コーパス検索結果 (1語後でソート)
通し番号をクリックするとPubMedの該当ページを表示します
1 susceptible waveform mimicking flow in human carotid sinus.
2 lation of the aortic arch and/or ipsilateral carotid sinus.
3 ve terminals innervating the aortic arch and carotid sinus.
4 10) developed atherosclerotic lesions in the carotid sinuses.
5 e coronary arteries, the aortic arch and the carotid sinuses.
7 otid baroreceptors via a vascularly isolated carotid sinus and anodal block of baroreceptor afferent
9 ng pressure increments (80-100 mm Hg) in the carotid sinus and by electrical stimulation (stimulus in
11 it and pressures distending the aortic arch, carotid sinus and coronary artery baroreceptors were con
13 two distinct regions of the carotid artery (carotid sinus and distal internal carotid artery) that a
14 g muscle contraction following disruption of carotid sinus and vagus nerves it is proposed that: (1)
15 espiratory input following disruption of the carotid sinus and vagus nerves significantly blunted, bu
17 ulation of baroreceptors in the aortic arch, carotid sinuses and coronary arteries, stimulation of re
18 ing baroreceptor regions in the aortic arch, carotid sinus, and right subclavian artery, as well as i
19 opulmonary bypass, reflexogenic areas of the carotid sinuses, aortic arch and coronary arteries and t
20 r signals from arterial baroreceptors in the carotid sinuses are processed within the brain and contr
21 f interaction between pulmonary arterial and carotid sinus baroreceptor reflexes; physiological and p
23 of the total sensory input conveyed from the carotid sinus baroreceptors to the dlNTS is mediated by
25 nous oxyradicals produced in atherosclerotic carotid sinuses contribute to baroreceptor dysfunction.
28 l syncope (227 [35%]); other causes included carotid sinus hypersensitivity (37 [5.8%]), and a group
31 ion for non-life-threatening situations (eg, carotid sinus hypersensitivity), most device advisories
32 Of the 10 studies, 4 addressed patients with carotid sinus hypersensitivity, and the remaining 6 addr
33 subjects with a positive response to either carotid sinus massage (CSM) or lower body negative press
34 influenced by the method of execution of the carotid sinus massage and the coexistence of the cardioi
35 ch patient underwent a head-up tilt test and carotid sinus massage during continuous electrocardiogra
39 climatization, which leads to an increase in carotid sinus nerve (CSN) activity and ensuing hypervent
40 e responses to electrical stimulation of the carotid sinus nerve (CSN) and steady-state relationships
42 r, Ba2+ (3 and 5 mM) significantly increased carotid sinus nerve (CSN) discharge over baseline firing
43 lotrimazole and miconazole), we measured the carotid sinus nerve (CSN) discharge using an in vitro pe
44 e have traced the central projections of the carotid sinus nerve (CSN) in the cat by utilizing the tr
46 a by increasing chemosensory activity in the carotid sinus nerve (CSN), causing hyperventilation and
47 t inputs, steady-state relationships between carotid sinus nerve (electrical) stimulation frequency a
48 e to stimuli that evoked robust increases in carotid sinus nerve activity in five out of five control
58 ese mechanisms are as follows: (1) increased carotid sinus nerve discharge rate to the respiratory ce
59 Baroreceptor activity was measured from the carotid sinus nerve during pressure ramps in isolated ca
61 that TRPC1/3/4/5/6 proteins localize to the carotid sinus nerve fibers, some of which were immunorea
67 tor function was examined by recording whole carotid sinus nerve responses to cessation of ventilatio
72 These studies support the concept that brief carotid sinus nerve stimulations cause a transient outwa
73 respiratory oscillator's phase responses to carotid sinus nerve stimulations in cats to the phase re
74 nerve, brief repeated carotid occlusions and carotid sinus nerve stimulations were performed to deter
75 block of baroreceptor afferent fibers in the carotid sinus nerve to examine the medullary projections
76 and transmit this sensory information in the carotid sinus nerve to the brain via neurons in the petr
77 y responses to electrical stimulation of the carotid sinus nerve were enhanced by CIH (p < 0.05).
78 ATP to activate chemoafferent fibres of the carotid sinus nerve which transmit this information to t
79 e in the number of unmyelinated axons in the carotid sinus nerve, compared with age-matched normoxic
83 bolished following interruption of vagal and carotid sinus nerves (from 0.301 +/- 0.012 to 0.311 +/-
84 nated by prior bilateral transections of the carotid sinus nerves or by prior inhibition of neurones
87 roreceptor innervation (aortic depressor amd carotid sinus nerves) was intact, rhythms correlated to
91 ated atherosclerotic plaque formation at the carotid sinus of Adamts13(-/-)/ApoE(-/-) mice compared w
94 agonist, unilateral pressure changes in the carotid sinus, or occlusion of the descending aorta in 1
95 agonist, unilateral pressure changes in the carotid sinus, or occlusion of the descending aorta in t
96 +/- 2.2 impulses s(-1)) whereas increases in carotid sinus pressure (CSP) induced significant decreas
100 on of baroreflex activity through electrical carotid sinus stimulation influences insulin sensitivity
102 nd mean arterial pressure (MAP) responses to carotid sinus stimulation were used to develop reflex fu
104 Two groups of 34 consecutive patients with carotid sinus syndrome as the sole cause of falls and sy
107 Assessment of the vasodepressor reflex in carotid sinus syndrome is influenced by the method of ex
110 he clinical characteristics of patients with carotid sinus syndrome who presented with falls with tho
112 nsitive nerve endings in the aortic arch and carotid sinus that play a critical role in acute regulat
117 d dogs, the splenic pedicle was tied and the carotid sinuses were vascularly isolated and perfused at
WebLSDに未収録の専門用語(用法)は "新規対訳" から投稿できます。