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1 lex ultrasound peak systolic velocity of the cavernosal arteries increased from baseline by 14.4 +/-
2 Relaxation of the smooth muscle cells in the cavernosal arterioles and sinuses results in increased b
3 f both of the cavernous arteries and a right cavernosal artery pseudoaneurysm was detected on angiogr
4 Doppler ultrasonographic examination of the cavernosal artery revealed a 1.5 cm-diameter pseudoaneur
5 1.5 cm-diameter pseudoaneurysm at the right cavernosal artery, together with a high-velocity shunt b
6 was associated with a marked augmentation of cavernosal cGMP levels, suggesting that, although lower
10 se and MYPT-1 phosphorylation at a time when cavernosal eNOS protein, constitutive NOS activity, and
11 mediated penile fibrosis, we purified corpus cavernosal fibroblast cells (CCFCs), the major cell type
12 significant decrease in erectile response to cavernosal nerve stimulation in the STZ-diabetic rat.
13 crease in cavernosal pressure in response to cavernosal nerve stimulation was enhanced in animals tra
14 and often invisible anatomical course of the cavernosal nerves continues to engender innovations in s
16 s, is considered the principle stimulator of cavernosal smooth muscle relaxation, however, the inhibi
17 f penile rehabilitation is to prevent corpus cavernosal smooth muscle structural alterations to not o
18 -galactosidase reporter gene was observed in cavernosal tissue 1 day after intracavernosal administra
19 for the prevention or at least reduction of cavernosal tissue ischemia and potential damage conseque
20 IS grafts (SIS-ADSC) resulted in significant cavernosal tissue preservation and maintained erectile r
21 inase protein and mRNA have been detected in cavernosal tissue, the role of Rho-kinase in the regulat
23 , 18), we examined the role of Rho-kinase in cavernosal tone, based on the hypothesis that antagonism
24 inase pathway may play a synergistic role in cavernosal vasoconstriction to maintain penile flaccidit
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