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1 s morphological findings or mild to moderate cardiomegaly.
2 ase was progressive, causing weight loss and cardiomegaly.
3 trophic signals and exhibit stress-dependent cardiomegaly.
4 eading secondarily to hepatosplenomegaly and cardiomegaly.
5 s ablated and W(sh) mice develop symptoms of cardiomegaly.
6                         The presence of both cardiomegaly and CHF in the two affected males and atria
7 e creatine kinase-Cre;Vps34(f/f) mice led to cardiomegaly and decreased contractility.
8 nd of the first month of life, she developed cardiomegaly and signs of cardiac failure.
9 ce of severe myocarditis, with dysrhythmias, cardiomegaly, and cardiogenic shock.
10 on are abnormal apical impulse, radiographic cardiomegaly, and q waves or left bundle branch block on
11 isted mobility, presence of heart failure or cardiomegaly any time before discharge, presence of peri
12  majority of Galphaq/beta2ARH mice died with cardiomegaly at 5 weeks.
13                      AdTNFRI did not reverse cardiomegaly but abrogated myocardial inflammation.
14    All FK506 and CsA children and adults had cardiomegaly by HW, HW/BW (P(FK506 peds) <0.024, P(CsA p
15                                              Cardiomegaly by HW, HW/BW, and histology was uniformly p
16 ectual disability (ID), atrial fibrillation, cardiomegaly, congestive heart failure (CHF), some somat
17 f the central pulmonary arteries and massive cardiomegaly due to right ventricular and right atrial e
18 vascular hemolysis, exuberant hematopoiesis, cardiomegaly, glomerulosclerosis, visceral congestion, h
19  by a range of anatomical defects, including cardiomegaly, hyperflexibility of the joints, hypertrich
20                                Hypertension, cardiomegaly, increased creatinine, overdose (primarily
21 riteria variables (dyspnea, pulmonary rales, cardiomegaly, interstitial or pulmonary edema on chest r
22                     Radiographic evidence of cardiomegaly is common in patients with congenital compl
23 overexpression of IGF-1 in myocytes leads to cardiomegaly mediated by an increased number of cells in
24 oled sensitivity, 76%; 95% CI, 62%-90%), and cardiomegaly on chest radiograph (pooled sensitivity, 89
25 cardia, elevated jugular venous pressure, or cardiomegaly on chest radiograph.
26 ourth heart sound on examination, absence of cardiomegaly or signs of congestive heart failure on che
27 ratio (p < 0.001), subjective impressions of cardiomegaly (p < 0.01), and increased VPW (p = 0.02).
28 ochondrodysplasia, patent ductus arteriosus, cardiomegaly, pericardial effusion, and lymphoedema.
29 eft ventricular dysfunction, increasing age, cardiomegaly, peripheral vascular disease, chronic renal
30 werful overall predictors, with only age and cardiomegaly showing a better independent association wi
31 ed blood cells, consistent with the observed cardiomegaly, splenomegaly, elevated bilirubin levels an
32 ation, increased vascularization, and causes cardiomegaly through persistent addition of wall myocard
33 to deposition of immunocomplexes followed by cardiomegaly with ventricular dilation and hypertrophy,

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