ライフサイエンスコーパス: carotid atherosclerosis

1 (CT) can assess both anatomy and biology of carotid atherosclerosis.

2 childhood to adulthood had increased risk of carotid atherosclerosis.

3 on to the clinical and ultrasound markers of carotid atherosclerosis.

4 ne correlated with the presence or extent of carotid atherosclerosis.

5 rticulate matter exposures on development of carotid atherosclerosis.

6 (n=362) in a prevalent case-control study of carotid atherosclerosis.

7 tudy the pathogenesis and natural history of carotid atherosclerosis.

8 ic core and intraplaque hemorrhage, in human carotid atherosclerosis.

9 y for suspected NASH; 427 were evaluated for carotid atherosclerosis.

10 Carotid atherosclerosis accounts for approximately 10% o

11 was generally associated with higher risk of carotid atherosclerosis and clinical cardiovascular dise

12 We investigated the relationship between carotid atherosclerosis and CRP and their joint roles in

13 rdiovascular disease, with evidence of early carotid atherosclerosis and moderately elevated LDL chol

14 ment is associated with an increased risk of carotid atherosclerosis and stroke, determinants of cogn

15 cipal method for determining the severity of carotid atherosclerosis and the need for endarterectomy.

16 idence that auscultatory gaps are related to carotid atherosclerosis and to increased arterial stiffn

17 in elderly men and postmenopausal women with carotid atherosclerosis, as well as with risk of stroke

18 These findings indicate excess coronary and carotid atherosclerosis at ABI values below 1.10 (men) a

19 endovascular techniques for the treatment of carotid atherosclerosis, beginning with balloon angiopla

20 Baseline carotid atherosclerosis but not atherosclerotic CVD risk

21 cholesterol were associated only weakly with carotid atherosclerosis but were associated strongly wit

22 arotid endarterectomy underwent screening of carotid atherosclerosis by ultrasound and MR.

23 We evaluated the prevalence of carotid atherosclerosis compared with a control populati

24 In nine subjects with carotid atherosclerosis, double-oblique, contrast materi

25 pausal women and 1124 men (>/=45 years) with carotid atherosclerosis, from prospective population-bas

26 An association between fatty liver and carotid atherosclerosis has been established; however, i

27 plex roles of pre-existing disease (frailty, carotid atherosclerosis, hypertension, low diastolic blo

28 plasma fatty acid pattern is associated with carotid atherosclerosis in a direction generally consist

29 ation between antioxidant vitamin status and carotid atherosclerosis in a group of elderly persons.

30 onomic reward, and the 4-year progression of carotid atherosclerosis in a population-based sample of

31 osis is associated with a high risk of early carotid atherosclerosis in G1 CHC patients.

32 vational studies have reported less frequent carotid atherosclerosis in healthy women taking postmeno

33 erresponsivity to accelerated development of carotid atherosclerosis in human subjects and to exacerb

34 ter repeat polymorphism were associated with carotid atherosclerosis in humans, an effect that was ex

35 history of major depression and subclinical carotid atherosclerosis in middle-aged women.

36 man subjects and to exacerbated coronary and carotid atherosclerosis in monkeys.

37 e used to identify a subset of patients with carotid atherosclerosis in need of intensified medical t

38 strogen use has been associated with reduced carotid atherosclerosis in observational studies, but th

39 t of RRR-alpha-tocopherol supplementation on carotid atherosclerosis in patients with stable coronary

40 RA inflammation contribute significantly to carotid atherosclerosis in RA, and may modify one anothe

41 the extent to which ACS are associated with carotid atherosclerosis in RA.

42 levels in women with and without significant carotid atherosclerosis in the Atherosclerosis Risk in C

43 f antioxidant vitamins and either measure of carotid atherosclerosis in the women.

44 entify the lipid-rich necrotic core in human carotid atherosclerosis in vivo with high sensitivity an

45 us caps from thin and ruptured caps in human carotid atherosclerosis in vivo.

46 abstinence are associated with lower odds of carotid atherosclerosis in women.

47 The effect of ezetimibe on carotid atherosclerosis is indeterminate, but ezetimibe

48 nopause and hormone replacement therapy with carotid atherosclerosis is limited.

49 lus statins showed that modest regression of carotid atherosclerosis is possible in individuals with

50 A total of 37 subjects with carotid atherosclerosis (mean age, 69.9 years; age range

51 6 was negatively associated to the extent of carotid atherosclerosis measured as intima-media thickne

52 uneck study with long-term follow-up data on carotid atherosclerosis measured by high-resolution dupl

53 Carotid atherosclerosis, measured as carotid intima-medi

54 ing endarterectomized patients with advanced carotid atherosclerosis (n=92) and myocardial infarction

55 llele was associated not only with prevalent carotid atherosclerosis (odds ratio [OR]: 1.46 [95% conf

56 ant of FABP4 on cardiovascular morbidity and carotid atherosclerosis on a population level (n=7491) a

57 g similar pathogenesis for both coronary and carotid atherosclerosis, one could interpret risk factor

58 pertrophy, systolic dysfunction, coronary or carotid atherosclerosis, or other vascular abnormalities

59 Carotid atherosclerosis, SLE duration, damage index, ser

60 0, 10.7) after adjustment for age, race, and carotid atherosclerosis status.

61 released February 1991) and the Asymptomatic Carotid Atherosclerosis Study (ACAS clinical alert relea

62 erectomy Trial (NASCET) and the Asymptomatic Carotid Atherosclerosis Study (ACAS) demonstrated the ef

63 Although the Asymptomatic Carotid Atherosclerosis Study (ACAS) reported that carot

64 erectomy Trial (NASCET) and the Asymptomatic Carotid Atherosclerosis Study (ACAS) showed significant

65 The Asymptomatic Carotid Atherosclerosis Study (ACAS) showed that carotid

66 osterone and SHBG to be inversely related to carotid atherosclerosis, suggesting their potential impo

67 rd have significantly greater progression of carotid atherosclerosis than more advantaged men.

68 Addition of CRP or carotid atherosclerosis to conventional risk factors mod

69 We measured carotid atherosclerosis using high-resolution ultrasound

70 Carotid atherosclerosis was assessed by ultrasound, wher

71 The prevalence and severity of carotid atherosclerosis was greatest in those with the l

72 Two measures of subclinical carotid atherosclerosis were assessed using B-mode ultra

73 Eight patients with symptomatic carotid atherosclerosis were imaged using 18FDG-PET and

74 pite the significant associations of CRP and carotid atherosclerosis with CVD, these measures modestl