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   1 ddition, disruption of a conserved structure proximal to A1 caused a 10-fold reduction in all transcripts that utilized A
     2 ts in the pristine forest interior, and (iii) nest building caused a 62% decline in forest tree sapling density over the 
  
  
  
  
     7 n contrast, treatment of macrophages with recombinant Wnt5a caused a decrease in the load of antimony-sensitive and -resi
     8                   It was also observed that ion irradiation caused a decrease in the Si-O-Si bond angle of silica, simila
  
  
  
  
  
    14 s, L-AP4 slightly reduced cone ICa ( approximately 10%) and caused a larger reduction in cone-driven EPSCs ( approximatel
    15 ctivation in conjunction with response-outcome conditioning caused a later decay in goal-directed responding-that is, mic
  
  
    18 m of a patient with a V262F missense mutation in Eda, which caused a milder form of X-linked HED (XLHED), contained low l
    19 eltaK1L is the first vaccinia virus construct reported that caused a muted innate immune gene expression profile and decr
  
  
    22  gene in astrocytes, or in astrocytes and oligodendrocytes, caused a persistent hypomyelination, as did deletion from ast
  
    24                  However, repeated 15 min daily stimulation caused a persistent increase in spontaneous locomotion to eme
    25                   The deletion of genes from the wca operon caused a persistent-infection E. coli strain to become sensit
    26 served in young lean CT-1(-/-) mice (2 mo), CT-1 deficiency caused a phase shift of the acrophase.                       
    27 HK4, a key regulator of the pyruvate dehydrogenase complex, caused a profound cell growth inhibition in tumour cells harb
    28 at both the in vitro digestion and the colonic fermentation caused a pronounced decrease in 3,5-O-dicaffeoylquinic acid a
    29 en WT mice were treated with a CB2-specific antagonist that caused a pronounced inhibition of HDM-induced airway inflamma
    30              Exposure to 13 physicochemically distinct ENMs caused a range of mast degranulation responses, with smaller 
    31 hat in vivo inhibition using beta-aminopropionitrile (BAPN) caused a rapid, global regression of the entire network, with
  
  
  
    35  of hypertrophied myocytes to the Orai channel blocker BTP2 caused a reduction of AP duration and reduced diastolic Ca(2+
  
    37 oft agar assay, treatment of HC11-Int3 cells with P50-siRNA caused a significant decrease in colony formation.           
    38 the agent inhibited pERK1/2 phosphorylation in all tumours, caused a significant decrease in proliferation in 5/23 (22%) 
    39              Moreover, both neonatal and adult THS exposure caused a significant increase in percentage of B-cells and si
    40  activities (V max ) but short-term drying and waterlogging caused a significant increase of dissolved organic carbon (DO
    41                      Mutations P79R, C231Y, G483R and M705V caused a significant reduction in glutamate and glycine agoni
    42            In agreement with these results, USP39 depletion caused a significant reduction in pre-mRNA splicing efficienc
  
    44   Removing the branched network junctions from the analysis caused a slight increase in the average persistence length to
    45 technology or silencing of the polyadenylation factor CPSF1 caused a splice switch that inhibited expression of AR varian
    46 nt treatment of PHHs with IFN-lambda4, but not IFN-lambda3, caused a strong and sustained induction of SOCS1 and refracto
  
    48 tion, the addition of palmitic, stearic or eicosanoic acids caused a transition from a one-step to two-step process.     
    49 isruption of Met (Lgr5(Creert2)/Met(fl/fl)/Apc(fl/fl) mice) caused a twofold increase in apoptosis in microadenomas, resu
  
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