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1 me sequencing, with relatively low yield for cardiovascular abnormalities.
2 nsulin sensitivity, glucose intolerance, and cardiovascular abnormalities.
3 PCD have heterotaxy, and most of those have cardiovascular abnormalities.
4 altered feeding behavior and metabolism, and cardiovascular abnormalities.
5 age is associated with chronic metabolic and cardiovascular abnormalities.
6 is because they may be at increased risk for cardiovascular abnormalities.
7 that children infected with HIV-1 might have cardiovascular abnormalities.
8 pleiotropic defects, the most striking being cardiovascular abnormalities.
9 vironment has an important role in postnatal cardiovascular abnormalities.
10 associated with human pathologies involving cardiovascular abnormalities.
11 r the diagnosis and characterization of most cardiovascular abnormalities.
12 essels, and that Madh6 mutants have multiple cardiovascular abnormalities.
13 this deletion are normal and do not exhibit cardiovascular abnormalities.
14 null mutants also have a similar spectrum of cardiovascular abnormalities.
15 care physicians for the purpose of excluding cardiovascular abnormalities.
16 ed in its power to detect potentially lethal cardiovascular abnormalities.
17 tory of cardiovascular disease or structural cardiovascular abnormalities.
18 imited value in identification of underlying cardiovascular abnormalities.
19 hr after antibody treatment displayed severe cardiovascular abnormalities.
20 free of stroke and cardiac disease from the Cardiovascular Abnormalities and Brain Lesions (CABL) st
21 evaluated in 950 participants from the CABL (Cardiovascular Abnormalities and Brain Lesions) study by
23 dysfunction may contribute to metabolic and cardiovascular abnormalities and subsequent increases in
24 ography database, to characterize associated cardiovascular abnormalities, and to describe long-term
25 umerous structural, electrical, and acquired cardiovascular abnormalities are capable of causing SCD,
26 riety of unsuspected, clinically significant cardiovascular abnormalities are common in long-term sur
27 now a standard for management decisions when cardiovascular abnormalities are identified in trained a
29 hes for the treatment of selected congenital cardiovascular abnormalities associated with diabetes.
30 ential to detect (or raise the suspicion of) cardiovascular abnormalities capable of causing sudden d
31 ase of the homologous region in mouse causes cardiovascular abnormalities characteristic of the human
34 IV-1 infection is associated with persistent cardiovascular abnormalities identifiable shortly after
36 y deficiency of vitamin A is associated with cardiovascular abnormalities in avian and murine systems
37 rovide a synopsis of the current research on cardiovascular abnormalities in children with obesity an
40 y of, course of, and factors associated with cardiovascular abnormalities in pediatric HIV are incomp
42 lain the higher incidence of respiratory and cardiovascular abnormalities in the former condition.
43 nation among themselves, responsible for the cardiovascular abnormalities in velocardiofacial/DiGeorg
44 vasive detection and assessment of autonomic cardiovascular abnormality in obstructive sleep apnea sy
46 age analysis of Nt3-/- animals reveal severe cardiovascular abnormalities including atrial and ventri
47 ral crest results in perinatal lethality and cardiovascular abnormalities, including interrupted aort
48 idgestation with extreme hydrops fetalis and cardiovascular abnormalities, including overdeveloped ve
49 nalyses of Cited2(-/-) embryos reveal severe cardiovascular abnormalities, including pulmonic arteria
50 complete null phenotype, including multiple cardiovascular abnormalities involving the endocardial c
51 f 61 decedents (44%) had clinically relevant cardiovascular abnormalities, most frequently atheroscle
52 ibe heterozygously deleted (Df1/+) mice with cardiovascular abnormalities of the same type as those a
53 consequence of abnormal lung development and cardiovascular abnormalities, often leading to grave neu
55 ed of having cardiovascular disease, and the cardiovascular abnormality responsible for sudden death
56 moval of the cardiac neural crest results in cardiovascular abnormalities suggesting the lack of rege
57 ing cardiotoxic treatments nevertheless have cardiovascular abnormalities, systemic inflammation, and
58 more acutely lethal to salmon, a spectrum of cardiovascular abnormalities was consistently evident in
59 In addition to the previously documented cardiovascular abnormalities, we find that the somites a
61 Progress has been quite impressive for some cardiovascular abnormalities, whereas in other areas the
62 eotide injection caused a wavy notochord and cardiovascular abnormalities with a reduced heart rate a
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