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1 s been associated with chronic damage to the cardiovascular system.
2 M in general with specific references to the cardiovascular system.
3  the control of vascular tone throughout the cardiovascular system.
4 in the physiology and pathophysiology of the cardiovascular system.
5 of elastic fibers in the skin, eyes, and the cardiovascular system.
6 hanism of disease and redox signaling in the cardiovascular system.
7 as HFHS diets are largely deleterious to the cardiovascular system.
8 en to tissues is the primary function of the cardiovascular system.
9  new understanding of how COX-2 protects the cardiovascular system.
10  decreased the anti-inflammatory EETs in the cardiovascular system.
11 lood is coordinated with the response of the cardiovascular system.
12 chemic diseases, particularly of the cerebro-cardiovascular system.
13 e context of remote organ connections of the cardiovascular system.
14  and post-transcriptional events also in the cardiovascular system.
15 (CSE) produces hydrogen sulfide (H2S) in the cardiovascular system.
16  endothelial MEF2 function in the developing cardiovascular system.
17 elopmental and pathological processes in the cardiovascular system.
18 timerin genes, EMILIN-3 was not found in the cardiovascular system.
19 nctions they have been shown to exert in the cardiovascular system.
20 n, and the increased oxidative stress in the cardiovascular system.
21  II in the endothelium is detrimental to the cardiovascular system.
22 ge of physiological roles, especially in the cardiovascular system.
23 or to the development and homeostasis of the cardiovascular system.
24 sted whether ozone exposure can modulate the cardiovascular system.
25 actor (NGF) exerts protective effects on the cardiovascular system.
26 the adverse effects of dietary sodium on the cardiovascular system.
27 ck the beneficial effects of estrogen on the cardiovascular system.
28 orted beneficial effects of vitamin D in the cardiovascular system.
29 possess important beneficial activity in the cardiovascular system.
30 ely smooth muscle-specific expression in the cardiovascular system.
31 hat is being explored more frequently in the cardiovascular system.
32 ne monophosphate (cAMP) signaling within the cardiovascular system.
33      Estrogen has pleiotropic effects on the cardiovascular system.
34  major source of superoxide (O(2)(-)) in the cardiovascular system.
35  emerging evidence of its involvement in the cardiovascular system.
36 otential adverse effects of hyperoxia on the cardiovascular system.
37 n virtually all major functions of NO in the cardiovascular system.
38  understanding of the molecular basis of the cardiovascular system.
39 he immune system, the nervous system and the cardiovascular system.
40 quired for development and adaptation of the cardiovascular system.
41 rimary manifestations in the skin, eyes, and cardiovascular system.
42 damental significance to the function of the cardiovascular system.
43 ic activity in the skeleton and in the renal-cardiovascular system.
44 ifestations primarily in the skin, eyes, and cardiovascular system.
45  the adverse effects of air pollution on the cardiovascular system.
46 mbryogenesis, particularly in the developing cardiovascular system.
47 ilatory and antiproliferative actions in the cardiovascular system.
48 neficial effects of a high-K(+) diet for the cardiovascular system.
49 y for survival, as well as the health of the cardiovascular system.
50 le in the development and homeostasis of the cardiovascular system.
51 ecting neurons implicated in fine-tuning the cardiovascular system.
52 nd necrosis) as they relate to the heart and cardiovascular system.
53 ic oxide (NO) is a potent vasodilator in the cardiovascular system.
54  functional or structural alterations in the cardiovascular system.
55 P is an important endogenous mediator in the cardiovascular system.
56 hysiology, and diseases such as those of the cardiovascular system.
57 bute to endothelial dysfunction in the aging cardiovascular system.
58 in how selective COX2 inhibition impacts the cardiovascular system.
59 or role in regulating cAMP metabolism in the cardiovascular system.
60 al targets for therapeutic modulation of the cardiovascular system.
61 IV protease inhibitors negatively impact the cardiovascular system.
62 fy the effect of metal-rich particles on the cardiovascular system.
63 selected imaging technologies focused on the cardiovascular system.
64 critical for preserving the integrity of the cardiovascular system.
65  role in the development and function of the cardiovascular system.
66 e associated with detrimental effects on the cardiovascular system.
67 iotropic effects of statins that benefit the cardiovascular system.
68 are major mediators of cGMP signaling in the cardiovascular system.
69 ant site modulating autonomic outflow to the cardiovascular system.
70 her singly or in combination, can affect the cardiovascular system.
71 emands, and this places an extra load on the cardiovascular system.
72  in RGS-regulated G protein signaling in the cardiovascular system.
73 ebrate mesoderm leading to the origin of the cardiovascular system.
74  organisms, specifically in the case for the cardiovascular system.
75 lar phenotypes that characterize the failing cardiovascular system.
76 t role in modulating oxidative stress in the cardiovascular system.
77 This was found in many organs, including the cardiovascular system.
78 variation underlying giraffe's long neck and cardiovascular system.
79 e respiratory system and vasodilation in the cardiovascular system.
80 focuses on the role of miRs in ageing of the cardiovascular system.
81 nervous system to other sites, including the cardiovascular system.
82 arditis, a life-threatening infection of the cardiovascular system.
83 ith primary aldosteronism (PA) impairs their cardiovascular system.
84 P4 and the effects of DPP4 inhibitors on the cardiovascular system.
85 in various tissues and organs, including the cardiovascular system.
86 lterations in the structure of the mammalian cardiovascular system.
87 ange--supposedly mediated by reflexes to the cardiovascular system.
88 mited regenerative capacity of the mammalian cardiovascular system.
89 n factor that is critical for patterning the cardiovascular system.
90 se, implying important reorganization of the cardiovascular system.
91 s that are involved in the regulation of the cardiovascular system.
92 ntral role in the delicate regulation of the cardiovascular system.
93 cling is critical for normal function of the cardiovascular system.
94 as become a major model for the study of the cardiovascular system.
95  of these sizes may harm the respiratory and cardiovascular systems.
96 entially protecting the immune, nervous, and cardiovascular systems.
97  manifestations in the ocular, skeletal, and cardiovascular systems.
98  and homeostasis of the nervous, immune, and cardiovascular systems.
99  multiple diseases affecting the nervous and cardiovascular systems.
100 t not exclusively affect central nervous and cardiovascular systems.
101 x interaction exists between the nervous and cardiovascular systems.
102 ory and excitatory events in the nervous and cardiovascular systems.
103 tanding of the anatomy and physiology of the cardiovascular system, along with advances in imaging te
104 es TGF-beta1-induced PAI-1 expression in the cardiovascular system and adipose tissue.
105 oRNAs (miRNA) as fine-tune regulators in the cardiovascular system and bone biology has gained accept
106  receptors in nonreproductive tissues, e.g., cardiovascular system and bones, implies that the increa
107 processes integral to the development of the cardiovascular system and cardiovascular diseases.
108 ction and a variety of other benefits in the cardiovascular system and elsewhere.
109                               Effects on the cardiovascular system and endothelial function were asse
110 e mechanistic effects of aging per se on the cardiovascular system and focus on the prolongevity bene
111 aling is critical for normal function in the cardiovascular system and is currently the primary targe
112 his novel pathway is widely expressed in the cardiovascular system and is emerging as an important me
113 tabolic control, is crucial for cells of the cardiovascular system and is emerging to play key roles
114            VPO1 is expressed in cells of the cardiovascular system and is secreted into the bloodstre
115 rogress and the growing understanding of the cardiovascular system and its pathophysiology refined al
116          Statins promote fibrinolysis in the cardiovascular system and may play a role in the prevent
117 asis and possibly acts as a link between the cardiovascular system and metabolism, especially in meta
118  reconcile opposing views of n-6 PUFA on the cardiovascular system and present evidence supporting a
119 are based on the architectural design of the cardiovascular system and presume sibling vessels are sy
120 asorelaxation in response to oxidants in the cardiovascular system and provides a molecular explantio
121 ht into the roles of the KATP channel in the cardiovascular system and suggests novel therapeutic pos
122 malian circulatory system comprises both the cardiovascular system and the lymphatic system.
123  the beneficial effects of L-arginine in the cardiovascular system and thus provide new potential ave
124 quired for gene expression in the developing cardiovascular system and underscore the potential of re
125 how alcohol affects different aspects of the cardiovascular system and, hence, the overall effects of
126 ge that exists on aldosterone actions in the cardiovascular system and, in providing the translationa
127  roles of nitric oxide in neurotransmission, cardiovascular systems and immune responses.
128 ent and function of the nervous, immune, and cardiovascular systems and kidney, bone, and skeletal mu
129 mias, assessment of cardiac function and the cardiovascular system, and available treatment options.
130 lar processes in the central nervous system, cardiovascular system, and gastrointestinal tract.
131             Hypertension involves the entire cardiovascular system, and hypertensive vascular disease
132 development, adverse effects on bone and the cardiovascular system, and infertility.
133 d, focusing on somatosensation, hearing, the cardiovascular system, and interactions between TRP chan
134 organ system development, in particular, the cardiovascular system, and its potential implications in
135 l described anti-inflammatory actions in the cardiovascular system, and mimetic peptides that retain
136 2 that drives prostacyclin production in the cardiovascular system, and that urinary metabolites do n
137 e (NO) is an important signaling molecule in cardiovascular system, and the sustained release of NO b
138 en sensing, delivery, and utilization in the cardiovascular system, and we highlight examples of aber
139 xerts a number of deleterious changes in the cardiovascular system, and, in particular, on the large
140 ne congenital anomaly, 89.5% (88.4-90.6) for cardiovascular system anomalies, 79.1% (76.7-81.3) for c
141 markers that reflect molecular states of the cardiovascular system are central for clinical decision
142                 The effects of leptin on the cardiovascular system are discussed in detail, including
143  no doubt that the effects of ghrelin in the cardiovascular system are mediated not only via its grow
144 forms supporting their production within the cardiovascular system are not fully understood.
145 of GPCR trafficking, particularly within the cardiovascular system, as well as highlights the recent
146  induce protective effects on the kidney and cardiovascular system beyond blood glucose control.
147 enal axis, which might negatively affect the cardiovascular system both acutely-by precipitating myoc
148 itamin D deficiency may adversely affect the cardiovascular system, but data from longitudinal studie
149 tant intracellular signaling molecule in the cardiovascular system, but its spatiotemporal dynamics i
150 mechanisms underlying the development of the cardiovascular system, but we are just beginning to unde
151 t activates platelets and other cells of the cardiovascular system by cleaving its G-protein-coupled
152 ce-enhancing drugs may have an effect on the cardiovascular system by means of directly altering the
153 nd designed to exploit NO's influence on the cardiovascular system, cancer biology, the immune respon
154 Bloodstream Infections and Infections of the Cardiovascular System, Central Nervous System Infections
155                                       In the cardiovascular system, changes in oxidative balance can
156 d inflammatory nature of this disease of the cardiovascular system complicates targeted cellular ther
157 cal relevance of this species of RNA for the cardiovascular system, concentrating mainly on recent fi
158 fect on biological mechanisms related to the cardiovascular system, data on their clinical effects ar
159                       The nervous system and cardiovascular system develop in concert and are functio
160 r growth, cell cycle, cell death, cancer and cardiovascular system development.
161  instrumental in further delineating how the cardiovascular system develops and how perturbations in
162 ified population suggests that the mammalian cardiovascular system develops from multipotential proge
163 ecent studies of the functions of TSP in the cardiovascular system, diabetes and aging, which placed
164 sociated with 1D blood flow modelling in the cardiovascular system, discuss model encoding standards,
165 n the dlPAG plays a differential role on the cardiovascular system during heat- and cold-mediated noc
166 ng of the physiological control of the fetal cardiovascular system during hypoxic stress.
167 c insults involving both the airways and the cardiovascular system during oxidant-driven inflammatory
168 structural and functional alterations to the cardiovascular system during physiological and premature
169 reproductive tissue remodeling and the renal cardiovascular system during pregnancy.
170 ensin system (RAS) is a key regulator of the cardiovascular system, electrolyte, and water balance.
171 chanistic effects of secondhand smoke on the cardiovascular system, emphasizing research published si
172 caine's sympathomimetic actions on the human cardiovascular system, even in individuals carrying the
173                                              Cardiovascular system-focused circulating miRNA expressi
174 e current literature on ROS signaling in the cardiovascular system, focusing on the role of ROS in no
175  Here we discuss macrophages residing in the cardiovascular system, focusing particularly on their de
176 e deletion is embryonic lethal with abnormal cardiovascular system formation, and its role in cardiov
177 n, and glucagon-mediated delivery spares the cardiovascular system from adverse T3 action.
178  the highest recognition of integrins in the cardiovascular system from drugs used to inhibit platele
179  data suggest that adenosine may protect the cardiovascular system from ischemia/reperfusion injury,
180 hanism that protects critical tissues of the cardiovascular system from nutrient-induced injury.
181 nergic receptors (ARs) are key regulators of cardiovascular system function that increase blood press
182                                 Although the cardiovascular system has been a focus of intense study,
183 ibed in failing hearts, autophagy within the cardiovascular system has been characterized widely in c
184 ibed in failing hearts, autophagy within the cardiovascular system has been widely characterized in c
185 role of different micro-RNAs (miRNAs) in the cardiovascular system has been widely recognized.
186 ein O-GlcNAcylation in the regulation of the cardiovascular system has increased rapidly in recent ye
187 ts precise role in various cell types of the cardiovascular system has not been extensively investiga
188 the effects of neurological disorders on the cardiovascular system has only expanded in the past 2 de
189 ential benefits of tomato-rich diets for the cardiovascular system have been related to plasma concen
190 he wideranging functions of microRNAs in the cardiovascular system have provided new perspectives on
191               Lumped parameter models of the cardiovascular system have the potential to assist resea
192 veiled new aspects of estrogen action in the cardiovascular system; however, clinical trials have not
193 ive stress responses and their impact on the cardiovascular system; however, researchers are just beg
194 idase is a major source of superoxide in the cardiovascular system; however, whether this enzyme cont
195 mation on the effects of SHS exposure on the cardiovascular system in animal and pediatric studies, i
196           Some of these drugs may affect the cardiovascular system in detrimental ways and others per
197 actor Foxc2 is an essential regulator of the cardiovascular system in development and disease.
198                                          The cardiovascular system in general, and the heart in parti
199 tool to counteract heme toxic effects on the cardiovascular system in hemolytic diseases.
200 enafil treatment directly protects the fetal cardiovascular system in hypoxic development, and that t
201 ms biology between the kidney, skeleton, and cardiovascular system in native and transplant kidney di
202  well known to affect the development of the cardiovascular system in utero including over/under mate
203  paralleling the increased complexity of the cardiovascular system in vertebrates compared with inver
204 uld affect development of fetuses as well as cardiovascular systems in the offspring.
205     Statins exert pleiotropic effects on the cardiovascular system, in part through an increase in ni
206     Various S1P receptors are present in the cardiovascular system, including cardiac tissue.
207 dentary aging has deleterious effects on the cardiovascular system, including decreased left ventricu
208 ing many other biological actions within the cardiovascular system, including effects on cardiac cont
209 f NO that exerts beneficial effects upon the cardiovascular system, including reductions in inflammat
210 cification may occur at several sites in the cardiovascular system, including the intima and media of
211 s review, we discuss the role of AMPK in the cardiovascular system, including the molecular basis of
212         Vitamin D and calcium may affect the cardiovascular system independently and interactively.
213  The abundance of long noncoding RNAs in the cardiovascular system indicates that they may be part of
214                        The patterning of the cardiovascular system into systemic and pulmonic circula
215                                          The cardiovascular system is broadly composed of the heart,
216    Real-time imaging of the embryonic murine cardiovascular system is challenging due to the small si
217 ardiac dysfunction, the role of TWEAK in the cardiovascular system is completely unknown.
218 ,25-dihydroxy-vitamin D3; 1,25-VitD3) in the cardiovascular system is controversial.
219               This review focuses on how the cardiovascular system is regulated when exercising in th
220 rmation available about mTOR function in the cardiovascular system is related to the role of mTORC1 i
221              Bacterial dissemination via the cardiovascular system is the most common cause of infect
222                       The role of TES in the cardiovascular system is unknown.
223 re of interactions between the pulmonary and cardiovascular systems is becoming increasingly apprecia
224 2, a major proinflammatory prostanoid in the cardiovascular system, is a potent stimulus in inducing
225 ystem disorder affecting the skin, eyes, and cardiovascular system, is caused by mutations in the ABC
226 tissue mineralization of the skin, eyes, and cardiovascular system, is caused by mutations in the ABC
227 s of thyroid hormone action on the heart and cardiovascular system, it is possible to explain the cha
228 luid back to the bloodstream, but unlike the cardiovascular system, lacks a centralized pump-the hear
229                     Birds have high-pressure cardiovascular systems like mammals but generate nucleat
230                                       In the cardiovascular system, MAGP1;MAGP2 double knockout mice
231 crease the risk of overall birth defects and cardiovascular system malformation among live births, an
232 y approximately 2.71%, and the proportion of cardiovascular system malformation rose by 0.92% from 20
233 ide (H2S) exhibits beneficial effects in the cardiovascular system, many of which depend on nitric ox
234 ocaine exerts various adverse effects on the cardiovascular system, many times with grave results.
235 iety of stressors on skeletal muscle and the cardiovascular system: mechanical, metabolic, oxidative.
236                                       In the cardiovascular system, miRNAs have been shown to be crit
237                                       In the cardiovascular system, mitochondrial ROS have been linke
238 ritical position in the bioenergetics of the cardiovascular system, mitophagy is particularly importa
239                            In the vertebrate cardiovascular system, multiple Notch family receptors a
240  the function of myocardin in the developing cardiovascular system, Myocd(F/F)/Wnt1-Cre(+) and Myocd(
241 biochemistry of Nox enzymes expressed in the cardiovascular system (Nox1, 2, 4, and 5), their roles i
242 5 mug kg(-1)) caused profound effects on the cardiovascular system of anesthetized rats, whereas CfTX
243 chicken embryo the effects on the developing cardiovascular system of changes in oxygenation, nutriti
244  the functioning of both the otolith and the cardiovascular system of the astronauts before and after
245 % O(2)) pregnancy with an antioxidant on the cardiovascular system of the offspring at the end of ges
246 and real-time monitoring of pulse signals of cardiovascular system on a smart phone/PC.
247 s for PKD-mediated signaling pathways in the cardiovascular system, particularly in the regulation of
248    Evidence is rapidly accumulating that the cardiovascular system--platelet and endothelial function
249 tly showed that cocaine stimulates the human cardiovascular system primarily by acting in the brain t
250 f neuronal activity patterns controlling the cardiovascular system, pulse-modulated activity exists i
251 but their longer-term adverse effects on the cardiovascular system raise concerns.
252 alterations in function and structure of the cardiovascular system ranging from transient and benign
253  CPAP treatment is accompanied by changes in cardiovascular system-related miRNAs that may potentiall
254 on of Rac1 will be associated with extensive cardiovascular system-related side effects and identify
255 ermore, certain other diseases affecting the cardiovascular system remain prevalent and important cau
256           Development and homeostasis of the cardiovascular system require intimate interactions betw
257 tate-of-the-art workshop on "Alcohol and the Cardiovascular System: Research Challenges and Opportuni
258  been established for decades, how the fetal cardiovascular system responds to chronic hypoxia has be
259 e at increased risk for malformations of the cardiovascular system (risk ratio, 3.72; 95 percent conf
260 ge in heart rate is a major component of the cardiovascular system's ability to adjust cardiac output
261 from myocardial dysfunction that impairs the cardiovascular system's function.
262 ltiple developmental programs, including the cardiovascular system, seems to have a significant funct
263 hypothesis that vestibular influences on the cardiovascular system serve to protect against the occur
264                                       In the cardiovascular system, studies have detected and charact
265 open intervention for diseases affecting the cardiovascular system such as twin-twin transfusion synd
266  in virtually all cell types relevant to the cardiovascular system (such as endothelial cells, cardia
267 physiological and pathological states in the cardiovascular system; such states include myocardial in
268 narily conserved among organisms with modern cardiovascular systems, suggesting that RGS2 arose from
269 ysiological systems including breathing, the cardiovascular system, temperature, and sleep-wake cycle
270 caused by the compensatory mechanisms in the cardiovascular system that are activated by 1) higher en
271                              Diseases of the cardiovascular system that cause sudden cardiac deaths a
272  is the continuous single-cell lining of the cardiovascular system that forms a critical interface be
273 SE) is a major H(2)S-producing enzyme in the cardiovascular system that uses cysteine as the main sub
274                During the development of the cardiovascular system, the UPS regulates cell signaling
275 onexcitable tissues, such as the nervous and cardiovascular systems, the kidney and the liver, and ce
276 tiate rapid, non-TRE-mediated effects in the cardiovascular system through cross-coupling to the phos
277 ficial effects of estrogen in protecting the cardiovascular system through ER-dependent mechanisms wi
278 ears that HIV protease inhibitors affect the cardiovascular system through several distinct mechanism
279 d that Hx prevented heme-iron loading in the cardiovascular system, thus limiting the production of r
280  autoregulatory malfunction occur within the cardiovascular system; thus, it seems a daunting challen
281  places severe demands on the ability of the cardiovascular system to deliver oxygen to cells.
282 iovascular development and adaptation of the cardiovascular system to hemodynamic stress.
283 n vivo function of ELA peptides in the adult cardiovascular system, to seek evidence for alteration i
284  important voltage-gated K(+) channel in the cardiovascular system underlying the ultra-rapid rectify
285 ide complex at loading rates relevant to the cardiovascular system, varied from the lowest-affinity H
286  development of the pharyngeal apparatus and cardiovascular system via Tbx1.
287  physiological roles for Kv7 channels in the cardiovascular system warrant increased attention becaus
288 ed pharmacokinetic (PBPK) model of the human cardiovascular system was incorporated into 58 extended
289 To further determine the role of Klf4 in the cardiovascular system, we herein derived mice deficient
290  a bird's-eye view of S-nitrosylation in the cardiovascular system, we provide a conceptual framework
291  representation of afferent signals from the cardiovascular system, were investigated in 34 medicatio
292                   This is exemplified in the cardiovascular system where sGC governs smooth muscle to
293 ed for adipogenesis but is also found in the cardiovascular system, where it has been proposed to opp
294  (KATP) channels are widely expressed in the cardiovascular system, where they regulate a range of bi
295 luding the brain, kidneys, lungs, bones, and cardiovascular system, which becomes apparent with incre
296      This defence is contingent on the fetal cardiovascular system, which in late gestation adopts st
297         This is particularly relevant to the cardiovascular system, which uses mechanical signals fro
298 nowledge about the actions of H2S within the cardiovascular system with an emphasis on the therapeuti
299 ystem disorder affecting skin, eyes, and the cardiovascular system with progressive pathological mine
300 ys predicted adverse outcomes in nervous and cardiovascular systems, with more pronounced changes at

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