ライフサイエンスコーパス: carotid artery thrombosis

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1 s, we also performed ferric chloride-induced carotid artery thrombosis.

2 times and impaired mesenteric arteriole and carotid artery thrombosis.

3 re protected from TF-dependent FeCl3-induced carotid artery thrombosis.

4 del of ferric chloride induced non-occlusive carotid artery thrombosis.

5 FeCl3 was used to induce carotid artery thrombosis.

6 mostasis in a tail bleeding model and normal carotid artery thrombosis.

7 aluated in rat models of mural and occlusive carotid artery thrombosis.

8 -) mice have long bleeding times and delayed carotid artery thrombosis, 78 +/- 6.7 minutes, versus 31

9 l microscopy, we evaluated susceptibility to carotid artery thrombosis after FeCl3-induced injury in

10 beta3(Delta760-762) mice were protected from carotid artery thrombosis after vessel injury with FeCl(

11 UT7 activity was examined in mouse models of carotid artery thrombosis and collagen/epinephrine-induc

12 ant in both the rat model of FeCl(2)-induced carotid artery thrombosis and the rabbit model of jugula

13 inal fragment protects against FeCI3-induced carotid artery thrombosis as well as cerebral infarction

14 d only a partial correction, but in an FeCl3 carotid artery, thrombosis assay correction was equivale

15 d VWF had defective hemostasis and defective carotid artery thrombosis, but experienced significant c

16 ce showed nearly complete protection against carotid artery thrombosis caused by low FeCl(3) injury.

17 ive vehicle or drug immediately after common carotid artery thrombosis (CCAT).

18 rosclerotic lesion area, and delayed time to carotid artery thrombosis in a photochemical injury mode

19 re resistant to the development of occlusive carotid artery thrombosis in the absence of systemic fib

20 ) mice showed retardation in FeCl(3)-induced carotid artery thrombosis in vivo.

21 -bleeding times and impaired FeCl(3)-induced carotid artery thrombosis in vivo.

22 large vessel thrombus formation, we studied carotid artery thrombosis in wild-type mice, mice lackin

23 arterioles (P<0.05) but not from accelerated carotid artery thrombosis induced by the HM/LF diet.

24 sis (P < .01) in an electrolytically induced carotid artery thrombosis model in dogs.

25 Finally, we showed with an in vivo carotid artery thrombosis model that genetic deletion of

26 Using a murine carotid artery thrombosis model we demonstrated CD36-dep

27 time) and in vivo (e.g. rat FeCl(2)-induced carotid artery thrombosis model).

28 n overt bleeding diathesis, but in a FeCl(3) carotid artery thrombosis model, all showed impaired thr

29 In a carotid artery thrombosis model, both AbetaPP(-/-) and A

30 In a mouse carotid artery thrombosis model, non-targ-CD39, although

31 In an FeCl3 injury-induced carotid artery thrombosis model, thrombus growth rate an

32 in is more potent than hirulog-1 in a murine carotid artery thrombosis model.

33 s antithrombotic effect examined in a rabbit carotid artery thrombosis model.

34 platelet VWF showed normal tail bleeding and carotid artery thrombosis, similar to wild-type mice.

35 ptor-deleted mice (Bdkrb2(-/-)) have delayed carotid artery thrombosis times and prolonged tail bleed

36 Susceptibility to carotid artery thrombosis was first examined in wild-typ

37 Carotid artery thrombosis was not accelerated in mice de

38 KIN59 significantly inhibited FeCl3-induced carotid artery thrombosis without affecting hemostasis.

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