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1 s morphological findings or mild to moderate cardiomegaly.
2 ase was progressive, causing weight loss and cardiomegaly.
3 trophic signals and exhibit stress-dependent cardiomegaly.
4 eading secondarily to hepatosplenomegaly and cardiomegaly.
5 s ablated and W(sh) mice develop symptoms of cardiomegaly.
10 on are abnormal apical impulse, radiographic cardiomegaly, and q waves or left bundle branch block on
11 isted mobility, presence of heart failure or cardiomegaly any time before discharge, presence of peri
14 All FK506 and CsA children and adults had cardiomegaly by HW, HW/BW (P(FK506 peds) <0.024, P(CsA p
16 ectual disability (ID), atrial fibrillation, cardiomegaly, congestive heart failure (CHF), some somat
17 f the central pulmonary arteries and massive cardiomegaly due to right ventricular and right atrial e
18 vascular hemolysis, exuberant hematopoiesis, cardiomegaly, glomerulosclerosis, visceral congestion, h
19 by a range of anatomical defects, including cardiomegaly, hyperflexibility of the joints, hypertrich
21 riteria variables (dyspnea, pulmonary rales, cardiomegaly, interstitial or pulmonary edema on chest r
23 overexpression of IGF-1 in myocytes leads to cardiomegaly mediated by an increased number of cells in
24 oled sensitivity, 76%; 95% CI, 62%-90%), and cardiomegaly on chest radiograph (pooled sensitivity, 89
26 ourth heart sound on examination, absence of cardiomegaly or signs of congestive heart failure on che
27 ratio (p < 0.001), subjective impressions of cardiomegaly (p < 0.01), and increased VPW (p = 0.02).
28 ochondrodysplasia, patent ductus arteriosus, cardiomegaly, pericardial effusion, and lymphoedema.
29 eft ventricular dysfunction, increasing age, cardiomegaly, peripheral vascular disease, chronic renal
30 werful overall predictors, with only age and cardiomegaly showing a better independent association wi
31 ed blood cells, consistent with the observed cardiomegaly, splenomegaly, elevated bilirubin levels an
32 ation, increased vascularization, and causes cardiomegaly through persistent addition of wall myocard
33 to deposition of immunocomplexes followed by cardiomegaly with ventricular dilation and hypertrophy,
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