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1 longer than the glutamate-evoked increase in cation conductance.
2 dies and investigations of single-channel or cation conductance.
3 hat some mutant band 3 protein can mediate a cation conductance.
4 independent, Ca(2+)-regulated, non-selective cation conductance.
5 dney contain an ATP-activated, non-selective cation conductance.
6 epolarization arise from the activation of a cation conductance.
7 duces an L-canavanine-activated nonselective cation conductance.
8 o NK3-mediated activation of a non-selective cation conductance.
9 as associated with a Ca2+- and Na+-permeable cation conductance.
10  that results in the observed alterations in cation conductance.
11  activation of a hyperpolarization-activated cation conductance.
12 egmental area primarily by increasing such a cation conductance.
13 electivity and gain of a depolarizing inward cation conductance.
14  mediated by either chloride or nonselective cation conductances.
15 ocker of the calcium-activated non-selective cation conductance, abolished the effect on eupnoeic act
16  outward currents mediated by a nonselective cation conductance and a calcium-activated potassium con
17 nductance, which may include a non-selective cation conductance and a K+ conductance, appears to be i
18  rest of a hyperpolarization-activated mixed-cation conductance and a low-threshold, depolarization-a
19 vide a molecular explanation for p7-mediated cation conductance and its inhibition by adamantane deri
20 obilization and activation of a nonselective cation conductance; and cAMP increases mediated by G(s).
21 ocytes, vasopressin activated a nonselective cation conductance at concentrations that enhanced slow
22 er and by the calcium-activated nonselective cation conductance blocker flufenamic acid.
23 tion of a voltage-independent, non-selective cation conductance, but the timing mechanism responsible
24  (10 microM) caused inhibition of whole-cell cation conductance by approximately 55 %.
25 zation of the neurons initiated by increased cation conductance by cAMP-gated cation channels.
26 l 2 (TPC2) as the first reported melanosomal cation conductance by directly patch-clamping skin and e
27  key role of a Ca(2+)-activated nonselective cation conductance ((CaN)) in generating the plateau pot
28  Cl(-) solutions, and an inwardly rectifying cation conductance (cationic I(ATP)).
29             Our data suggest that a specific cation conductance composed of acid-sensing ion channels
30              In smooth muscle, non-selective cation conductances contribute to agonist-evoked depolar
31 tassium channels and that the cAMP-dependent cation conductance does not contribute significantly to
32                                 The block of cation conductances during the internal dialysis of Cs(+
33  for cation binding, cation selectivity, and cation conductance efficiency.
34 ts, the free-energy barriers and the maximum cation conductance for the permeation of various cations
35 (GIRK/K(G)) conductance and a cAMP-dependent cation conductance have both been implicated in this eff
36 e K+, and hyperpolarization-activated, mixed cation conductances have varying distributions in hippoc
37 mediated by a calcium-activated nonselective cation conductance (I(CAN)) that is activated by calcium
38 tively active Ca(2+)-permeable non-selective cation conductance (I(cat)) in rabbit ear artery smooth
39 the hyperpolarization-activated nonselective cation conductance (I(h)) attenuates EPSPs mainly by red
40 t, I(T), and the hyperpolarization-activated cation conductance, I(sag).
41  presence of the hyperpolarization-activated cation conductance, I(sag).
42 nist for the calcium-activated non-selective cation conductance (ICAN ) abolished the effect of PGE2
43 n glucocorticoid-free medium by activating a cation conductance identical to the glucocorticoid-induc
44 yl borate (2-APB) to activate a nonselective cation conductance in its STIM1-independent mode.
45 fied that is essential for the light-induced cation conductance in photoreceptor cells.
46  production of a PLC-dependent, nonselective cation conductance in pontine neurons.
47 imulus for catfish, activated a nonselective cation conductance in some bilayers, which was antagoniz
48 bitors transiently increase the paracellular cation conductance in the thick ascending limb.
49 muscarinic receptor responsive non-selective cation conductance in Trp6 cells that was absent in cont
50 dy is to investigate the effect of Ang II on cation conductances in freshly dispersed rabbit mesenter
51  indicate that Ang II activates two distinct cation conductances in mesenteric artery myocytes by sti
52 trated that 5-HT and SP modulated background cation conductances in pre-BotC and motor neurons, inclu
53 ecrease and a Cs+-insensitive, non-selective cation conductance increase may account for the two type
54  activation of a hyperpolarization-activated cation conductance is a key mechanism through which sync
55 tion is Ca2+, indicating that influx via the cation conductance is another source of the increases in
56 polarization and so an amiloride-insensitive cation conductance is present.
57 group of ICC expressed a basal non-selective cation conductance (NSCC) that was inhibited by an incre
58 nderlie the diversity of amiloride-sensitive cation conductances observed in a wide variety of tissue
59                            Slow nonselective cation conductances play a central role in determining t
60 alysis of the novel P2Y1 receptor-associated cation conductance revealed that the open channel curren
61 -100 microM) stimulates a novel nonselective cation conductance seen only in oocytes expressing human
62 ent entering the cell through a non-specific cation conductance that continuously varied in amplitude
63 4 channel agonist, activated a non-selective cation conductance that coupled to activation of SK chan
64 nhibited, voltage-independent, non-selective cation conductance that has similar properties to the co
65 ill via a novel non-selective store-operated cation conductance that is blocked by nifedipine.
66   In voltage clamp, high [Ca2+]0 activates a cation conductance that underlies the depolarization.
67 gramicidins form well-defined channels, with cation conductances that are approximately 60% of those
68 MP, evoked a robust activation of whole-cell cation conductance to 220 % of control.
69                               A non-specific cation conductance was reversibly induced upon deoxygena
70 ompanied by up-regulation of a non-selective cation conductance with TRPM4-like properties and appare

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