ライフサイエンスコーパス: castanospermine

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1 serotypes of dengue virus were inhibited by castanospermine.

2 ion induced by the ER glucosidase inhibitor, castanospermine.

3 crase activity is abolished by the inhibitor castanospermine.

4 alpha-subunits expressed in the presence of castanospermine.

5 ies by evaluating the inhibitory activity of castanospermine against a panel of clinically important

6 Using castanospermine (an alpha-glucosidase inhibitor) and yea

7 ed that alpha-glucosidase inhibitors such as castanospermine and deoxynojirimycin inhibit dengue viru

8 glucosidase inhibitors, such as 6-O-butanoyl castanospermine and N-nonyl deoxynorjirimycin, resulted

9 However, both prolonged incubation with castanospermine and removal of N-glycosylation sites do

10 kedly decreased secretion of meprin, whereas castanospermine and swainsonine had little effect on sec

11 ication to the potent glycosidase inhibitors castanospermine and swainsonine.

12 nzymes with active site-directed inhibitors (castanospermine, conduritol B epoxide and deoxynojirimyc

13 An inhibitor of glucose trimming, castanospermine (CST), abolished binding to Cnx/Crt but

14 Indeed, one glucosidase inhibitor, castanospermine (CST), and two mannosidase inhibitors, k

15 l inhibition of ER glucosidase activity with castanospermine (CST), enhanced apo(a)-CNX/CRT interacti

16 was inhibited using a glucosidase inhibitor, castanospermine (CST), the rate of disulfide formation a

17 However, treatment with castanospermine does not appear to alter subunit folding

18 co-treatment with proteasome inhibitors and castanospermine enhances the accumulation of polyubiquit

19 Addition of the glucosidase inhibitor, castanospermine, failed to significantly influence the a

20 Correspondingly, castanospermine had no adverse or protective effect on W

21 Overall, our data suggest that castanospermine has a strong antiviral effect on dengue

22 d alpha-subunits, and generally reverses the castanospermine induced loss of alpha-subunit protein.

23 Castanospermine inhibited dengue virus infection at the

24 Swainsonine and castanospermine, inhibitors of glycoprotein processing t

25 s were blocked by the glucosidase inhibitors castanospermine or N-butyldeoxynojirimycin.

26 rom a pivotal sp(2)-iminosugar-type reducing castanospermine precursor is reported.

27 Importantly, castanospermine prevented mortality in a mouse model of

28 The ER glucosidase inhibitor, castanospermine, prevented apo(a) secretion, but did not

29 tion between endogenous calnexin and AChR by castanospermine resulted in decreased AChR subunit foldi

30 In contrast, cells treated with castanospermine show a moderate increase in GRP78 mRNA (

31 lycan-calnexin interactions are prevented by castanospermine, showing that N-glycan-mediated calnexin

32 calnexin and calreticulin by the addition of castanospermine significantly increased fragment secreti

33 revention of glycan-calnexin interactions by castanospermine slightly increases ER retention of beta(

34 We used the glucosidase inhibitor castanospermine to block oligosaccharide processing, and

35 Moreover, castanospermine treatment failed to affect the infectivi

36 Castanospermine treatment reduces the association of cal

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