ライフサイエンスコーパス: caudate

1 in the right primary motor cortex and right caudate.

2 arning rate and BOLD activity in the ventral caudate.

3 ith the neural representation of risk in the caudate.

4 in the medial prefrontal cortex and ventral caudate.

5 opercularis), left temporal pole, and right caudate.

6 poral gyrus, frontal regions, cerebellum and caudate.

7 s well as left anterior cingulate cortex and caudate.

8 b regions including the cingulate cortex and caudate.

9 end level relative to baseline in the dorsal caudate.

10 e significantly increased fALFF in the right caudate.

11 ctivity between the dorsal ACC and bilateral caudate.

12 e type of learning processes mediated by the caudate.

13 t sizes for BPND and SBRHR were found in the caudate (0.6), putamen (1.7 and 1.4), ventral striatum (

14 frontal white matter (-11.4%; P < .001), and caudate (-10.6%; P = .04), while the Cho:Cr ratio was hi

15 wer than those in controls (P < 0.05) in the caudate (2.54 +/- 0.79 vs. 3.68 +/- 0.56), putamen (1.39

16 ion of 20 ng/ml and D2/3 occupancies of 43% (caudate), 25% (putamen), 43% (thalamus).

17 on of 60 ng/ml, and D2/3 occupancies of 61% (caudate), 49% (putamen) and 69% (thalamus).

18 iated with high D2/3 occupancies (65 +/- 8%, caudate; 67 +/- 11%, thalamus; 52 +/- 11%, putamen).

19 tribution volume ratio (DVR) relative to the caudate (a pseudoreference region).

20 s found that subjects with greater dlPFC and caudate activation to nocebo-induced itch also demonstra

21 ced itch also demonstrated greater dlPFC and caudate activation, respectively, for real allergen itch

22 These results suggest that, as indexed by caudate activity, successful service dogs generalize ass

23 ly decreased in the right ventral and dorsal caudate after prolonged abstinence.

24 utamen, p<0.0001) and (18)F-FDOPA uptake (in caudate: age </=50 years, p=0.0002; all other age ranges

25 iatum, parietal lobe, dorsal putamen, dorsal caudate, amygdala, and hippocampus.

26 Using anatomically defined ROIs in the caudate, amygdala, and visual cortex, we developed a cla

27 mbens, and putamen activations and increased caudate-amygdala and caudate-hippocampus connectivity.

28 and superior temporal gyrus, as well as left caudate and anterior cingulate cortex.

29 d functional connectivity between the dorsal caudate and both the thalamus and midbrain bilaterally.

30 ght hippocampus, on average, but lower right caudate and corpus callosum volume, relative to 22q-del

31 at shape asymmetry in hippocampus, amygdala, caudate and cortex is predictive of disease onset.

32 Recording from downstream neurons in the caudate and from thalamic neurons projecting to the medi

33 motor area fibers (M2/M3/M4) arched over the caudate and lateral motor area fibers (M1/LPMCv) curved

34 Greater activation in the left caudate and left frontal pole was associated with abstin

35 activated bilateral insula, bilateral dorsal caudate and left precentral gyrus.

36 d right caudate, and increased ReHo in right caudate and left putamen.

37 ly associated with reductions in activity in caudate and limbic structures, respectively.

38 associated with lower gray matter volumes in caudate and medial orbitofrontal cortex.

39 Greater caudate and medial prefrontal cortex reactivity to gain

40 functional connectivity between the ventral caudate and medial temporal cortex increased as a functi

41 signal during food picture evaluation in the caudate and nucleus accumbens.

42 r within the left uncinate fasciculus, right caudate and occipital regions (p < 0.05).

43 (anterior insula), and learning and memory (caudate and parahippocampal gyrus).

44 ed to distractor processing within the right caudate and posterior putamen.

45 (SI) at follow-up, most substantially in the caudate and putamen (P < .001 for both).

46 wnregulated and 822 upregulated genes in the caudate and putamen (striatum) of TS individuals.

47 (SMD) of PET uptakes in the whole striatum, caudate and putamen in manifest and premanifest HDGECs c

48 temporal lobe, and occipital lobe as well as caudate and putamen nuclei, after adjusting for age (P <

49 ls from the anterior cingulate cortex (ACC), caudate and putamen of 16 RC BD-I, 34 non-RC BD-I and 44

50 nglia transcriptome by RNA sequencing in the caudate and putamen of nine TS and nine matched normal c

51 The age-by-diagnosis interaction in the caudate and putamen supports the relevance of different

52 Alterations in total gray matter and caudate and putamen volumes in unaffected siblings sugge

53 d asymmetry for thalamus, lateral ventricle, caudate and putamen volumes, and rightward asymmetry for

54 , and ventral striatum) and with ex-smokers (caudate and putamen).

55 For the caudate and putamen, LL showed higher DRD2 availability

56 amine transporter binding (all age ranges in caudate and putamen, p<0.0001) and (18)F-FDOPA uptake (i

57 ons showed a single relationship between the caudate and QSU.

58 Reduced functional connectivity between the caudate and the ventrolateral prefrontal cortex was sele

59 r multiple system atrophy in the putamen and caudate, and increased for progressive supranuclear pals

60 levated fALFF in bilateral putamen and right caudate, and increased ReHo in right caudate and left pu

61 ne showed increased GBCr in the lateral PFC, caudate, and insula.

62 efrontal and parietal regions, the amygdala, caudate, and mid-brain.

63 ight anterior insula, putamen, thalamus, and caudate, and midbrain and pons.

64 inferior frontal and superior temporal gyri, caudate, and other structures is affirmed.

65 e frontal gyri, precuneus, cingulate cortex, caudate, and postcentral gyrus (all regions: p < .001, e

66 otor area (pre-SMA), inferior frontal gyrus, caudate, and subthalamic nucleus (STN).

67 In fMRI analyses, during inhibition, right caudate anomalies reflected a childhood ADHD history and

68 salience circuitry (ventral striatum, dorsal caudate, anterior cingulate cortex) during processing of

69 rtex), and control demand-selective (insula, caudate, anterior cingulate, and parietal cortex) event

70 dities, particularly in the anterior insula, caudate, anterior cingulate, medial frontal gyrus, and d

71 prefrontal cortex/anterior cingulate cortex, caudate, anterior insula, and thalamus were more likely

72 d middle temporal gyrus, olfactory gyrus and caudate are all related to learning, suggesting that the

73 This was not caused by altered size of the caudate, as its cross-sectional surface areas were simil

74 Decreased mean diffusivity in the left caudate at follow-up was seen (P < .001).

75 ents in patients with VCSL disruption due to caudate atrophy (e.g., Huntington's disease, HD), the re

76 acrostructural neuroimaging measures such as caudate atrophy and ventricular expansion were significa

77 er of specific neuroimaging measures such as caudate atrophy in disease-modifying trials, we propose

78 that impulsivity positively correlated with caudate baseline BPND in PG only.

79 t expansion, most marked in the ventromedial caudate bilaterally, the right pulvinar thalamic nucleus

80 Dopamine synthesis was 16% higher in the caudate body, 17% higher in the dorsal putamen, and 17%

81 and 3) to unexpected reward omission in the caudate body.

82 activation in multiple regions (e.g., in the caudate, cingulate, and precentral gyrus) and decreased

83 ent, patients exhibited higher fALFF in left caudate compared with controls.

84 riatal connectivity, which included abnormal caudate connections with a distributed set of associativ

85 Importantly, dorsal caudate connectivity with the ventrolateral thalamus and

86 unctional coupling between the dlPFC and the caudate correlates with the degree of susceptibility to

87 mbens (Cohen's d=-0.15), amygdala (d=-0.19), caudate (d=-0.11), hippocampus (d=-0.11), putamen (d=-0.

88 0.19 vs -0.10), amygdala (d=-0.18 vs -0.14), caudate (d=-0.13 vs -0.07), hippocampus (d=-0.12 vs -0.0

89 Caudate D2DR availability was positively associated with

90 Hippocampal and caudate D2DR availability were interrelated, and functio

91 l temporal cortex increased as a function of caudate D2DR availability.

92 ing and was linked to OT, whereas a stronger caudate-dACC connectivity was associated with increase i

93 smaller over the shorter intervals, although caudate diffusivity metrics performed strongly over 9 an

94 egulate dopamine release in the dorsolateral caudate (DLC) and nucleus accumbens (NAc) core.

95 saline demonstrated greater fMRI response in caudate, dorsolateral prefrontal cortex (dlPFC), and int

96 e striatal value representations by applying caudate electrical stimulation in macaque monkeys (n = 3

97 ype on DRD2 availability at baseline for the caudate (F(2,90) = 8.2, p = 0.001) and putamen (F(2,90)

98 igue and brain activation was evident in the caudate for this task as well.

99 ectivity between the eventual lesion and the caudate (for responders vs nonresponders, mean [SD] grou

100 Furthermore, caudate functional connectivity patterns differentiated

101 These value signals in the caudate guide the orienting of gaze differently: volunta

102 erently: flexible (short-term) values by the caudate head and stable (long-term) values by the caudat

103 ate groups of dopamine neurons innervate the caudate head and tail and may selectively guide the flex

104 gaze differently: voluntary saccades by the caudate head circuit and automatic saccades by the cauda

105 lidal volume (PV) in males, and (2) relative caudate head expansion and ventral striatum contraction

106 The plane immediately superior to the caudate head was chosen for analysis.

107 synthesis capacity in the dorsal putamen and caudate head was positively correlated with gambling dis

108 d the same Task X Fatigue interaction in the caudate head.

109 ngulate cortex, thalamus, putamen, pallidum, caudate, hippocampus, and brain stem.

110 brain measures: nucleus accumbens, amygdala, caudate, hippocampus, globus pallidus, putamen, thalamus

111 structures: the nucleus accumbens, amygdala, caudate, hippocampus, pallidum, putamen and thalamus.

112 cal structures (nucleus accumbens, amygdala, caudate, hippocampus, pallidum, putamen, thalamus, and l

113 tivations and increased caudate-amygdala and caudate-hippocampus connectivity.

114 dorsal mPFC and a region in posterior insula/caudate in which female but not male PG participants sho

115 l prefrontal cortex (DLPFC), hippocampus and caudate) in a much larger set of postmortem samples from

116 nt induction in the number of GAD67-cells in caudate-kindled rats in the dentate gyrus and CA3 hippoc

117 upt the excitation/inhibition balance in the caudate leading to dysfunctional corticostriatal circuit

118 neural responses to smoking cues in the left caudate, left inferior frontal gyrus, and left frontal p

119 n in the striatum, especially in the rostral caudate, manifesting as excess synthesis and release.

120 us mapped to executive function tasks; right caudate mapped to both executive function tasks and musi

121 We argue that caudate microstimulation can differentially increase sti

122 wn to induce neural plasticity [10, 11], and caudate microstimulation in primates has been shown to a

123 lenge effects were most profound in putamen, caudate, midbrain, thalamus, and cerebellum.

124 Strikingly, both amygdala-mPFC and caudate-mPFC coupling during active coping trials covari

125 Caudate neural recordings (n = 1) show that changes in v

126 vity network, with decreased connectivity in caudate nuclei and thalami and increased connectivity in

127 th nonsmokers, K was 15% to 20% lower in the caudate nuclei of consuming smokers.

128 s with CIS developed atrophy of the thalami, caudate nuclei, cerebellum, and frontal, parietal, and t

129 es in K in the right dorsal and left ventral caudate nuclei.

130 Dense bilateral connections were to caudate nuclei.

131 the prefrontal cortex (PFC), the head of the caudate nucleus (CN), and the ventral anterior nucleus (

132 fronto-striato-thalamic circuits-head of the caudate nucleus (hCaud), putamen, globus pallidus, thala

133 = 10), superior cerebellar peduncle (n = 7), caudate nucleus (n = 4), whole thalamus (n = 3), and put

134 left, but the opposite pattern in the right, caudate nucleus (P < .001).

135 11-labeled [11C]PMP acetylcholinesterase and caudate nucleus [11C]DTBZ monoaminergic positron-emissio

136 f synchronously generated S cells within the caudate nucleus adjoining the ganglionic eminence, poten

137 We found that spike activity in the caudate nucleus after each trial corresponded to an inte

138 The medial caudate nucleus also shows hyperactivity in humans lacki

139 two bilateral convergence zones (one in the caudate nucleus and another in the putamen) that consist

140 ividuals exhibit abnormal development of the caudate nucleus and associative cortical areas, suggesti

141 tivity between the right DLPFC and the right caudate nucleus and bilateral (para)cingulate gyrus incr

142 The caudate nucleus and cortical regions with connections to

143 Furthermore, increased activity in bilateral caudate nucleus and hippocampus for the cued relative to

144 Functional connectivity between the caudate nucleus and hippocampus was also increased after

145 ned period, volume reduction occurred in the caudate nucleus and hippocampus, but iron content increa

146 (p<0.05; corrected) abnormalities within the caudate nucleus and planum temporale.

147 n methamphetamine users from controls in the caudate nucleus and putamen and higher D1-receptor densi

148 D was positively correlated with BPND in the caudate nucleus and putamen in nonsmokers and female smo

149 based methods in both the DAT-rich striatum (caudate nucleus and putamen) and the SERT-rich extrastri

150 ce of multipolar ChAT-ir interneurons in the caudate nucleus and putamen, whereas monkeys have a more

151 between the connectivity profile between the caudate nucleus and the lateral prefrontal cortex and di

152 significant linear effects of the ARs in the caudate nucleus and the orbitofrontal cortex for all of

153 tington's disease, HD), the relevance of the caudate nucleus and VCSL on cortical visual processing i

154 The results revealed the caudate nucleus as the key brain structure involved in s

155 er, model-based fMRI analyses identified the caudate nucleus as the key structure involved in selecti

156 Higher iron content in the caudate nucleus at baseline predicted lesser improvement

157 hippocampus, thalamus, globus pallidus, and caudate nucleus compared with 26 control males (effect s

158 sed connectivity between the DN and the left caudate nucleus could play a role in balance impairment

159 lassifier based on the specialization of the caudate nucleus distinguished patients from controls wit

160 d both independent cognitive predictions for caudate nucleus dopaminergic (F = 7.25; P = .008) and co

161 ts with neocortical cholinergic deficits had caudate nucleus dopaminergic deficits.

162 </= -2) global cognitive impairment scores, caudate nucleus dopaminergic denervation was relatively

163 pically occurs in the context of significant caudate nucleus dopaminergic denervation.

164 s overlooked specialization of the posterior caudate nucleus for executive functions, often considere

165 otonergic degeneration in human ventromedial caudate nucleus from individuals with an APOE epsilon4 a

166 and cortical regions with connections to the caudate nucleus had markedly abnormal hemispheric specia

167 Similar results were seen in mouse caudate nucleus homozygous for APOE epsilon4 via targete

168 ebellum (granule cell layer), as well as the caudate nucleus in humans and chimpanzees.

169 crease in dopamine innervation of the medial caudate nucleus in humans is a species-typical character

170 e medial nucleus accumbens shell (NAcSh) and caudate nucleus in postmortem human brains.

171 ese results underscore the importance of the caudate nucleus in relation to cognitive fatigue.

172 tex, and midcingulate cortex, as well as the caudate nucleus involved in the reward system.

173 nd was associated with a reduced dentate and caudate nucleus iron content compared to placebo.

174 The caudate nucleus is a part of the visual corticostriatal

175 has demonstrated that neural activity in the caudate nucleus is modulated by task-relevant action val

176 The caudate nucleus may be involved in the repetitive moveme

177 striatal (123)I-FP-CIT binding ratios in the caudate nucleus of PSP patients than in that of both PD

178 striatal (123)I-FP-CIT binding ratios in the caudate nucleus of PSP patients than in that of both PD

179 specific bottom-up cues, and they place the caudate nucleus of the dorsal striatum at the center of

180 ution of visual corticostriatal loop and the caudate nucleus on generating selective response within

181 re was no evidence of altered variability of caudate nucleus or frontal lobe volumes.

182 re was no evidence of altered mean volume of caudate nucleus or putamen.

183 te cortex and between the right amygdala and caudate nucleus predicted the magnitude of reduction in

184 Thus, the caudate nucleus provides interpretive monitoring of ongo

185 have revealed that the head and tail of the caudate nucleus selectively and differentially process f

186 ofrontal cortex, anterior temporal lobe, and caudate nucleus than PCA, and PCA showed more asymmetric

187 Clozapine treatment led to reductions in caudate nucleus volume in three separate studies.

188 nal capsule adjacent to the head of the left caudate nucleus was found in PD-ICB, but not surviving c

189 ents, the decreased connectivity in the left caudate nucleus was related with worse balance performan

190 FA in the SN and CBF in the caudate nucleus were inversely correlated with motor dys

191 subset of these regions (PCC, thalamus, and caudate nucleus) covaried with the level of arousal.

192 gered endogenous opioid release in thalamus, caudate nucleus, and anterior insula.

193 binding sites in the hippocampus, thalamus, caudate nucleus, and cerebellum but not in the corpus ca

194 tral (head) and caudal (tail) regions of the caudate nucleus, both of which target the superior colli

195 timulations of the hippocampus, amygdala, or caudate nucleus, followed by sacrifice and immunohistoch

196 rtical regions (nucleus accumbens, amygdala, caudate nucleus, globus pallidus, hippocampus, putamen,

197 ing in functional connectivity involving the caudate nucleus, insula, medial prefrontal cortex and ot

198 Differences were detected in the dorsal caudate nucleus, putamen, and globus pallidus but the ob

199 served in the amygdala, raphe nuclei region, caudate nucleus, putamen, hippocampus, and anterior cing

200 inding potential in the raphe nuclei region, caudate nucleus, putamen, thalamus, and insula cortex (P

201 d in the substantia nigra (SNc), dentate and caudate nucleus, red nucleus, putamen and globus pallidu

202 ratios were 0.97 and 0.76 in the putamen and caudate nucleus, respectively.

203 mate dorsal striatum, within the putamen and caudate nucleus, signal the uncertainty of object-reward

204 The effect was specific to caudate nucleus, where growth rate was doubled.

205 d via a neural representation of risk in the caudate nucleus, whereas the representations of other de

206 ervation selectively localized to the medial caudate nucleus.

207 s, and decreased in the hippocampus, but not caudate nucleus.

208 the prediction of forthcoming demand in the caudate nucleus.

209 mplicated in the pathophysiology of OCD, the caudate nucleus.

210 the cortical regions with connections to the caudate nucleus.

211 us, putamen, pallidum, nucleus accumbens, or caudate nucleus.

212 temporal gyrus, insula, fusiform gyrus, and caudate nucleus.

213 s influencing the volumes of the putamen and caudate nucleus.

214 dorsal striatum, including activation in the caudate nucleus.

215 g frontal and parietal cortex, thalamus, and caudate nucleus.

216 lus, the thalamic reticular nucleus, and the caudate nucleus.

217 luding the hippocampus, the thalamus and the caudate nucleus.

218 teral and dorsomedial prefrontal cortex, and caudate nucleus.

219 did not differ across species in the medial caudate nucleus.

220 rom -0.13 in the dorsal raphe to 0.88 in the caudate nucleus.

221 involved in reward processing, including the caudate, nucleus accumbens, amygdala, anterior insula, a

222 ls, also exhibited significantly potentiated caudate, nucleus accumbens, and putamen activations and

223 was investigated in three striatal regions: caudate, nucleus accumbens, and putamen.

224 error regression within the caudate, ventral caudate/nucleus accumbens, and anterior and posterior in

225 mPFC and, for the first time, in the dorsal caudate of antipsychotic-naive patients with FEP, which

226 ere found both in the mPFC and in the dorsal caudate of patients with FEP compared with healthy contr

227 y elevated GPC+PC levels in ACC, putamen and caudate of RC BD-I patients compared to healthy controls

228 R=0.97, p=0.05), a greater decrease in right caudate (OR=4.03, p=0.01) and mean striatal (OR=6.90, p=

229 The TSPO VT was measured in the dorsal caudate, orbitofrontal cortex, thalamus, ventral striatu

230 ne whether TSPO VT is elevated in the dorsal caudate, orbitofrontal cortex, thalamus, ventral striatu

231 showed that older age was linked to smaller caudate (P < .001) and putamen (P = .01) volumes (both c

232 ontal cortex (P < .05) and hypoactivation in caudate (P < .01) across aggregated tasks; hyperactivati

233 s showed lower mean (11)C-IMA107 BPND in the caudate (P < 0.001), putamen (P < 0.001) and globus pall

234 , no dopamine increases were observed in the caudate (p = 0.1) or putamen (p = 0.8) following methylp

235 rable atrophy, particularly in the thalamus, caudate, pallidum and putamen, and this was most apparen

236 middle frontal gyrus, anterior cingulate and caudate parcellations and with white matter lesion volum

237 ing the mPFC and a second region, the dorsal caudate, patients with FEP were treated with oral risper

238 all ps < .05) lower in SCZs in the amygdala, caudate, posterior cingulate cortex, hippocampus, hypoth

239 Greater caudate prediction error response when underweight was a

240 nsitivity correlated positively with ventral caudate prediction error response.

241 ns or in healthy subjects: a deactivation of caudate-prefrontal circuits accompanied by hyperactivati

242 distribution volume ratio with respect to a caudate pseudo-reference region.

243 underlying basis for differences between the caudate putamen (CPu) and nucleus accumbens (NAc).

244 e effect of NBQX (0, 0.3 mug/0.3 mul) in the caudate putamen (CPu) on CS responding in the non-alcoho

245 describe pharmacokinetic-occupancy curves in caudate, putamen and thalamus.

246 increases and decreases in subregions of the caudate, putamen, and hippocampus in 22q-dup relative to

247 ssexuals increased SERT binding in amygdala, caudate, putamen, and median raphe nucleus.

248 l D2R availability compared with nonsmokers (caudate, putamen, and ventral striatum) and with ex-smok

249 any of the brain regions studied (thalamus, caudate, putamen, nucleus accumbens, globus pallidus, an

250 as intracranial volume, but larger bilateral caudate, putamen, pallidum and lateral ventricle volumes

251 olume and surface-based shape metrics of the caudate, putamen, pallidum, and nucleus accumbens in 53

252 Regions of interest for the caudate, putamen, ventral striatum, SN, and cerebellum w

253 Regions of interest for the caudate, putamen, ventral striatum, substantia nigra (SN

254 ctive interfering particles (DIPs), into the caudate-putamen (CP) and scored for an innate immune res

255 Optogenetic stimulation in the caudate-putamen and neocortex of "histaminergic" axonal

256 ) release in the nucleus accumbens (NAc) and caudate-putamen through an indirect mechanism that invol

257 cortical brain regions (cortex, hippocampus, caudate-putamen, nucleus accumbens, thalamus, and hypoth

258 nked as follows: cingulate cortex > insula > caudate/putamen > frontal cortex > temporal cortex > tha

259 he amygdala, and decreased metabolism in the caudate/putamen and medial geniculate nucleus.

260 s treated with methadone exhibited increased caudate/putamen metabolism, whereas buprenorphine produc

261 croglial activation and apoptosis, including caudate/putamen, white matter, and, in juvenile-onset ca

262 ity (r=-0.289, p=0.0264), and brain atrophy (caudate r=0.178, p=0.0087; whole-brain r=0.602, p<0.0001

263 rrelated with lower (11)C-IMA107 BPND in the caudate (r = -0.54; P = 0.011), putamen (r = -0.48; P =

264 rrelated with lower (11)C-IMA107 BPND in the caudate (r = -0.65; P = 0.005), putamen (r = -0.51; P =

265 rrelated with lower (11)C-IMA107 BPND in the caudate (r = -0.73; P = 0.031) and putamen (r = -0.74; P

266 These two caudate regions encode the reward values of visual objec

267 e flexible and stable learning/memory in the caudate regions.

268 density, show a positive association between caudate response and weight change.

269 possessing the TaqIA A2/A2 allele, but lower caudate response predicted body fat gain for adolescents

270 Replicating an earlier finding, elevated caudate response to milkshake receipt predicted body fat

271 = 2.7), similarly moderated the relation of caudate response to milkshake receipt to future body fat

272 The rostromedial caudate (rmCD) of primates is thought to contribute to r

273 The caudate's prediction of control demand subsequently guid

274 Consistent with this functional link, the caudate showed a higher response to scenes compared with

275 eries of fMRI experiments, we found that the caudate showed a stronger functional connection to parah

276 iatum, specifically the ventral striatum and caudate, striatal nodes implicated in motivational goal-

277 ed two pathways originating from the primate caudate tail (CDt).

278 e head circuit and automatic saccades by the caudate tail circuit.

279 p showed a Task X Fatigue interaction in the caudate tail resulting from a positive correlation betwe

280 to the caudal-lateral SNc and project to the caudate tail, which encodes long-term value memories of

281 te head and stable (long-term) values by the caudate tail.

282 ponents of cortical-subcortical circuits-the caudate, thalamic, striatal, orbitofrontal, anterior cin

283 omparisons, in interconnected regions of the caudate, thalamus and right orbitofrontal cortex.

284 ogressive supranuclear palsy in the putamen, caudate, thalamus, and vermis, and decreased in the supe

285 r density of calretinin+ interneurons in the caudate that was driven by loss of small calretinin+ neu

286 significantly dysregulated modules in the HD caudate, the most prominently affected brain region, and

287 n activity in three regions of interest (the caudate, the putamen, and the medial orbitofrontal corte

288 In the caudate, the reduction in BOLD signal was driven specifi

289 VT) (mLcm(-3)) values ranged from 1.5 in the caudate to 11 in the cerebellum.

290 Functional connectivity of the right caudate to the left dorsolateral prefrontal cortex was n

291 airment (age, UPSIT, RBDSQ, CSF Abeta42, and caudate uptake on DAT imaging) allowed prediction of cog

292 ) for prediction error regression within the caudate, ventral caudate/nucleus accumbens, and anterior

293 h-analysis model indicated that the parental caudate-vmPFC connectivity in infancy predicted lower ch

294 table coparental behavioral styles; stronger caudate-vmPFC connectivity was associated with more coll

295 ens volume and local changes in pallidum and caudate volume occurred in patients defined as treatment

296 antipsychotic medication users showed larger caudate volumes in MDD patients compared with controls.

297 The magnitude of response in the caudate was positively correlated with a successful outc

298 h was associated with hyperactivation in the caudate, was observed.

299 Because we had prior hypotheses about the caudate, we performed a confirmatory analysis of a separ

300 ished findings, the VS and adjacent anterior caudate were associated with evaluating the value of rew