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1 ed as intact, moderately carious, or grossly carious.
2 were associated with the number of untreated carious and missing tooth surfaces in adulthood.
3                      The number of untreated carious and missing tooth surfaces were associated with
4 32 y), oral health outcomes (e.g., number of carious and missing tooth surfaces), and oral health-rel
5 ess for components of hydrated dentin from 8 carious and non-carious human teeth.
6                                    Permanent carious and sound teeth indicated for extraction were ex
7                                              Carious and sound teeth indicated for extraction were ra
8 reparative mechanisms that operate following carious and traumatic dental injury are critical for pul
9 eth restored with crowns and presenting with carious cavitation were excluded.
10  gingival recession (GR) associated with non-carious cervical lesions (NCCLs) treated by connective t
11  of disease states, ranging from sub-surface carious changes through to more advanced lesions.
12 xpression was also evident in dental pulp of carious compared with noncarious teeth.
13 on) annually averted 0.29 (95%CI: 0.16-0.42) carious coronal and 0.22 (95%CI: 0.08-0.37) carious root
14 sable mixture which could be used to restore carious defects in teeth followed a tortuous path of neg
15 ed this hypothesis by bonding to transparent carious dentin containing occluded dentinal tubules.
16 bide bur, and both were lower than flat, non-carious dentin controls.
17                                              Carious dentin from extracted human molars was removed w
18 e physical and microstructural properties of carious dentin in the 4 different zones to determine imp
19 gnize signals from TGF-beta1 and bacteria in carious dentin.
20 nated during bonding to occluded transparent carious dentin.
21 omposites have the potential to remineralize carious enamel lesions.
22 were placed in holders that exposed only the carious enamel surface.
23          Tooth slices were prepared from non-carious human molars and treated with 0-50 ng/mL rhVEGF(
24 th the presence of Gram-positive bacteria in carious human teeth.
25 ts of hydrated dentin from 8 carious and non-carious human teeth.
26 ls the direction of neurite outgrowth toward carious injuries by modulating the secretion of brain-de
27 ntal pulp fibroblasts, localized beneath the carious injury site, do express this receptor.
28  nerve fiber's terminal branches beneath the carious injury site.
29 l orchestrating pulp nerve sprouting beneath carious injury, a critical step in dentin-pulp regenerat
30  secretion of nerve growth factor (NGF) upon carious injury.
31 n of BDNF by pulp fibroblasts under sites of carious injury.
32 ucture, human dental pulp, following chronic carious insult.
33 a severe recession defect and its associated carious lesion were managed using the combination of a l
34 d to promote remineralization and harden the carious lesion.
35 lesions (CAE), and caries active with dentin carious lesions (CA).
36  from enamel carious lesions (PE) and dentin carious lesions (PD) were collected.
37 ies-free tooth surfaces (PF) and from enamel carious lesions (PE) and dentin carious lesions (PD) wer
38 all accuracy of visual methods for detecting carious lesions and to identify possible sources of hete
39                                 For proximal carious lesions confined to enamel (not reaching the ena
40 ther Thio-GLU or GLU had significantly fewer carious lesions in the buccal enamel or dentinal surface
41          Here, for the first time, we report carious lesions preserved in specimens of A. simus, reco
42  fabM strain exhibited fewer and less severe carious lesions than those observed in the wild-type str
43 ficantly lower number of enamel and dentinal carious lesions was observed for the mutant-infected rat
44                  The depth of the artificial carious lesions was significantly affected by "adhesive
45 llus casei, which often predominates in deep carious lesions where B cells and plasma cells predomina
46 ould intervene invasively (restoratively) on carious lesions where evidence and clinical recommendati
47 type c strain originally isolated from human carious lesions, which is extensively used as a laborato
48 issue and reduced signal from de-mineralized carious lesions.
49 thogenic potential of S. mutans in advancing carious lesions.
50 aggressive onset of the disease with rampant carious lesions.
51 ence factor responsible for the formation of carious lesions.
52 invasive/nonrestorative treatment of "early" carious lesions: those confined to enamel or reaching th
53                        Their source was: (1) carious material from advanced root lesions (ARL), (2) p
54 Exposed root surfaces frequently exhibit non-carious notches representing material loss by abrasion,
55 Teeth were categorized as intact, moderately carious, or grossly carious.
56 uited 534 children aged 6-10 yrs with >or= 2 carious posterior teeth.
57 Hall Technique (HT) is a method for managing carious primary molars.
58  carious coronal and 0.22 (95%CI: 0.08-0.37) carious root surfaces.
59 nic whites (0.6) having the fewest untreated carious root surfaces.
60                            The mean cost per carious surface avoided was estimated at pound251 (95% c
61 aces in MA users was quite distinctive, with carious surface involvement being highest for the maxill
62 es (95%CI = 1.0-1.9), or approximately 1 new carious surface per person per year.
63         The DE was 1.4 for the clustering of carious surfaces within teeth, 6.0 for carious teeth wit
64 ous teeth within an individual, and 38.0 for carious surfaces within the individual.
65  outcomes were only detected with respect to carious surfaces.
66 n, is activated at the injured site of human carious teeth and plays an important role in dental-pulp
67  molecules present in pulp cell lysates from carious teeth specifically activated PAR-2, but those fr
68               Twenty human extracted and non-carious teeth were divided into 4 groups: 5 primary and
69  be isolated from inflamed pulp derived from carious teeth with symptomatic irreversible pulpitis (I-
70 ng of carious surfaces within teeth, 6.0 for carious teeth within an individual, and 38.0 for carious
71 l: < 1 and high-risk school: >/= 1 untreated carious teeth).
72 ne and treat patients with a matched pair of carious teeth, and each pair of teeth was treated in a r
73                         The neural status of carious teeth, particularly those associated with a pain
74 nitial mechanisms regulating this process in carious teeth.
75 tensive caries, as measured by the number of carious teeth.
76 of caries progression in sealed vs. unsealed carious teeth.
77 g was 2.6% for sealed and 12.6% for unsealed carious teeth.
78  of BDNF secretion by pulp fibroblasts under carious teeth.
79 sis was performed with the outcome variables carious tooth or surface.
80 new caries outcomes defined as the number of carious tooth surfaces within each cluster.

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