ライフサイエンスコーパス: carotid artery injury

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1 an occlusive thrombus after FeCl(3)-induced carotid artery injury.

2 well as neointima formation, after ZO common carotid artery injury.

3 lets, formed unstable thrombi in response to carotid artery injury.

4 ically upregulated following balloon-induced carotid artery injury.

5 ation and neointimal thickening after murine carotid artery injury.

6 hrombus formation on ferric chloride-induced carotid artery injury.

7 tiplatelet agent developed stroke related to carotid artery injuries.

8 One hundred fourteen patients had 157 carotid artery injuries (43 bilateral), and 79 patients

9 2) (PAMAM G-3) prevents thrombosis following carotid artery injury and pulmonary thromboembolism in m

10 endothelial matrix, mice were subjected to a carotid artery injury assay in which ferric chloride adm

11 d with higher risk of stroke due to internal carotid artery injuries, but monitoring was not useful f

12 FVIII had no effect after longer (3-minute) carotid artery injury, but it shortened the TTO after sh

13 One patient with grade II carotid artery injuries (by CTA and DSA) on antiplatelet

14 Angiography identified 24 patients with carotid artery injuries (CAI) and 43 patients with verte

15 Whole-blood clotting times and FeCl3 carotid artery injury correction demonstrated that plate

16 Among those with isolated internal carotid artery injuries, five of nine with delayed strok

17 g for ipsilateral and contralateral internal carotid artery injury grade (adjusted risk ratio, 2.91;

18 Ferric chloride was used to induce carotid artery injury in 97 wild-type (WT), 84 PAI-1-/-,

19 After carotid artery injury in CD40 ligand knockout (CD40L(-/-

20 Following wire-induced carotid artery injury in mice, the majority of circulati

21 s neointimal formation after balloon-induced carotid artery injury in rats.

22 After common carotid artery injury, knockdown of Nox4 by adenoviral N

23 red with wild-type (WT) in a FeCl(3)-induced carotid artery injury model.

24 ibitor significantly reduced thrombosis in a carotid artery injury model.

25 r wild-type (WT) littermates using the mouse carotid artery injury model.

26 lls and promoted reendothelialization in the carotid artery injury model.

27 In 2 carotid artery injury models (FeCl(3) and Rose Bengal/la

28 After carotid artery injury, Nf1(+/-) mice demonstrated increa

29 d markedly impaired thrombus formation after carotid artery injury or inferior vena cava ligation.

30 ient mice have reduced thrombus growth after carotid artery injury relative to conventionally raised

31 e found that after mesenteric arteriolar and carotid artery injury, Slit2 delayed vessel occlusion ti

32 By using a ferric chloride (FeCl3)-induced carotid artery injury thrombosis model, we found time to

33 Thrombus generation after photochemical carotid artery injury was accelerated in iron-loaded mic

34 The time to occlusion after FeCl(3)-induced carotid artery injury was delayed (11.8 +/- 3.6 minutes,

35 Left common carotid artery injury was induced with a guidewire in ap

36 able occlusive thrombi after ferric chloride carotid artery injury, whereas the majority of wild-type

37 Recognition of brain injury in asymptomatic carotid artery injuries with conventional methods can be

38 Among patients who sustained an internal carotid artery injury with or without additional vessel

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