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1 ependently associated with increased risk of cataractogenesis.
2 define radiation-hormone interactions during cataractogenesis.
3 age-related protein aggregation in lens and cataractogenesis.
4 cell processes implicated in steroid-induced cataractogenesis.
5 alpha A-crystallins occurs during aging and cataractogenesis.
6 eins are formed in the lens during aging and cataractogenesis.
7 s suggest a role for dietary carbohydrate in cataractogenesis.
8 ation is considered a factor responsible for cataractogenesis.
9 tance to oxidative stress, and, potentially, cataractogenesis.
10 linked to desmin related cardiomyopathy and cataractogenesis.
11 ity and thus contribute to oxidation-induced cataractogenesis.
12 taractous human lenses and may contribute to cataractogenesis.
13 rectly during normal lens development and/or cataractogenesis.
14 or and whole lenses has been associated with cataractogenesis.
15 delling the biological processes involved in cataractogenesis.
16 fertility and indirectly, through increased cataractogenesis.
17 f lens crystallins associated with aging and cataractogenesis.
18 here to be a key initiator of the process of cataractogenesis.
19 his pathway contributes to lens function and cataractogenesis.
20 as been thought to play an important role in cataractogenesis.
21 ve stress-induced apoptosis is implicated in cataractogenesis.
22 ls such as copper and iron are implicated in cataractogenesis.
23 and the kinetics of molecular change during cataractogenesis.
24 lization of the protein is not necessary for cataractogenesis.
25 tion therapy could be beneficial in delaying cataractogenesis.
26 ese crystallins is correlated with aging and cataractogenesis.
27 n in intercellular communication, leading to cataractogenesis.
28 ination showed the early histologic signs of cataractogenesis.
29 echanism of the initial stages of UV-induced cataractogenesis.
30 deficits contributing to diabetes-associated cataractogenesis.
31 tible to oxidative stress and, therefore, to cataractogenesis.
32 ed to detecting lens changes associated with cataractogenesis.
33 ular lens has been shown to prevent or delay cataractogenesis.
34 16-25 to determine if it was altered during cataractogenesis.
35 st that dietary carbohydrates play a role in cataractogenesis.
36 y explain their accumulation on aging and in cataractogenesis.
38 indings may have relevance for understanding cataractogenesis and other degenerative conditions in wh
39 eful for investigations of radiation-induced cataractogenesis and other studies of lens toxicity.
40 ine protease inhibitor, dramatically delayed cataractogenesis and prevented proteolytic cleavage of b
41 ested to be a common underlying mechanism of cataractogenesis, and augmentation of the antioxidant de
42 omized rat serves as a model for age-related cataractogenesis, and observation of a clear protective
43 present study aimed to determine the risk of cataractogenesis associated with radiation exposure from
44 ovel molecular insights into mouse and human cataractogenesis caused by alpha 8 and alpha 3 mutations
46 in 3 gene is involved in age-related nuclear cataractogenesis in alpha3Cx46 knockout mice, 129alpha3C
52 n inheritance patterns and the mechanism for cataractogenesis in these mutants are poorly understood.
53 ng changes in protease activities throughout cataractogenesis indicated that Lp82 activity was dramat
57 otype are independent of each other and that cataractogenesis is under the influence of an unidentifi
58 the use of a rat model of radiation-induced cataractogenesis, it was found that estrogen can either
59 osis of lens epithelial cells contributes to cataractogenesis, the proteasome may be a therapeutic ta
60 lied for studies of lens differentiation and cataractogenesis using induced pluripotent stem (iPS) ce
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