

20 ues of impaired healing or increased risk of cardiac rupture or failed to show any additional benefit

21 creased day 7 mortality because of increased cardiac rupture post-MI.

22 Cardiac pseudoaneurysm, a contained cardiac rupture, predisposes patients to further rupture

23 ggests that thrombolytic therapy accelerates cardiac rupture, typically to within 24 to 48 h of treat

24 Although the incidence of cardiac rupture was low (<1.0%), it was responsible for

25 A diagnosis of cardiac rupture was made clinically in patients with sud

26 urred within 48 h of treatment Patients with cardiac rupture were older, of lower body weight and sta

27 No further cardiac ruptures were documented.

28 Of the Agtr2-/Y mice, 63.6% died of cardiac rupture, whereas 23.5% of the WT mice died of th

29 mice displayed 100% mortality resulting from cardiac rupture within 12 days after MI compared with ap

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