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1 s 8 [0-83], P<0.0001; similar for hard/total cardiovascular events).
2  individuals >/=30 years of age with a prior cardiovascular event.
3 ular function, which may lower the risk of a cardiovascular event.
4 raphy/computed tomography in vivo and future cardiovascular events.
5 tabolic activity predicts risk of subsequent cardiovascular events.
6    Secondary outcomes included major adverse cardiovascular events.
7 with significantly lower mortality and fewer cardiovascular events.
8 d 6 months were at the lowest risk of future cardiovascular events.
9 ne particle (PM2.5) pollution triggers acute cardiovascular events.
10 d discrimination index, especially for total cardiovascular events.
11 s infections, malignancies, or major adverse cardiovascular events.
12 of medication, and in-hospital and long-term cardiovascular events.
13 signed to improve lipid profiles and prevent cardiovascular events.
14 superior to clopidogrel for the reduction of cardiovascular events.
15 was associated with a >30% increased risk of cardiovascular events.
16 rs, dual antiplatelet therapy, and long-term cardiovascular events.
17 activity are associated with a lower risk of cardiovascular events.
18 use in all-cause mortality and major adverse cardiovascular events.
19 d intolerance to statins have a high risk of cardiovascular events.
20 ears; 58% women) who experienced 13821 major cardiovascular events.
21 ole in the pathogenesis of platelet-mediated cardiovascular events.
22                    Odds ratio (OR) for major cardiovascular events.
23  Magmaris will result in reductions in major cardiovascular events.
24  and that PCTP expression is correlated with cardiovascular events.
25 al stiffness is an important risk factor for cardiovascular events.
26 ignificant coronary artery disease (CAD) and cardiovascular events.
27 these agents neither increased nor decreased cardiovascular events.
28 FR, and angiographic CAD severity on adverse cardiovascular events.
29 tion and treatment gaps increase the risk of cardiovascular events.
30 bo did not reduce the risk of major ischemic cardiovascular events.
31 o improve adherence may significantly reduce cardiovascular events.
32 the only independent factors associated with cardiovascular events.
33 ol (LDL-C) predicts both cerebrovascular and cardiovascular events.
34 er-intensity statins in adults without prior cardiovascular events.
35 luated aspirin for the primary prevention of cardiovascular events.
36 therosclerosis, coronary artery disease, and cardiovascular events.
37 as independent predictors of device-oriented cardiovascular events.
38 w-density lipoprotein cholesterol (LDL-C) on cardiovascular events.
39 urn could lead to reduced risk of downstream cardiovascular events.
40 in RA restores autonomic balance and reduces cardiovascular events.
41 ctor 15 identify patients at highest risk of cardiovascular events.
42 rgine with respect to the incidence of major cardiovascular events.
43 ements related to the prevention of clinical cardiovascular events.
44 g a perilous concurrence of risk factors for cardiovascular events.
45 0.78 mmol per liter) and reduced the risk of cardiovascular events.
46 t is likely to be recovered by prevention of cardiovascular events.
47 infarction, stroke, heart failure, and major cardiovascular events.
48 d cohort for risk prediction of coronary and cardiovascular events.
49 t could modulate the risk of atherosclerotic cardiovascular events.
50 %) hard cardiovascular, and 241 (7.3%) total cardiovascular events.
51  a higher frequency of diabetes mellitus and cardiovascular events.
52  (HR 0.53 [95% CI 0.40-0.71]), major adverse cardiovascular events (0.78 [0.69-0.87]), and hospital e
53 n cardiac death [1.70 (1.34 to 2.15)], first cardiovascular event (1.15 [1.01 to 1.32]), and any-caus
54 d high rates of hard coronary and hard/total cardiovascular events (10-year risk: 12.0%, 13.5%, and 3
55 es, 82 and 94 diabetes mellitus, 114 and 129 cardiovascular events, 119 and 147 non-AIDS malignancies
56 , 0.57; 95% CI, 0.50-0.65) and major adverse cardiovascular events (2.31 versus 3.45 events per 100 p
57 ce interval [CI], 1.01 to 1.07; P=0.01), any cardiovascular event (2727 events; hazard ratio, 1.04; 9
58  outcomes included in-hospital major adverse cardiovascular events, 30-day mortality, and 1-year mort
59 ave lower rates of in-hospital major adverse cardiovascular events, 30-day mortality, and 1-year mort
60 th angiography), with no difference in major cardiovascular event (8.0% versus 11.6%; P=0.20) or symp
61  with ACS as in patients with non-ACS (major cardiovascular event, 8.0% versus 8.5%; P=0.83; revascul
62 coronary event was 64% higher, the risk of a cardiovascular event 85% higher, death 124% higher, myoc
63                  Secondary outcomes were any cardiovascular event (a composite of any coronary event,
64                The primary outcome was major cardiovascular events (a composite of death, myocardial
65 dently associated with a 24% greater risk of cardiovascular events (adjusted hazard ratio [aHR], 1.24
66 zetimibe, when added to simvastatin, reduces cardiovascular events after acute coronary syndrome.
67                   However, current trends in cardiovascular events after kidney transplantation are p
68 anada, to determine whether the incidence of cardiovascular events after kidney transplantation has c
69 ial disease (PAD) have high rates of adverse cardiovascular events after percutaneous coronary interv
70 ng stroke after PVI, and explore the risk of cardiovascular events after PVI in patients with and wit
71 , when added to simvastatin therapy, reduces cardiovascular events after recent acute coronary syndro
72 the hazard ratio for all-cause mortality and cardiovascular events against the mean on-treatment SBP
73 of significant absolute increases of 1.2% in cardiovascular events and 0.4% in mortality with torcetr
74                          Those with interval cardiovascular events and a dilated LV (increased LV end
75 es that nowadays patients more often survive cardiovascular events and a major number dies from cance
76 pared with <140 mm Hg reduced rates of major cardiovascular events and all-cause death without eviden
77 of the J curve was present, with a nadir for cardiovascular events and all-cause mortality just below
78 ent SBP levels are associated with increased cardiovascular events and all-cause mortality.
79 ted with reduced mortality and incidences of cardiovascular events and cancer in obese individuals.
80 pproximately 2-fold increased risk of future cardiovascular events and cardiovascular death.
81  new insights into stratification of risk of cardiovascular events and death among patients with chro
82 ntly lower rates of fatal and nonfatal major cardiovascular events and death from any cause.
83 F had markedly increased risk for subsequent cardiovascular events and death, highlighting the import
84 eported a reduction in 3-point major adverse cardiovascular events and HF hospitalization risk.
85 ization for heart failure, and major adverse cardiovascular events and higher risk of below-knee lowe
86  essential hypertension, the excess risk for cardiovascular events and mortality was limited to patie
87 ified by cardiac imaging, is associated with cardiovascular events and predisposes to the development
88 cy is associated with decreased incidence of cardiovascular events and VTE.
89  SYNTAX scores predict higher rates of major cardiovascular events and were associated with more favo
90 s (defined by age and occurrence of previous cardiovascular event) and 1 study with admixed populatio
91 ion, urinary albumin concentration, previous cardiovascular event, and hypertension treatment time (o
92  for cardiovascular events but with no prior cardiovascular event, and the secondary prevention cohor
93  respect to baseline IHD, outcomes (IHD, HF, cardiovascular events, and all-cause death), and EF chan
94 association between on-treatment SBP levels, cardiovascular events, and all-cause mortality in patien
95 iabetes or type 2 diabetes], hyperlipidemia, cardiovascular events, and chronic kidney disease) (mean
96  of CHB patients in terms of hepatic events, cardiovascular events, and death remains unknown.
97 ween AKI and cardiovascular mortality, major cardiovascular events, and disease-specific events: cong
98 D increased the risk of death, major adverse cardiovascular events, and major bleeding but did not af
99 e potassium concentrations with arrhythmias, cardiovascular events, and mortality.
100  reported all-cause mortality, major adverse cardiovascular events, and other outcomes of interest.
101        Patients with PAD are at high risk of cardiovascular events, and PCSK9 inhibition with evolocu
102 or posttransplant metabolic syndrome (PTMS), cardiovascular events, and renal insufficiency.
103 on is critical for the reduction of repeated cardiovascular events, and the control of cardiovascular
104                            Hard coronary and cardiovascular events, and total cardiovascular events i
105                                              Cardiovascular events are associated with significant mo
106 y have a similar crude rate of major adverse cardiovascular events as those with type 1 myocardial in
107 sation, and associated risk of major adverse cardiovascular events at 1 year.
108 fts with regard to mortality or the rates of cardiovascular events at 5 years of follow-up.
109                    The rate of major adverse cardiovascular events at 5 years was 31.0% in the off-pu
110 ovides moderately strong evidence of reduced cardiovascular events at the expense of increased bleedi
111 ions with TFA restrictions experienced fewer cardiovascular events, beyond temporal trends, compared
112 /=50 years of age with >/=2 risk factors for cardiovascular events but with no prior cardiovascular e
113 sured with ultrasound correlates with future cardiovascular events, but conventional ultrasound imagi
114 ose did not reduce the risk of major adverse cardiovascular events, but did reduce the incidence of d
115 sis are crucially involved in development of cardiovascular events, but little is known about the inf
116 ; 95% CI, 0.89-1.37) and individual risks of cardiovascular events, but the risk of sternal wound inf
117 t disease by 27% (P=0.002) and major adverse cardiovascular events by 25% (P=0.004) consistently amon
118 t disease by 27% (P=0.033) and major adverse cardiovascular events by 25% (P=0.037) during the initia
119  we investigated the effect of evolocumab on cardiovascular events by diabetes status at baseline, de
120 bute to the clinically observed reduction in cardiovascular events by evaluating its effect on inflam
121  Statins are highly effective for preventing cardiovascular events by reducing low-density lipoprotei
122            Many assume that the reduction in cardiovascular events can be attributed to the anti-infl
123 were cardiovascular mortality, major adverse cardiovascular events (cardiovascular mortality, myocard
124 s class can reduce three-point major adverse cardiovascular events, cardiovascular mortality, and all
125 cts of canakinumab on rates of major adverse cardiovascular events, cardiovascular mortality, and all
126 e did not significantly reduce major adverse cardiovascular events compared with asprin alone, but re
127  neither increased nor decreased the risk of cardiovascular events compared with placebo in patients
128 use associated with increased 30-day adverse cardiovascular events compared with the lowest tertile.
129 frequently displayed target organ damage and cardiovascular events compared with those without PA, in
130 owed until 31 December 2015 for incidence of cardiovascular events (composite of myocardial infarctio
131 9 (PCSK9) has been shown to be predictive of cardiovascular events (CVEs) in patients who are at high
132 y (32.2%) patients experienced intrahospital cardiovascular events (CVEs) including 281 (23.8%) with
133 the first occurrence of an adjudicated major cardiovascular event (death from cardiovascular causes,
134 prospectively followed for the occurrence of cardiovascular events (death, heart failure, hospitaliza
135 ion, the increase in work disability after a cardiovascular event decreases close to the pre-event le
136 ation for heart failure event, major adverse cardiovascular events (defined as all-cause mortality, n
137          The primary outcome was an incident cardiovascular event, defined as a composite of incident
138 use and a composite outcome of major adverse cardiovascular events, defined as death from any cause,
139 tients with type 2 diabetes at high risk for cardiovascular events, degludec was noninferior to glarg
140 ttack (TIA) are at increased risk for future cardiovascular events despite current preventive therapi
141 tic vascular disease remain at high risk for cardiovascular events despite effective statin-based tre
142 e population and improved treatment of acute cardiovascular events, despite the efficacy of many ther
143 ge, lower income, and an absence of previous cardiovascular events, diabetes, obesity, or alcohol use
144 tients with low SYNTAX scores (</=22), major cardiovascular events did not differ significantly betwe
145 ular disease, the incidence of major adverse cardiovascular events did not differ significantly betwe
146 summary, compared to TASC, the proportion of cardiovascular events did not markedly decrease over the
147                               (Prevention of Cardiovascular Events [e.g., Death From Heart or Vascula
148    Secondary outcomes included major adverse cardiovascular events (eg, nonfatal myocardial infarctio
149  MF noted on CMR had a higher probability of cardiovascular events (event rate, 10 of 55 [18.2%] vs 0
150 8 (17%) patients, with the most common being cardiovascular events (four patients [4%]).
151 85% of the overall study population) averted cardiovascular events from a behavioural intervention ai
152 ues are associated with an increased risk of cardiovascular events, giving rise to the so-called J-cu
153 ents with type 2 diabetes mellitus and prior cardiovascular events had higher rates of cardiovascular
154 -year cumulative incidence of death or major cardiovascular event has remained stable over time.
155 ; 95% confidence interval, 1.10 to 1.72) and cardiovascular events (hazard ratio, 1.23; 95% confidenc
156 ) was associated with a higher risk of major cardiovascular events (hazard ratio: 1.36, confidence in
157 ndependently associated with secondary major cardiovascular events (hazard ratio=2.28; 95% confidence
158 ), and 3.09 (0.96 to 8.78) for major adverse cardiovascular events, hospitalizations, and vascular ac
159 the study medications) a lower risk of major cardiovascular events; however, they also had lower isch
160  Patients with SVRs also had a lower risk of cardiovascular events (HR, 0.42; 95% CI, 0.25-0.69; P =
161  [CI]:1.26 to 1.56; p <0.001), major adverse cardiovascular events (HR: 1.29; 95% CI: 1.16 to 1.43; p
162 s associated with a significant reduction in cardiovascular events in 18144 patients after acute coro
163 ndependently associated with secondary major cardiovascular events in a severely atherosclerotic popu
164 T, and growth-differentiation factor 15 with cardiovascular events in ACHD.
165 ctor of future major adverse atherosclerotic cardiovascular events in asymptomatic individuals.
166 ether acarbose could reduce the frequency of cardiovascular events in Chinese patients with establish
167 poprotein (HDL) cholesterol levels to reduce cardiovascular events in clinical trials have led to inc
168 in (hs-CRP) is independently associated with cardiovascular events in coronary artery disease (CAD) p
169 lude that TMAO concentration associates with cardiovascular events in hemodialysis patients but the e
170 r the prevention of stroke and other adverse cardiovascular events in IMPROVE-IT (Improved Reduction
171 e that plasma MGO levels are associated with cardiovascular events in individuals with type 1 diabete
172               In comparison with adjudicated cardiovascular events in MESA, the concordance rates for
173  Considering the high risk for major adverse cardiovascular events in patients receiving hemodialysis
174 n aspirin in preventing recurrent stroke and cardiovascular events in patients with acute cerebral is
175 We aimed to investigate the risk of incident cardiovascular events in patients with primary aldostero
176                               (Prevention of Cardiovascular Events in Patients with Prior Heart Attac
177              The PEGASUS-TIMI (Prevention of Cardiovascular Events in Patients with Prior Heart Attac
178  14,355) from PEGASUS-TIMI 54 (Prevention of Cardiovascular Events in Patients With Prior Heart Attac
179 alongside the PEGASUS-TIMI 54 (Prevention of Cardiovascular Events in Patients With Prior Heart Attac
180 orapaxar) and PEGASUS-TIMI 54 (Prevention of Cardiovascular Events in Patients With Prior Heart Attac
181 osis factor-alpha blockade appears to reduce cardiovascular events in patients with severe psoriasis.
182 Evolocumab was equally effective in reducing cardiovascular events in patients with stable atheroscle
183 tries have shown reductions in major adverse cardiovascular events in psoriasis patients and rheumato
184 haemodialysis (HD) have an increased risk of cardiovascular events in the first year after starting H
185 uced low-density lipoprotein cholesterol and cardiovascular events in the FOURIER trial (Further Card
186 bitor evolocumab reduced LDL cholesterol and cardiovascular events in the FOURIER trial.
187 ation for the primary prevention of clinical cardiovascular events in the general population have not
188 onocyte-derived macrophages, predicts future cardiovascular events in the general population.
189 logy, could help to explain residual risk of cardiovascular events in the general population.
190                         There were 2 adverse cardiovascular events in the metoprolol group and none i
191                     There were modestly more cardiovascular events in the MMF/SRL group.
192 sociation between atopic dermatitis (AD) and cardiovascular events in the Nurses' Health Study 2, a c
193 had no benefit with respect to major adverse cardiovascular events in the trial involving lower-risk
194 as an independent predictor of major adverse cardiovascular events in those with type 2 myocardial in
195                                              Cardiovascular events including myocardial infarction, i
196 oronary and cardiovascular events, and total cardiovascular events including revascularization, as we
197 ral, these patients have elevated risk for a cardiovascular event (including acute coronary syndrome,
198 HF had markedly increased risk of subsequent cardiovascular events, including a 27-fold increase (P<0
199 ) for a composite end point of major adverse cardiovascular events, including cardiovascular death an
200                                        Major cardiovascular events, including nonfatal myocardial inf
201            We also noticed that the risk for cardiovascular events increased with increasing number o
202 dimensional speckle tracking predicts future cardiovascular events independent of conventional risk f
203 d with higher mortality and a higher rate of cardiovascular events independent of traditional cardiov
204  reduced regenerative capacity contribute to cardiovascular events, independent of conventional risk
205 ipid and lipoprotein levels, and the risk of cardiovascular events involving 102837 participants from
206 onation, the risk of all-cause mortality and cardiovascular events is no higher than in healthy nondo
207                          Whether it prevents cardiovascular events is uncertain.
208 elial dysfunction, a major predictor of late cardiovascular events, is linked to the severity of obst
209 owever, an abnormal FT was more specific for cardiovascular events, leading to overall similarly mode
210 identified those patients at highest risk of cardiovascular events (log-rank P<0.0001).
211 control (<120 mm Hg) had fewer major adverse cardiovascular events (MACE) and deaths but higher rates
212 formed multivariable model for major adverse cardiovascular events (MACE) and determined the continuo
213       We prospectively studied major adverse cardiovascular events (MACE) at 2 years in 607 patients
214 t is associated with increased major adverse cardiovascular events (MACE) in the setting of acute cor
215                                Major adverse cardiovascular events (MACE) were defined as the primary
216 of a family history of AF with major adverse cardiovascular events (MACE).
217  incident type 1 and T2MI, and major adverse cardiovascular events (MACE, a composite of all-cause de
218                Cardiovascular (major adverse cardiovascular events [MACE], cardiovascular mortality,
219 ar AF were recruited and rates of thrombotic/cardiovascular events, major bleeding and mortality were
220 he efficacy of NHE inhibitors on the risk of cardiovascular events may be enhanced when heart failure
221 ale (ESS) scores, blood pressure, mortality, cardiovascular events, motor vehicle crashes, quality of
222  2 mg/L had a 25% reduction in major adverse cardiovascular events (multivariable adjusted hazard rat
223 ite outcome of posttransplant death or major cardiovascular event (myocardial infarction, coronary an
224 o compare all-cause mortality, major adverse cardiovascular events, myocardial infarction (MI), or ta
225  (n = 1094; 21%), and those with preexisting cardiovascular events (n = 1620; 35%).
226 .3 pmol/L) was most strongly associated with cardiovascular events (n=165, adjusted hazard ratio, 9.0
227 ed diabetes mellitus, chronic renal failure, cardiovascular events, NLR-NAR cancer, bone events, and
228 , 1.34-1.41), corresponding to an additional cardiovascular event observed per year in 1 of every 74
229      During a median follow-up of 3.3 years, cardiovascular events occurred in 1014 patients (7.3%),
230 nd the median follow-up was 7 months), major cardiovascular events occurred in 173 patients each in t
231 d the median follow-up was 12 months), major cardiovascular events occurred in 179 and 224 patients,
232                             However, serious cardiovascular events occurred in 22.8% of aspirin users
233                                        Major cardiovascular events occurred in 57 patients (2.5%) who
234                             No major adverse cardiovascular events occurred in either group.
235 in one who received placebo, and adjudicated cardiovascular events occurred in five who received tofa
236 g a median of 3.0 years of follow-up, 62 690 cardiovascular events occurred.
237  Despite finding no significant reduction in cardiovascular events on average, it is possible that so
238 re no deaths and four positively adjudicated cardiovascular events, one (3%) among patients on aphere
239 increase, respectively) but not with risk of cardiovascular events or graft failure.
240 on between sodium intake and cardiovascular (cardiovascular events or mortality) and renal (end-stage
241 rval [CI], 0.55-1.12; P=0.19), major adverse cardiovascular events (OR, 0.73, CI, 0.51-1.05; P=0.09),
242 ding attenuated nonsignificant risk of major cardiovascular events (OR, 0.985 [95% CI, 0.955-1.015]).
243 e of the composite outcome or death-censored cardiovascular events over time (P = 0.41 and 0.92, resp
244  profile (and numerically the lowest rate of cardiovascular events) over a 6-year period compared wit
245 5), history of smoking (P = 0.01), and prior cardiovascular events (P = 0.0004).
246 d risk of cardiovascular mortality and major cardiovascular events, particularly heart failure and ac
247 rum calcium levels, may increase the risk of cardiovascular events, particularly myocardial infarctio
248 tatins under both guidelines experienced 9.6 cardiovascular events per 1000 patient-years, those indi
249 on between the HMGCR score and risk of major cardiovascular events per unit change in levels of LDL-C
250 incidence, and (3) identify risk factors for cardiovascular events post-liver transplantation.
251 sease were the most consistent predictors of cardiovascular events posttransplant (significant in 8/2
252  phenotyping improves prediction accuracy in cardiovascular event prediction in an initially asymptom
253 rs-has been reported to decrease the risk of cardiovascular events primarily by reducing the risk of
254 k reduction in the three-point major adverse cardiovascular event primary outcome (cardiovascular mor
255                    Major adverse cardiac and cardiovascular event rate at 30 days was low (4%) and oc
256 ignificant step-up increase in major adverse cardiovascular events rate was observed across the 3 FFR
257 is finding, the observed crude major adverse cardiovascular event rates were similar between groups (
258  interleukin-1beta, reduces inflammation and cardiovascular event rates with no effect on lipid conce
259  progression is associated with coronary and cardiovascular event rates, but adds only weakly to risk
260  SBP level of lower than 120 mm Hg decreases cardiovascular event rates.
261 L-C (and other lipoproteins) and the risk of cardiovascular events related to variants in the CETP ge
262                                              Cardiovascular events represent a major source of morbid
263 d risk of cardiovascular mortality and major cardiovascular events, respectively ([RR 1.86; 95% confi
264 +)CD133(+): low versus high levels predicted cardiovascular events, restenosis after endovascular int
265 nd comorbidities, the risk of death or major cardiovascular event steadily declined across the years
266 cant associations were found between vWF and cardiovascular events, stroke, mortality and bleeding.
267 ypertension, atherosclerosis, and associated cardiovascular events such as myocardial infarction, str
268 eased plasma TMAO concentrations and adverse cardiovascular events, such as myocardial infarction, st
269  cholesterol (LDL-C) levels without reducing cardiovascular events, suggesting that the clinical bene
270 erence in all-cause mortality, major adverse cardiovascular events, target vessel revascularization,
271  clopidogrel monotherapy had a lower risk of cardiovascular events than those receiving aspirin.
272 ho discontinued aspirin had a higher rate of cardiovascular events than those who continued (multivar
273 eated with canagliflozin had a lower risk of cardiovascular events than those who received placebo bu
274  and apoB levels and a corresponding risk of cardiovascular events that was proportional to the atten
275 s 0.55 [0.34-0.90]), while for major adverse cardiovascular events the HR in the group with cardiovas
276  baseline and 6 months, and the incidence of cardiovascular events through 24 months (P<0.001 for eac
277                         Examining cumulative cardiovascular events (total burden of disease) gives a
278 o identify surgical subtypes associated with cardiovascular events using a large administrative datab
279 es Study (DPPOS, n=1018; 1996-2001), and for cardiovascular events using data from the Action for Hea
280 activity, the adjusted hazard ratio (HR) for cardiovascular events was 0.52 (95% confidence interval
281                             The incidence of cardiovascular events was higher in patients with primar
282 rated fatty acid supplementation on clinical cardiovascular events, we update prior recommendations f
283 ng percentages for the composite of death or cardiovascular event were 4.9% and 7.1% at 3-year follow
284  on coronary heart disease and major adverse cardiovascular events were assessed over the 4.9-year ra
285 rospectively followed SCAD cohort, long-term cardiovascular events were common.
286 ed as stenosis greater than or equal to 50%; cardiovascular events were defined as a composite of car
287                         Trends in deaths and cardiovascular events were estimated with Cox regression
288                        The associations with cardiovascular events were externally validated in 18953
289                                              Cardiovascular events were identified with the Swedish i
290  with mid/high SYNTAX scores, however, major cardiovascular events were lower after revascularization
291 lar risk, but without diabetes, the rates of cardiovascular events were lower among those who were as
292                                     Incident cardiovascular events were observed among 70 patients (2
293       The rates of in-hospital major adverse cardiovascular events were significantly lower in patien
294 rwent surgical adrenalectomy, had a previous cardiovascular event, were not treated with MR antagonis
295  study progression of subclinical disease to cardiovascular events where participants were initially
296 provided evidence of benefit in reduction of cardiovascular events with these agents.
297 ant reduction in time to first major adverse cardiovascular event) with the GLP-1R agonists liragluti
298 3 m2), and a composite of all-cause death or cardiovascular event, with surveillance every 3 months.
299 ecipients (9.0%) died or experienced a major cardiovascular event within 3 years of transplantation.
300 igh school, and college and above], previous cardiovascular events [yes or no], current smoker [yes o

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