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1 roponin (the NH(2) terminus (TnT-(1-153)) of cardiac troponin T).
2 coronary syndrome (NSTE-ACS) and an elevated cardiac troponin T.
3 (endo)plasmic reticulum Ca(2+) ATPase 1, and cardiac troponin T.
4 2a, and very little of the mature isoform of cardiac troponin T.
5 gy in patients with NSTE-ACS and an elevated cardiac troponin T.
6 n heavy chain, myosin binding protein C, and cardiac troponin T.
7 1,200 patients with NSTE-ACS and an elevated cardiac troponin T.
8 -day mortality, better than high-sensitivity cardiac troponin T.
9 exon 5 with the adjoining exon 6 from avian cardiac troponin-T.
10 atric dosing resulted in fewer elevations of cardiac troponin T (0 of 12 piglets vs. 6 of 11 piglets,
11 144 patients with elevated high-sensitivity cardiac troponin T, 31 had signs of MI and 113 did not.
14 segment results in conformational changes in cardiac troponin T, an altered binding affinity for trop
15 protein expression of cardiomyogenic markers cardiac troponin T and alpha-smooth muscle actin in CPCe
16 urs within the tropomyosin-binding domain of cardiac troponin T and alters the charge of the residue.
18 onal antibodies (mAbs) raised against bovine cardiac troponin T and chicken tropomyosin to visualize
19 assess the usefulness of baseline levels of cardiac troponin T and CK-MB and the electrocardiographi
21 he 2 well-known biomarkers (high-sensitivity cardiac troponin T and N-terminal pro-brain natriuretic
22 ment of beating sheets of cells that express cardiac troponin T and show a full range of action poten
23 this study was to investigate the utility of cardiac troponin T and troponin I for predicting outcome
28 defects in beta-cardiac myosin heavy chain, cardiac troponin T, and alpha-tropomyosin account for >
29 B-type natriuretic peptide, high-sensitivity cardiac troponin T, and high-sensitivity C-reactive prot
30 B-type natriuretic peptide, high-sensitivity cardiac troponin T, and high-sensitivity C-reactive prot
31 min, beta-myosin heavy chain, alpha-actinin, cardiac troponin T, and phospholamban at levels comparab
35 ype natriuretic peptide and high-sensitivity cardiac troponin T are frequently elevated in severe sep
36 coding the thin-filament contractile protein cardiac troponin T are responsible for 15% of all cases
37 per reference limit for the high-sensitivity cardiac troponin T assay (hs-cTnT) in 3 large independen
38 With the widespread use of high-sensitive cardiac troponin T assays, positive tests become frequen
39 ide, alpha- and beta-myosin heavy chain, and cardiac troponin T) by day 3 with subsequent progression
41 onal analysis of all protein coding exons of cardiac troponin T, cardiac troponin I, alpha-tropomyosi
42 17.8 ng/L and a perioperative high-sensitive cardiac troponin T change greater than or equal to 6.3 n
43 The risk predictive power of high-sensitive cardiac troponin T change in addition to the Revised Car
46 type natriuretic peptide or high-sensitivity cardiac troponin T concentrations were independently ass
47 ans, and patients were masked to study serum cardiac troponin-T concentrations and echocardiographic
48 s, serial endomyocardial biopsies, and serum cardiac troponin-T concentrations were obtained from 68
50 intronic elements (called MSEs) flanking the cardiac troponin T (cTNT) alternative exon 5 and promote
55 f two pre-mRNAs that are misregulated in DM, cardiac troponin T (cTNT) and insulin receptor (IR).
60 r group previously identified elevated serum cardiac troponin T (cTnT) as the most powerful predictor
63 eins including myosin heavy chain (MyHC) and cardiac troponin T (cTnT) cause a dominant genetic heart
67 (ETR-3/NAPOR/BRUNOL3) promotes inclusion of cardiac troponin T (cTNT) exon 5 via binding between pos
68 to determine whether there is immunoreactive cardiac troponin T (cTnT) expression in diseased skeleta
70 nother purine-rich enhancer from the chicken cardiac troponin T (cTNT) gene for the ability to regula
71 ative exon 5 of the striated muscle-specific cardiac troponin T (cTNT) gene is included in mRNA from
72 Striated muscle-specific expression of the cardiac troponin T (cTNT) gene is mediated through two M
75 ical and biological significance of elevated cardiac troponin T (cTnT) in patients with neuromuscular
84 e investigated the physiological role of the cardiac troponin T (cTnT) isoforms in the presence of hu
87 prospectively evaluated the relation between cardiac troponin T (cTnT) level, the presence and severi
89 erified assumption that chronically elevated cardiac troponin T (cTnT) levels fluctuate randomly arou
93 a splice donor site mutation (trunc) in the cardiac troponin T (cTnT) model familial hypertrophic ca
94 of established interstitial fibrosis in the cardiac troponin T (cTnT) mouse model of human hypertrop
95 eporting the functional effects of the first cardiac troponin T (CTnT) mutation linked to infantile R
97 ial necrosis was assessed by measurements of cardiac troponin T (cTnT) on admission and 12 h after ad
99 MBNL1 controls the splicing of exon 5 in the cardiac troponin T (cTNT) pre-mRNA by competing directly
100 Regulated alternative splicing of avian cardiac troponin T (cTNT) pre-mRNA requires multiple int
101 st-procedure CK-MB, an isolated elevation in cardiac troponin T (cTnT) predicts long-term survival.
103 s to determine whether different profiles of cardiac troponin T (cTnT) values assessed over time woul
105 esent study was to evaluate whether elevated cardiac troponin T (cTnT) was independently associated w
109 iac injury are cardiac troponin I (cTnI) and cardiac troponin T (cTnT) which have been considered as
110 NT photodetector for an on-chip detection of cardiac troponin T (cTnT) with a detection limit of 12 p
112 splicing of TNNT2, the gene that encodes for cardiac troponin T (cTnT), a biomarker of myocardial inj
113 f body mass, heart rate, blood pressures and cardiac troponin T (cTnT), a biomarker of myocyte damage
114 interaction between LVH, low but detectable cardiac troponin T (cTnT), and elevated N-terminal pro-B
115 s insensitive to reconstitution of cTnI with cardiac troponin T (cTnT), cTnC, or cTnC and cTnT in the
117 t of expression of a mutant (Arg92Gln) human cardiac troponin T (cTnT), known to cause HCM in humans,
119 50%, and improved diastolic function in the cardiac troponin T (cTnT)-Q92 transgenic mouse model of
128 tiation factor-15 (GDF-15), high-sensitivity cardiac troponin T (cTnT-hs) and haemoglobin, age, and p
130 cardiograms and serial serum measurements of cardiac troponin T (cTnT; cardiac injury biomarker), N-t
131 the pathologic free light chains (p < 0.05), cardiac troponin-T (cTnT) (p < 0.01), and the Karnofsky
134 nano-gap device provides the capability for cardiac-troponin T (cTnT) measurements with co-existed 1
135 s screening of cardiac Troponin-I (cTnI) and cardiac-Troponin-T (cTnT) in a point-of-care sensor form
137 (Ser532Pro and Phe764Leu) and a deletion in cardiac troponin T (deltaLys210) caused early-onset vent
138 eletion of the N-terminal variable region of cardiac troponin T demonstrates a novel mechanism by whi
141 ic cardiomyopathy caused by mutations in the cardiac troponin T gene (TNNT2) has been associated with
143 f a 9-bp segment from intron 7 of the turkey cardiac troponin T gene may be responsible for the weake
145 mano-Ward syndrome and long-QT syndrome, and cardiac troponin T gene, tnnt2, affected in human cardio
149 deaths compared with 29.5% among those with cardiac troponin T > or = 0.01 microg/L (p < .001).
150 43.7%, 33.8%, and 25.7% among patients with cardiac troponin T > or = 0.01 microg/L and 75.3%, 67.6%
151 GST) affinity tag at the N-terminus of human cardiac troponin T (hcTnT) and an intervening tobacco et
152 pe natriuretic peptide, and high-sensitivity cardiac troponin T, higher levels of Cp were associated
153 anterior circulation and a high-sensitivity cardiac troponin T (hs-cTnT) acquired on the day of admi
154 ween serial measurements of high-sensitivity cardiac troponin T (hs-cTnT) and future events in HF.
155 Although small elevations of high-sensitive cardiac troponin T (hs-cTnT) are associated with inciden
157 tional level, with elevated high-sensitivity cardiac troponin T (hs-cTnT) concentrations (>/=14 ng/L)
158 tic peptide (NT-proBNP) and high-sensitivity cardiac troponin T (hs-cTnT) concentrations were measure
159 rognostic value of baseline high-sensitivity cardiac troponin T (hs-cTnT) elevation in SCAD patients
160 ated hemoglobin (HbA1c) and high-sensitivity cardiac troponin T (hs-cTnT) in 9,661 participants free
161 sponse to mental stress and high-sensitivity cardiac troponin T (hs-cTnT) in healthy older individual
162 implications of introducing high-sensitivity cardiac troponin T (hs-cTnT) into clinical practice and
164 f an undetectable (<5 ng/l) high-sensitivity cardiac troponin T (hs-cTnT) level and an electrocardiog
165 hesized that any detectable high-sensitivity cardiac troponin T (hs-cTnT) level is associated with ad
166 s are preferable when using high-sensitivity cardiac troponin T (hs-cTnT) levels in the diagnosis of
168 econdary endpoints included high-sensitivity cardiac troponin T (hs-cTnT) on day 4, left ventricular
169 A 1-h algorithm based on high-sensitivity cardiac troponin T (hs-cTnT) testing at presentation and
170 Thus, we assessed whether high-sensitivity cardiac troponin T (hs-cTnT), a marker of subclinical my
171 B-type natriuretic peptide, high-sensitivity cardiac troponin T (hs-cTnT), and high-sensitivity cardi
172 uretic peptide (NT-proBNP), high-sensitivity cardiac troponin T (hs-cTnT), and low-density lipoprotei
173 uretic peptide (NT-proBNP), high-sensitivity cardiac troponin T (hs-cTnT), Cystatin-C (Cys-C), high-s
174 oponin T, measured by a new highly sensitive cardiac troponin T (hs-cTnT), may reflect ischemia witho
176 rdial Infarction) evaluated high-sensitivity cardiac troponin-T (hs-cTnT) in a 1-hour acute myocardia
177 ning with a biomarker (e.g. high sensitivity cardiac troponin T [hs-cTnT] or B-type natriuretic pepti
178 th myocardial damage (using high-sensitivity cardiac troponin-T [hs-cTnT]) and with coronary heart di
179 chain, myosin light chain 1/2, tropomyosin, cardiac troponins (T, I, C), and the trimeric troponin c
180 of wild-type (WT) cTn and cTn containing WT cardiac troponin T/I + cardiac troponin C (cTnC) D65A (a
181 ponin I was measured in 61,379 patients, and cardiac troponin T in 7880 patients (both proteins were
182 n 97.4% of the patients and high-sensitivity cardiac troponin T in 84.5%, with higher concentrations
185 was defined as an absolute high-sensitivity cardiac troponin T increase of >/=14 ng/L from preoperat
186 l prospective measurements were performed of cardiac troponin T, indexes of NO production (NO(2)(-) a
190 n of force development, the four known human cardiac troponin T isoforms, TnT1 (all exons present), T
191 Deletion of the 14 C-terminal residues of cardiac troponin T leads to hypertrophic cardiomyopathy.
193 level of 1.0 microg per liter or higher or a cardiac troponin T level of 0.1 microg per liter or high
198 acrophages, almost complete normalization of cardiac troponin T levels in serum and of left ventricul
203 ng hospitalization, 12.5% of patients with a cardiac troponin T < 0.01 microg/L suffered deaths compa
204 /L and 75.3%, 67.6%, and 62.9% in those with cardiac troponin T < 0.01 microg/L, respectively (p < .0
205 a mutation of the 5'-flanking region of the cardiac troponin T M-CAT-1 element upregulated expressio
206 We also found that Max was a part of the cardiac troponin T M-CAT-TEF-1 complex even when the DNA
209 eri-procedural increases of high-sensitivity cardiac troponin T (mean: 9.9 ng/ml, range: 2.7 to 19.0
211 ive and 24-hour postoperative high-sensitive cardiac troponin T measurements and the respective chang
216 eins specifically activate exon inclusion of cardiac troponin T minigenes in vivo via muscle-specific
217 lure express an unusual low molecular weight cardiac troponin T missing 11 amino acids due to the spl
218 tide enhancer that has similarities with the cardiac Troponin T MSE3 enhancer, and a potentially nove
224 native splicing patterns (for example, human cardiac troponin T) or affects other aspects of RNA biol
225 e have asked whether serum concentrations of cardiac troponin-T predict development of coronary arter
227 combinase expressed under the control of the cardiac troponin T promoter resulted in death by E12.5;
228 s to dissect M-CAT elements derived from the cardiac troponin T promoter, whose regulation is highly
229 rdiac-selective expression of EcSOD from the cardiac troponin-T promoter after systemic administratio
230 ellular superoxide dismutase (EcSOD) via the cardiac troponin-T promoter would protect the mouse hear
233 expression, transgenic mice carrying the rat cardiac troponin T proximal promoter (-497 bp from the t
234 igenic mice, turned on and off expression of cardiac troponin T-Q92 (cTnT-Q92), responsible for human
236 and methylprednisolone significantly reduced cardiac troponin T release and the number of allograft i
237 nhanced myocardial damage evidenced by serum cardiac troponin T release in the rat and mouse cardiac
238 nges in markers of cardiac (high-sensitivity cardiac troponin T), renal (creatinine and cystatin-C),
239 ific antigens cardiac myosin heavy chain and cardiac troponin T, respectively (immunocytochemistry),
241 We examined the value of serum levels of cardiac troponin T, serum creatine kinase MB (CK-MB) lev
242 pro-brain natriuretic peptide (pro-BNP), and cardiac troponin T showed significant linear trends for
244 ificantly more cardiomyocytes, determined by cardiac troponin-T staining, in the MI zone of the QHG21
245 tions between or near residues 112 to 136 of cardiac troponin-T, the crucial TnT1 (N-terminal domain
246 enes encoding cardiac troponin I (TNNI3) and cardiac troponin T (TNNT2) caused altered troponin prote
247 RNAs including the Insulin Receptor (Insr), Cardiac Troponin T (Tnnt2), Lim Domain Binding 3 (Ldb3)
248 ted splicing of three CELF target pre-mRNAs, cardiac troponin T (Tnnt2), myotubularin-related 1 gene
250 ardiac troponin I (TNNI3p.98truncation ) and cardiac troponin T (TNNT2p.K217deletion ; also known as
251 estigated the prognostic value of detectable cardiac troponin T (TnT) and elevated N-terminal pro-B-t
252 ausing mutations associated with a truncated cardiac troponin T (TnT) and missense mutations in the b
258 t to evaluate the prognostic significance of cardiac troponin T (TnT) serum levels after noncardiac s
260 be caused by dominant missense mutations in cardiac troponin T (TnT), alpha-tropomyosin, C-protein,
262 h no cardiovascular disease in our study had cardiac troponin T values above the current myocardial i
263 emoglobin A(1c), detectable high-sensitivity cardiac troponin T was associated with subsequent CVD (m
268 ype natriuretic peptide and high-sensitivity cardiac troponin T were measured 1, 2, and 7 days after
270 o cause HCM, both wild-type and mutant human cardiac troponin T were overexpressed in Escherichia col
271 Left ventricular structure and function and cardiac troponin-T were among the top predictors for inc
272 defect in the interactions between CTnC and cardiac troponin T, which are known to be necessary for
274 hich most patients would have had a negative cardiac troponin T with older assays); and Group 4, thos
275 rmined pre- and postoperative high-sensitive cardiac troponin T with the occurrence of major adverse
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