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1 longer than the glutamate-evoked increase in cation conductance.
2 dies and investigations of single-channel or cation conductance.
3 hat some mutant band 3 protein can mediate a cation conductance.
4 independent, Ca(2+)-regulated, non-selective cation conductance.
5 dney contain an ATP-activated, non-selective cation conductance.
6 epolarization arise from the activation of a cation conductance.
7 duces an L-canavanine-activated nonselective cation conductance.
8 o NK3-mediated activation of a non-selective cation conductance.
9 as associated with a Ca2+- and Na+-permeable cation conductance.
10 that results in the observed alterations in cation conductance.
11 activation of a hyperpolarization-activated cation conductance.
12 egmental area primarily by increasing such a cation conductance.
13 electivity and gain of a depolarizing inward cation conductance.
14 mediated by either chloride or nonselective cation conductances.
15 ocker of the calcium-activated non-selective cation conductance, abolished the effect on eupnoeic act
16 outward currents mediated by a nonselective cation conductance and a calcium-activated potassium con
17 nductance, which may include a non-selective cation conductance and a K+ conductance, appears to be i
18 rest of a hyperpolarization-activated mixed-cation conductance and a low-threshold, depolarization-a
19 vide a molecular explanation for p7-mediated cation conductance and its inhibition by adamantane deri
20 obilization and activation of a nonselective cation conductance; and cAMP increases mediated by G(s).
21 ocytes, vasopressin activated a nonselective cation conductance at concentrations that enhanced slow
23 tion of a voltage-independent, non-selective cation conductance, but the timing mechanism responsible
26 l 2 (TPC2) as the first reported melanosomal cation conductance by directly patch-clamping skin and e
27 key role of a Ca(2+)-activated nonselective cation conductance ((CaN)) in generating the plateau pot
31 tassium channels and that the cAMP-dependent cation conductance does not contribute significantly to
34 ts, the free-energy barriers and the maximum cation conductance for the permeation of various cations
35 (GIRK/K(G)) conductance and a cAMP-dependent cation conductance have both been implicated in this eff
36 e K+, and hyperpolarization-activated, mixed cation conductances have varying distributions in hippoc
37 mediated by a calcium-activated nonselective cation conductance (I(CAN)) that is activated by calcium
38 tively active Ca(2+)-permeable non-selective cation conductance (I(cat)) in rabbit ear artery smooth
39 the hyperpolarization-activated nonselective cation conductance (I(h)) attenuates EPSPs mainly by red
42 nist for the calcium-activated non-selective cation conductance (ICAN ) abolished the effect of PGE2
43 n glucocorticoid-free medium by activating a cation conductance identical to the glucocorticoid-induc
47 imulus for catfish, activated a nonselective cation conductance in some bilayers, which was antagoniz
49 muscarinic receptor responsive non-selective cation conductance in Trp6 cells that was absent in cont
50 dy is to investigate the effect of Ang II on cation conductances in freshly dispersed rabbit mesenter
51 indicate that Ang II activates two distinct cation conductances in mesenteric artery myocytes by sti
52 trated that 5-HT and SP modulated background cation conductances in pre-BotC and motor neurons, inclu
53 ecrease and a Cs+-insensitive, non-selective cation conductance increase may account for the two type
54 activation of a hyperpolarization-activated cation conductance is a key mechanism through which sync
55 tion is Ca2+, indicating that influx via the cation conductance is another source of the increases in
57 group of ICC expressed a basal non-selective cation conductance (NSCC) that was inhibited by an incre
58 nderlie the diversity of amiloride-sensitive cation conductances observed in a wide variety of tissue
60 alysis of the novel P2Y1 receptor-associated cation conductance revealed that the open channel curren
61 -100 microM) stimulates a novel nonselective cation conductance seen only in oocytes expressing human
62 ent entering the cell through a non-specific cation conductance that continuously varied in amplitude
63 4 channel agonist, activated a non-selective cation conductance that coupled to activation of SK chan
64 nhibited, voltage-independent, non-selective cation conductance that has similar properties to the co
67 gramicidins form well-defined channels, with cation conductances that are approximately 60% of those
70 ompanied by up-regulation of a non-selective cation conductance with TRPM4-like properties and appare
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