ライフサイエンスコーパス: carotid artery

1 tion followed by ligation of the left common carotid artery.

2 or pediatric cardiac catheterization via the carotid artery.

3 creased 90% 14 d after balloon injury in rat carotid artery.

4 ia a diagnostic catheter located in a common carotid artery.

5 revealed unilateral aplasia of the internal carotid artery.

6 erotic plaque formation and stability in the carotid artery.

7 of plasma lipids and stiffness of the common carotid artery.

8 nd platelets in athero-prone segments of the carotid artery.

9 ted and a flow probe was placed on the right carotid artery.

10 ima, media, or atherosclerotic plaque in the carotid artery.

11 were implanted unilaterally in the internal carotid artery.

12 therosclerosis has been mainly researched in carotid arteries.

13 out mice showed enhanced constriction in the carotid arteries.

14 ma compared with normal tissue from the same carotid arteries.

15 ery conditions, were observed in stented rat carotid arteries.

16 d positive CACS was stronger in femoral than carotid arteries.

17 placing semiconstrictive collars around both carotid arteries.

18 rch, extracranial, and intracranial internal carotid arteries.

19 bilateral paragangliomas around branches of carotid arteries.

20 occlusive thrombi following FeCl3 injury of carotid arteries.

21 substantial CaCCs in VSMCs of the aorta and carotid arteries.

22 x vivo perfusion or intravital microscopy of carotid arteries.

23 c calcification in the intracranial internal carotid arteries.

24 l carotid artery) in both the left and right carotid arteries.

25 ed macrophage content and lesion size in the carotid arteries.

26 lar cell adhesion molecule 1 in wire-injured carotid arteries.

27 ical properties in phantom and human cadaver carotid arteries.

28 velocities and low blood volume flow in the carotid arteries after ligation in FVB mice.

29 TSPAN2 expression is attenuated in mouse carotid arteries after ligation injury and in failed hum

30 Internal carotid artery agenesis is a rare anomaly that can be cl

31 downregulation of miR-494 expression in the carotid arteries, although miR-494 target genes were upr

32 underwent magnetic resonance imaging of the carotid arteries and aorta.

33 ium-dependent relaxations in renal arteries, carotid arteries and aortae, and flow-mediated dilatatio

34 Moreover, studies of experimental murine carotid arteries and cultured EC revealed that TWIST1 wa

35 liac arteries but not the brachial or common carotid arteries and not correlated significantly with b

36 expression of 5-HTT was elevated in injured carotid arteries and over-expression of 5-HTT induced pr

37 iR-181b reduced thrombus formation by 73% in carotid arteries and prolonged time to occlusion by 1.6-

38 Herein, the anatomy of the carotid arteries and the Doppler sonography and CT angio

39 induced by endothelial abrasion of the right carotid artery and abdominal aorta of 7 rabbits fed an a

40 induced by endothelial abrasion of the right carotid artery and abdominal aorta of 7 rabbits fed an a

41 The left internal carotid artery and both internal jugular veins were cann

42 for GPIbalpha have delayed thrombosis after carotid artery and cremaster microvascular injury withou

43 Carotid artery and echocardiographic abnormalities, and

44 rticles enhanced miR-126 availability in the carotid artery and improved reendothelialization of inju

45 gnal significantly increased in the inflamed carotid artery and in the aorta.

46 cine model of synthetic graft placed between carotid artery and ipsilateral jugular vein was used to

47 etween relative radiotherapy exposure to the carotid artery and prevalence of WML volume and CMBs.

48 e of the atherosclerotic plaques in both the carotid artery and the aortic root.

49 cclusion after stroke (intracranial internal carotid artery and/or middle cerebral artery M1 and/or M

50 hoGEF tyrosine phosphorylation in rat common carotid arteries, and siRNA-mediated down-regulation of

51 26 +/- 12 mm Hg; P<0.01), distension of the carotid artery, and carotid arterial wall tension (P<0.0

52 restenosis was induced by ligating the left carotid artery, and VSMCs were pretreated with platelet-

53 rrupted aortic arches, large brachiocephalic/carotid artery aneurysms and cardiac septation defects,

54 large or giant wide-necked proximal internal carotid artery aneurysms.

55 ents (nine men, two women) undergoing common carotid artery angiography.

56 munoreactive cells in segments of the common carotid artery, aorta, and pulmonary artery appears to r

57 moreceptors have been isolated to the common carotid artery, aorta, and pulmonary artery of turtles.

58 The PMCA4a:4b ratio in uninjured carotid arteries ( approximately 1:1) was significantly

59 ing those the endothelium experiences in the carotid artery are responsible for determining the fate

60 The renal and cephalocervical (mainly carotid arteries) arterial beds are classically involved

61 tic plaques of the abdominal aorta and right carotid artery as compared with normal control arteries

62 tic plaques of the abdominal aorta and right carotid artery as compared with normal control arteries

63 In ligated carotid arteries at 4 d, SPM treatment was associated wi

64 nonclassical monocytes patrol inside healthy carotid arteries at a velocity of 36 mum/min, 3x faster

65 Extracranial internal carotid artery atherosclerotic occlusive disease is a co

66 We used identical carotid artery B-mode ultrasonographic methods in 5 coho

67 ar carotid interventions on the extracranial carotid artery between 2009 and 2014.

68 l of the common carotid artery (CCA-IMT) and carotid artery bifurcation (BIF-IMT) between 2010 and 20

69 he carotid body, a chemosensory organ at the carotid artery bifurcation that monitors blood oxygen an

70 Chronic reductions in carotid artery blood flow are associated with increased

71 hibited endothelial recovery in wire-injured carotid arteries, but this effect was also abrogated by

72 , paraganglioma is located around the common carotid artery, but may also be located within the middl

73 he carotid plaques and contralateral plaques/carotid arteries by an experienced radionuclide radiolog

74 Intracranial carotid artery calcification contributed to 75% of all s

75 ll strokes; for aortic arch and extracranial carotid artery calcification this incidence was only 45%

76 iated with progressive calcification; in the carotid arteries, calcification appears to influence vas

77 ow increased mean blood pressure measured by carotid artery cannulation and increased microvascular r

78 otid IMT was measured at 3 locations (common carotid artery, carotid bulb, internal carotid artery) i

79 body by the injection of adenosine into the carotid artery causes a dose-dependent increase in minut

80 Common carotid artery (CCA) balloon angioplasty injury was perf

81 hickness of the right far wall of the common carotid artery (CCA-IMT) and carotid artery bifurcation

82 neointima and adventitia of the ligated left carotid arteries compared with the right.

83 in expression were observed in ligated mouse carotid arteries, correlating closely with expression of

84 ion analysis of 5 additional atherosclerotic carotid arteries demonstrated biofilm bacteria within al

85 nsibility and Young's elastic modulus at the carotid artery, derived from carotid artery ultrasonogra

86 bal cerebral tissue injury, and cognition in carotid artery disease (CAD).

87 To compare prevalence of carotid artery disease and its various types of lesions

88 ress the risks and benefits of screening for carotid artery disease as well as how to apply the guide

89 olism and hemodynamic disturbances caused by carotid artery disease in these disorders is discussed.

90 gnificantly more likely to have coronary and carotid artery diseases.

91 The left common carotid artery divided into the internal carotid, extern

92 iPhone camera was held in a cradle over the carotid artery during iPhone measurements.

93 ter Embozene(R) embolization of the external carotid artery (ECA).

94 improved per-patient rate of weight gain or carotid artery echodensity; 71.0% of participants succee

95 emic events (AAE), and extracranial internal carotid artery (eICA) stenoses, detectable via submandib

96 iveness of carotid artery stenting (CAS) and carotid artery endarterectomy (CEA) for the prevention o

97 osed to chronic disturbed flow, and in mouse carotid artery endothelia subjected to surgically induce

98 ry, comprehensive ophthalmic evaluation, and carotid artery evaluation (by Doppler/angiography) on th

99 1 was enriched in calcified regions of human carotid arteries, examined by immunohistochemistry.

100 Ligated carotid arteries from diabetic mice developed more exten

101 versus nondiabetic patients and in aorta and carotid arteries from streptozotocin-diabetic versus non

102 umber and size of atherosclerotic plaques in carotid artery, heart, aortic arch and aorta in acute an

103 lood flow (CBF) was measured at the internal carotid artery (ICA) and vertebral artery (VA) and CBF v

104 neurysm in the cavernous segment of internal carotid artery (ICA) is a relatively rare entity.

105 graphy to distinguish true cervical internal carotid artery (ICA) occlusion from pseudo-occlusion (de

106 emic events (AAEs) and extracranial internal carotid artery (ICA) stenosis as risk factors for silent

107 eginning at the cervical segment of internal carotid artery (ICA) together with a dissecting aneurysm

108 and improved reendothelialization of injured carotid arteries in vivo.

109 ommon carotid artery, carotid bulb, internal carotid artery) in both the left and right carotid arter

110 d with higher risk of stroke due to internal carotid artery injuries, but monitoring was not useful f

111 Among those with isolated internal carotid artery injuries, five of nine with delayed strok

112 g for ipsilateral and contralateral internal carotid artery injury grade (adjusted risk ratio, 2.91;

113 ient mice have reduced thrombus growth after carotid artery injury relative to conventionally raised

114 Among patients who sustained an internal carotid artery injury with or without additional vessel

115 hrombus formation on ferric chloride-induced carotid artery injury.

116 stocompatibility complex mismatched aorta to carotid artery interposition grafts, using wild type (WT

117 ing; 200-250 g/d) intakes slows 12-mo common carotid artery intima media thickness (CCA IMT) progress

118 Outcomes included changes in right common carotid artery intima-media thickness (CCA-IMT) and new

119 Data on common carotid artery intima-media thickness (CCA-IMT) were ava

120 We investigated the association of common carotid artery intima-media thickness (CCA-IMT) with sno

121 D) and evaluated the association with common carotid artery intima-media thickness (cCIMT) using mult

122 pants of the Children's Health Study on whom carotid artery intima-media thickness (CIMT) measurement

123 erosis, as measured by progression of common carotid artery intima-media thickness (cIMT), in adults

124 Coprimary outcomes included common carotid artery intima-media thickness and coronary arter

125 Carotid artery intima-media thickness and inter-adventit

126 gression of coronary artery calcium and mean carotid artery intima-media thickness and long-term expo

127 ected adults and evaluated associations with carotid artery intima-media thickness and plaque.

128 ans (fully-adjusted P=0.004) and with common carotid artery intima-media thickness in the Framingham

129 ence in the rates of change in either common carotid artery intima-media thickness or coronary artery

130 er high-density lipoprotein cholesterol with Carotid artery intima-media thickness strengthened with

131 METHODS AND Carotid artery intima-media thickness was measured at ba

132 Common carotid artery intima-media thickness was measured by ul

133 al aortic calcium score, common and internal carotid artery intima-media thickness, and ankle-brachia

134 condary outcomes included vascular function, carotid artery intima-media thickness, augmentation inde

135 ession of coronary artery calcium and common carotid artery intima-media thickness.

136 brachial artery flow-mediated dilatation and carotid artery intima-medial hyperplasia.

137 he primary outcome was the rate of change in carotid-artery intima-media thickness (CIMT), which was

138 markers, 24-h ambulatory blood pressure, and carotid artery intimamedia thickness.

139 Trifurcation of the common carotid artery is an unusual variation.

140 formation, we induced neointima formation by carotid artery ligation in Nf1(+/-) and WT mice with gen

141 In a mouse carotid artery ligation model, genetic knockdown of the

142 ice, disturbed flow as the result of partial carotid artery ligation rapidly suppressed endothelial C

143 Furthermore, in Tcad/ApoE-DKO mice, carotid artery ligation resulted in a significant increa

144 In vivo, adult FVB mice underwent unilateral carotid artery ligation with administration of RvD2, MaR

145 ditioning ischemic insult (unilateral common carotid artery ligation) to 12- to 14-week-old mice and

146 formation and vascular remodelling following carotid artery ligation.

147 avated intimal hyperplasia following partial carotid artery ligation.

148 e devised a procedure to stabilize the mouse carotid artery mechanically without altering blood dynam

149 compared with the contralateral asymptomatic carotid arteries (median: 1.4; Q1-Q3, 1.3-1.6).

150 En face staining of the murine aorta or carotid arteries modified with flow-altering cuffs demon

151 dle cerebral artery to extracranial internal carotid artery more than or equal to 3.

152 al ischemia produced by transient, bilateral carotid artery occlusion (BCAO).

153 sion and VaD was induced by bilateral common carotid artery occlusion (BCCAO) in adult male Sprague D

154 Permanent bilateral common carotid artery occlusion (BCCAO) was used as a model of

155 similar in HRG-deficient and wild-type mice, carotid artery occlusion after FeCl3 injury was accelera

156 Klkb1(-/-) mice have delayed carotid artery occlusion times on the rose bengal and fe

157 middle cerebral artery or terminal internal carotid artery occlusion using computed tomographic angi

158 ed in the AQP4(-/-) vs. AQP4(+/+) mice after carotid artery occlusion, as were blood-brain barrier (B

159 2/3(-/-)) were subjected to bilateral common carotid artery occlusion, followed by reperfusion and tr

160 hemic injury was induced by bilateral common carotid artery occlusion, whereas severe focal stroke in

161 method to monitor retinopathy in a bilateral carotid artery occlusion-induced ocular ischemia, we obs

162 lts demonstrate that P. acnes can infect the carotid arteries of humans with atherosclerosis as a com

163 monocrotaline, whereas it was upregulated in carotid arteries of Macaca fascicularis subjected to ath

164 sly with a fast intra-vascular sensor in the carotid artery of anaesthetized, mechanically ventilated

165 e left external and internal branches of the carotid artery of male FVB mice and performed sham opera

166 A catheter was inserted into the right carotid artery of mice, which acted as a vascular graft.

167 vein opacification in patients with internal carotid artery or middle cerebral artery (MCA) stroke an

168 All consecutive patients with internal carotid artery or middle cerebral artery occlusions tran

169 with occlusion of the intracranial internal carotid artery or proximal middle cerebral artery who ha

170 pophyseal opening accommodating the internal carotid arteries) or osteichthyans (facial nerve exiting

171 thrombus formation was analyzed in a murine carotid artery photochemical injury model.

172 d coronary artery calcification (CARDIA) and carotid artery plaque burden (BioImage).

173 tima-media thickness (CCA-IMT) and new focal carotid artery plaque formation (IMT >1.5 mm) over media

174 dy compares predictive uses of CAC presence, carotid artery plaque presence, and high IMT for inciden

175 ient was greater than 0.8 in all measures of carotid artery/plaque uptake (SUV) and greater than 0.6

176 carriers showed reduced CXCR4 expression in carotid artery plaques (n=188), which was furthermore as

177 Presence of carotid artery plaques on the involved side was signific

178 c plaques and the asymptomatic contralateral carotid arteries/plaques showed no significant differenc

179 % decrease in clotting time) was observed in carotid artery preparations of fat-fed mice subjected to

180 i-viral delivery of miR-30c into injured rat carotid arteries prevented the injury-induced increase i

181 idence of peripheral artery disease), of the carotid arteries (previous carotid artery revascularisat

182 Surgical cutdown for access to the common carotid artery provides a more direct route for certain

183 Trauma is the most common cause of carotid artery pseudoaneurysms.

184 Unstable atherosclerotic lesions in carotid arteries require surgical endarterectomy to redu

185 disease), of the carotid arteries (previous carotid artery revascularisation or asymptomatic carotid

186 tional Cardiovascular Data Registry's (NCDR) Carotid Artery Revascularization and Endarterectomy Regi

187 cular Registry (1999 CAS; 3255 CEA) and 4055 Carotid Artery Revascularization and Endarterectomy Regi

188 ery's Vascular Registry: 96.7% versus 44.5%; Carotid Artery Revascularization and Endarterectomy Regi

189 Tissue lysates of mouse carotid arteries revealed that cLDL induced the expressi

190 cnes 16S rRNA gene was detectable in 4 of 15 carotid artery samples, and viable P. acnes was one amon

191 The variations of the carotid arteries should be known to avoid and reduce the

192 P values were adjusted for age, sex, carotid artery site, and family relations.

193 tid disease was defined as cervical internal carotid artery stenosis (>50%) or occlusion.

194 Of 10579 individuals with a diagnosis of carotid artery stenosis (4615 women and 5964 men; mean [

195 hemia (OR, 7.67; CI, 5.31-11.07; P < 0.001), carotid artery stenosis (OR, 7.52; CI, 6.22-9.09; P < 0.

196 TBRmax was not significantly correlated to carotid artery stenosis (rho=0.506, P=0.135).

197 The incidence of carotid artery stenosis and plaques, cardiac embolic sou

198 ars, range, 59.4-69.7) with ipsilateral >70% carotid artery stenosis and who underwent carotid endart

199 tid artery stenosis with 70%-89% and 50%-69% carotid artery stenosis at presentation.

200 y was queried for individuals diagnosed with carotid artery stenosis between October 1, 2006, and Sep

201 carotid endarterectomy only with substantial carotid artery stenosis disease progression.

202 tool to identify patients with asymptomatic carotid artery stenosis most likely to benefit from caro

203 tid artery revascularisation or asymptomatic carotid artery stenosis of at least 50%), or coronary ar

204 Thirty-one (40%) had carotid artery stenosis of at least 70%.

205 The degree of carotid artery stenosis on histology correlated well wit

206 Carotid artery stenosis on the involved side was worse i

207 l/6J mice were subjected to bilateral common carotid artery stenosis or a sham operation and fed norm

208 ndard surgical risk with severe asymptomatic carotid artery stenosis randomly assigned to carotid art

209 ompared CAS against CEA for the treatment of carotid artery stenosis were selected.

210 were modeled for patients with asymptomatic carotid artery stenosis with 70%-89% and 50%-69% carotid

211 s, the benefits of treatment of asymptomatic carotid artery stenosis with carotid endarterectomy (CEA

212 ich the subset of patients with asymptomatic carotid artery stenosis with IPH on MR images would unde

213 ternative to endarterectomy for treatment of carotid artery stenosis, but long-term efficacy is uncer

214 ugh many factors influence the management of carotid artery stenosis, it is not well understood wheth

215 n significant variation in classification of carotid artery stenosis, likely leading to differences i

216 my (CEA) for the prevention of stroke due to carotid artery stenosis.

217 ical guideline on screening for asymptomatic carotid artery stenosis.

218 provider-induced demand in the management of carotid artery stenosis.

219 most meaningful risk factor was ipsilateral carotid artery stenosis.

220 Among patients with carotid-artery stenosis who had been randomly assigned t

221 The impact of carotid artery stent fractures on the incidence of adver

222 nt, and one patient underwent placement of a carotid artery stent.

223 lict regarding the relative effectiveness of carotid artery stenting (CAS) and carotid artery endarte

224 four randomised controlled trials comparing carotid artery stenting (CAS) and carotid endarterectomy

225 Despite increased carotid artery stenting (CAS) dissemination following th

226 h following carotid endarterectomy (CEA) and carotid artery stenting (CAS) on a national level in Ger

227 Effectiveness of carotid artery stenting (CAS) relative to carotid endart

228 Clinical trials demonstrated the efficacy of carotid artery stenting (CAS) relative to carotid endart

229 ted imaging (DWI) are frequently found after carotid artery stenting (CAS), but their clinical releva

230 ts following carotid endarterectomy (CEA) or carotid artery stenting (CAS), the applicability of thes

231 undergoing carotid endarterectomy (CEA) and carotid artery stenting (CAS); to describe hospital vari

232 75.8 years; 43% women) and 231077 underwent carotid artery stenting (mean age, 75.4 years; 49% women

233 Carotid endarterectomy and carotid artery stenting are the leading approaches to re

234 Of 1021 patients treated with carotid artery stenting during a mean follow-up of 3.1+/

235 g-term effectiveness in stroke prevention by carotid artery stenting in a large number of patients in

236 ed from controlled clinical trials undergoes carotid artery stenting in daily clinical practice.

237 ggest that independent modular filter use in carotid artery stenting in high surgical risk patients i

238 nrandomized, open-label, single-arm study of carotid artery stenting in high surgical risk patients w

239 rom 1999 to 2014, whereas the performance of carotid artery stenting increased until 2006 and then de

240 Long-term stroke prevention by carotid artery stenting is effective in experienced cent

241 carotid artery stenosis randomly assigned to carotid artery stenting or carotid endarterectomy (Abbot

242 Embolic protection during carotid artery stenting reduces the rate of thromboembol

243 ural intervention (carotid endarterectomy or carotid artery stenting) compared with medical managemen

244 ata outside of controlled clinical trials in carotid artery stenting.

245 lic Protection System in patients undergoing carotid artery stenting.

246 Carotid endarterectomy and carotid artery stenting.

247 Previous clinical trials have suggested that carotid-artery stenting with a device to capture and rem

248 In this trial, we compared carotid-artery stenting with embolic protection and caro

249 The effects of HIV on carotid artery structure may differ across the lifespan,

250 rcle of Willis upon stenosis of the internal carotid arteries, supply blood to the anterior cerebral

251 ersus control, 2.12+/-0.27; P=0.001) and the carotid arteries (TBRmax: CKD, 2.45+/-0.65 versus contro

252 P < 0.05, respectively) in the ligated left carotid arteries than the right carotid or healthy arter

253 dioluminescence signal from the ligated left carotid artery than the nonligated right carotid: 1.63 x

254 Intravital microscopy of the carotid artery, the jugular vein, and cremasteric arteri

255 l microscopy, we evaluated susceptibility to carotid artery thrombosis after FeCl3-induced injury in

256 rosclerotic lesion area, and delayed time to carotid artery thrombosis in a photochemical injury mode

257 In an FeCl3 injury-induced carotid artery thrombosis model, thrombus growth rate an

258 in is more potent than hirulog-1 in a murine carotid artery thrombosis model.

259 d VWF had defective hemostasis and defective carotid artery thrombosis, but experienced significant c

260 platelet VWF showed normal tail bleeding and carotid artery thrombosis, similar to wild-type mice.

261 del of ferric chloride induced non-occlusive carotid artery thrombosis.

262 s, we also performed ferric chloride-induced carotid artery thrombosis.

263 mostasis in a tail bleeding model and normal carotid artery thrombosis.

264 aluated in rat models of mural and occlusive carotid artery thrombosis.

265 nd neutrophilia that precedes development of carotid artery thrombus formation.

266 st magnetic resonance imaging of basilar and carotid arteries to measure cerebral blood flow.

267 Exposure of the carotid artery to radiation was not associated with WML

268 injected with microembolic materials in the carotid artery to serve as positive controls.

269 In mice, the carotid artery to the ipsilateral jugular vein was conne

270 We report a case of left common carotid artery trifurcation in a 74-year-old man.

271 d CT angiography findings of the left common carotid artery trifurcation were described with images.

272 modulus at the carotid artery, derived from carotid artery ultrasonography (n = 6,531 and 6,528); an

273 S Cohort Study who underwent repeated B-mode carotid artery ultrasound imaging in 2004-2013.

274 S Cohort Study who underwent repeated B-mode carotid artery ultrasound imaging in 2004-2013.

275 ic stroke propensity through the left common carotid artery using an idealized aortic arch model.

276 ke Scale score), site of occlusion (internal carotid artery vs M1 segment of middle cerebral artery v

277 The change in mean carotid artery wall area was -3.37 mm(2) after 12 months

278 Median carotid artery wall echodensity and carotid-femoral puls

279 Carotid artery wall imaging was performed in 10 healthy

280 and intima-media-adventitia thickness], and carotid artery wall stress [CAWS]).

281 , patients with CGD had a 22% lower internal carotid artery wall volume compared with control subject

282 s was measured based on displacements of the carotid artery wall, and Young's modulus was 2-fold grea

283 ue is a promising new tool for MR imaging of carotid artery walls.

284 The endothelial damage in atherosclerotic carotid arteries was assessed by electron microscopy and

285 Wire-induced injury of the carotid artery was performed in mice with a ubiquitous,

286 In the present study, human atherosclerotic carotid arteries were examined following endarterectomy

287 After noninvasive imaging, the murine carotid arteries were imaged in situ and ex vivo, follow

288 performed in vivo experiments where rat left carotid arteries were injured using balloon angioplasty

289 Carotid arteries were isolated from newborn (postnatal d

290 Plaque areas from both carotid arteries were summed as the carotid plaque burde

291 visfatin in mice with partially ligated left carotid artery were found to have significantly increase

292 Extradural aneurysms in the internal carotid artery were included; fusiform aneurysms, infund

293 B-mode ultrasound measurements of the carotid artery were made in 196 children with FH and 64

294 and a high number of microembolic signals in carotid artery were observed during sheath/catheter mani

295 rotid plaques and contralateral asymptomatic carotid arteries, which was corrected for background act

296 rein, we show that neointima formation after carotid artery wire injury reduces markedly in CD40(-/-)

297 METHODS AND In mouse carotid arteries with established intimal lesions tailor

298 decellularized vessels obtained from porcine carotid arteries with poly (ethylmethacrylate-co-diethyl

299 low-up, 40 of 42 patients (95%) had a normal carotid artery, with 2 instances of mild stenosis.

300 IUGR group was seen in the iliac but not the carotid arteries without between-sex differences.