ライフサイエンスコーパス: catabolite repression

1 ntermediary metabolism in Escherichia coli ("catabolite repression").

2 lated by agmatine induction and carbohydrate catabolite repression.

3 tabolism, are subject to direct Crp-mediated catabolite repression.

4 or limiting, a phenomenon known as nitrogen catabolite repression.

5 Synthesis of this activity is subject to catabolite repression.

6 r exclusion mechanisms of succinate-mediated catabolite repression.

7 least some competence genes are regulated by catabolite repression.

8 is is a major transcription factor mediating catabolite repression.

9 The tdc operon is subject to CRP-controlled catabolite repression.

10 opic developmental processes, but not carbon catabolite repression.

11 on of an array of functions including carbon catabolite repression.

12 tified in the promoter region, indicative of catabolite repression.

13 or citrate synthase and is subject to carbon catabolite repression.

14 enase, which is regulated in both species by catabolite repression.

15 dia with much agitation, CcpA alone mediated catabolite repression.

16 ional cofactor controlling the phenomenon of catabolite repression.

17 cally induced by gluconate and repressed via catabolite repression.

18 oth of which contribute to the phenomenon of catabolite repression.

19 lis cells to screen for mutants resistant to catabolite repression.

20 hat the toxin genes are subject to a form of catabolite repression.

21 insensitive to transcriptional regulation by catabolite repression.

22 on, CcpB and CcpA both proved to function in catabolite repression.

23 ariations in cell growth rate rather than to catabolite repression.

24 ption mutant is highly sensitive to nitrogen catabolite repression.

25 es in appropriate locations to exert glucose catabolite repression.

26 phosphorylation at His-15 can prevent carbon catabolite repression.

27 lated by the SOS response, anaerobiosis, and catabolite repression.

28 gluconate and is subject to fourfold glucose catabolite repression.

29 table alanyl residue are resistant to carbon catabolite repression.

30 s operon is also believed to be regulated by catabolite repression.

31 s regulatory phenomenon is defined as carbon catabolite repression.

32 efficient microbial conversion due to carbon catabolite repression.

33 source, glucose, in a process called carbon catabolite repression.

34 g very weakly transcribed genes under strong catabolite repression.

35 others through a regulatory mechanism termed catabolite repression.

36 h gene, the DMML encoding gene is subject to catabolite repression.

37 ns with a hpr(H22A) allele exhibited relaxed catabolite repression.

38 rating that HPr-His22-P is needed for strong catabolite repression.

39 trol region cloned in E. coli was subject to catabolite repression.

40 scriptome appeared to be regulated by carbon catabolite repression.

41 r region showed that both CREs contribute to catabolite repression.

42 activity were shown to be refractory to such catabolite repression.

43 grown in media containing sugars that cause catabolite repression.

44 oreover, consistent with a classical role in catabolite repression, a cAMP-CRP-dependent reporter sho

45 CcpA (catabolite control protein A)-mediated catabolite repression, a global regulatory mechanism in

46 implications for mechanisms of CRP-dependent catabolite repression acting in conjunction with a membe

47 es), chemotaxis (methyl-accepting proteins), catabolite repression (adenylate cyclases), and modulati

48 he chromosome was barely sensitive to carbon catabolite repression, although the H15A mutant HPr can

49 These results suggest that catabolite repression and anaerobic repression of citZ a

50 e in gntT expression which is independent of catabolite repression and binding of GntR to the operato

51 ) operon of Escherichia coli is regulated by catabolite repression and by tryptophan-induced inhibiti

52 ) operon of Escherichia coli is regulated by catabolite repression and by tryptophan-induced transcri

53 ) operon of Escherichia coli is regulated by catabolite repression and by tryptophan-induced transcri

54 iration, in addition to their known roles in catabolite repression and carbon source utilization in o

55 nase/phosphorylase, are primary mediators of catabolite repression and catabolite activation in Bacil

56 ) is a global regulatory protein involved in catabolite repression and glucose activation in Gram-pos

57 ritical role for the PTS in CcpA-independent catabolite repression and induction of cel gene expressi

58 ssion of fruA is under the control of carbon catabolite repression and is induced by growth in fructa

59 itoring and use that information to regulate catabolite repression and related responses.

60 in stable environments, with more stringent catabolite repression and slower transcriptional reprogr

61 t that His-15 of HPr is important for carbon catabolite repression and that either mutation or phosph

62 ted that CcpA was essential for carbohydrate catabolite repression and that Flp was required for opti

63 s finding suggests that ure1 is regulated by catabolite repression and that limiting glucose in urine

64 moter was previously shown to be a target of catabolite repression and the binding site for a putativ

65 metabolic phenotype depends on the level of catabolite repression and the metabolic state-dependent

66 ) operon in Escherichia coli is regulated by catabolite repression and tryptophan-induced transcripti

67 e operon of Escherichia coli is regulated by catabolite repression and tryptophan-induced transcripti

68 ) operon of Escherichia coli is regulated by catabolite repression and tryptophan-induced transcripti

69 ) operon of Escherichia coli is regulated by catabolite repression and tryptophan-induced transcripti

70 ) operon of Escherichia coli is regulated by catabolite repression and tryptophan-induced transcripti

71 cpA), a highly conserved regulator of carbon catabolite repression and virulence in a number of gram-

72 atabolism, chemotaxis, glycogen utilization, catabolite repression, and inducer exclusion.

73 dependent of the SOS response, anaerobiosis, catabolite repression, and integration host factor as we

74 ulating central carbon metabolism and carbon catabolite repression, and is a frequent target of metab

75 ng mechanisms of signal transduction, carbon catabolite repression, and quorum-sensing.

76 ed in Bacillus subtilis global carbon source catabolite repression, and the ptsH1 mutation in the gen

77 d thus Rho-dependent termination rather than catabolite repression appears to be the event influenced

78 role in abpA expression, other mechanisms of catabolite repression are present.

79 Genes subject to CcpA-independent catabolite repression are primarily concerned with sporu

80 tion of HPr at Ser-46 is required for carbon catabolite repression as ptsH1 mutants in which Ser-46 o

81 is protein is a major factor responsible for catabolite repression at the nrf promoter, and Fis can o

82 In order to identify the mechanism of catabolite repression by glucose, a mutation was introdu

83 In addition to catabolite repression by glucose, l-leucine acts by inhi

84 s of gluconate (gnt) and xylose (xyl) operon catabolite repression by glucose, mannitol, and sucrose.

85 er finding that tdc expression is subject to catabolite repression by intermediary metabolites, stron

86 suggests that Pyk may participate in glucose catabolite repression by serving among all of the factor

87 nd G6PDH activity are known to be subject to catabolite repression by succinate.

88 ate that propionate metabolism is subject to catabolite repression by the global transcriptional regu

89 Deletion of hprK in S. meliloti enhanced catabolite repression caused by succinate, as did an S53

90 Carbon catabolite repression (CCR) allows bacteria to alter met

91 s mannose and glucose, is involved in carbon catabolite repression (CCR) and regulates the expression

92 olite control protein (CcpA) mediates carbon catabolite repression (CCR) by controlling expression of

93 REs), which have been shown to govern carbon catabolite repression (CCR) by functioning as negative c

94 riptional regulator that accounts for carbon catabolite repression (CCR) control of the anaerobic cat

95 CcpA is the global mediator of carbon catabolite repression (CCR) in gram-positive bacteria, a

96 , two regulatory genes that carry out carbon catabolite repression (CCR) in staphylococci and other G

97 Carbohydrate catabolite repression (CCR) in Streptococcus mutans can

98 Carbon catabolite repression (CCR) is a regulatory phenomenon i

99 Carbon catabolite repression (CCR) is a regulatory phenomenon o

100 In many bacteria, carbon catabolite repression (CCR) is central to such regulatio

101 Carbon catabolite repression (CCR) is one of the most fundament

102 In Bacillus subtilis, CcpA-dependent carbon catabolite repression (CCR) mediated at several cis-acti

103 In Bacillus subtilis, carbon catabolite repression (CCR) of many genes is mediated at

104 btilis mutant that partially relieves carbon catabolite repression (CCR) of the hut operon was isolat

105 ilm formation were under some form of carbon catabolite repression (CCR), a regulatory network in whi

106 so includes inhibited glycolysis, and carbon catabolite repression (CCR)-mediated carbohydrate-depend

107 ntegral to pneumococcus's strategy of carbon catabolite repression (CCR).

108 ated that CcpA plays a direct role in carbon catabolite repression (CCR).

109 , is known as carbon catabolite and nitrogen catabolite repression (CCR, NCR), and has been shown to

110 3 in-frame deletion mutants show a relief of catabolite repression compared to the wild type.

111 nce participate in the formation of the CcpA catabolite repression complex at cre sites.

112 ossible that induction of plcH is subject to catabolite repression control (CRC) by tricarboxylic cyc

113 We show that the catabolite repression control (Crc) protein, which plays

114 is was apparently not due to a defect in the catabolite repression control (Crc) protein.

115 In this study, the role of Crc in catabolite repression control has been studied in Pseudo

116 The gene (crc) responsible for catabolite repression control in Pseudomonas aeruginosa

117 The effect of growth in 2xYT medium on catabolite repression control in Pseudomonas putida has

118 This suggests that the mechanism of catabolite repression control in rich media by Crc invol

119 slocator (ArsB), superoxide dismutase (SOD), catabolite repression control protein (Crc), or glutathi

120 Crc (catabolite repression control protein) was shown to be r

121 Crc (catabolite repression control) protein of Pseudomonas ae

122 a xylose catabolic activation independent of catabolite repression control.

123 For succinate and lactose, the loss of catabolite repression could be attributed to the constit

124 Consistent with catabolite repression (CR), a DNA sequence with high hom

125 tor (arcA and etrA [fnr homolog]) and carbon catabolite repression (crp and cya) proteins affect arse

126 ative intensities of CcpA- and CcpB-mediated catabolite repression depended on growth conditions.

127 sing conditions, demonstrating that nitrogen catabolite repression does not influence dimorphism.

128 The cyclic AMP (cAMP)-dependent catabolite repression effect in Escherichia coli is amon

129 lted in partial relief of succinate-mediated catabolite repression, extreme sensitivity to cobalt lim

130 in the nucleus, thereby activating nitrogen catabolite repression genes.

131 cally relieved glucose- and sucrose-promoted catabolite repression, (ii) reduced the growth rate in m

132 a transcriptional regulator known to mediate catabolite repression in a number of low-G+C-content gra

133 tein IIA(Glc) plays a key regulatory role in catabolite repression in addition to its role in the vec

134 he sole signaling molecule for CcpA-mediated catabolite repression in B. subtilis.

135 g the process of sugar import via the PTS to catabolite repression in bacilli.

136 ant physiological roles, ranging from carbon catabolite repression in bacteria to mediating the actio

137 se-pairing RNA Spot 42 plays a broad role in catabolite repression in Escherichia coli by directly re

138 ENR to the regulatory network behind carbon catabolite repression in Escherichia coli is presented.

139 lect an aspect of a more global mechanism of catabolite repression in L. monocytogenes.

140 The loss of catabolite repression in lacR mutants was seen in cells

141 tate of which is the control point of carbon catabolite repression in low G+C Gram-positive bacteria.

142 These mutants suggested that catabolite repression in pseudomonads might, in part, in

143 gether with the Hfq protein, participates in catabolite repression in pseudomonads, helping to coordi

144 ne-22 residue, and that HPr-His22-P enhances catabolite repression in the presence of succinate.

145 although the carbon sources responsible for catabolite repression in the two species differ.

146 luorescens, suggesting a common mechanism of catabolite repression in these three species.

147 n enteric bacteria, the key player of carbon catabolite repression is a component of the glucose-spec

148 In Sinorhizobium meliloti, catabolite repression is influenced by a noncanonical ni

149 CcpA to inhibit transcription suggests that catabolite repression is not simply caused by CcpA bindi

150 Here, we show that this catabolite repression is relieved by mutations that weak

151 mpared with a relatively weak role in carbon catabolite repression, is similar to the role of RCO1 in

152 c archaeon Sulfolobus solfataricus employs a catabolite repression-like regulatory system to control

153 ction seen in this organism indicates that a catabolite repression-like system is present in a member

154 The archaeon Sulfolobus solfataricus uses a catabolite repression-like system to control production

155 nes are substrate inducible and sensitive to catabolite repression, mediated through ArcR and CcpA, r

156 the parent H26 and glpK mutant strains, with catabolite repression more pronounced in the glycerol ki

157 circuit responsible for regulating nitrogen catabolite repression (NCR) in yeast.

158 Nitrogen catabolite repression (NCR) is the basis of this selecti

159 en use is mediated in large part by nitrogen catabolite repression (NCR), which results in the repres

160 gnal transduction pathway regulates nitrogen catabolite repression (NCR)-sensitive (GAP1, GAT1, DAL5)

161 and Deh1p mediate the regulation of nitrogen catabolite repression (NCR)-sensitive gene expression in

162 hey bind GATA sequences upstream of nitrogen catabolite repression (NCR)-sensitive genes and activate

163 of Gln3 and Gat1, the activators of nitrogen catabolite repression (NCR)-sensitive genes whose produc

164 ities is transcriptional control of nitrogen catabolite repression (NCR)-sensitive genes.

165 ressors (Dal80p and Deh1p) regulate nitrogen catabolite repression (NCR)-sensitive transcription in S

166 Gln3 intracellular localization and nitrogen catabolite repression (NCR)-sensitive transcription in S

167 Nitrogen catabolite repression (NCR)-sensitive transcription is a

168 Gln3, the major activator of nitrogen catabolite repression (NCR)-sensitive transcription, is

169 riptional activator responsible for nitrogen catabolite repression (NCR)-sensitive transcription, per

170 ulates in the nucleus, and mediates nitrogen catabolite repression (NCR)-sensitive transcription.

171 Expression of these latter genes is nitrogen catabolite repression (NCR)-sensitive, i.e. expression i

172 This regulation is designated nitrogen catabolite repression (NCR).

173 a physiological process designated nitrogen catabolite repression (NCR).

174 r ones through a process designated nitrogen catabolite repression (NCR).

175 es whose expression is sensitive to nitrogen catabolite repression (NCR).

176 ogical response has been designated nitrogen catabolite repression (NCR).

177 ogical response has been designated nitrogen catabolite repression (NCR).

178 Catabolite repression of a number of catabolic operons i

179 CcpA, which is responsible for catabolite repression of a number of secondary carbon so

180 ) may be involved in but is not required for catabolite repression of alpha-amylase, indicating that

181 Catabolite repression of ctaBCD'-lacZ was partly depende

182 f the ccpA gene had no discernible effect on catabolite repression of fruA.

183 ans-acting factors involved in induction and catabolite repression of fruA.

184 Catabolite repression of galactose by glucose is one of

185 Additionally, Crp mediates strong indirect catabolite repression of many cytoplasmic stress respons

186 lot experiments also showed that there is no catabolite repression of nanE-nanA transcription by gluc

187 d growth phenotype was reflected in a strong catabolite repression of pauA promoter activation by CAD

188 protein, but was not necessary for nitrogen catabolite repression of peptide import or PTR2 expressi

189 for the HPr protein was reported to relieve catabolite repression of several genes.

190 ent work has shown that in Bacillus subtilis catabolite repression of several operons is mediated by

191 and that this mechanism in part accounts for catabolite repression of sigma(L)-directed levD operon e

192 rulation in C. perfringens, glucose-mediated catabolite repression of sporulation is not due to the a

193 CreA-dependent carbon catabolite repression of starch and ethanol utilization

194 Synergistic carbon catabolite repression of the Bacillus subtilis aconitase

195 Carbon catabolite repression of the Bacillus subtilis citrate s

196 hromosomal bkdR-lacZ fusion, suggesting that catabolite repression of the bkd operon was the result o

197 enes; (iii) CcpA plays little direct role in catabolite repression of the cel regulon, but loss of sp

198 Carbon catabolite repression of the gnt operon of Bacillus subt

199 endent phosphorylation, also prevents carbon catabolite repression of the gnt operon.

200 mutants that had abnormal succinate-mediated catabolite repression of the melA-agp genes, which are r

201 Catabolite repression of the tricarboxylic acid (TCA) cy

202 f highly expressed genes that are subject to catabolite repression or activation mediated by the cycl

203 richia coli growing on sugars that result in catabolite repression or amino acids that feed into glyc

204 on this enzymatic activity or the canonical catabolite repression pathway, but likely does require s

205 form of a negative regulator of the nitrogen catabolite repression pathway, Ure2p.

206 substitution into HPr alleviated the strong catabolite repression phenotypes of strains carrying Del

207 se or casamino acids, suggesting that carbon catabolite repression plays a role in regulating xynA.

208 by negative feedback on glpK expression via catabolite repression, possibly to prevent methylglyoxal

209 al abscess formation, indicating that carbon catabolite repression presents an important pathogenesis

210 We linked this to indirect regulation of the catabolite repression protein Crc via the non-coding RNA

211 as luxS and ompX and provide a link between catabolite repression, quorum sensing, and nitrogen assi

212 Catabolite repression regulates transcription initiation

213 e master transcriptional regulator of carbon catabolite repression/regulation (CCR).

214 wth on non-glucose substrates as part of the catabolite-repression response.

215 The other mutants display less stringent catabolite repression, resulting in leaky expression of

216 l (PRC1) vacuolar protease genes is nitrogen catabolite repression sensitive and is regulated by the

217 -responsive) and TorC1-independent (nitrogen catabolite repression-sensitive and methionine sulfoximi

218 Gln3p is a nitrogen catabolite repression-sensitive GATA-type transcription

219 sequences situated upstream of all nitrogen catabolite repression-sensitive genes that encode enzyme

220 eving Gln3 nuclear localization and nitrogen catabolite repression-sensitive transcription in respons

221 family DNA binding proteins mediate nitrogen catabolite repression-sensitive transcription in Sacchar

222 ontaining UASNTR sites that mediate nitrogen catabolite repression-sensitive transcription, and the s

223 Gln3-Myc13 nuclear accumulation and nitrogen catabolite repression-sensitive transcription, generate

224 s leads to an increase in succinate-mediated catabolite repression (SMCR).

225 carbon sources is termed succinate-mediated catabolite repression (SMCR).

226 s of various mechanisms for glucose control (catabolite repression, specific repression, and inducer

227 in carbon source can favor different carbon catabolite repression strategies.

228 icomponent phosphorelay system that controls catabolite repression, sugar transport and carbon metabo

229 The nanATEK operon is controlled by catabolite repression, suggesting that diminished expres

230 concluded that the components of the carbon catabolite repression system are essential to regulating

231 utations also increase glycerol-induced auto-catabolite repression that reduces glpK transcription in

232 tions to illustrate systemic effects such as catabolite repression, the aerobic/anaerobic diauxic shi

233 PTS) of gram-positive bacteria and regulates catabolite repression through phosphorylation/dephosphor

234 nd uptake in Escherichia coli are subject to catabolite repression through the cyclic AMP (cAMP)-CRP

235 l a key physiological role of cAMP-dependent catabolite repression: to ensure that proteomic resource

236 f some cellulases and hemicellulases, that a catabolite repression type of mechanism regulates cellul

237 preferred carbon source, succinate can exert catabolite repression upon genes needed for the utilizat

238 d by glucose but not by fructose, suggesting catabolite repression via two cre-like sequences identif

239 Sucrose-dependent catabolite repression was also evident in strains contai

240 otein 30% identical to CcpA, and relief from catabolite repression was shown to be due to the absence

241 o glucose-promoted but not mannitol-promoted catabolite repression were identified.