ライフサイエンスコーパス: cardiotoxin

1 es before and after muscle injury induced by cardiotoxin.

2 d dramatically from 1 to 3 d after injury by cardiotoxin.

3 generation, we injured wild-type muscle with cardiotoxin and found that Akt induced a faster regenera

4 protected muscle from both acute injury with cardiotoxin and from chronic muscle disease in the mdx o

5 ted in PDGFRbeta(+) cells are protected from cardiotoxin and laceration-induced skeletal muscle fibro

6 nin-alpha2-deficient muscle was damaged with cardiotoxin and muscle regeneration quantified.

7 Duchenne muscular dystrophy, and 2 myotoxin (cardiotoxin and notexin) muscle injury models.

8 ble disulfide-containing proteins, including cardiotoxin and prion aggregates.

9 After cardiotoxin (CTX) injury, regenerating myofibers express

10 ng and in regenerating muscle in response to cardiotoxin (CTX)-induced injury.

11 bustly in regenerating skeletal muscle after cardiotoxin (CTX)-induced muscle injury in vivo and diff

12 ythroid-derived-2)-like 2 (Nrf2), aggravates cardiotoxin (CTX)-induced tibialis anterior (TA) muscle

13 We investigated the cardiotoxin (CTX)-mediated transient acute mouse model o

14 Cobra cardiotoxins (CTX) are a family of three-fingered basic

15 Cardiotoxins (CTXs) from cobra venom show cytotoxicity t

16 ffect skeletal muscle regeneration following cardiotoxin damage.

17 the discovery of a new insulinotropic agent, cardiotoxin-I (CTX-I), from the Naja kaouthia snake veno

18 rvival and wound healing in a mouse model of cardiotoxin induced muscle injury.

19 MyoR and miR-378 were anticorrelated during cardiotoxin-induced adult muscle regeneration in mice.

20 lt in impaired muscle regeneration following cardiotoxin-induced injury in mice.

21 ed cells and macrophages were assessed after cardiotoxin-induced injury of chimeric mice produced by

22 l when ablation occurred at the same time as cardiotoxin-induced injury.

23 wth and impaired regenerative capacity after cardiotoxin-induced injury.

24 o the tibialis anterior muscle 3 days before cardiotoxin-induced injury.

25 Two hours after cardiotoxin-induced muscle damage, local activin A prote

26 dels, we studied regeneration consecutive to cardiotoxin-induced muscle injury and observed a signifi

27 ers (MyoD, myogenin, and active-Notch) after cardiotoxin-induced muscle injury in vivo and in SCs cul

28 In a cardiotoxin-induced muscle injury model, lack of MKP-1 i

29 splayed attenuated muscle regeneration after cardiotoxin-induced muscle injury.

30 In vivo, acute cardiotoxin-induced muscle regeneration was enhanced in

31 licited by immunization with pVCL/MSP1a into cardiotoxin-induced regenerating muscle were evaluated i

32 ntiation and prolonged Pax7 expression after cardiotoxin-induced skeletal muscle injury, while single

33 ments revealed that Staufen1 increases after cardiotoxin injection before returning to the low levels

34 Following cardiotoxin injection, a known trigger for satellite cel

35 pha(-/-)) mice after injury by intramuscular cardiotoxin injection.

36 g in mouse tibialis anterior muscles after a cardiotoxin injection.

37 ersion of marrow cells to skeletal muscle in cardiotoxin-injured anterior tibialis muscle in a green

38 c gene expression and muscle regeneration in cardiotoxin-injured beta3-integrin-null mice are impaire

39 neration and that gene transfer of GEFT into cardiotoxin-injured mouse tibialis anterior muscle exert

40 ed that SP cells isolated from dystrophic or cardiotoxin-injured muscle fail to undergo myogenesis.

41 ute to muscle regeneration when grafted into cardiotoxin-injured muscle.

42 obust muscle regeneration when injected into cardiotoxin-injured skeletal muscle.

43 ransplantation of these committed cells into cardiotoxin-injured skeletal muscles of NOD/SCID mice re

44 d damage, exhibit delayed regeneration after cardiotoxin injury and suffer from defective myoblast fu

45 Rgamma-null mice in both critical defect and cardiotoxin injury models.

46 Timing of the cardiotoxin injury relative to the transplantation was c

47 Muscle regeneration was induced by cardiotoxin injury, and we evaluated satellite cell acti

48 njury and improved muscle regeneration after cardiotoxin injury, as well as increased satellite cell

49 vivo model of muscle regeneration following cardiotoxin injury, ectopic miR-431 injection greatly im

50 Within 3 days of cardiotoxin injury, GFP-positive mononuclear cells were

51 effect on muscle regeneration in response to cardiotoxin injury.

52 ubstantially delayed regeneration induced by cardiotoxin injury.

53 Here, we injected cardiotoxin into gastrocnemius muscle of Hmox1(+/+) and

54 as a delay of muscle regeneration following cardiotoxin-mediated injury.

55 Early ablation (day 1 after cardiotoxin) resulted in the smallest regenerated myofib

56 und delay in satellite cell activation after cardiotoxin treatment in alpha7 integrin-null animals wh

57 beta1 integrin plays in muscle regeneration, cardiotoxin was used to induce damage in the tibialis an

58 In an experimental mouse model, cardiotoxin was used to induce muscle injury and repair,