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1 tudied in vitro using isolated rabbit corpus cavernosum.
2 d the beneficial actions of RSV on aorta and cavernosum.
3 nt relaxation to acetylcholine for aorta and cavernosum.
4 hich causes cGMP to accumulate in the corpus cavernosum.
5 was present in the endothelium of the corpus cavernosum.
6 tric oxide is present in the diabetic corpus cavernosum.
7 r rosuvastatin (RSV) were examined on corpus cavernosum and aorta from streptozotocin-induced diabeti
8 osphate hydrolysis [corrected] in the corpus cavernosum and therefore increases the penile response t
9 O synthase is found in human clitoral corpus cavernosum and vagina, we hypothesized that human argina
10 ents underlie detumescent tone in the corpus cavernosum, and that modulation of this mechanism by the
11 Many tonic SMCs (vascular, airway and corpus cavernosum) are driven by a cytosolic Ca2+ oscillator th
12      A Rho-kinase inhibitor increases corpus cavernosum (CC) pressure in an in vivo rat model suggest
13  but not in rat primary corpus smooth muscle cavernosum cells; and (iv) in situ hybridization of glan
14  and this effect was reproduced in WT corpus cavernosum exposed to NOS inhibitors.
15                      In rabbit penile corpus cavernosum, intracellular cGMP was determined to be 18 +
16 g the eNOS gene (AdCMVeNOS) into the corpora cavernosum of the aged rat.
17 s, including the smooth muscle of the corpus cavernosum of the penis.
18 ed blood flow into the penis, raising corpus cavernosum pressure to culminate in penile erection.
19 sm of Rho-kinase results in increased corpus cavernosum pressure, initiating the erectile response in
20            Relaxation of arterial and corpus cavernosum smooth muscle (CCSM) is necessary to increase
21  membrane currents in isolated rabbit corpus cavernosum smooth muscle cells.
22 bility to NO synthase in human penile corpus cavernosum smooth muscle, the inhibition of human argina
23 oth muscle relaxation in human penile corpus cavernosum tissue that is required for erection.
24 t arginase is present in human penile corpus cavernosum tissue, and that the arginase inhibitor BEC c
25 functional role of BK channels in the corpus cavernosum utilizing a knock-out mouse lacking the Slo g
26                               Relaxations of cavernosum were in the nondiabetic range following preve
27 noncholinergic nerve-mediated relaxations of cavernosum were reduced 25-33% by diabetes.

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