1 ed as intact, moderately carious, or grossly
carious.
2 were associated with the number of untreated
carious and missing tooth surfaces in adulthood.
3 The number of untreated
carious and missing tooth surfaces were associated with
4 32 y), oral health outcomes (e.g., number of
carious and missing tooth surfaces), and oral health-rel
5 ess for components of hydrated dentin from 8
carious and non-carious human teeth.
6 Permanent
carious and sound teeth indicated for extraction were ex
7 Carious and sound teeth indicated for extraction were ra
8 reparative mechanisms that operate following
carious and traumatic dental injury are critical for pul
9 eth restored with crowns and presenting with
carious cavitation were excluded.
10 gingival recession (GR) associated with non-
carious cervical lesions (NCCLs) treated by connective t
11 of disease states, ranging from sub-surface
carious changes through to more advanced lesions.
12 xpression was also evident in dental pulp of
carious compared with noncarious teeth.
13 on) annually averted 0.29 (95%CI: 0.16-0.42)
carious coronal and 0.22 (95%CI: 0.08-0.37) carious root
14 sable mixture which could be used to restore
carious defects in teeth followed a tortuous path of neg
15 ed this hypothesis by bonding to transparent
carious dentin containing occluded dentinal tubules.
16 bide bur, and both were lower than flat, non-
carious dentin controls.
17 Carious dentin from extracted human molars was removed w
18 e physical and microstructural properties of
carious dentin in the 4 different zones to determine imp
19 gnize signals from TGF-beta1 and bacteria in
carious dentin.
20 nated during bonding to occluded transparent
carious dentin.
21 omposites have the potential to remineralize
carious enamel lesions.
22 were placed in holders that exposed only the
carious enamel surface.
23 Tooth slices were prepared from non-
carious human molars and treated with 0-50 ng/mL rhVEGF(
24 th the presence of Gram-positive bacteria in
carious human teeth.
25 ts of hydrated dentin from 8 carious and non-
carious human teeth.
26 ls the direction of neurite outgrowth toward
carious injuries by modulating the secretion of brain-de
27 ntal pulp fibroblasts, localized beneath the
carious injury site, do express this receptor.
28 nerve fiber's terminal branches beneath the
carious injury site.
29 l orchestrating pulp nerve sprouting beneath
carious injury, a critical step in dentin-pulp regenerat
30 secretion of nerve growth factor (NGF) upon
carious injury.
31 n of BDNF by pulp fibroblasts under sites of
carious injury.
32 ucture, human dental pulp, following chronic
carious insult.
33 a severe recession defect and its associated
carious lesion were managed using the combination of a l
34 d to promote remineralization and harden the
carious lesion.
35 lesions (CAE), and caries active with dentin
carious lesions (CA).
36 from enamel carious lesions (PE) and dentin
carious lesions (PD) were collected.
37 ies-free tooth surfaces (PF) and from enamel
carious lesions (PE) and dentin carious lesions (PD) wer
38 all accuracy of visual methods for detecting
carious lesions and to identify possible sources of hete
39 For proximal
carious lesions confined to enamel (not reaching the ena
40 ther Thio-GLU or GLU had significantly fewer
carious lesions in the buccal enamel or dentinal surface
41 Here, for the first time, we report
carious lesions preserved in specimens of A. simus, reco
42 fabM strain exhibited fewer and less severe
carious lesions than those observed in the wild-type str
43 ficantly lower number of enamel and dentinal
carious lesions was observed for the mutant-infected rat
44 The depth of the artificial
carious lesions was significantly affected by "adhesive
45 llus casei, which often predominates in deep
carious lesions where B cells and plasma cells predomina
46 ould intervene invasively (restoratively) on
carious lesions where evidence and clinical recommendati
47 type c strain originally isolated from human
carious lesions, which is extensively used as a laborato
48 issue and reduced signal from de-mineralized
carious lesions.
49 thogenic potential of S. mutans in advancing
carious lesions.
50 aggressive onset of the disease with rampant
carious lesions.
51 ence factor responsible for the formation of
carious lesions.
52 invasive/nonrestorative treatment of "early"
carious lesions: those confined to enamel or reaching th
53 Their source was: (1)
carious material from advanced root lesions (ARL), (2) p
54 Exposed root surfaces frequently exhibit non-
carious notches representing material loss by abrasion,
55 Teeth were categorized as intact, moderately
carious,
or grossly carious.
56 uited 534 children aged 6-10 yrs with >or= 2
carious posterior teeth.
57 Hall Technique (HT) is a method for managing
carious primary molars.
58 carious coronal and 0.22 (95%CI: 0.08-0.37)
carious root surfaces.
59 nic whites (0.6) having the fewest untreated
carious root surfaces.
60 The mean cost per
carious surface avoided was estimated at pound251 (95% c
61 aces in MA users was quite distinctive, with
carious surface involvement being highest for the maxill
62 es (95%CI = 1.0-1.9), or approximately 1 new
carious surface per person per year.
63 The DE was 1.4 for the clustering of
carious surfaces within teeth, 6.0 for carious teeth wit
64 ous teeth within an individual, and 38.0 for
carious surfaces within the individual.
65 outcomes were only detected with respect to
carious surfaces.
66 n, is activated at the injured site of human
carious teeth and plays an important role in dental-pulp
67 molecules present in pulp cell lysates from
carious teeth specifically activated PAR-2, but those fr
68 Twenty human extracted and non-
carious teeth were divided into 4 groups: 5 primary and
69 be isolated from inflamed pulp derived from
carious teeth with symptomatic irreversible pulpitis (I-
70 ng of carious surfaces within teeth, 6.0 for
carious teeth within an individual, and 38.0 for carious
71 l: < 1 and high-risk school: >/= 1 untreated
carious teeth).
72 ne and treat patients with a matched pair of
carious teeth, and each pair of teeth was treated in a r
73 The neural status of
carious teeth, particularly those associated with a pain
74 nitial mechanisms regulating this process in
carious teeth.
75 tensive caries, as measured by the number of
carious teeth.
76 of caries progression in sealed vs. unsealed
carious teeth.
77 g was 2.6% for sealed and 12.6% for unsealed
carious teeth.
78 of BDNF secretion by pulp fibroblasts under
carious teeth.
79 sis was performed with the outcome variables
carious tooth or surface.
80 new caries outcomes defined as the number of
carious tooth surfaces within each cluster.