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1 e before giving rise to a long and extensive cauda equina.
2 tonin, and dopamine or by stimulation of the cauda equina.
3 ficits at admission and 17.3% presented with cauda equina.
4 stimulated through their motor roots in the cauda equina.
5 dehyde and removal of the sciatic nerves and cauda equina.
6 c spinal cord and nodular enhancement of the cauda equina.
7 vidence of leptomeningeal enhancement in the cauda equina although CSF cytology was negative, and one
8 ging studies demonstrated enhancement of the cauda equina and leptomeninges of the lower spinal cord.
9 5% CI 0.85-2.75; p=0.15), and spinal cord or cauda equina compressions were reported in seven (2%) of
12 on that future repair of brachial plexus and cauda equina injuries will include spinal cord surgery t
18 sacral ventral root avulsion injury model of cauda equina injury to investigate the lesion effect in
19 peated, they demonstrated enhancement of the cauda equina nerve roots, trigeminal nerve, and pachymen
22 rotoxicity, including seizures, papilledema, cauda equina syndrome (n = 2), and encephalitis after a
24 s presented with meningitis alone (73%), the cauda equina syndrome or focal infection (15%), or poste
25 anglia and brainstem, spinal cord, conus and cauda equina to disorders of peripheral innervation.
26 e ventral tail artery in rats that had their cauda equina transected to eliminate the somatic innerva
27 (17%) of the 18 lesions, enhancement in the cauda equina was similar to that seen in Guillain-Barre
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