ライフサイエンスコーパス: caval

1 portal-caval, and 5 H-shaped (H-type portal-caval)], 2 had portal-to-hepatic vein shunts (portohepat

2 unt [subdivided in 5 end-to-side-like portal-caval, 7 side-to-side-like portal-caval, and 5 H-shaped

3 The wide-open triangular caval anastomosis is easy to perform, allowing short imp

4 VC) stenosis in a patient with a "piggyback" caval anastomosis.

5 Extracorporeal bypass, including both caval and portal venous return, produced significant inc

6 Caval and pulmonary veins were occluded.

7 ike portal-caval, 7 side-to-side-like portal-caval, and 5 H-shaped (H-type portal-caval)], 2 had port

8 stance, and lowering cerebral, superior vena caval, and pulmonary blood flows.

9 This study describes the first use of caval-aortic access and closure to enable transcatheter

10 Caval-aortic access and tract closure were successful in

11 Caval-aortic access has been successful in animals.

12 Caval-aortic access refers to percutaneous entry into th

13 ibitive-risk patients who underwent TAVR via caval-aortic access.

14 There were no deaths attributable to caval-aortic access.

15 January 2014, 19 patients underwent TAVR via caval-aortic access; 79% were women.

16 witching from an inferior to a superior vena caval approach; 5) use of a 60-cm guiding sheath; 6) det

17 ity pressure during transient inferior, vena caval balloon occlusions.

18 was established postasystole via aortic and caval cannulation and maintained for 2 h.

19 ovenous bypass, portocaval decompression, or caval clamping in 11 recipients and describe the modific

20 ovenous bypass, portocaval decompression, or caval clamping.

21 In the present models, the caval connections were offset through a range of 0.0 to

22 Chronic thoracic inferior vena caval constriction (TIVCC) is a model of reduced cardiac

23 unloading produced by thoracic inferior vena caval constriction (TIVCC).

24 Fontan physiology includes knowledge of the caval contributions to right (RPA) and left (LPA) pulmon

25 cipal diagnosis of proximal or inferior vena caval deep vein thrombosis and treated with CDT from 200

26 donor vena cava was too short to bridge the caval defect for interposition.

27 ight ventricular function, and inferior vena caval diameters.

28 ing the technique of liver resection without caval excision (the piggyback technique).

29 nt percutaneous placement of a superior vena caval filter for prevention of PE.

30 ere was a fivefold increase in the number of caval filter implants.

31 Except for one randomized trial, the vena caval filter literature consists of case series or conse

32 Eleven patients received anticoagulants or caval filter placement as a result of CT findings.

33 Inferior vena caval filters (IVCFs) may prevent recurrent pulmonary em

34 ary therapy for venous thromboembolism, vena caval filters are an important alternative when anticoag

35 ndications to anticoagulation, inferior-vena-caval filters can be considered, but their use needs car

36 to establish the appropriate place for vena caval filters in the treatment of venous thromboembolic

37 Inferior vena caval filters provide protection from life-threatening P

38 Vena caval filters represent a potentially important but poor

39 safety, effectiveness, numbers, and types of caval filters.

40 (POH-DCM); they were compared to VOH (aorta-caval fistula).

41 Myocardial infarcted rats and aorto-caval fistulated rats were used as a low output HF model

42 After the method was validated, caval flow contributions were quantified in patients.

43 imaging enable in vivo quantification of the caval flow distribution to the PAs in patients with Font

44 All patients had persistent aorto-caval flow immediately post-procedure.

45 by using primarily endobronchial forceps for caval fragments and snares for cardiac and pulmonary fra

46 epatic venous gas, and two had inferior vena caval gas.

47 ess to the abdominal aorta by electrifying a caval guidewire and advancing it into a pre-positioned a

48 erations (52%) which were performed with the caval interposition approach to liver transplantation, c

49 gery in managing these complex patients with caval involvement.

50 rterial pressure catheters and inferior vena caval (IVC) occluders; four had placement of thoracic ao

51 studied four dogs before and during inferior caval (IVC) occlusion at five different inotropic stages

52 epatic venous (HV), subhepatic inferior vena caval (IVC), and portal venous (PV) flow rates were meas

53 rior margin of the inferior vena cava (hilar-caval line) on lateral radiographs; this line correspond

54 er (n = 66) or anterior to (n = 2) the hilar-caval line.

55 Using the aorto-caval model of an AV fistula model in the rat, we demons

56 Transient inferior vena caval obstruction was used to determine PV relations.

57 bdominal compression, nitroglycerin, or vena caval obstruction).

58 plotted versus end-diastolic volume during a caval occlusion (preload-independent recruitable systoli

59 Vena caval occlusion (VCO) was used to reduce left ventricula

60 ystolic pressure-volume relationships during caval occlusion and was used as the gold standard of LV

61 iastolic pressure-volume relationship during caval occlusion at baseline, after sildenafil, and BNP i

62 The piggyback technique without caval occlusion is possible in the majority of patients.

63 and outcome of a piggyback technique without caval occlusion or veno-venous bypass (VB), we retrospec

64 ants, and an animal with acute inferior vena caval occlusion to produce portal hypertension.

65 es (strain) from successive diastoles during caval occlusion were used to evaluate LV/RV diastolic me

66 Isolated Pringle maneuver and caval occlusion with Pringle maneuver produced significa

67 t increases in MAP and cardiac output during caval occlusion with Pringle maneuver, while atriocaval

68 after caval occlusion, Pringle maneuver, and caval occlusion with Pringle maneuver.

69 nditions were altered by saline infusion and caval occlusion, and lusitropic state was changed by dob

70 For inferior vena caval occlusion, control biopsy specimens lost 1.23 g, w

71 recorded at baseline and at intervals after caval occlusion, Pringle maneuver, and caval occlusion w

72 ular P/Q, created by transient inferior vena caval occlusion, under basal and endotoxic conditions.

73 modified utilizing fluid administration and caval occlusion, whereas dobutamine and esmolol were use

74 Loading conditions were altered by caval occlusion, whereas lusitropic state was changed by

75 Preload independence during vena caval occlusions was achieved by preload adjustment (1/[

76 esults strongly suggest the incorporation of caval offsets in future total cavopulmonary connections.

77 portal-caval shunts patients have a 1-stage caval partition, and the others have a 1-stage ligation.

78 reas the 5 others had a 1-stage longitudinal caval partition.

79 The first 2 side-to-side-like portal-caval patients had a successful 2-stage closure whereas

80 All 5 end-to-side-like portal-caval patients had a threadlike intrahepatic portal veno

81 vidence of IVC thrombosis, device migration, caval penetration, or pulmonary embolism.

82 ahepatic portosystemic shunts, H-type portal-caval, portohepatic, and patent ductus venosus patients

83 estimate the agreement between superior vena caval pressure (SVCP) and femoroiliac venous pressure (F

84 rterial pressure (AP) and intrathoracic vena caval pressure (VP).

85 here were trends toward higher superior vena caval pressure early after the operation and at follow-u

86 rences between right atrial or inferior vena caval pressures among the groups.

87 od pressure, right atrial, and inferior vena caval pressures were measured continuously.

88 Total caval pulmonary anastomosis was performed in 53 patients

89 an ADV and an accompanying alternative porto-caval shunt between the right portal vein and inferior v

90 120 min (n=8) liver warm ischemia in splenic-caval shunt group survived for over 1 day, 6/8 for over

91 with or without portosystemic shunt (splenic-caval shunt).

92 s produced in 7-day-old rabbits via an aorto-caval shunt, after which, the rabbits were treated with

93 e rats with liver warm ischemia plus splenic-caval shunt.

94 have a 2-stage closure, side-to-side portal-caval shunts patients have a 1-stage caval partition, an

95 es are good provided end-to-side-like portal-caval shunts patients have a 2-stage closure, side-to-si

96 inated contrast-enhanced material showed the caval size to be within 3 mm in all 119 patients.

97 bypass in 11 operations (41%) performed with caval sparing (piggyback) surgical technique.

98 iation, simultaneous arterial, superior vena caval (SsvcO2), and pulmonary venous (SpvO2) oximetry wa

99 nts identified with late portal vein or vena caval stenoses or thromboses from a cohort of 524 grafts

100 Hepatic arterial fraction obtained with caval subtraction agreed well with those with fluorescen

101 strated between TLBF in humans measured with caval subtraction and direct inflow phase-contrast MR im

102 Conclusion Caval subtraction phase-contrast MR imaging is a simple

103 Thereafter, consistency of caval subtraction phase-contrast MR imaging-derived TLBF

104 Purpose To validate caval subtraction two-dimensional (2D) phase-contrast ma

105 Arterial (SaO(2)) and superior vena caval (SvO(2)) co-oximetry and cerebral oxygen saturatio

106 eed in the case of an adult with unsuspected caval system obliteration.

107 according to the ending of the shunt in the caval system.

108 nnels between the superior and inferior vena caval systems after bidirectional cavopulmonary anastomo

109 tions between the superior and inferior vena caval systems were identified and measured.

110 Robotic surgery for selected level I and II caval thrombi is feasible.

111 d SNAD given for 7 days appeared to decrease caval thrombosis in this model of deep vein thrombosis.

112 The prevalence of observed post-filter caval thrombosis was 2.7%.

113 est this hypothesis a model of inferior vena caval thrombosis was used.

114 tal vein thrombosis, and 1 had inferior vena caval thrombosis.

115 A passive splenic and vena caval to jugular vein shunt with systemic heparinization

116 ding quantification of superior and inferior caval, total pulmonary artery, total pulmonary vein, asc

117 ) had complete closure of the residual aorto-caval tract.

118 tricle, atrioventricular septal defects, and caval vein abnormalities.

119 ur lineage analysis unequivocally shows that caval vein and atrial myocardium share a common origin a

120 tracing has suggested a distinct origin for caval vein myocardium, from a proposed third heart field

121 Inferior caval vein thrombosis was induced in cohorts of adult wi

122 caval veins, and a persistent left inferior caval vein.

123 are created by abnormal localization of the caval veins combined with ectopic pericardial cavity for

124 y reduced sinus horn myocardium, hypoplastic caval veins, and a persistent left inferior caval vein.

125 ialization, alignment, and morphology of the caval veins.

126 us horn myocardium, and the alignment of the caval veins.

127 Vena caval venting decreased the release of potassium into th

128 conclude that portal vein flush without vena caval venting provided a lower incidence of PRS than any

129 The patients in groups 3 and 4 (vena caval venting) demonstrated smaller percentage increases

130 oup 4 (n=19), hepatic artery flush with vena caval venting.

131 group 3 (n=29), portal vein flush with vena caval venting; and group 4 (n=19), hepatic artery flush

132 : group 1 (n=31), portal vein flush, no vena caval venting; group 2 (n=21), hepatic arterial flush, n

133 up 2 (n=21), hepatic arterial flush, no vena caval venting; group 3 (n=29), portal vein flush with ve

134 fragments of polydioxanone suture within the caval wall at 32 weeks.