ライフサイエンスコーパス: cardiovascular physiology

1 tion may provide valuable insights into IUGR cardiovascular physiology.

2 INTS: Wave travel plays an important role in cardiovascular physiology.

3 nsive role of these small regulatory RNAs in cardiovascular physiology.

4 ant regulators of renal sodium transport and cardiovascular physiology.

5 r (GPCR) Agtrl1b, a known regulator of adult cardiovascular physiology.

6 he wider context of vascular development and cardiovascular physiology.

7 re consequences of fundamental principles of cardiovascular physiology.

8 s (betaARs) are critical regulators of acute cardiovascular physiology.

9 gh altitude and developed the field of fetal cardiovascular physiology.

10 ous-flow LVADs alter blood flow dynamics and cardiovascular physiology and can induce progression of

11 gical molecules that play important roles in cardiovascular physiology and contribute to disease init

12 fine the key roles that mitochondria play in cardiovascular physiology and disease and provide insigh

13 iotensin II (Ang II) plays a central role in cardiovascular physiology and disease.

14 nt statins were shown to improve biomarkers, cardiovascular physiology and lung function in animal mo

15 ing the effects exerted by mTOR signaling in cardiovascular physiology and pathological states.

16 R pathway plays a key regulatory function in cardiovascular physiology and pathology.

17 icate that sex has an important influence on cardiovascular physiology and pathology.

18 , content, and transmission, with a focus on cardiovascular physiology and pathophysiology.

19 dase (Nox) enzymes are critical mediators of cardiovascular physiology and pathophysiology.

20 ones have emerged as important modulators of cardiovascular physiology and pathophysiology.

21 indered studies on the role of endothelin in cardiovascular physiology and pathophysiology.

22 use technology provide novel models to study cardiovascular physiology and pathophysiology.

23 ese pathways, RGS proteins are regulators of cardiovascular physiology and potentially novel drug tar

24 nderstood how altered ACTC structure affects cardiovascular physiology and results in the development

25 tability, hypothalamic-pituitary signalling, cardiovascular physiology and sympathetic function.

26 r actions of PARs that play a role in normal cardiovascular physiology and that are likely to contrib

27 he short-term effects of estrogen central to cardiovascular physiology are mediated by ERalpha functi

28 role of exosomes and microvesicles in normal cardiovascular physiology, as well as during cardiovascu

29 Cardiovascular physiology at rest and during acute stres

30 ions or polymorphisms in genes that regulate cardiovascular physiology, blood coagulation, lipid meta

31 ssociated with acute, unfavorable effects on cardiovascular physiology, but few studies have examined

32 Cardiovascular physiology changes during embryonic devel

33 ct evidence for a role of Cyp 4a isoforms in cardiovascular physiology, establish Cyp 4a14 (-/-) mice

34 x differences are apparent at every level of cardiovascular physiology from action potential duration

35 nsation), sound (hearing), and shear stress (cardiovascular physiology); however, the identity of the

36 hours every day) on neural, peripheral, and cardiovascular physiology in Drosophila melanogaster.

37 inflammation and thrombosis and measures of cardiovascular physiology in healthy young persons.

38 pothyroidism often cause opposing effects on cardiovascular physiology in the short term.

39 ing PKA phosphorylation at this site impairs cardiovascular physiology in vivo, leading to reduced ex

40 xpress estrogen receptors, but their role in cardiovascular physiology is not well understood.

41 clear that, in addition to effects on adult cardiovascular physiology, lipids also play key roles in

42 The role played by nitric oxide (NO) in cardiovascular physiology remains highly controversial.

43 de (NO) signaling regulates key processes in cardiovascular physiology, specifically vasodilation, pl

44 hibits pharmacological properties related to cardiovascular physiology that are distinct from nitric

45 NO is a major regulator of cardiovascular physiology that reduces vascular and card