ライフサイエンスコーパス: cardiorespiratory

1 These results suggest that persistent cardiorespiratory abnormalities caused by LT-IH are medi

2 vance of this dual chemoreceptor feedback to cardiorespiratory abnormalities present in diseases in w

3 mbedded in the brainstem networks regulating cardiorespiratory activity and the response to glucopriv

4 er into the medullary raphe had no effect on cardiorespiratory activity or the chemoreflex.

5 n vivo, ATP injection into the NTS increased cardiorespiratory activity; however, injection of a P2-r

6 spiratory control and may underlie important cardiorespiratory adjustments for gas exchange improveme

7 rimary termination and integration point for cardiorespiratory afferents in the brainstem.

8 Patients in the multimodal group had less cardiorespiratory and anastomotic complications but more

9 hly polluted air resulted in weight gain and cardiorespiratory and metabolic dysfunction.

10 deaths but also may increase mortality from cardiorespiratory and other causes.

11 Cardiorespiratory and perceptual responses were measured

12 tween postictal generalized EEG suppression, cardiorespiratory arrest and sudden death following a se

13 a (n=1), intussusception of the graft (n=1), cardiorespiratory arrest during anesthesia (n=1), and co

14 Preventable cardiorespiratory arrest from underlying cardiac dysrhyt

15 he suffered a pulseless electrical activity cardiorespiratory arrest from which he could not be resu

16 at RRSs are associated with reduced rates of cardiorespiratory arrest outside of the intensive care u

17 d unrelated to study treatment was reported (cardiorespiratory arrest).

18 al hemorrhage, and 30% of patients following cardiorespiratory arrest.

19 e significantly for tachycardia, seizure, or cardiorespiratory arrest.

20 photobleaching, was not affected just after cardiorespiratory arrest; and (iii) Aqp4 gene deletion d

21 There were three sudden unexplained cardiorespiratory arrests in the propofol group (3 of 31

22 hough the cause of SIDS is unknown, immature cardiorespiratory autonomic control and failure of arous

23 ized rates for medicine, surgery/gynecology, cardiorespiratory, cardiovascular, and neurology cohorts

24 nfected neurons, especially those located in cardiorespiratory centers in the medulla.

25 oradrenergic cell groups nor within the main cardiorespiratory centers of the dorsolateral pons.

26 de sufficient afferent input to initiate the cardiorespiratory changes consistent with the nasopharyn

27 s, including especially pulmonary oedema and cardiorespiratory collapse.

28 arization (SD) in dorsal medulla, leading to cardiorespiratory collapse.

29 encephalitis (n = 21), and encephalitis with cardiorespiratory compromise (n = 11).

30 valuable tool to rescue children with severe cardiorespiratory compromise related to myocarditis.

31 rticipants (2.0%) were found to have serious cardiorespiratory conditions that had been previously mi

32 Arch (PRSx8-ArchT-EYFP-LVV) and measured the cardiorespiratory consequences of Arch activation (10 s)

33 and peripheral feedback mechanisms governing cardiorespiratory control and may underlie important car

34 he carotid body (CB) chemoreceptors improves cardiorespiratory control and survival during heart fail

35 EAAT2 modulates cardiorespiratory control and tempers excitatory cardior

36 also noted in ventral areas associated with cardiorespiratory control, including the gigantocellular

37 information to brainstem nuclei involved in cardiorespiratory control.

38 ed with abnormalities of 5-HT neurons and of cardiorespiratory control.

39 ral chemoreceptors play an important role in cardiorespiratory control.

40 tus solitarii (nTS), an important nucleus in cardiorespiratory control.

41 ation has a significantly stronger effect on cardiorespiratory coupling than healthy aging.

42 ration increased ICU admissions triggered by cardiorespiratory criteria, whereas admissions triggered

43 n the late 2000s, with a larger increase for cardiorespiratory deaths than for deaths from other caus

44 rlier decades, for older populations and for cardiorespiratory deaths.

45 Glu5 signaling (i.e. seizures), or affecting cardiorespiratory defects in RS model mice.

46 d side effects, including sedation, amnesia, cardiorespiratory depression, and anticonvulsive toleran

47 was assessed using a home-based multichannel cardiorespiratory device.

48 Chronic cardiorespiratory disease is associated with low birthwe

49 ed as a cause of hypertension in a number of cardiorespiratory diseases states and has therefore been

50 ion of autonomic morbidities associated with cardiorespiratory diseases, such as sleep-disordered bre

51 the role of the carotid body chemoreflex in cardiorespiratory diseases.

52 chemoreflex function is strongly related to cardiorespiratory disorders and disease progression in h

53 tion might be of therapeutic value to reduce cardiorespiratory dysfunction and improve survival durin

54 This ultimately results in cardiorespiratory dysfunction, which is the predominant

55 sessed short-term associations between daily cardiorespiratory ED visit counts and daily levels of 24

56 showed particularly strong associations with cardiorespiratory ED visit outcomes.

57 Their complex cardiorespiratory effects are presumably mediated by the

58 Neuronal excitation and cardiorespiratory effects following EAAT2 inhibition wer

59 These cardiorespiratory effects were prevented via ganglionic

60 e conducted a time-series study of PM2.5 and cardiorespiratory emergency department (ED) visits in th

61 ophysiological pathways linking exposure and cardiorespiratory events.

62 ge of 66 years (range 26-86 years) underwent cardiorespiratory exercise testing before major hepatobi

63 s, the natural history is characteristically cardiorespiratory failure and death in the first year of

64 Cardiorespiratory failure is the leading cause of death

65 Cardiorespiratory failure is the most common cause of su

66 g the study (pulmonary artery thrombosis and cardiorespiratory failure); neither death was judged to

67 ures frequently led to apneas, brainstem SD, cardiorespiratory failure, and death.

68 n is a rescue therapy used to support severe cardiorespiratory failure.

69 nd/or sudden death due to apnea episodes and cardiorespiratory failure.

70 nd monitored cases, as well as human seizure cardiorespiratory findings related to SUDEP, and SUDEP a

71 Standardized surveys of cardiorespiratory findings were conducted among male tel

72 To determine the association among cardiorespiratory fitness ("fitness"), adiposity, and mo

73 erage running distance (kilometers per day), cardiorespiratory fitness (10-km footrace performance),

74 cal care unit as younger people with similar cardiorespiratory fitness (13 vs 12; P = 0.08 and 1 vs 1

75 The primary outcome measures were weight and cardiorespiratory fitness (as measured with the 6-minute

76 The present review focuses on cardiorespiratory fitness (commonly measured by maximal

77 is study sought to determine the capacity of cardiorespiratory fitness (CRF) algorithms without exerc

78 A positive association between cardiorespiratory fitness (CRF) and white matter integri

79 ce has firmly established that low levels of cardiorespiratory fitness (CRF) are associated with a hi

80 Cardiorespiratory fitness (CRF) as assessed by formalize

81 features around the home and workplace with cardiorespiratory fitness (CRF) based on a treadmill tes

82 <.05) associated with BF%, diastolic BP, and cardiorespiratory fitness (CRF) for the Chu et al PT onl

83 has been shown to be effective in improving cardiorespiratory fitness (CRF) in patients with systoli

84 Poor cardiorespiratory fitness (CRF) is an independent risk f

85 It is well established that cardiorespiratory fitness (CRF) is inversely associated

86 Cardiorespiratory fitness (CRF) is not routinely measure

87 Low cardiorespiratory fitness (CRF) may contribute to CV ris

88 Evidence on the effect of cardiorespiratory fitness (CRF) on age-related longitudi

89 An increase in cardiorespiratory fitness (CRF) through exercise trainin

90 Prospective data relating cardiorespiratory fitness (CRF) with nonfatal cardiovasc

91 ave examined the association between LTL and cardiorespiratory fitness (CRF), an enduring trait influ

92 the individual and joint associations among cardiorespiratory fitness (CRF), body mass index, and he

93 ted the association between overall diet and cardiorespiratory fitness (CRF).

94 45.9 +/- 12.7 years) completed assessment of cardiorespiratory fitness (CRF, VO(2peak)), muscle stren

95 d self-reported physical activity, predicted cardiorespiratory fitness (cycle ergometer test), obesit

96 Cardiorespiratory fitness (fitness) is associated with c

97 s well known, there is a lack of data on how cardiorespiratory fitness (hereafter referred to as fitn

98 /m(2)), physical activity (in km/d run), and cardiorespiratory fitness (in m/s during 10-km footrace)

99 tive functioning predisposed to better adult cardiorespiratory fitness (neuroselection).

100 Move and OnTrack resulted in less decline in cardiorespiratory fitness (P < .001), better physical fu

101 piratory fitness compared to adequate or low cardiorespiratory fitness (p < 0.001 for both).

102 >/=2 compared to those with METs gain <2 in cardiorespiratory fitness (p < 0.001 for both).

103 g program (25+/-9 miles/wk) led to increased cardiorespiratory fitness (peak oxygen consumption, 44.6

104 ciation was found between sedentary time and cardiorespiratory fitness (r = -.13, p>.05).

105 Cardiorespiratory fitness (standardized odds ratio: 0.33

106 Secondary outcomes measured were cardiorespiratory fitness (VO2 peak) and body compositio

107 Exercise improved cardiorespiratory fitness (VO2 peak) compared with the C

108 Cardiorespiratory fitness also declines with age, and th

109 Effects on cardiorespiratory fitness and abdominal obesity are both

110 udy aimed to define the relationship between cardiorespiratory fitness and age in the context of post

111 independent and linked associations between cardiorespiratory fitness and age on postsurgical mortal

112 nsity exercise training was found to improve cardiorespiratory fitness and attenuate cutaneous vasodi

113 The combined associations of changes in cardiorespiratory fitness and body mass index (BMI) with

114 ficant epidemiologic observations connecting cardiorespiratory fitness and cancer.

115 The association between cardiorespiratory fitness and covariate adjusted decline

116 We investigated the impact of cardiorespiratory fitness and exercise training on physi

117 elationships between hard physical activity, cardiorespiratory fitness and health parameters.

118 re is a graded, inverse relationship between cardiorespiratory fitness and incident AF, especially am

119 ey were used to describe the distribution of cardiorespiratory fitness and its association with obesi

120 ave demonstrated strong associations between cardiorespiratory fitness and lower cardiovascular disea

121 udy, we investigated the association between cardiorespiratory fitness and measures of gray matter at

122 2-hour glucose level (primary outcomes) and cardiorespiratory fitness and measures of insulin action

123 tive tests, there was no association between cardiorespiratory fitness and midlife cognitive function

124 inverse, dose-dependent association between cardiorespiratory fitness and mortality is well-establis

125 High maternal BMI and low levels of cardiorespiratory fitness and physical activity independ

126 ge, during and after treatment) and improves cardiorespiratory fitness and physical function.

127 nd no weight loss demonstrate that increased cardiorespiratory fitness and reduced intra-abdominal ad

128 We found a positive association between cardiorespiratory fitness and regional gray matter volum

129 The primary outcomes were cardiorespiratory fitness and skeletal muscle (vastus la

130 ned the effects of simvastatin on changes in cardiorespiratory fitness and skeletal muscle mitochondr

131 Simvastatin attenuates increases in cardiorespiratory fitness and skeletal muscle mitochondr

132 This study sought to evaluate the role of cardiorespiratory fitness and the incremental benefit of

133 High levels of cardiorespiratory fitness and/or habitual physical activ

134 Moreover, cardiorespiratory fitness appears to be one of the stron

135 Low levels of cardiorespiratory fitness are associated with high risk

136 Physical activity and cardiorespiratory fitness are not currently recognized a

137 no evidence for a neuroprotective effect of cardiorespiratory fitness as of midlife.

138 oper Center Longitudinal Study who underwent cardiorespiratory fitness assessment at a mean age of 49

139 Improvement in cardiorespiratory fitness augments the beneficial effect

140 rences were observed in daily step counts or cardiorespiratory fitness between the groups.

141 Exercise training improved cardiorespiratory fitness by 5.0 ml kg(-1) min(-1) (95%

142 trategies was greatest in patients with high cardiorespiratory fitness compared to adequate or low ca

143 vity (>/=9 METs) holds greater potential for cardiorespiratory fitness compared to physical activity

144 s additional improvements in S(I), S(G), and cardiorespiratory fitness compared with a sedentary life

145 Higher cardiorespiratory fitness during childhood is associated

146 hard physical activity were associated with cardiorespiratory fitness for boys (F = 5.64, p<.01) whe

147 ty decreased significantly in the group with cardiorespiratory fitness gain >/=2 METs as compared to

148 ts effect on AF recurrence or the benefit of cardiorespiratory fitness gain is unknown.

149 Impact of cardiorespiratory fitness gain was ascertained by the ob

150 Participants with better cardiorespiratory fitness had higher cognitive test scor

151 rbidity and mortality, but the prevalence of cardiorespiratory fitness has not been quantified in rep

152 atory fitness and the incremental benefit of cardiorespiratory fitness improvement on rhythm control

153 Further adjustment for cardiorespiratory fitness in a subset of 572610 men with

154 chronic diseases and is associated with low cardiorespiratory fitness in adults.

155 ive breastfeeding has a beneficial effect on cardiorespiratory fitness in children and adolescents.

156 ysical activity subcomponents are related to cardiorespiratory fitness in children.

157 ercise testing as an objective assessment of cardiorespiratory fitness in clinical oncology research

158 se relationship between cancer incidence and cardiorespiratory fitness in large population studies.

159 Higher cardiorespiratory fitness in middle age is strongly asso

160 tion of health care costs in later life with cardiorespiratory fitness in midlife after adjustment fo

161 e, fat-free mass (FFM), and fat mass (FM) on cardiorespiratory fitness in pediatric renal transplant

162 ctivity, exercise training (ET), and overall cardiorespiratory fitness in the prevention and treatmen

163 Cardiorespiratory fitness increased by 10% (p < 0.05) in

164 Cardiorespiratory fitness is an independent predictor of

165 On the other hand, high cardiorespiratory fitness is known to be a strong predic

166 ramming effect of exclusive breastfeeding on cardiorespiratory fitness is of public health interest.

167 Although cardiorespiratory fitness is protective against incident

168 Cardiorespiratory fitness levels were assessed between 1

169 well as the effect of physical activity and cardiorespiratory fitness on cardiovascular diseases.

170 , there are few data regarding the effect of cardiorespiratory fitness on health care costs independe

171 activity/exercise training, and increases in cardiorespiratory fitness on the prognosis of obese pati

172 Cardiorespiratory fitness predicts arrhythmia recurrence

173 Patients older than 75 years with low cardiorespiratory fitness spent a median of 11 days long

174 Older people with normal cardiorespiratory fitness spent the same number of days

175 nsitions revealed little variability between cardiorespiratory fitness tertiles.

176 axial accelerometers and undertook a maximal cardiorespiratory fitness test.

177 Cardiorespiratory fitness was assessed by a maximal trea

178 Adult cardiorespiratory fitness was assessed using a submaxima

179 To determine a dose response, cardiorespiratory fitness was categorized as: low (<85%)

180 Cardiorespiratory fitness was estimated by maximal metab

181 To determine whether cardiorespiratory fitness was longitudinally associated

182 In addition, men with greater cardiorespiratory fitness were at significantly less ris

183 sociations of habitual physical activity and cardiorespiratory fitness with IHTG and the prevalence o

184 The implications of cardiorespiratory fitness with prognosis are discussed,

185 muscle (low percentage of lean mass and low cardiorespiratory fitness) are likely to contribute thes

186 ody mass index, systolic blood pressure, and cardiorespiratory fitness).

187 wer incident AMD risk independent of weight, cardiorespiratory fitness, and cigarette use.

188 ther, and to what extent, physical activity, cardiorespiratory fitness, and obesity at age 16 mediate

189 factors influence physical activity levels, cardiorespiratory fitness, and risk of death.

190 djustment or not for key confounders such as cardiorespiratory fitness, and to the lack of consensus

191 hanges in myocardial structure and function, cardiorespiratory fitness, and traditional cardiac risk

192 study was to examine the association between cardiorespiratory fitness, body mass index (BMI), and wa

193 ardial infarction, type 2 diabetes mellitus, cardiorespiratory fitness, body mass index, systolic blo

194 re made for height, weight, body mass index, cardiorespiratory fitness, cognitive ability, and socioe

195 demonstrated benefits in muscular strength, cardiorespiratory fitness, functional task performance,

196 ellitus, abnormal resting ECG responses, and cardiorespiratory fitness, hazard ratios (95% confidence

197 Cardiorespiratory fitness, homeostasis model assessment

198 One consequence of inactivity, low cardiorespiratory fitness, is an established risk factor

199 improvements in SI and that only EX improved cardiorespiratory fitness, mitochondrial respiration and

200 Exercise training has been shown to improve cardiorespiratory fitness, physical capacity, and qualit

201 ercise ECG responses, with stratification by cardiorespiratory fitness, quantified as treadmill test

202 It is also possible that low cardiorespiratory fitness, rather than overweight or obe

203 included physical activity (steps per day), cardiorespiratory fitness, self-efficacy, healthy living

204 ity, objectively measured physical activity, cardiorespiratory fitness, self-reported sports particip

205 BMI, greater physical activity, and greater cardiorespiratory fitness, the latter being statisticall

206 itively associated (beta = .45, p<.001) with cardiorespiratory fitness.

207 ociated with 10-14 year-old schoolchildren's cardiorespiratory fitness.

208 relationship between sedentary behaviour and cardiorespiratory fitness.

209 1) when compared with patients with adequate cardiorespiratory fitness.

210 behaviour as a means to maintain or improve cardiorespiratory fitness.

211 ence interval = -0.039, -0.011), but not via cardiorespiratory fitness.

212 ow-up duration between the groups defined by cardiorespiratory fitness.

213 en childhood cognitive functioning and adult cardiorespiratory fitness.

214 trics of physiological performance including cardiorespiratory function (heart rate [fH ] and ventila

215 Evidence of improved cardiorespiratory function after pectus excavatum repair

216 ng the constellation of factors that bear on cardiorespiratory function and that become intricately e

217 spiratory testing and techniques to preserve cardiorespiratory function before elective surgery in ol

218 oxygenation (ECMO) has been used to support cardiorespiratory function during pediatric cardiopulmon

219 Whereas cardiorespiratory function has long been applied by card

220 uronal activity and thereby basal and reflex cardiorespiratory function is unknown.

221 nistration to achieve supranormal indices of cardiorespiratory function, which has led to the advent

222 lays on buffering nTS excitation and overall cardiorespiratory function.

223 produced neuronal excitation to alter basal cardiorespiratory function.

224 operties, and ultimately on basal and reflex cardiorespiratory function.

225 Cardiorespiratory functions in mammals are exquisitely s

226 ry reflex response, with profound effects on cardiorespiratory functions.

227 channels in the spontaneous firing in these cardiorespiratory GABAergic neurons that possess a pacem

228 nception and Nov 28, 2016, investigating the cardiorespiratory health effects of particulate ambient

229 We aimed to investigate the cardiorespiratory health effects of particulate ambient

230 l in the nucleus tractus solitarii (nTS) for cardiorespiratory homeostasis and initiation of sensory

231 nsory ganglia (NGs), structures critical for cardiorespiratory homeostasis, and may be linked to the

232 for maintenance of carotid body function and cardiorespiratory homeostasis.

233 less than 10 mum (PM10) and daily emergency cardiorespiratory hospitalizations in Hong Kong, China,

234 his review provide objective evidence of the cardiorespiratory impairment associated with severe pect

235 Cardiorespiratory indices, including breath duration (TT

236 nursing alert was associated with decreased cardiorespiratory instability concern criteria in step-d

237 (INDEX) correlated with our single-parameter cardiorespiratory instability concern criteria, and whet

238 g system INDEX correlated significantly with cardiorespiratory instability concern criteria, usually

239 red parameters even transiently beyond local cardiorespiratory instability concern triggers (heart ra

240 altered sensory gating in nTS contributes to cardiorespiratory instability in RTT and that nTS is a s

241 Cardiorespiratory instability may be undetected in monit

242 applied to address earlier identification of cardiorespiratory insufficiency and direct focused, pati

243 niques can be used to parsimoniously predict cardiorespiratory insufficiency.

244 This cardiorespiratory interaction may arise from interaction

245 f H is evidenced by a respiratory influence (cardiorespiratory interaction) on heart rate variability

246 rrhythmia represent different aspects of the cardiorespiratory interaction, and that key physiologic

247 ho did not panic displayed signs of impaired cardiorespiratory interoception, including a complete ab

248 ions: a panic induction and an assessment of cardiorespiratory interoception.

249 n collaterals that arborize into neighboring cardiorespiratory locations and likely release norepinep

250 Cardiorespiratory measures showed that apnea improved du

251 inary assessment of efficacy using automated cardiorespiratory measures, EEG, a set of RTT-oriented c

252 ir pollution is associated with increases in cardiorespiratory morbidity and mortality in LMIC's, wit

253 articulate matter (PM2.5) is associated with cardiorespiratory morbidity and mortality, but the mecha

254 ne the effects of hypercapnia on the central cardiorespiratory network, we used an in vitro medullary

255 mergency department (ED) visits for selected cardiorespiratory outcomes were obtained for the five-co

256 ) was associated with ED visits for multiple cardiorespiratory outcomes, providing support for the ut

257 eleased by a subset of PVN neurons modulates cardiorespiratory output via V(1A) receptors in the RVLM

258 le, there were no significant differences in cardiorespiratory parameters between the CFS only group

259 tional analyses excluded that measured basic cardiorespiratory parameters or interoceptive sensitivit

260 Serial evaluations of vital signs, cardiorespiratory parameters, blood cultures, inflammato

261 We investigate whether and how cardiorespiratory phase synchronization (CRPS) responds

262 of SNA is important - being recruited under cardiorespiratory reflex conditions and elevated in the

263 ence sympathetic nervous system activity and cardiorespiratory reflex function in health and disease.

264 wo classes of SPNs in situ to their roles in cardiorespiratory reflex integration and have shown that

265 late these characteristics to their roles in cardiorespiratory reflex integration.

266 is involved in the operation of several key cardiorespiratory reflexes, contributes to central proce

267 ng than Wt in the hindbrain, most notably in cardiorespiratory regions of the nucleus tractus solitar

268 , and acidosis and play an important role in cardiorespiratory regulation.

269 Hypercapnia evokes a strong cardiorespiratory response including gasping and a prono

270 le of human brain structures controlling the cardiorespiratory response to exercise ('central command

271 cortical area in the neural circuitry of the cardiorespiratory response to exercise, since stimulatio

272 of three types of feedback afferents on the cardiorespiratory response to voluntary, rhythmic exerci

273 ifferences among the groups for any measured cardiorespiratory response, but perceptual differences i

274 hogenesis, how PNE affects the SLCF-mediated cardiorespiratory responses remains unexplored.

275 iorespiratory control and tempers excitatory cardiorespiratory responses to activation of the periphe

276 role of ORX in the anxiety-like behavior and cardiorespiratory responses to acute exposure to a thres

277 With matching for aerobic fitness, cardiorespiratory responses to exercise in patients with

278 n maternal smoking and SIDS, we examined the cardiorespiratory responses to hypercapnia in animals ex

279 PNE rat pups at postnatal days 11-14: 1) the cardiorespiratory responses to intralaryngeal applicatio

280 The persistent cardiorespiratory responses to LT-IH were associated wit

281 ts continuously rated the intensity of their cardiorespiratory sensation using a dial.

282 terenol elicited dose-dependent increases in cardiorespiratory sensation, with all participants repor

283 ympathetic arousal, heart rate increase, and cardiorespiratory sensation.

284 y a role in dynamically detecting changes in cardiorespiratory sensation.

285 Changes in cardiorespiratory status associated with postnatal CMV i

286 cemia, infection, and hypotension and elicit cardiorespiratory stimulation, adrenaline and adrenocort

287 imulation of these neurons produces vigorous cardiorespiratory stimulation, sighing, and arousal from

288 most effects of acute hypoxia, specifically cardiorespiratory stimulation, sighs, and arousal.

289 erentially respond during the peak period of cardiorespiratory stimulation.

290 nable period of time after the withdrawal of cardiorespiratory support (WCRS).

291 eath in less than 60 min after withdrawal of cardiorespiratory support conducted in 28 accredited int

292 Death within 60 minutes of withdrawal of cardiorespiratory support occurred in 377 (49.3%).

293 th within 60 minutes following withdrawal of cardiorespiratory support.

294 ing death within 60 minutes of withdrawal of cardiorespiratory support.

295 ients die within 60 minutes of withdrawal of cardiorespiratory support.

296 oxia (CIH), alterations in the regulation of cardiorespiratory system become persistent because of ch

297 e importance of therapeutic targeting of the cardiorespiratory system.

298 should consider both the prognostic value of cardiorespiratory testing and techniques to preserve car

299 Clinical assessment, diagnostic imaging, and cardiorespiratory testing of patients with pectus excava

300 Cardiorespiratory variables were continuously recorded t