ライフサイエンスコーパス: cation transport

1 lation and reordering, and thus a slowing of cation transport.

2 led receptor pathway, membrane potential and cation transport.

3 ittle is known about how disorder influences cation transport.

4 contributes to forming a pathway for organic cation transport.

5 occurs as part of the catalytic cycle during cation transport.

6 e examined further for phenotype relating to cation transport.

7 e 11 are responsible for altering monovalent cation transport.

8 to the membrane-embedded part that mediates cation transport.

9 raction with PTS rather than eserine-induced cation transport.

10 nt energy barrier, thus permitting efficient cation transport.

11 rmal surface area in RBCs and for normal RBC cation transport.

12 may be required for energy transduction and cation transport.

13 Glu(206) (E206Q) resulted in loss of organic cation transport activity, whereas conserving the negati

14 alanine or tryptophan fully restored organic cation transport activity.

15 nthesis, carbohydrate uptake and metabolism, cation transport, amino acid metabolism, ubiquinone and

16 L5 of NHE1 as important elements involved in cation transport and inhibitor sensitivity, which may in

17 t remains unclear how TRPM6 affects divalent cation transport and whether this involves functional ho

18 tive Neosepta CMS is known to block divalent cations transport and can therefore mitigate reductions

19 rs implicated genes and pathways involved in cation transport, angiotensin production, and regulators

20 ring xylem refilling and for the activity of cation transport as having a significant role in the gen

21 lation and cation binding domains in various cation transport ATPase.

22 ATP4 is thought to be a cation-transporting ATPase responsible for maintaining l

23 ATP-binding cassette (ABC) transporter and a cation-transporting ATPase were upregulated in GECs.

24 These results suggest that PepO, a cation-transporting ATPase, and an ABC transporter are r

25 The gastric H,K-ATPase is related to other cation transport ATPases, for example, Na,K-ATPase and C

26 high degree of sequence homology with other cation transport ATPases.

27 TPase, which belongs to the P-type family of cation-transporting ATPases, is activated up to 10-fold

28 evisiae, which belongs to the P2 subgroup of cation-transporting ATPases, is encoded by the PMA1 gene

29 Within the large family of P-type cation-transporting ATPases, members differ in the numbe

30 ) RBCs demonstrate an abnormal regulation of cation transport by cell volume.

31 t 5 (M5) is thought to play a direct role in cation transport by the sarcoplasmic reticulum Ca2+-ATPa

32 Proton and sodium cation transport by these compounds has been demonstrate

33 Pump-mediated K(+)-like organic cation transport challenges the concept of rigid structu

34 ress in understanding altered red blood cell cation transport characteristics of SCD.

35 not undergo large-scale movements during the cation transport cycle.

36 hibits the Na, K-ATPase by disruption of the cation transport domain rather than the catalytic domain

37 carnitine transport function and the organic cation transport function of OCTN2.

38 mutations may not interfere with the organic cation transport function.

39 ion but significantly stimulated the organic cation transport function.

40 These couple ATP hydrolysis with cation transport, generating cation gradients across mem

41 les induce coupled chloride anion and sodium cation transport in both liposomal models and cells, and

42 ive channel regulation, leading to increased cation transport in erythroid cells.

43 r understanding of the mechanisms of organic cation transport in rat liver, little is known about the

44 n inhibitor of NKCC1, alters transepithelial cation transport in rat OMCD.

45 oposed role of this protein as a mediator of cation transport in RBC.

46 that the filament growth can be dominated by cation transport in the dielectric film.

47 that the cAMP pathway regulates PC2-mediated cation transport in the hST.

48 for elucidation of the mechanisms of organic cation transport in the human liver and understanding of

49 tle is known about the mechanisms of organic cation transport in the human liver.

50 ransporter, which may play a role in organic cation transport in vivo.

51 resulted in (i) greater pyrophosphate-driven cation transport into root vacuolar fractions, (ii) incr

52 Available data indicate that cation transport is impaired in many cells in chronic re

53 Electrogenic cation transport is known to reside in the principal cel

54 transport is Na(+)-dependent whereas organic cation transport is Na(+)-independent, we investigated t

55 d human organic cation transporters, organic cation transport kinetics differed markedly.

56 diated Fe acquisition, Fe storage, and other cation transport mechanisms.

57 ctance is accompanied by a sharp increase in cation transport number and by pronounced open-channel l

58 utated in genes involved in gene regulation, cation transport or stress tolerance were shown to be hi

59 ode a protein of 732 amino acids, similar to cation transport P-type ATPases in the Cpx-type family.

60 the Saccharomyces cerevisiae Atx1 is Ccc2, a cation transporting P-type ATPase located in secretory v

61 ne encodes a highly homologous member of the cation-transport P-type ATPase family.

62 st Ccc2 protein, which are integral membrane cation-transporting P-type ATPases involved in copper tr

63 ses resembles that of the well-characterized cation-transporting P-type ATPases, and it is unknown wh

64 rotein (WNDP) belongs to the large family of cation-transporting P-type ATPases, however, the detaile

65 endocytic compartments, but their kinship to cation-transporting P-type transporters raised doubts ab

66 s 5 and 8 of the P-ATPases contribute to the cation transport pathway and that the fundamental mechan

67 A carrier-mediated organic cation transport process appears to exist in the conjunc

68 ine, but not by substrates for other organic cation transport processes identified in blLPM vesicles,

69 bbit (rb) exhibit differences in citrate and cation transport properties.

70 d the phylogenetic relationships of over 150 cation transport proteins.

71 extramembraneous ATP binding domain and the cation transport regions of the Na,K-ATPase.

72 analysis predicted that the hub gene CHAC1 (cation transport regulator homolog 1) was regulated by t

73 that an Arabidopsis thaliana protein with a cation transport regulator-like domain, hereafter referr

74 CHAC1 (cation transport regulator-like protein 1), a novel gene

75 eexisting networks designed for adhesion and cation transport/response and that its regulation occurs

76 The cation transport selectivities of the Ca2+ ionophores A2

77 Furthermore, the structure reveals that cation transport site II is occupied by Mg(2+), and crys

78 hrough conformational changes induced at the cation transport site located within the membrane or at

79 the carnitine transport site and the organic cation transport site were not identical.

80 n the cardiac glycoside binding site and the cation transport sites of the Na,K-ATPase transpires giv

81 ubnetwork was enriched for genes involved in cation transport, synaptic transmission, and transmissio

82 or the components of an ATP-binding cassette cation-transport system (troABCD) and a DtxR-like transc

83 W solution as assessed by tetraethylammonium cation transport (TEA).

84 with the previously known role of SLC11A1 in cation transport, these effects were enhanced by elevati

85 nt adsorption and hydrodynamic dispersion on cation transport through a reactive porous medium with a

86 x 10(-14) cm(2)/s for ambipolar electron and cation transport throughout the film.

87 n gerO spores but not the ability to restore cation transport to E. coli cells defective in K(+) upta

88 The electrodriven cation transport together with the use of highly Cs+ sel

89 In peripheral tissues, organic cation transport via some OCTs is inhibited by corticost

90 Plots of the log of the rate of cation transport vs the log of the ionophore concentrati

91 Long-distance radical cation transport was studied in DNA condensates.

92 d without external Na(+) and K(+) represents cation transport when normal occlusion at sites I and II

93 compared with SGLT1 and the sugar-activated cation transport without sugar transport that occurs in