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1 gyrii, as well as the anterior cingulate and caudate nucleus.
2 lications to the whole striatum, putamen, or caudate nucleus.
3 ted in reversal learning, such as the medial caudate nucleus.
4 me in insular cortex and increased volume of caudate nucleus.
5 teral and dorsomedial prefrontal cortex, and caudate nucleus.
6  cortices, with mild involvement also of the caudate nucleus.
7  right lentiform nucleus and extended to the caudate nucleus.
8 atients, mainly in the nucleus accumbens and caudate nucleus.
9 thway might elicit neuritic outgrowth to the caudate nucleus.
10 allele in the left anterior insula and right caudate nucleus.
11 odulating neural signals in the amygdala and caudate nucleus.
12  did not differ across species in the medial caudate nucleus.
13 eral nucleus of the amygdala and tail of the caudate nucleus.
14 ergic modulation of neuronal activity in the caudate nucleus.
15 arison subjects in metabolic activity in the caudate nucleus.
16 the prefrontal and parietal cortices and the caudate nucleus.
17 es reached only marginal significance in the caudate nucleus.
18 rom -0.13 in the dorsal raphe to 0.88 in the caudate nucleus.
19 en, smaller in anterior putamen, smallest in caudate nucleus.
20 and in volumes of the right frontal lobe and caudate nucleus.
21 ffective connection from ventral midbrain to caudate nucleus.
22 d uptake are similar for the putamen and the caudate nucleus.
23 jects with single-voxel (1)H-MRS of the left caudate nucleus.
24 medication, however, affects the size of the caudate nucleus.
25 zophrenia suggest high choline levels in the caudate nucleus.
26 ippocampus, mammillary bodies, thalamus, and caudate nucleus.
27 ervation selectively localized to the medial caudate nucleus.
28 s, and decreased in the hippocampus, but not caudate nucleus.
29  the prediction of forthcoming demand in the caudate nucleus.
30 mplicated in the pathophysiology of OCD, the caudate nucleus.
31 the cortical regions with connections to the caudate nucleus.
32 us, putamen, pallidum, nucleus accumbens, or caudate nucleus.
33  temporal gyrus, insula, fusiform gyrus, and caudate nucleus.
34 s influencing the volumes of the putamen and caudate nucleus.
35 dorsal striatum, including activation in the caudate nucleus.
36 g frontal and parietal cortex, thalamus, and caudate nucleus.
37 lus, the thalamic reticular nucleus, and the caudate nucleus.
38 luding the hippocampus, the thalamus and the caudate nucleus.
39 upregulation of dopamine (DA) release in the caudate nucleus.
40 11-labeled [11C]PMP acetylcholinesterase and caudate nucleus [11C]DTBZ monoaminergic positron-emissio
41 36%), left posterior putamen (18%), and left caudate nucleus (12%) of the patients than in the compar
42         There was mild hypometabolism in the caudate nucleus (-8.4% vs. control, F((2,22)) = 3.2, P <
43 usion within and outside MCA territory, e.g. caudate nucleus (9+/-6 ml per 100 g per min at 60 min vs
44  extent of signal reduction in the bilateral caudate, nucleus accumbens and midbrain correlating with
45 o sucrose in dopamine-related brain regions (caudate, nucleus accumbens) were large in older adults.
46 involved in reward processing, including the caudate, nucleus accumbens, amygdala, anterior insula, a
47 ls, also exhibited significantly potentiated caudate, nucleus accumbens, and putamen activations and
48  was investigated in three striatal regions: caudate, nucleus accumbens, and putamen.
49  not smoke, particularly in the left ventral caudate/nucleus accumbens and left ventral putamen (rang
50               An overall decrease in ventral caudate/nucleus accumbens binding potential in those who
51 error regression within the caudate, ventral caudate/nucleus accumbens, and anterior and posterior in
52 tchoice changes in preference are tracked in caudate nucleus activity.
53 f synchronously generated S cells within the caudate nucleus adjoining the ganglionic eminence, poten
54          We found that spike activity in the caudate nucleus after each trial corresponded to an inte
55                                   The medial caudate nucleus also shows hyperactivity in humans lacki
56  an increase in tonic dopamine levels in the caudate nucleus and a corresponding reduction in D2/D3 r
57 s with schizophrenia would display preserved caudate nucleus and abnormal prefrontal cortex activatio
58  two bilateral convergence zones (one in the caudate nucleus and another in the putamen) that consist
59 ural representation of aversive value in the caudate nucleus and anterior cingulate cortex.
60 ividuals exhibit abnormal development of the caudate nucleus and associative cortical areas, suggesti
61 tivity between the right DLPFC and the right caudate nucleus and bilateral (para)cingulate gyrus incr
62 volumetric differences in the frontal lobes, caudate nucleus and cerebellum, and hyperactivity was re
63 lored in the major functional domains of the caudate nucleus and compared with the putamen, using fas
64 ed away from the injection tract towards the caudate nucleus and corpus callosum.
65                                          The caudate nucleus and cortical regions with connections to
66 tients with PD showed less activation in the caudate nucleus and greater activation in a region of pr
67 Furthermore, increased activity in bilateral caudate nucleus and hippocampus for the cued relative to
68          Functional connectivity between the caudate nucleus and hippocampus was also increased after
69 ned period, volume reduction occurred in the caudate nucleus and hippocampus, but iron content increa
70 tal cortex bilaterally, the head of the left caudate nucleus and in bilateral putamen.
71 entified within the peri-infarct zone in the caudate nucleus and in ipsilateral cingulate cortex (pos
72 m contiguous slices were used to measure the caudate nucleus and lateral ventricles in 15 right-hande
73 occipital-parietal lobes, but a larger right caudate nucleus and lateral ventricles.
74 tigate the contributions of the hippocampus, caudate nucleus and midbrain dopaminergic system to diff
75 ly with activity in the body and tail of the caudate nucleus and negatively with activity in the hipp
76 iatal D(2)/D(3) receptor availability in the caudate nucleus and nucleus accumbens that reached stati
77 l activity in the inferior frontal gyrus and caudate nucleus and performance of a go/nogo task in par
78 (p<0.05; corrected) abnormalities within the caudate nucleus and planum temporale.
79 es striatal-frontal connectivity between the caudate nucleus and prefrontal cortex during motor timin
80    While diffuse plaques, including those in caudate nucleus and presubiculum, were less prominently
81 ping (P < .05), and group differences in the caudate nucleus and pulvinar thalamus, compared with con
82 , AcH3 and AcH4 expression from cells in the caudate nucleus and Purkinje cells of the cerebellum in
83 n methamphetamine users from controls in the caudate nucleus and putamen and higher D1-receptor densi
84 ificantly increased, up to 130% in the right caudate nucleus and putamen and up to 116% in both the r
85                                          The caudate nucleus and putamen exhibited the highest uptake
86 D was positively correlated with BPND in the caudate nucleus and putamen in nonsmokers and female smo
87 r DAT nor D2/D3 receptor availability in the caudate nucleus and putamen was predictive of social ran
88 based methods in both the DAT-rich striatum (caudate nucleus and putamen) and the SERT-rich extrastri
89 ce of multipolar ChAT-ir interneurons in the caudate nucleus and putamen, whereas monkeys have a more
90 utamen, and long finger-like branches in the caudate nucleus and putamen.
91                                     The left caudate nucleus and right cerebellum were more active in
92 d to D(2)/D(3) receptor availability in left caudate nucleus and right lateral putamen/claustrum (p <
93 e experiments indicate a causal role for the caudate nucleus and thalamus in automatic inhibitory mot
94                                              Caudate nucleus and thalamus volumes were unaffected by
95  left prefrontal cortex, anterior cingulate, caudate nucleus and thalamus, and recruited more areas,
96                               We found both, caudate nucleus and the hippocampus to be activated by p
97 between the connectivity profile between the caudate nucleus and the lateral prefrontal cortex and di
98 significant linear effects of the ARs in the caudate nucleus and the orbitofrontal cortex for all of
99 in the anterior cingulate gyrus, head of the caudate nucleus and the posterolateral orbitofrontal cor
100  intrastriatal cell bodies were found in the caudate nucleus and the precommissural putamen, consider
101         Abnormalities were also found in the caudate nucleus and the putamen.
102 osis, mediated by aberrant activation of the caudate nucleus and the temporo-parietal junction.
103 i bilaterally (L>R), anterior cingulate, and caudate nucleus and the temporo-parietal-occipital junct
104 tington's disease, HD), the relevance of the caudate nucleus and VCSL on cortical visual processing i
105 la (P = .007), and rostral head of the right caudate nucleus and ventral putamen and globus pallidus
106 -level-dependent response differences in the caudate nucleus and ventral striatum during reward antic
107    Here, we show that neurons in the primate caudate nucleus and ventral striatum modulate their acti
108 d by prior-enhanced connectivity between the caudate nucleus and ventrolateral prefrontal cortex, whi
109                 Presupplementary motor area, caudate nucleus, and anterior insula activation was asso
110 gered endogenous opioid release in thalamus, caudate nucleus, and anterior insula.
111 icant decreases in the mediodorsal thalamus, caudate nucleus, and cerebellum as well as increases in
112  binding sites in the hippocampus, thalamus, caudate nucleus, and cerebellum but not in the corpus ca
113 eceptors, such as the hippocampus, thalamus, caudate nucleus, and cerebellum.
114 putamen, internal capsule, external capsule, caudate nucleus, and globus pallidus.
115 parison of transcriptomes from frontal pole, caudate nucleus, and hippocampus of multiple adult human
116 insula, cuneus, fusiform gyrus, thalamus and caudate nucleus, and increased GM predominantly in tempo
117 ppocampi, temporal and parietal lobes, right caudate nucleus, and insulae in patients with MCI to AD
118 f gray matter (cortex, thalamus, head of the caudate nucleus, and lentiform nucleus).
119 e anterior cingulate cortex, the head of the caudate nucleus, and medial temporal lobe structures.
120 subiculum, hippocampus, hypothalamus, medial caudate nucleus, and nucleus accumbens as well as the ma
121 amine into synaptosomes from frontal cortex, caudate nucleus, and nucleus accumbens from wild-type, N
122 r dopamine release in the nucleus accumbens, caudate nucleus, and putamen among Ser23 carriers, after
123 onoamine levels in the orbitofrontal cortex, caudate nucleus, and putamen could explain variance in b
124 ncreased in the ipsilateral globus pallidus, caudate nucleus, and putamen.
125  nucleus accumbens, the rostral ventromedial caudate nucleus, and rostral ventromedial putamen.
126 gulate cortex (PCC), superior frontal gyrus, caudate nucleus, and substantia nigra.
127 ation (MFC) in the globus pallidus, putamen, caudate nucleus, and thalamus for 22 patients with ADHD
128  recruitment of the superior temporal gyrus, caudate nucleus, and thalamus.
129 sts between the caudal two-thirds of Pf, the caudate nucleus, and the SNr.
130 dial pain system (medial thalamus, amygdala, caudate nucleus, anterior cingulate gyrus, insular corte
131       (11)C-DTBZ binding was measured in the caudate nucleus, anterior putamen, and posterior putamen
132 les of the serotonin and dopamine within the caudate nucleus are poorly understood, this study compar
133                     The results revealed the caudate nucleus as the key brain structure involved in s
134 er, model-based fMRI analyses identified the caudate nucleus as the key structure involved in selecti
135 rward planning are expressed in the anterior caudate nucleus as values of individual branching steps
136                   Higher iron content in the caudate nucleus at baseline predicted lesser improvement
137 measurable effects after injections into the caudate nucleus, basolateral amygdala, hippocampus, vent
138 er an ideal group for the measurement of the caudate nucleus because they may be genetically related
139 tral (head) and caudal (tail) regions of the caudate nucleus, both of which target the superior colli
140 d (1.0 mg/kg, i.v.) dopamine overflow in the caudate nucleus but not in the nucleus accumbens.
141 n between prefrontal cortex and ventromedial caudate nucleus (CAUD) activity] suggest that the post-a
142 nglia circuit through the tail of the monkey caudate nucleus (CDt) guides such object-directed saccad
143           We now report that the tail of the caudate nucleus (CDt) may serve to control a visuomotor
144 imental, has been applied to the cerebellum, caudate nucleus, centromedian thalamus, anterior thalamu
145            Four brain regions were analyzed: caudate nucleus, cerebellum, prefrontal association cort
146 etabolism in the frontal and temporal lobes, caudate nucleus, cingulate gyrus, and mediodorsal nucleu
147 he striatum, which includes a differentiated caudate nucleus (CN) and the putamen in rabbits.
148 n neurons of the prefrontal cortex (PFC) and caudate nucleus (CN) have not been studied in awake, fre
149 ese experiments was to determine whether the caudate nucleus (CN) plays a role in retrieval of previo
150 ccumbens (NAc), prefrontal cortex (PFC), and caudate nucleus (CN) under general anesthesia.
151 the prefrontal cortex (PFC), the head of the caudate nucleus (CN), and the ventral anterior nucleus (
152 gulate cortex (PCC), upper precuneus (UPCU), caudate nucleus (CN), cingulate motor area (CMA), supple
153 neuronal dysmaturation response to HI in the caudate nucleus (CN), which contributes to motor and cog
154  hippocampus, thalamus, globus pallidus, and caudate nucleus compared with 26 control males (effect s
155 s pallidus, pulvinar thalamus, thalamus, and caudate nucleus, compared with control subjects with R2*
156  found that distinct circuits of the primate caudate nucleus control behavior selectively in the flex
157      Patients' reduced activation within the caudate nucleus correlated negatively with paranoia scor
158 sed connectivity between the DN and the left caudate nucleus could play a role in balance impairment
159  subset of these regions (PCC, thalamus, and caudate nucleus) covaried with the level of arousal.
160 ted from previous studies, activation of the caudate nucleus differentiated between positive and nega
161 lassifier based on the specialization of the caudate nucleus distinguished patients from controls wit
162 n the right ventral tegmental area and right caudate nucleus, dopamine-rich areas associated with mam
163 d both independent cognitive predictions for caudate nucleus dopaminergic (F = 7.25; P = .008) and co
164 ts with neocortical cholinergic deficits had caudate nucleus dopaminergic deficits.
165  </= -2) global cognitive impairment scores, caudate nucleus dopaminergic denervation was relatively
166 pically occurs in the context of significant caudate nucleus dopaminergic denervation.
167 ed changes in neuronal activity occur in the caudate nucleus during trace EBC.
168 ry input structure of the basal ganglia, the caudate nucleus, encode three aspects of decision making
169                      However, neurons in the caudate nucleus encoded the difference in the temporally
170 ntially expressed mRNAs were detected in the caudate nucleus, followed by motor cortex, then cerebell
171 timulations of the hippocampus, amygdala, or caudate nucleus, followed by sacrifice and immunohistoch
172 nctional and effective connectivity of human caudate nucleus following sulpiride treatment, which is
173 s overlooked specialization of the posterior caudate nucleus for executive functions, often considere
174 otonergic degeneration in human ventromedial caudate nucleus from individuals with an APOE epsilon4 a
175 egions (amygdala, anterior cingulate cortex, caudate nucleus, frontal cortex, hippocampus, insula, th
176                                 In contrast, caudate nucleus gene expression is highly conserved.
177 cerebral white matter, hippocampus-amygdala, caudate nucleus, globus pallidus plus putamen, and dienc
178 rtical regions (nucleus accumbens, amygdala, caudate nucleus, globus pallidus, hippocampus, putamen,
179 and cortical regions with connections to the caudate nucleus had markedly abnormal hemispheric specia
180 fronto-striato-thalamic circuits-head of the caudate nucleus (hCaud), putamen, globus pallidus, thala
181  multidimensional discrimination task in the caudate nucleus, hippocampus, anterior and posterior cin
182           Similar results were seen in mouse caudate nucleus homozygous for APOE epsilon4 via targete
183 he DRD1 transcript in DLPFC, hippocampus and caudate nucleus in a large cohort of subjects (~700), in
184 , was markedly elevated in the neocortex and caudate nucleus in HD patients.
185 a2+ signaling and degeneration of MSN in the caudate nucleus in HD.
186 ebellum (granule cell layer), as well as the caudate nucleus in humans and chimpanzees.
187 crease in dopamine innervation of the medial caudate nucleus in humans is a species-typical character
188 metry revealed atrophy of the cerebellum and caudate nucleus in manifesting patients (P = 0.04 and 0.
189         We recorded neuronal activity in the caudate nucleus in monkeys during categorization behavio
190 al ganglia, we recorded from the FEF and the caudate nucleus in monkeys performing an asymmetrically
191 re centred on the left frontal operculum and caudate nucleus in non-fluent primary progressive aphasi
192 e medial nucleus accumbens shell (NAcSh) and caudate nucleus in postmortem human brains.
193 ese results underscore the importance of the caudate nucleus in relation to cognitive fatigue.
194 ontrols, and was significantly higher in the caudate nucleus in secondary progressive MS (12.9/s vs 1
195 icantly lower bilaterally in the head of the caudate nucleus in the group with diabetes and depressio
196 eral and inferolateral prefrontal cortex and caudate nucleus in the Tourette group relative to compar
197 n interaction between cortical areas and the caudate nucleus, in which the caudate body supports both
198 at electrical microstimulation in the monkey caudate nucleus influences both choice and saccade respo
199 ing in functional connectivity involving the caudate nucleus, insula, medial prefrontal cortex and ot
200 tex, and midcingulate cortex, as well as the caudate nucleus involved in the reward system.
201 nd was associated with a reduced dentate and caudate nucleus iron content compared to placebo.
202                                          The caudate nucleus is a part of the visual corticostriatal
203                                          The caudate nucleus is commonly active when learning relatio
204                                 The anterior caudate nucleus is essential for goal-directed behavior
205                                          The caudate nucleus is known to have a role in behavioral mo
206 has demonstrated that neural activity in the caudate nucleus is modulated by task-relevant action val
207  No significant changes were detected in the caudate nucleus (lateral or medial) or in the cingulate,
208 te matter, cortex, cerebellum, thalamus, and caudate nucleus, many of which could compromise the cont
209  that focal biophysical abnormalities in the caudate nucleus may be central to the pathophysiology of
210                                          The caudate nucleus may be involved in the repetitive moveme
211 components of these circuits, such as in the caudate nucleus, may result in neurobehavioral syndromes
212     Rats were then given hippocampus, medial caudate nucleus (MCN), or cortical control lesions.
213 urves showed high accumulation in the insula/caudate nucleus, moderate uptake in the thalamus, and th
214 = 10), superior cerebellar peduncle (n = 7), caudate nucleus (n = 4), whole thalamus (n = 3), and put
215 low in the nucleus accumbens (n = 4) and the caudate nucleus (n = 5) using in vivo microdialysis.
216  dopaminergic compartment of the putamen and caudate nucleus not only in manifest SCA17 patients (P =
217 g lasting memory, including the hippocampus, caudate nucleus, nucleus basalis and cortex.
218    Robust and differential activation of the caudate nucleus occurred only when a perception of conti
219 neurons, were increased significantly in the caudate nucleus of CR monkeys, suggesting a role for gli
220 tracranial administration of MV-CEA into the caudate nucleus of Ifnar(ko) CD46 Ge did not result in c
221 on of the BG by muscimol injections into the caudate nucleus of monkeys and assessing behavior of Par
222 s in the lateral prefrontal cortex (PFC) and caudate nucleus of monkeys as they performed a trial-and
223  indwelling cannula implanted into the right caudate nucleus of normal rats or i.t. in rats bearing e
224 striatal (123)I-FP-CIT binding ratios in the caudate nucleus of PSP patients than in that of both PD
225 striatal (123)I-FP-CIT binding ratios in the caudate nucleus of PSP patients than in that of both PD
226 e of Neuron, demonstrate that neurons in the caudate nucleus of the basal ganglia signal which dimens
227  and white matter for the 4 major lobes, and caudate nucleus of the brain were compared in patients a
228  specific bottom-up cues, and they place the caudate nucleus of the dorsal striatum at the center of
229 d human lymphoblastoid B-cell lines into the caudate nucleus of the nude rat resulted in lethal CNS t
230 omatic L1 insertions, in the hippocampus and caudate nucleus of three individuals.
231  300 single neurons from cerebral cortex and caudate nucleus of three normal individuals, recovering
232 ution of visual corticostriatal loop and the caudate nucleus on generating selective response within
233 paminergic depletions of the marmoset medial caudate nucleus on serial discrimination reversal learni
234 re was no evidence of altered variability of caudate nucleus or frontal lobe volumes.
235 re was no evidence of altered mean volume of caudate nucleus or putamen.
236 left, but the opposite pattern in the right, caudate nucleus (P < .001).
237 han did control subjects (putamen, P = .012; caudate nucleus, P = .008; thalamus, P = .012) and psych
238 -medicated ADHD patients (putamen, P = .006; caudate nucleus, P = .010; thalamus, P = .021).
239 pretreatment glucose metabolism in the right caudate nucleus (partial r=-0.53), improvement of major
240 rtex, and right inferior parietal lobule and caudate nucleus, perhaps reflecting conflict experienced
241 te cortex and between the right amygdala and caudate nucleus predicted the magnitude of reduction in
242                                    Thus, the caudate nucleus provides interpretive monitoring of ongo
243                               Volumes of the caudate nucleus, putamen, and globus pallidus and gray a
244      Differences were detected in the dorsal caudate nucleus, putamen, and globus pallidus but the ob
245 wer necrotic lesions in the cerebral cortex, caudate nucleus, putamen, and in hippocampal sectors CA1
246 served in the amygdala, raphe nuclei region, caudate nucleus, putamen, hippocampus, and anterior cing
247  two groups did not differ in volumes of the caudate nucleus, putamen, or frontal white matter region
248 inding potential in the raphe nuclei region, caudate nucleus, putamen, thalamus, and insula cortex (P
249 al, occipital, parietal, and temporal lobes; caudate nucleus; putamen; and thalamus.
250 d in the substantia nigra (SNc), dentate and caudate nucleus, red nucleus, putamen and globus pallidu
251 n and/or 6-hydroxydopamine (6-OHDA) into the caudate nucleus, respectively, modeling the lesions seen
252 ratios were 0.97 and 0.76 in the putamen and caudate nucleus, respectively.
253 ori region of interest analysis of the right caudate nucleus, right temporo-parietal junction and med
254 significant metabolic decreases in the right caudate nucleus, right ventrolateral prefrontal cortex (
255  have revealed that the head and tail of the caudate nucleus selectively and differentially process f
256    However the lateral prefrontal cortex and caudate nucleus showed a non-linear U-shape relationship
257 mate dorsal striatum, within the putamen and caudate nucleus, signal the uncertainty of object-reward
258 tly, mitochondrial PCR array profiling in HD caudate nucleus specimens showed increased mRNA expressi
259 ty in the ventral prefrontal cortex, ventral caudate nucleus, thalamus, and hippocampus.
260 amo-cortical network comprising the putamen, caudate nucleus, thalamus, supplementary motor area, pre
261 m disorders had greater volumes of the right caudate nucleus than comparison subjects as well as a re
262 ofrontal cortex, anterior temporal lobe, and caudate nucleus than PCA, and PCA showed more asymmetric
263       Here we identify neurons in the monkey caudate nucleus that create a spatially selective respon
264             Activity was also present in the caudate nucleus, the thalamus, and the cerebellum.
265                                      For the caudate nucleus, there was a significant game x group in
266 radient of increasing Ki values from head of caudate nucleus to rostral putamen to caudal putamen.
267 ns known to track expected value such as the caudate nucleus, together with anterior cingulate cortic
268 itry that includes the prefrontal cortex and caudate nucleus, two regions that show prominent changes
269 s were found for the target regions putamen, caudate nucleus, ventral striatum, and substantia nigra
270 9 for the respective target regions putamen, caudate nucleus, ventral striatum, and substantia nigra,
271         Volumes of interest for the putamen, caudate nucleus, ventral striatum, substantia nigra, tha
272      The 2T model VT values for the putamen, caudate nucleus, ventral striatum, substantia nigra, tha
273 nd significant, inverse correlations between caudate nucleus volume and the severity of perseveration
274     Clozapine treatment led to reductions in caudate nucleus volume in three separate studies.
275  are consistent with the findings of reduced caudate nucleus volume reported in studies of neurolepti
276                                              Caudate nucleus volume was unrelated to BDNF levels or s
277 ute (13.1%, 13.2%) and relative (9.1%, 9.2%) caudate nucleus volumes in never-medicated subjects with
278                                      Reduced caudate nucleus volumes may be a good candidate marker f
279                                              Caudate nucleus volumes were initially abnormal for pati
280                                              Caudate nucleus volumes were significantly (P =.008) sma
281                                          The caudate nucleus was also segmented and examined in relat
282 alysis established that activity in the left caudate nucleus was associated with increased activity i
283 nal capsule adjacent to the head of the left caudate nucleus was found in PD-ICB, but not surviving c
284                   Functional connectivity of caudate nucleus was modulated specifically by dopaminerg
285                                          The caudate nucleus was reduced only after symptomatic onset
286 ents, the decreased connectivity in the left caudate nucleus was related with worse balance performan
287                            The volume of the caudate nucleus was significantly correlated with the pe
288  our study, the MTR in the head of the right caudate nucleus was significantly lower in the MDD group
289 al ventromedial prefrontal regions and right caudate nucleus (washing); putamen/globus pallidus, thal
290                  FA in the SN and CBF in the caudate nucleus were inversely correlated with motor dys
291  putamen in both genders and the putamen and caudate nucleus were significantly smaller in older than
292 opaminergic system, the hippocampus, and the caudate nucleus were to different degrees significantly
293                   The effect was specific to caudate nucleus, where growth rate was doubled.
294 d via a neural representation of risk in the caudate nucleus, whereas the representations of other de
295 uent trial in both prefrontal cortex and the caudate nucleus which is correlated with behavioral adju
296  We recorded neural activity in the anterior caudate nucleus while monkeys made saccades to multiple
297 corded from phasically active neurons in the caudate nucleus while monkeys performed a probabilistica
298  in dopamine transmission in the left medial caudate nucleus while simultaneously producing significa
299 he association of intrinsic pathology in the caudate nucleus with abnormalities in working memory in
300                                       In the caudate nucleus with increased BMI we saw the upregulati

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