ライフサイエンスコーパス: carotid

1 Carotid (18)F-fluoride uptake was increased in clinicall

2 Carotid (18)F-fluorodeoxyglucose uptake appeared to be i

3 herosclerosis was measured in right and left carotids, abdominal aorta, right and left iliofemoral ar

4 95% CI = -0.053 to 0.049; P = 0.916) and the carotids (adalimumab: TBR = 0.031, 95% CI = -0.005 to 0.

5 modest increase in vascular inflammation in carotids after 52 weeks of treatment with adalimumab.

6 Bilateral carotid and femoral territories were explored by 3DVUS t

7 eficient (apoE(-/-)) mice with CaCl2-induced carotid aneurysm (n = 11).

8 specificity were shown by autoradiography in carotid aneurysm.

9 sis and adverse outcomes in the ACT-1 trial (Carotid Angioplasty and Stenting Versus Endarterectomy i

10 In IUGR baboons there was increased carotid arterial blood flow velocity during late systole

11 idence of peripheral artery disease), of the carotid arteries (previous carotid artery revascularisat

12 ersus control, 2.12+/-0.27; P=0.001) and the carotid arteries (TBRmax: CKD, 2.45+/-0.65 versus contro

13 velocities and low blood volume flow in the carotid arteries after ligation in FVB mice.

14 TSPAN2 expression is attenuated in mouse carotid arteries after ligation injury and in failed hum

15 expression of 5-HTT was elevated in injured carotid arteries and over-expression of 5-HTT induced pr

16 nonclassical monocytes patrol inside healthy carotid arteries at a velocity of 36 mum/min, 3x faster

17 he carotid plaques and contralateral plaques/carotid arteries by an experienced radionuclide radiolog

18 En face staining of the murine aorta or carotid arteries modified with flow-altering cuffs demon

19 monocrotaline, whereas it was upregulated in carotid arteries of Macaca fascicularis subjected to ath

20 The endothelial damage in atherosclerotic carotid arteries was assessed by electron microscopy and

21 METHODS AND In mouse carotid arteries with established intimal lesions tailor

22 decellularized vessels obtained from porcine carotid arteries with poly (ethylmethacrylate-co-diethyl

23 IUGR group was seen in the iliac but not the carotid arteries without between-sex differences.

24 hoGEF tyrosine phosphorylation in rat common carotid arteries, and siRNA-mediated down-regulation of

25 hibited endothelial recovery in wire-injured carotid arteries, but this effect was also abrogated by

26 1 was enriched in calcified regions of human carotid arteries, examined by immunohistochemistry.

27 l carotid artery) in both the left and right carotid arteries.

28 ed macrophage content and lesion size in the carotid arteries.

29 lar cell adhesion molecule 1 in wire-injured carotid arteries.

30 ical properties in phantom and human cadaver carotid arteries.

31 c plaques and the asymptomatic contralateral carotid arteries/plaques showed no significant differenc

32 Common carotid artery (CCA) balloon angioplasty injury was perf

33 ter Embozene(R) embolization of the external carotid artery (ECA).

34 lood flow (CBF) was measured at the internal carotid artery (ICA) and vertebral artery (VA) and CBF v

35 neurysm in the cavernous segment of internal carotid artery (ICA) is a relatively rare entity.

36 graphy to distinguish true cervical internal carotid artery (ICA) occlusion from pseudo-occlusion (de

37 induced by endothelial abrasion of the right carotid artery and abdominal aorta of 7 rabbits fed an a

38 cine model of synthetic graft placed between carotid artery and ipsilateral jugular vein was used to

39 e of the atherosclerotic plaques in both the carotid artery and the aortic root.

40 cclusion after stroke (intracranial internal carotid artery and/or middle cerebral artery M1 and/or M

41 ing those the endothelium experiences in the carotid artery are responsible for determining the fate

42 tic plaques of the abdominal aorta and right carotid artery as compared with normal control arteries

43 Extracranial internal carotid artery atherosclerotic occlusive disease is a co

44 Chronic reductions in carotid artery blood flow are associated with increased

45 ow increased mean blood pressure measured by carotid artery cannulation and increased microvascular r

46 bal cerebral tissue injury, and cognition in carotid artery disease (CAD).

47 To compare prevalence of carotid artery disease and its various types of lesions

48 gnificantly more likely to have coronary and carotid artery diseases.

49 iPhone camera was held in a cradle over the carotid artery during iPhone measurements.

50 iveness of carotid artery stenting (CAS) and carotid artery endarterectomy (CEA) for the prevention o

51 ry, comprehensive ophthalmic evaluation, and carotid artery evaluation (by Doppler/angiography) on th

52 d with higher risk of stroke due to internal carotid artery injuries, but monitoring was not useful f

53 Among those with isolated internal carotid artery injuries, five of nine with delayed strok

54 g for ipsilateral and contralateral internal carotid artery injury grade (adjusted risk ratio, 2.91;

55 ient mice have reduced thrombus growth after carotid artery injury relative to conventionally raised

56 Among patients who sustained an internal carotid artery injury with or without additional vessel

57 D) and evaluated the association with common carotid artery intima-media thickness (cCIMT) using mult

58 erosis, as measured by progression of common carotid artery intima-media thickness (cIMT), in adults

59 ected adults and evaluated associations with carotid artery intima-media thickness and plaque.

60 METHODS AND Carotid artery intima-media thickness was measured at ba

61 sion and VaD was induced by bilateral common carotid artery occlusion (BCCAO) in adult male Sprague D

62 Permanent bilateral common carotid artery occlusion (BCCAO) was used as a model of

63 hemic injury was induced by bilateral common carotid artery occlusion, whereas severe focal stroke in

64 sly with a fast intra-vascular sensor in the carotid artery of anaesthetized, mechanically ventilated

65 e left external and internal branches of the carotid artery of male FVB mice and performed sham opera

66 A catheter was inserted into the right carotid artery of mice, which acted as a vascular graft.

67 All consecutive patients with internal carotid artery or middle cerebral artery occlusions tran

68 with occlusion of the intracranial internal carotid artery or proximal middle cerebral artery who ha

69 d coronary artery calcification (CARDIA) and carotid artery plaque burden (BioImage).

70 disease), of the carotid arteries (previous carotid artery revascularisation or asymptomatic carotid

71 P values were adjusted for age, sex, carotid artery site, and family relations.

72 tid disease was defined as cervical internal carotid artery stenosis (>50%) or occlusion.

73 Of 10579 individuals with a diagnosis of carotid artery stenosis (4615 women and 5964 men; mean [

74 The incidence of carotid artery stenosis and plaques, cardiac embolic sou

75 y was queried for individuals diagnosed with carotid artery stenosis between October 1, 2006, and Sep

76 tid artery revascularisation or asymptomatic carotid artery stenosis of at least 50%), or coronary ar

77 Carotid artery stenosis on the involved side was worse i

78 l/6J mice were subjected to bilateral common carotid artery stenosis or a sham operation and fed norm

79 ndard surgical risk with severe asymptomatic carotid artery stenosis randomly assigned to carotid art

80 ompared CAS against CEA for the treatment of carotid artery stenosis were selected.

81 ugh many factors influence the management of carotid artery stenosis, it is not well understood wheth

82 my (CEA) for the prevention of stroke due to carotid artery stenosis.

83 provider-induced demand in the management of carotid artery stenosis.

84 lict regarding the relative effectiveness of carotid artery stenting (CAS) and carotid artery endarte

85 75.8 years; 43% women) and 231077 underwent carotid artery stenting (mean age, 75.4 years; 49% women

86 Carotid endarterectomy and carotid artery stenting are the leading approaches to re

87 Of 1021 patients treated with carotid artery stenting during a mean follow-up of 3.1+/

88 ggest that independent modular filter use in carotid artery stenting in high surgical risk patients i

89 nrandomized, open-label, single-arm study of carotid artery stenting in high surgical risk patients w

90 rom 1999 to 2014, whereas the performance of carotid artery stenting increased until 2006 and then de

91 carotid artery stenosis randomly assigned to carotid artery stenting or carotid endarterectomy (Abbot

92 Embolic protection during carotid artery stenting reduces the rate of thromboembol

93 ural intervention (carotid endarterectomy or carotid artery stenting) compared with medical managemen

94 Carotid endarterectomy and carotid artery stenting.

95 lic Protection System in patients undergoing carotid artery stenting.

96 in is more potent than hirulog-1 in a murine carotid artery thrombosis model.

97 s, we also performed ferric chloride-induced carotid artery thrombosis.

98 nd neutrophilia that precedes development of carotid artery thrombus formation.

99 In mice, the carotid artery to the ipsilateral jugular vein was conne

100 ic stroke propensity through the left common carotid artery using an idealized aortic arch model.

101 s was measured based on displacements of the carotid artery wall, and Young's modulus was 2-fold grea

102 Extradural aneurysms in the internal carotid artery were included; fusiform aneurysms, infund

103 rein, we show that neointima formation after carotid artery wire injury reduces markedly in CD40(-/-)

104 ommon carotid artery, carotid bulb, internal carotid artery) in both the left and right carotid arter

105 otid IMT was measured at 3 locations (common carotid artery, carotid bulb, internal carotid artery) i

106 umber and size of atherosclerotic plaques in carotid artery, heart, aortic arch and aorta in acute an

107 ima, media, or atherosclerotic plaque in the carotid artery.

108 were implanted unilaterally in the internal carotid artery.

109 ient was greater than 0.8 in all measures of carotid artery/plaque uptake (SUV) and greater than 0.6

110 ndarterectomy and 8 controls without culprit carotid atheroma.

111 was generally associated with higher risk of carotid atherosclerosis and clinical cardiovascular dise

112 Baseline carotid atherosclerosis but not atherosclerotic CVD risk

113 Carotid atherosclerosis was assessed by ultrasound, wher

114 in elderly men and postmenopausal women with carotid atherosclerosis, as well as with risk of stroke

115 pausal women and 1124 men (>/=45 years) with carotid atherosclerosis, from prospective population-bas

116 (CT) can assess both anatomy and biology of carotid atherosclerosis.

117 approaches to the management of extracranial carotid atherosclerotic occlusive disease and the basis

118 DS AND LKB1 expression was examined in human carotid atherosclerotic plaques and in western diet-fed

119 ccumulated lipids in endarterectomized human carotid atherosclerotic plaques using three-dimensional

120 its the vascular response to injury in a rat carotid balloon angioplasty model.

121 Carotid baroreflex activation lowers blood pressure and

122 in the cross-sectional areas of the internal carotid, basilar, and middle cerebral arteries on the fi

123 ke in the Netherlands (MR CLEAN) in whom the carotid bifurcation could be assessed (n = 443) were inc

124 rical signals from the chemoreceptors of the carotid bifurcation to the central nervous system where

125 Conclusion Carotid webs at the symptomatic carotid bifurcation were observed in 2.5% of the patient

126 The presence of a carotid web at the carotid bifurcations was evaluated at computed tomograph

127 principal peripheral chemoreceptors are the carotid bodies (CBs) and alteration in their function ha

128 ing on the circumstance, the activity of the carotid bodies and that of RTN vary in the same or the o

129 normalities present in diseases in which the carotid bodies are hyperactive at rest, e.g. essential h

130 vivo adenoviral transfection of KLF2 to the carotid bodies in CHF rabbits restored KLF2 expression,

131 are silent and the excitatory input from the carotid bodies is suppressed.

132 apeutic approaches that increase KLF2 in the carotid bodies may be efficacious in the treatment of re

133 The carotid bodies stimulate the respiratory pattern generat

134 Clinical studies suggest that abnormal carotid body (CB) activity may be a driver of sleep apne

135 Enhanced carotid body (CB) chemoreflex function is strongly relat

136 SA), have been shown to exhibit a heightened carotid body (CB) chemosensory reflex and hypertension.

137 a (IH) on blood pressure (BP), breathing and carotid body (CB) chemosensory reflex were examined in a

138 t K(+) channels (Kv) are highly expressed in carotid body (CB) glomus cells, but their role in hypoxi

139 Augmented sensory neuronal activity from the carotid body (CB) has emerged as a principal cause of hy

140 s enzyme may offer a new target for reducing carotid body activity in selected cardiovascular disease

141 quickly triggers a compensatory increase in carotid body activity.

142 ery blood flow are associated with increased carotid body chemoreceptor activity.

143 Increased carotid body chemoreflex (CBC) sensitivity plays a role

144 KEY POINTS: Enhanced carotid body chemoreflex activity contributes to develop

145 transmission is an important element of the carotid body chemotransduction pathway.

146 Conversely, lentiviral KLF2 siRNA in the carotid body decreased KLF2 expression, increased chemor

147 KEY POINTS: Carotid body dysfunction is recognized as a cause of hyp

148 rived NECs were retained as PNECs, while the carotid body evolved via the aggregation of neural crest

149 that trigger amniote respiratory reflexes - carotid body glomus cells, and 'pulmonary neuroendocrine

150 n conclusion, down-regulation of KLF2 in the carotid body increases CBC sensitivity, oscillatory brea

151 The results indicate that restoring carotid body KLF2 in chronic heart failure reduces sympa

152 ur data show CD73 to be a novel regulator of carotid body sensory function and therefore suggest that

153 derpinnings of the oxygen sensitivity of the carotid body Type I cells are becoming better defined as

154 nd its current role in oxygen sensing by the carotid body; reactive oxygen species as key transducers

155 sured at 3 locations (common carotid artery, carotid bulb, internal carotid artery) in both the left

156 ff electrodes and the BP waves recorded with carotid catheters were ensemble averaged relative to the

157 x anti-inflammatory network was abrogated by carotid chemoreceptor denervation and by pharmacological

158 mechanism of neuromodulation mediated by the carotid chemoreceptors and involving both the sympatheti

159 The presence of extracranial carotid disease (ECD) is associated with less favorable

160 ase versus patients with acutely symptomatic carotid disease and recent ischemic stroke.

161 e strongly associated with femoral than with carotid disease burden, whereas hypertension and diabete

162 ved in patients with asymptomatic high-grade carotid disease versus patients with acutely symptomatic

163 Extracranial carotid disease was defined as cervical internal carotid

164 herapy is the preferred treatment option for carotid dissections.

165 grated with gene expression profiling of 121 carotid endarterectomies and an analysis of protein secr

166 domly assigned to carotid artery stenting or carotid endarterectomy (Abbott Vascular).

167 n abdominal aortic aneurysm repair (OAR) and carotid endarterectomy (CEA) performed by very low-volum

168 the study, 937111 unique patients underwent carotid endarterectomy (mean age, 75.8 years; 43% women)

169 vascular event between 5 and 180 days of the carotid endarterectomy [symptomatic]) confirmed elevatio

170 ients with culprit carotid stenosis awaiting carotid endarterectomy and 8 controls without culprit ca

171 Carotid endarterectomy and carotid artery stenting are t

172 Carotid endarterectomy and carotid artery stenting.

173 Human plaques were derived from carotid endarterectomy and stained against P2X7.

174 tional trends in performance and outcomes of carotid endarterectomy and stenting among Medicare benef

175 e Medicare beneficiaries, the performance of carotid endarterectomy declined from 1999 to 2014, where

176 hy, and the intervention in the 3 trials was carotid endarterectomy for patients with stenosis exceed

177 s) with the odds of procedural intervention (carotid endarterectomy or carotid artery stenting) compa

178 In a meta-analysis, carotid endarterectomy reduced rates of 1) perioperative

179 00 expression in >70 samples obtained during carotid endarterectomy revealed that local miR-100 expre

180 que instability as seen in humans, and human carotid endarterectomy samples.

181 lar matrix and associated molecules in human carotid endarterectomy specimens from 6 symptomatic vers

182 omatic Subjects Who Are at Standard Risk for Carotid Endarterectomy With Significant Extracranial Car

183 Of 366 patients with carotid endarterectomy, 61 exhibited some degree of LOY

184 ion to stroke or death in patients receiving carotid endarterectomy, a harm of screening included the

185 attack, stroke, or amaurosis fugax), due for carotid endarterectomy, were prospectively recruited.

186 haracteristics and outcome in men undergoing carotid endarterectomy.

187 event within the previous 5 days) undergoing carotid endarterectomy.

188 consenting patients with recent symptomatic carotid events (transient ischemic attack, stroke, or am

189 8)Ga-DOTATATE uptake in patients with recent carotid events, assessed inter- and intraobserver variab

190 be more conveniently measured compared with carotid-femoral pulse wave velocity (cfPWV).

191 We evaluated ultrasound-detected carotid, iliofemoral, and abdominal aortic plaques; coro

192 specificity varied from 25% (brain imaging; carotid imaging) to 99% (anticoagulation quality).

193 95% confidence interval [CI]: 0.3, 0.7) and carotid IMT (mean difference, 37 mum; 95% CI: 25, 49) we

194 The end-of-study difference in mean carotid IMT between children with HeFH and unaffected si

195 As a result, no difference in carotid IMT could be detected between the 2 groups after

196 significantly less progression of increased carotid IMT in children with HeFH than untreated unaffec

197 ect of 2-year treatment with rosuvastatin on carotid IMT in children with HeFH.

198 t was also associated with an increased mean carotid IMT of 15 mum (95% CI: 0, 29) but not after addi

199 During 2 years of follow-up, the change in carotid IMT was 0.0054 mm/y (95% confidence interval, 0.

200 Carotid IMT was assessed by ultrasonography at baseline

201 Carotid IMT was measured at 3 locations (common carotid

202 ildren with HeFH who were >/=6 years of age, carotid IMT was significantly greater at baseline compar

203 At baseline, mean+/-SD carotid IMT was significantly greater for the 197 childr

204 ntent with total and regional aortic PWV and carotid IMT while adjusting for several possible confoun

205 sociated with aortic pulse wave velocity and carotid IMT.

206 In a rat carotid injury model, periadventitial delivery of resver

207 educed platelet prothrombotic capacity after carotid injury.

208 lerosis measures (coronary calcium score and carotid intima media thickness) in an independent sample

209 WHRadjBMI was also associated with higher carotid intima-media thickness (39%; 95% CI, 9%-77% per

210 factors plus lipids for predicting high-risk carotid intima-media thickness (cIMT) in adulthood.

211 Carotid intima-media thickness (IMT) was acquired and me

212 s of atherosclerosis manifested by increased carotid intima-media thickness (IMT).

213 levels, and measures of cardiovascular risk (carotid intima-media thickness and levels of high-sensit

214 reas neither drug had significant effects on carotid intima-media thickness, other cardiovascular mar

215 METHODS AND An ex vivo porcine carotid jugular arteriovenous shunt was established and

216 -based miRNA array of plasma, sampled at the carotid lesion site, identified 8 deregulated miRNAs (mi

217 BRmax differentiated culprit from nonculprit carotid lesions (median difference: 0.12; IQR: 0.0 to 0.

218 ients with both asymptomatic and symptomatic carotid occlusive diseases.

219 out vascular disease had higher rates of any carotid plaque (34% versus 25%; P=0.04), noncalcified (1

220 We prospectively evaluated carotid plaque (68)Ga-DOTATATE uptake in patients with r

221 s with prediabetes had an increased risk for carotid plaque and adverse functional cardiac parameters

222 HIV, and the presence and characteristics of carotid plaque are associated with subsequent vascular e

223 E was associated with presence of vulnerable carotid plaque as well as increased risk of stroke in wo

224 An unstable carotid plaque at risk of stroke is characterized by low

225 spotty calcification and low attenuation) of carotid plaque by computerized tomography among PLWHIV w

226 f endogenous estradiol and testosterone with carotid plaque composition in elderly men and postmenopa

227 rthermore, we delivered let-7 mimic to human carotid plaque ex vivo and observed significant changes

228 e is accompanied by an acute decrease in the carotid plaque expression of micro-RNAs (miRs)-221 and m

229 3), and Gal-3 binding protein (Gal-3BP) with carotid plaque formation (focal intima-media thickness >

230 ts is increased; however, whether incidental carotid plaque is increased and there is an association

231 on and intraplaque neovascularization in the carotid plaque of symptomatic and asymptomatic patients.

232 d target:background ratio were higher in the carotid plaque of symptomatic patients (P=0.021 and P=0.

233 valuated for cardiovascular risk factors and carotid plaque on ultrasound.

234 characteristics as a biomarker indicative of carotid plaque rupture and stroke.

235 1 has potential as a diagnostic biomarker of carotid plaque rupture and stroke.

236 erotic human aorta tissue (n = 15) and human carotid plaque samples (n = 19).

237 Carotid plaque was independently associated with a 3-fol

238 of vessels was also significantly higher in carotid plaque with high-CE (P<0.001).

239 ns, microbleeds), cardiovascular parameters (carotid plaque, left ventricular function, and myocardia

240 There is an increase in incidental carotid plaque, noncalcified plaque, and high-risk plaqu

241 d there is an association between incidental carotid plaque, plaque characteristics, and vascular eve

242 and 9.7% higher (95% CI, 2.2-17.8) burden of carotid plaque.

243 hlights culprit and phenotypically high-risk carotid plaque.

244 in the majority of HIV-affected adults with carotid plaque.

245 sion in patients with stable versus unstable carotid plaque.

246 xyglucose can identify culprit and high-risk carotid plaque.

247 red by drawing regions of interest along the carotid plaques and contralateral plaques/carotid arteri

248 let-7 levels are decreased in diabetic human carotid plaques and in a model of diabetes-associated at

249 er, extracellular lipids accumulate in human carotid plaques as distinct 3D structures that include a

250 TE and TT were not associated with calcified carotid plaques in either sex.

251 TATE activity on PET in recently symptomatic carotid plaques is not significantly different from cont

252 miR-210 contributes to stabilizing carotid plaques through inhibition of APC, ensuring smoo

253 regulated complement-related genes in human carotid plaques treated ex vivo.

254 In human carotid plaques, MerTK cleavage correlated with plaque n

255 ization are not systematically associated in carotid plaques, suggesting a temporal separation betwee

256 etection and characterization of symptomatic carotid plaques.

257 -DOTATATE binding in macrophages and excised carotid plaques.

258 t significantly different from contralateral carotids/plaques.

259 ated to enable in vivo measurement of radial/carotid pulse signals in near-surface arteries.

260 n detect intimal changes in a mouse model of carotid remodeling.

261 emic stroke preventative strategies, such as carotid revascularization for asymptomatic carotid steno

262 Of the 10 studies, 4 addressed patients with carotid sinus hypersensitivity, and the remaining 6 addr

263 The carotid sinus nerve (CSN) conveys electrical signals fro

264 clinical interest in patients with unstable carotid stenosis at risk of stroke.

265 or ischemic stroke: 18 patients with culprit carotid stenosis awaiting carotid endarterectomy and 8 c

266 likely to undergo procedural management for carotid stenosis compared with those in the salary-based

267 ive intensification, antihypertensive class, carotid stenosis intervention, and substance abuse refer

268 METHODS AND Thirty patients with severe carotid stenosis underwent (18)F-fluorodeoxyglucose-posi

269 s carotid revascularization for asymptomatic carotid stenosis, require reassessment given advances in

270 ardiac surgery include giant cell arteritis, carotid stenosis, stroke, hypercoagulable state, and DM

271 leading approaches to revascularization for carotid stenosis, yet contemporary data on trends in rat

272 Endarterectomy With Significant Extracranial Carotid Stenotic Disease).

273 ate of embolic complications associated with carotid stent placement.

274 Carotid surgery was performed 1.6+/-1.8 days after compl

275 0.796), but there was an increase in TBR in carotids (TBR = 0.027, 95% CI = 0.000 to 0.054; P = 0.04

276 A model of photochemical-induced carotid thrombosis was applied to miR-223 deficient mice

277 did not change in the weight-loss group, but carotid-to-femoral pulse wave velocity tended to decreas

278 left ventricular geometry were measured with carotid tonometry and cardiac magnetic resonance imaging

279 Fasting glucose levels and carotid ultrasonography measures were important predicto

280 The principal screening test was carotid ultrasonography, and the intervention in the 3 t

281 angiography prompted by abnormal results on carotid ultrasonography.

282 ports of all patients with reported internal carotid, vertebral, or suspected intracranial artery ane

283 trasound could not detect differences in the carotid wall even at 2 weeks post-surgery.

284 MRI examination and noninvasive capture of a carotid waveform using an iPhone camera (The waveform is

285 Analysis of carotid waveforms using intrinsic frequency methods can

286 Regardless of neck morphology, carotid waveforms were captured in all subjects, within

287 linical and imaging profile of patients with carotid web as well as their association with ischemic s

288 The presence of a carotid web at the carotid bifurcations was evaluated at

289 Nine patients with a symptomatic carotid web did not have major risk factors or other cau

290 To investigate the prevalence of symptomatic carotid web in patients with acute ischemic stroke due t

291 nt (kappa, 0.72) was observed for diagnosing carotid webs at CT angiography.

292 Conclusion Carotid webs at the symptomatic carotid bifurcation were

293 Interobserver agreement in the detection of carotid webs was examined by using kappa statistics.

294 found at the symptomatic side and two (0.5%) carotid webs were found at the asymptomatic side.

295 Results Eleven (2.5%) carotid webs were found at the symptomatic side and two

296 interobserver agreement in the assessment of carotid webs.

297 s IPH remained present in 50 (94%) of the 53 carotids with IPH at baseline, and it developed in five

298 IPH volume was present in 14 (26%) of the 53 carotids with IPH at baseline, and regression was presen

299 baseline magnetic resonance (MR) imaging (53 carotids with IPH) were randomly selected and were match

300 ine, and it developed in five (7%) of the 40 carotids without IPH at baseline.

301 nd were matched with 27 control subjects (53 carotids without IPH) to undergo a second MR examination