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1 ifestations associated with the X-linked CGD carrier state.
2 for persistent viruses to establish a normal carrier state.
3 actors were associated with the C. difficile carrier state.
4 l genes involved in the establishment of the carrier state.
5 lbladder colonization and maintenance of the carrier state.
6 on on cholesterol gallstones and the chronic carrier state.
7 on for treatment of children in the inactive carrier state.
8 onferred against malaria by the heterozygous carrier state.
9 lly exerts anti-viral pressure during a LCMV carrier state.
10 nfection, relapse, or the establishment of a carrier state.
11 rior to neuroinvasion and establishment of a carrier state.
12 lbladder colonization and maintenance of the carrier state.
13 g determinants of disease variability in the carrier state.
14 ccurs upon establishment of the asymptomatic carrier state.
15 B or the hepatitis B surface antigen (HBsAg) carrier state.
16 programs of EBV latency associated with the carrier state.
17 establishment and maintenance of the chronic carrier state.
18 ge of 10(-7)%, which frequently occur in the carrier state.
19 ifestyle, (systemic infection), to a dormant carrier state.
20 tions were significantly associated with the carrier state; 100% (9/9) of the infected patients were
21 ependent fatal meningitis or an asymptomatic carrier state amenable to immunotherapeutic clearance.
22 d aid in the understanding of the Salmonella carrier state, an important but underresearched area of
27 HBeAg)-positive chronic hepatitis], inactive carrier state, and reactivation (HBeAg-negative chronic
28 fections picked up at birth that established carrier states) are almost eliminated from the urban env
29 years), 4 eyes of 2 women with choroideremia carrier state (both in mid-50s), and 6 eyes of 6 control
30 less was established as the most common MRSA carrier state, broth enrichment or PCR may be critical f
31 nt recipients often leads to an asymptomatic carrier state characterized by a persistently elevated c
34 morbidity and even mortality, and a chronic carrier state exists, accounting for chronic hepatitis i
35 both male and female chickens may develop a carrier state following infection but that the increases
38 part of the human nasal microbiome, and this carrier state has often been associated with some type 2
40 conditions studied and that causes a chronic carrier state in BALB/c mice after peroral inoculation.
41 escribed here elucidates aspects of the FMDV carrier state in cattle which may facilitate identificat
44 cantly enhances our understanding of the EAV carrier state in stallions by unequivocally identifying
45 , death in young foals, establishment of the carrier state in stallions, and trade restrictions impos
47 athogens establish persistent infection or a carrier state in the human nasopharynx without overt dis
48 sive diagnostic test for the disease and the carrier state in this population and confirms biochemica
49 irm that elements of the vaccine establish a carrier state in vaccinated animals and present evidence
50 directly to the outer membrane can establish carrier states in Escherichia coli or Salmonella typhimu
52 The existence of a prolonged, asymptomatic carrier state is a political impediment for control and
55 nmental reservoir, the chronic, asymptomatic carrier state is thought to be a key feature of continue
56 ction risk in X-linked CGD carriers, and the carrier state itself is associated with autoimmunity.
58 consistent with the suggestion that the STGD carrier-state may predispose to the development of AMD.
62 ted only a modest influence on the S. aureus carrier state of middle-aged and elderly individuals.
67 nsistent with a natural establishment of the carrier state previously described only for ruminants.
69 mice provide a useful model of the DeltaGAG carrier state that can be used to probe genetic and envi
70 used to model the development of the chronic carrier state that develops in humans after infection wi
71 e gallbladder and persist in an asymptomatic carrier state that is frequently associated with the pre
74 variation affected progression from chronic carrier state to hepatocellular carcinoma (HCC), we anal
75 vents that lead to the secondary subclinical carrier state typical of most lentivirus infections.
76 PH oxidase p22phox CC genotype, the T-allele carrier state was associated with 2.1-fold higher odds f
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