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1                 The reaction is specific for cariogenic actinomyces, and it can detect as few as 10(4
2  dental caries, is considered to be the most cariogenic among all oral streptococci.
3 luding non-mutans) provide binding sites for cariogenic and other organisms.
4 est a differential sensitivity to NO between cariogenic and periodontopathogenic bacteria with implic
5 t lipoteichoic acid (LTA) from Gram-positive cariogenic bacteria induces expression of vascular endot
6 nderstanding signaling pathways triggered by cariogenic bacteria may reveal novel therapeutic targets
7 iology that derives from the interplay among cariogenic bacteria on the dentition, the host diet, and
8 y period, putative periodontal pathogens and cariogenic bacteria were overabundant in the group that
9  influenced by host factors, dietary intake, cariogenic bacteria, and other microbes.
10  tooth surfaces against the damage caused by cariogenic bacteria, because the bacteria can be easily
11 tyle-related factors such as high numbers of cariogenic bacteria, inadequate salivary flow, insuffici
12  for Streptococcus mutans, the most abundant cariogenic bacteria.
13                          In both experiments cariogenic bacterial recoveries were reduced relative to
14                                          The cariogenic bacterial species Streptococcus mutans metabo
15 ptococcus mutans is considered the principal cariogenic bacterium for dental caries.
16                         S. mutans is a major cariogenic bacterium in the multispecies bacterial biofi
17 ALB/c mice with immune complexes (IC) of the cariogenic bacterium Streptococcus mutans and mAbs again
18                                          The cariogenic bacterium Streptococcus mutans employs so-cal
19 ase system (AgDS) has been identified in the cariogenic bacterium Streptococcus mutans UA159.
20                                          The cariogenic bacterium Streptococcus mutans uses adhesin P
21  of extracellular polysaccharides (EPS) by a cariogenic bacterium, Streptococcus mutans (Cheng et al.
22                       Effectively inhibiting cariogenic biofilms and reducing secondary caries could
23 n building sticky glucan matrix to establish cariogenic biofilms by an important opportunistic pathog
24 sion could lead to new approaches to control cariogenic biofilms.
25 syltransferase (GtfBCD) enzymes to establish cariogenic biofilms.
26 rs associated with the evolution of virulent-cariogenic biofilms.
27 alistic role of S. mutans and C. albicans in cariogenic biofilms.
28 urring agents that affect the development of cariogenic biofilms.
29 amine fluoride, on human dental enamel under cariogenic challenge in situ.
30                                            A cariogenic challenge was provided in all cases by 5 dail
31 rvoir in plaque that can resist a subsequent cariogenic challenge.
32 it affords any protection against subsequent cariogenic challenges.
33  the experimental gum may help resist future cariogenic challenges.
34 s further support the concept that increased cariogenic conditions are associated with increased prop
35 ide protection during subsequent exposure to cariogenic conditions.
36 of the mineral ions were mobilized under non-cariogenic conditions.
37 rats that were infected with ACUS6 and fed a cariogenic diet with drinking water containing 25 mM ure
38         When challenged with S. mutans and a cariogenic diet, total smooth and sulcal surface lesions
39 eptococcus mutans (sobrinus) 6715, and fed a cariogenic diet.
40 e to extensive oral soft tissue damage and a cariogenic diet.
41  than a conventional gum in ameliorating the cariogenic effects of sucrose.
42 esponse to the pH decrease associated with a cariogenic episode are important components of the carie
43                      Frequent consumption of cariogenic foods and bacterial infection are risk factor
44 ically include oral hygiene level, counts of cariogenic micro-organisms in plaque and saliva, fluorid
45 s on infants' acquisition of a member of the cariogenic microbiota, and its potential effect on carie
46 ication that leads to the establishment of a cariogenic microflora and demineralization of the tooth.
47 t GTF to enhance protective immunity against cariogenic microorganisms.
48 ) worldwide and is considered to be the most cariogenic of all of the oral streptococci.
49  response to accumulated plaque select for a cariogenic or periopathogenic microbiota, respectively,
50                These provide a substrate for cariogenic oral bacteria to flourish and to generate ena
51  development of agriculture, mediated by the cariogenic oral bacterium Streptococcus mutans.
52         We determined the requirement in the cariogenic oral pathogen Streptocococcus mutans of the t
53                    Streptococcus mutans is a cariogenic oral pathogen whose virulence is determined l
54 mmunized mice maintained high levels of this cariogenic organism ( approximately 60% of the total ora
55 -ECC group containing higher levels of known cariogenic organisms.
56 onment of the oral cavity suggests that this cariogenic pathogen is capable of sensing and responding
57 ial colonization of the oral cavity with the cariogenic pathogen Streptococcus mutans and other bacte
58 ed for its ability to remove biofilms of the cariogenic pathogen Streptococcus mutans UA159, as well
59 robial peptide, was able to selectively kill cariogenic pathogen Streptococcus mutans with high effic
60 hts on how S. mutans may have become a major cariogenic pathogen.
61 ompositions via bioactive resins to suppress cariogenic/pathogenic species and promote benign species
62  solutions simulating resting (pH = 5.6) and cariogenic plaque fluid (pH = 4.8).
63 glucosyltransferase activity restores a less cariogenic plaque structure.
64 membrane potentials became more negative in "cariogenic plaque" solution for all types of sections: s
65 f the wells showing anion selectivity in the cariogenic "plaque-like" solution.
66 pic level, notably in the differences in the cariogenic potential between strains.
67 ls in post-sucrose plaque fluids reflect the cariogenic potential of dental plaque.
68 ropose that the absence of GbpA elevates the cariogenic potential of S. mutans by altering the struct
69                                          The cariogenic potential of S. mutans is related to its abil
70                                          The cariogenic potential of these organisms was evaluated in
71                      Processed starches have cariogenic potential when accompanying sucrose, but huma
72 dental plaque in such a way as to reduce its cariogenic potential.
73  biofilm was predicted to be always or never cariogenic, respectively.
74 s GBP-B can have a protective effect against cariogenic S. mutans infection and disease.
75 in every enamel well in either "resting" or "cariogenic" solution.
76 situ acid production and distribution by the cariogenic species S. mutans.
77 viously, we presented evidence that the oral cariogenic species Streptococcus mutans remains viable b
78 % of their oral bacterial spectra, including cariogenic species.
79 he immune response to a protein antigen from cariogenic streptococci, potentially through suppressive
80 uction of dental caries by interference with cariogenic streptococci.
81 ble bactericidal effect was observed against cariogenic Streptococcus mutans at pH 7.4, even when usi
82 occus gordonii, Streptococcus sanguinis, and cariogenic Streptococcus mutans.
83 fic permease, in the transport of sucrose by cariogenic Streptococcus mutans.
84 nd/or 11 plus 16, followed by infection with cariogenic Streptococcus sobrinus.
85 ce the level of dental caries caused by this cariogenic streptococcus.
86 nced by host genetic background, potentially cariogenic taxa are likely not controlled by genetic fac
87               BCS3-L1 was significantly less cariogenic than JH1140 in both gnotobiotic- and conventi
88 e-dependent adherence and significantly less cariogenic than the UA130 wild-type progenitor in germfr
89 ii colonization abundance: the former highly cariogenic, the latter not.
90 contributions on the ratio of aciduric (i.e. cariogenic) to non-aciduric bacteria to be unambiguously
91  low pH appears to be an important bacterial cariogenic trait.
92 h prevalence of caries to reliance on highly cariogenic wild plant foods in Pleistocene hunter-gather

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