ライフサイエンスコーパス: cardiac work

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1 sease, hypertension, or obesity) increase in cardiac work.

2 repair and may have the capacity to perform cardiac work.

3 s and reducing apical cavity compression and cardiac work.

4 consumption for comparable levels of CBF and cardiac work.

5 Despite similar increases in cardiac work and comparable sympathetic stimulation in C

6 correlated significantly with both increased cardiac work and degree of stenosis.

7 The beta 1 receptor blockade reduces cardiac work and may thereby lower myocardial blood flow

8 , because it shifts the relationship between cardiac work and myocardial oxygen consumption, suggesti

9 itions, recapitulating the linearity between cardiac work and respiration in the heart.

10 drenoceptor antagonist propranolol decreases cardiac work and resting energy expenditure while increa

11 butamine infusion and to correlate flow with cardiac work and severity of coronary artery disease.

12 spectroscopy) at baseline, during increased cardiac work, and during recovery.

13 in controls (5 ml/g SEM 0.6) after 5 min of cardiac work, and prolonged survival times up to 120 min

14 ute and chronic systolic function, increases cardiac work, and reduces mortality.

15 lowered below a certain level, increases in cardiac work become more "energetically costly" in terms

16 kJ/mol in M/MtCK-/- hearts during increased cardiac work but only slightly in wild-type (1.7 kJ/mol)

17 Resting MBF declined in proportion to cardiac work by approximately 20% from 0.61 +/- 0.09-0.5

18 c patient's inability to cope with increased cardiac work demand largely stems from mitochondrial red

19 ess correlated significantly with changes in cardiac work in healthy volunteers (r = 0.77; P < 0.001)

20 Despite higher levels of cardiac work in the Kv1.5(-/-) mice (versus WT mice at b

21 A high rate of cardiac work increases citric acid cycle (CAC) turnover

22 elating myocardial oxygen consumption to the cardiac work indices, stroke work, and pressure-volume a

23 We have previously shown that increased cardiac work initially caused a rapid Ca(2+)-independent

24 The response to a decrease in cardiac work is not known.

25 sents both an adaptive response to increased cardiac work load and a precursor state of heart failure

26 cantly diminished, indicating improvement of cardiac work load by RD.

27 h fibers, this new muscle may be suited to a cardiac work load.

28 aturation process that is independent of the cardiac working load.

29 stem is required for situations of increased cardiac work or acute stress.

30 flow increased appropriately for the greater cardiac work, there was a redistribution of regional blo

31 At high levels of cardiac work, tissue oxygen tension dropped significantl

32 ix Ca(2+) content, rapidly enough to support cardiac work transitions in vivo.

33 Cardiac work was estimated as the product of mean arteri

34 Cardiac work was measured by echocardiography, and effic

35 Energy substrate metabolism and cardiac work were determined ex vivo in a sequential pro

36 ed to the rate-pressure product, an index of cardiac work, were similar in both the patients with Kaw

37 ts the systolic pressure further, increasing cardiac work while reducing the diastolic pressure, on w

38 hysiologic characteristics include increased cardiac work with normal peripheral resistance, diffuse

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