ライフサイエンスコーパス: catabolite

1 accumulation of immunosuppressive tryptophan catabolites.

2 rther narrow down the structure of the found catabolites.

3 ient deprivation due to decreasing glutamine catabolites.

4 ecific biomarkers of interest were oxidative catabolites.

5 lterations in hexoses, lysolipids and purine catabolites.

6 When exposed to beta-lactams, however, PG catabolites (1,6-anhydroMurNAc-peptides) accumulate in t

7 at catalyzes oxidation of IAA to its primary catabolite 2-oxindole-3-acetic acid (oxIAA) remains unch

8 cation of AP at the N-terminus and the minor catabolite [29-270] by truncations of either side of the

9 ucuronide and the colonic microbiota-derived catabolite 3-(3'-hydroxy-4'-methoxyphenyl)hydracrylic ac

10 The major catabolite [3-285] was formed by truncation of AP at the

11 f drug-related material, whereas that of the catabolites [3-285] and [29-270] accounted for 66% and 9

12 Urinary excretion of MTX and its catabolite, 7-OH-MTX, was measured in 2 24-hour urine sp

13 In vitro, ACD2 can reduce red chlorophyll catabolite, a chlorophyll derivative.

14 K, but no such relationship was observed for catabolite-activated genes, suggesting that large number

15 imary mediators of catabolite repression and catabolite activation in Bacillus subtilis.

16 The catabolite activator protein (CAP) bends DNA in the CAP-

17 An E. coli catabolite activator protein (CAP) has been converted in

18 The catabolite activator protein (CAP) of Escherichia coli i

19 he binding of DNA to several variants of the catabolite activator protein (CAP) that differentially p

20 haracterize cyclic AMP (cAMP) binding to the catabolite activator protein (CAP), a transcriptional ac

21 tion complex comprising the Escherichia coli catabolite activator protein (CAP), RNA polymerase holoe

22 ranscription by preventing cyclic AMP (cAMP)-catabolite activator protein (CAP)-dependent activation.

23 we apply it to a challenging test case: the catabolite activator protein (CAP).

24 n TTHB099 (TAP) [homolog of Escherichia coli catabolite activator protein (CAP)], T. thermophilus RNA

25 e cAMP-mediated allosteric transition in the catabolite activator protein (CAP; also known as the cAM

26 ynechocystis sp. adenylyl cyclase (Cya1) and catabolite activator protein (SYCRP1) mutants to differe

27 terial transcription factors, including four catabolite activator protein homologues.

28 The catabolite activator protein is a dimer that consists of

29 ion curves for cells with and without active catabolite activator protein.

30 ulations of 5 ns on protein-DNA complexes of catabolite-activator protein (CAP), lambda-repressor, an

31 nosine triphosphate (ATP) and its first five catabolites: adenosine diphosphate (ADP), adenosine mono

32 Moreover, the pipecolate catabolite alpha-aminoadipate decreased 30-fold in RNA i

33 ements.We hypothesized that alpha-tocopherol catabolites alpha-carboxyethyl hydroxychromanol (alpha-C

34 work identifies C99 as the earliest betaAPP catabolite and main contributor to the intracellular bet

35 t niche-dependent manner, is known as carbon catabolite and nitrogen catabolite repression (CCR, NCR)

36 linker has multiple paths to produce active catabolites and that antibody and intracellular targets

37 h the accumulation of significantly more ABA catabolites and the complete restoration of normal wild-

38 Trp catabolites and their derivatives offer a new strategy f

39 e, decreased isovaleryl-carnitine (a leucine catabolite), and decreased tricarboxylic acid (TCA) cycl

40 When colon-derived phenolic catabolites are included with flavanone glucuronide and

41 findings demonstrate that kynurenine pathway catabolites are involved in the generation of the more s

42 poorly absorbed in the small intestine, but catabolites are very efficiently absorbed after microbia

43 ure elucidation of tetrapyrrolic chlorophyll catabolites, as well as by complementary biochemical and

44 rabidopsis and specifically demethylates Chl catabolites at the level of FCCs in the cytosol.

45 found that extracellular NAD(+), but not its catabolites, caused cell death (half-maximal effective c

46 for direct drug-to-antibody ratio (DAR) and catabolite characterization of antibody-drug conjugates

47 Two PC catabolites, choline and glycine betaine (GB), were suff

48 ilms, indicating active uptake, and arginine catabolites citrulline, ornithine, and putrescine were d

49 C46A3 silencing led to relative increases in catabolite concentrations in the lysosome.

50 amyloid beta-protein precursor (APP) and its catabolites contribute to the impaired synaptic plastici

51 ies expand our appreciation of CcpA-mediated catabolite control and provide insight into potential in

52 ofilm formation, as well as the fsr-mediated catabolite control of biofilm, is mediated via these pro

53 and the contributions of the CcpA and LacD.1 catabolite control pathways to the regulation of this re

54 in Bacillus subtilis for recognition of the catabolite control protein (CcpA) and consequential repr

55 n Streptococcus mutans can be independent of catabolite control protein A (CcpA) and requires specifi

56 a mutation in the transcriptional regulator catabolite control protein A (CcpA) demonstrated signifi

57 In Gram-positive bacteria, the catabolite control protein A (CcpA) functions as the mas

58 spx gene expression, we assessed the role of catabolite control protein A (CcpA) in spx expression co

59 We characterized the role of catabolite control protein A (ccpA) in the physiology an

60 The LacI/GalR family regulator catabolite control protein A (CcpA) is a global regulato

61 Catabolite control protein A (CcpA) is a highly conserve

62 ed us to analyze the role in pathogenesis of catabolite control protein A (CcpA), a GAS ortholog of a

63 Here we report that the catabolite control protein A (CcpA), a highly conserved

64 elements (cre) important for binding by the catabolite control protein A (CcpA), a mediator of CCR i

65 e regulation (CCR) is mediated by the carbon catabolite control protein A (CcpA), a member of the Lac

66 ment (cre) and affected the binding of CcpA (catabolite control protein A), a key regulator of many c

67 ve binding of the maltose repressor MalR and catabolite control protein A.

68 In Bacillus subtilis, the catabolite control protein C (CcpC) plays a critical rol

69 ory networks such as RALPs, Rgg/RopB and the catabolite control protein CcpA.

70 Previously, we described a novel protein, catabolite control protein E (CcpE) that functions as a

71 aureus, we identified a LysR-type regulator, catabolite control protein E (CcpE), with homology to th

72 n A (CcpA) is the master regulator of carbon catabolite control, which ensures optimal energy usage u

73 all, our data indicate that a cellular prion catabolite could interfere with Abeta-associated toxicit

74 roup of distinct dioxobilin-type chlorophyll catabolites (DCCs) as the major breakdown products in wi

75 NCCs are formyloxobilin-type catabolites derived from chlorophyll by oxygenolytic ope

76 nsumption should use in vivo metabolites and catabolites detected in this investigation at physiologi

77 Acetaldehyde, an alcohol catabolite detoxified by ALDH2, precipitates similar eff

78 no changes in epinephrine (Epi) or monoamine catabolites (DOPAC, 5-HIAA) at any ammonia concentration

79 bon scavenging from alpha-hydroxycarboxylate catabolites during the biochemical transition accompanyi

80 Concomitant formation of catabolites (e.g. 4-hydroxyphenylacetic acid) occurred w

81 was to identify and quantify metabolites and catabolites excreted in urine 0-24 h after the acute ing

82 sosomal ion homeostasis, membrane potential, catabolite export, membrane trafficking, and nutrient se

83 e of ABA homeostasis, as it is the major ABA catabolite exported from the cytosol.

84 transported out of the lysosome via specific catabolite exporters or via vesicular membrane trafficki

85 generated from their fluorescent chlorophyll catabolite (FCC) precursors by a nonenzymatic isomerizat

86 nverted to different fluorescent chlorophyll catabolites (FCCs) and nonfluorescent chlorophyll catabo

87 se from intermediary fluorescent chlorophyll catabolites (FCCs) by an acid-catalyzed isomerization in

88 entify bacteria expressing genes relevant to catabolite flow and to locate these genes within their e

89 timal utilization of androgen precursors and catabolites for DHT synthesis.

90 nt molecule while simultaneously identifying catabolites for recombinant fusion proteins.

91 3-acetic acid (oxIAA) is a major primary IAA catabolite formed in Arabidopsis thaliana root tissues.

92 The main focus was on catabolites formed by proteolysis of the fusion protein

93 th tmax of anthocyanins and other polyphenol catabolites from 1.0h to 6.33h in plasma and urine.

94 as a direct transporter of maytansine-based catabolites from the lysosome to the cytoplasm, promptin

95 In addition, these lipid species and their catabolites function as secondary signalling molecules i

96 ion start and is thought to promote the CAP (catabolite gene activation protein)-directed transcripti

97 The binding of cAMP to the Escherichia coli catabolite gene activator protein (CAP) produces a confo

98 yclic AMP receptor protein (CRP, also called catabolite gene activator protein or CAP) plays a key ro

99 tive cell killing with a cysteine-VC(R)-MMAE catabolite generated by lysosomal catabolism.

100 n of soluble heat-labile toxin is subject to catabolite (glucose) activation, and three binding sites

101 sis in an aldol condensation of the unstable catabolites glyceraldehyde 3-phosphate and dihydroxyacet

102 either rHuPH20 nor its directly generated HA catabolites have inflammatory properties in the air pouc

103 AMP is the predominant purine catabolite in erythrocytes deprived of glucose or expose

104 mino acid pipecolic acid (Pip), a common Lys catabolite in plants and animals, as a critical regulato

105 Consistent with a role for biotin and its catabolites in modulating these cell signals, greater th

106 lism, mTORC2 responds to declining glutamine catabolites in order to restore metabolic homeostasis.

107 The high levels of ABA and its catabolites in the senescing breeding line under long-te

108 NK cells with indoleamine 2,3-dioxygenase 1 catabolites in vitro ablated IL-17 production in a dose-

109 ivity but is rapidly metabolized to inactive catabolites in vivo.

110 is supported by biochemical measurements of catabolites in wild-type and mutant animals.

111 -1,6-bisphosphatase (FBPase) is subjected to catabolite inactivation and degradation when glucose-sta

112 ich results in aberrant lysosomal storage of catabolites, including the subunit c of mitochondrial AT

113 yanins were bioavailable, microbial phenolic catabolites increased approximately 10-fold more than an

114 both intact drug concentration and important catabolite information for this recombinant fusion prote

115 responsible for delivering noncleavable ADC catabolites into the cytoplasm.

116 Addition of the tryptophan catabolite kynurenine to DC cultures in which IDO activi

117 er, in the immune system, tryptophan and its catabolites, kynurenine and 3-hydroxyanthranilic acid (3

118 and a significant rise in the content of ATP-catabolites, malondialdehyde and ADP-ribose.

119 Some of these colon-derived catabolites may have a role in vivo in the potential pro

120 correlates with elevated levels of the toxic catabolite methylglyoxal.

121 immunomodulation by accumulating tryptophan catabolites, most notably kynurenine, appears to play an

122 representative of the energy state (ATP, ATP-catabolites), N-acetylaspartate (NAA), antioxidant defen

123 Nonfluorescent chlorophyll catabolites (NCCs) were described as products of chlorop

124 is broken down to nonfluorescent chlorophyll catabolites (NCCs).

125 olites (FCCs) and nonfluorescent chlorophyll catabolites (NCCs).

126 droxyvitamin D3 ((24R),25(OH)2D3) is a major catabolite of 25-hydroxyvitamin D metabolism and is an i

127 ncentrations of homovanillic acid (the major catabolite of dopamine) and the purine compound xanthine

128 ycolylglucosamine (GlcNGc) was shown to be a catabolite of NeuNGc.

129 Here, we show that a catabolite of the plant hormone abscisic acid (ABA), nam

130 sed production of kynurenine (Kyn) as a main catabolite of tryptophan (Trp) degradation is involved i

131 However, urinary excretion of 3 colonic catabolites of bacterial origin, most notably, 3-(3'-hyd

132 involved in the formation of dioxobilin-type catabolites of chlorophyll in Arabidopsis.

133 ne containing insulin and another containing catabolites of insulin.

134 Catabolites of Trp suppressed proliferation of myelin-sp

135 affect clearance dynamics and that access to catabolites only matters at low H2O2 concentrations.

136 hyrin-related molecules like red chlorophyll catabolite or exogenous protoporphyrin IX.

137 The CouR dimer bound two molecules of the catabolite p-coumaroyl-CoA (Kd = 11 +/- 1 muM).

138 The catabolite PA was positively associated with neopterin a

139 dent on sufficient amounts of glutaminolysis catabolites particularly alpha-ketoglutarate, which are

140 pheophorbide a to a primary fluorescent chl catabolite (pFCC) and it is catalyzed by two enzymes: ph

141 ane diterpenoid dehydroabietinal, the lysine catabolite pipecolic acid, a glycerol-3-phosphate-depend

142 A number of the catabolites produced along this pathway show neurotoxic

143 inary flavanone metabolites and ring fission catabolites produced by the action of the colonic microb

144 s to falling levels of glucose and glutamine catabolites, promoting glutaminolysis and preserving the

145 acids (BCAAs) (Leu, Ile, and Val) and their catabolites, propionylcarnitine and butyrylcarnitine, we

146 In Gram-positive bacteria, carbon catabolite protein A (CcpA) is the master regulator of c

147 ect regulator of phoPR transcription, carbon catabolite protein A, CcpA.

148 transcription by glucose required the carbon catabolite protein CcpA via an indirect mechanism.

149 : pheophorbide a oxygenase (PaO) and red chl catabolite reductase (RCCR).

150 In Gram-positive bacteria, carbon catabolite regulation (CCR) is mediated by the carbon ca

151 As a key component of carbon catabolite regulation, CcpA has been previously reported

152 apping pathways for global control of carbon catabolite regulation.

153 n of respiration genes, and CcpA, the carbon catabolite regulator protein.

154 rt-term anaerobiosis (2 generations) in both catabolite-repressed (glucose) and derepressed (galactos

155 vation of Gln3 and transcription of nitrogen catabolite-repressed (NCR) genes whose products function

156 These enzymes are universally catabolite-repressed and are further regulated by a rich

157 In addition, we observe that nitrogen catabolite-repressed genes are upregulated by Nab3 deple

158 lation was noted between the dependencies of catabolite-repressible gene expression on CcpA and HprK,

159 Carbon catabolite repression (CCR) allows bacteria to alter met

160 s mannose and glucose, is involved in carbon catabolite repression (CCR) and regulates the expression

161 olite control protein (CcpA) mediates carbon catabolite repression (CCR) by controlling expression of

162 riptional regulator that accounts for carbon catabolite repression (CCR) control of the anaerobic cat

163 CcpA is the global mediator of carbon catabolite repression (CCR) in gram-positive bacteria, a

164 , two regulatory genes that carry out carbon catabolite repression (CCR) in staphylococci and other G

165 Carbohydrate catabolite repression (CCR) in Streptococcus mutans can

166 Carbon catabolite repression (CCR) is a regulatory phenomenon i

167 Carbon catabolite repression (CCR) is a regulatory phenomenon o

168 In many bacteria, carbon catabolite repression (CCR) is central to such regulatio

169 Carbon catabolite repression (CCR) is one of the most fundament

170 ilm formation were under some form of carbon catabolite repression (CCR), a regulatory network in whi

171 so includes inhibited glycolysis, and carbon catabolite repression (CCR)-mediated carbohydrate-depend

172 ated that CcpA plays a direct role in carbon catabolite repression (CCR).

173 ntegral to pneumococcus's strategy of carbon catabolite repression (CCR).

174 , is known as carbon catabolite and nitrogen catabolite repression (CCR, NCR), and has been shown to

175 tor (arcA and etrA [fnr homolog]) and carbon catabolite repression (crp and cya) proteins affect arse

176 circuit responsible for regulating nitrogen catabolite repression (NCR) in yeast.

177 en use is mediated in large part by nitrogen catabolite repression (NCR), which results in the repres

178 of Gln3 and Gat1, the activators of nitrogen catabolite repression (NCR)-sensitive genes whose produc

179 ities is transcriptional control of nitrogen catabolite repression (NCR)-sensitive genes.

180 Gln3 intracellular localization and nitrogen catabolite repression (NCR)-sensitive transcription in S

181 Nitrogen catabolite repression (NCR)-sensitive transcription is a

182 Gln3, the major activator of nitrogen catabolite repression (NCR)-sensitive transcription, is

183 ogical response has been designated nitrogen catabolite repression (NCR).

184 This regulation is designated nitrogen catabolite repression (NCR).

185 carbon sources is termed succinate-mediated catabolite repression (SMCR).

186 s leads to an increase in succinate-mediated catabolite repression (SMCR).

187 implications for mechanisms of CRP-dependent catabolite repression acting in conjunction with a membe

188 nase/phosphorylase, are primary mediators of catabolite repression and catabolite activation in Bacil

189 ritical role for the PTS in CcpA-independent catabolite repression and induction of cel gene expressi

190 ssion of fruA is under the control of carbon catabolite repression and is induced by growth in fructa

191 itoring and use that information to regulate catabolite repression and related responses.

192 in stable environments, with more stringent catabolite repression and slower transcriptional reprogr

193 ted that CcpA was essential for carbohydrate catabolite repression and that Flp was required for opti

194 metabolic phenotype depends on the level of catabolite repression and the metabolic state-dependent

195 ) operon of Escherichia coli is regulated by catabolite repression and tryptophan-induced transcripti

196 cpA), a highly conserved regulator of carbon catabolite repression and virulence in a number of gram-

197 is protein is a major factor responsible for catabolite repression at the nrf promoter, and Fis can o

198 In order to identify the mechanism of catabolite repression by glucose, a mutation was introdu

199 In addition to catabolite repression by glucose, l-leucine acts by inhi

200 suggests that Pyk may participate in glucose catabolite repression by serving among all of the factor

201 nd G6PDH activity are known to be subject to catabolite repression by succinate.

202 ate that propionate metabolism is subject to catabolite repression by the global transcriptional regu

203 Deletion of hprK in S. meliloti enhanced catabolite repression caused by succinate, as did an S53

204 3 in-frame deletion mutants show a relief of catabolite repression compared to the wild type.

205 is was apparently not due to a defect in the catabolite repression control (Crc) protein.

206 slocator (ArsB), superoxide dismutase (SOD), catabolite repression control protein (Crc), or glutathi

207 a xylose catabolic activation independent of catabolite repression control.

208 The cyclic AMP (cAMP)-dependent catabolite repression effect in Escherichia coli is amon

209 tein IIA(Glc) plays a key regulatory role in catabolite repression in addition to its role in the vec

210 ant physiological roles, ranging from carbon catabolite repression in bacteria to mediating the actio

211 se-pairing RNA Spot 42 plays a broad role in catabolite repression in Escherichia coli by directly re

212 ENR to the regulatory network behind carbon catabolite repression in Escherichia coli is presented.

213 gether with the Hfq protein, participates in catabolite repression in pseudomonads, helping to coordi

214 ne-22 residue, and that HPr-His22-P enhances catabolite repression in the presence of succinate.

215 n enteric bacteria, the key player of carbon catabolite repression is a component of the glucose-spec

216 In Sinorhizobium meliloti, catabolite repression is influenced by a noncanonical ni

217 Here, we show that this catabolite repression is relieved by mutations that weak

218 the parent H26 and glpK mutant strains, with catabolite repression more pronounced in the glycerol ki

219 Catabolite repression of galactose by glucose is one of

220 Additionally, Crp mediates strong indirect catabolite repression of many cytoplasmic stress respons

221 d growth phenotype was reflected in a strong catabolite repression of pauA promoter activation by CAD

222 and that this mechanism in part accounts for catabolite repression of sigma(L)-directed levD operon e

223 enes; (iii) CcpA plays little direct role in catabolite repression of the cel regulon, but loss of sp

224 f highly expressed genes that are subject to catabolite repression or activation mediated by the cycl

225 on this enzymatic activity or the canonical catabolite repression pathway, but likely does require s

226 substitution into HPr alleviated the strong catabolite repression phenotypes of strains carrying Del

227 se or casamino acids, suggesting that carbon catabolite repression plays a role in regulating xynA.

228 al abscess formation, indicating that carbon catabolite repression presents an important pathogenesis

229 We linked this to indirect regulation of the catabolite repression protein Crc via the non-coding RNA

230 in carbon source can favor different carbon catabolite repression strategies.

231 concluded that the components of the carbon catabolite repression system are essential to regulating

232 utations also increase glycerol-induced auto-catabolite repression that reduces glpK transcription in

233 PTS) of gram-positive bacteria and regulates catabolite repression through phosphorylation/dephosphor

234 nd uptake in Escherichia coli are subject to catabolite repression through the cyclic AMP (cAMP)-CRP

235 Sucrose-dependent catabolite repression was also evident in strains contai

236 oreover, consistent with a classical role in catabolite repression, a cAMP-CRP-dependent reporter sho

237 atabolism, chemotaxis, glycogen utilization, catabolite repression, and inducer exclusion.

238 ulating central carbon metabolism and carbon catabolite repression, and is a frequent target of metab

239 ng mechanisms of signal transduction, carbon catabolite repression, and quorum-sensing.

240 lted in partial relief of succinate-mediated catabolite repression, extreme sensitivity to cobalt lim

241 nes are substrate inducible and sensitive to catabolite repression, mediated through ArcR and CcpA, r

242 by negative feedback on glpK expression via catabolite repression, possibly to prevent methylglyoxal

243 as luxS and ompX and provide a link between catabolite repression, quorum sensing, and nitrogen assi

244 The other mutants display less stringent catabolite repression, resulting in leaky expression of

245 The nanATEK operon is controlled by catabolite repression, suggesting that diminished expres

246 The archaeon Sulfolobus solfataricus uses a catabolite repression-like system to control production

247 -responsive) and TorC1-independent (nitrogen catabolite repression-sensitive and methionine sulfoximi

248 eving Gln3 nuclear localization and nitrogen catabolite repression-sensitive transcription in respons

249 Gln3-Myc13 nuclear accumulation and nitrogen catabolite repression-sensitive transcription, generate

250 s regulatory phenomenon is defined as carbon catabolite repression.

251 source, glucose, in a process called carbon catabolite repression.

252 g very weakly transcribed genes under strong catabolite repression.

253 others through a regulatory mechanism termed catabolite repression.

254 efficient microbial conversion due to carbon catabolite repression.

255 h gene, the DMML encoding gene is subject to catabolite repression.

256 ns with a hpr(H22A) allele exhibited relaxed catabolite repression.

257 rating that HPr-His22-P is needed for strong catabolite repression.

258 trol region cloned in E. coli was subject to catabolite repression.

259 scriptome appeared to be regulated by carbon catabolite repression.

260 r region showed that both CREs contribute to catabolite repression.

261 activity were shown to be refractory to such catabolite repression.

262 grown in media containing sugars that cause catabolite repression.

263 e master transcriptional regulator of carbon catabolite repression/regulation (CCR).

264 l a key physiological role of cAMP-dependent catabolite repression: to ensure that proteomic resource

265 wth on non-glucose substrates as part of the catabolite-repression response.

266 Carbon catabolite repressor (CCR)4 and CCR4-associated factor (

267 ex, containing the two key components carbon catabolite repressor 4 (CCR4) and CCR4-associated factor

268 The carbon catabolite repressor 4-CCR4 associated factor1 (CCR4-CAF

269 thermore, the inclusion of a deletion of the catabolite repressor gene, cre-1, in the triple beta-glu

270 he eight-subunit deadenylase complex "carbon catabolite repressor protein 4 (CCR4)-negative on TATA-l

271 r that is homologous to the Escherichia coli catabolite repressor protein, is thought to be the major

272 The role of the carbon catabolite repressor transcription factor homologue Bbcr

273 Mutation of two putative catabolite response elements (CREs) within the arc promo

274 r and binds to more than 50 operators called catabolite response elements (cres).

275 epression (CCR) by controlling expression of catabolite responsive (CR) genes or operons through inte

276 al xynA1 gene show significant similarity to catabolite responsive element (cre) defined in Bacillus

277 R) genes or operons through interaction with catabolite responsive elements (cres) located within or

278 The mutation was in a catabolite-responsive element (cre) and affected the bin

279 A catabolite-responsive element (CRE), a binding site for

280 s consensus identified a number of potential catabolite-responsive elements (cre) important for bindi

281 a bioinformatic search for additional carbon catabolite-responsive regulators in S. aureus, we identi

282 nd reveal critical roles for amino acid- and catabolite-sensing pathways in controlling gene expressi

283 The catabolites showed lowered antioxidant activity and cell

284 This result could be obtained without catabolite specific ELISAs or quantitative LC-MS assays.

285 Hydrogen sulfide is an essential catabolite that intervenes in the pathophysiology of sev

286 sts unwanted cellular components to generate catabolites that are required for housekeeping biosynthe

287 s indicate that syn-DHCA and syn-DHFA, colon catabolites that could be present in systemic circulatio

288 ryptophan to kynurenine and other downstream catabolites that inhibit T-cell proliferation and interl

289 ric acid (GABA), in the GABA shunt generates catabolites that may enter the tricarboxylic acid cycle,

290 the sum of TN-ApoA1, along with its two main catabolites, the individual PK profiles of all three com

291 Pheophorbide a (PhA), a chlorophyll catabolite, was shown to be an ABCG2 substrate based on

292 These catabolites were 3-(3'-methoxy-4'-hydroxyphenyl)propioni

293 The GC-MS analysis revealed that 8 urinary catabolites were also excreted in significantly higher q

294 Two pharmacologically active catabolites were identified with conserved fusion region

295 The drug and its catabolites were isolated from rabbit plasma by immunoca

296 The captured drug and catabolites were released from the streptavidin-coated m

297 gradation products, are abundant chlorophyll catabolites, which occur in fall leaves and in ripe frui

298 t, was also observed with OJ-derived colonic catabolites, which, after supplementation in the trained

299 Corresponding alterations in tryptophan catabolites with immunomodulatory properties in serum of

300 inary flavanone metabolites and (poly)phenol catabolites with the use of high-pressure liquid chromat