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1 ipal stress is important in the promotion of cavity formation.
2  bacteria is sufficient to trigger localized cavity formation.
3 cal scenarios that could result in localized cavity formation.
4 uent collapse of both neurogenesis and nasal cavity formation.
5 on during occlusion, which is accompanied by cavity formation.
6  spinal cord or brain injury often result in cavity formation.
7 GF receptor and is important in FGF2 central cavity formation.
8 giogenesis diminishes concurrent with cystic cavity formation.
9 solid caseous material and in the subsequent cavity formation.
10 rrier associated with solvent relaxation and cavity formation.
11 aval veins combined with ectopic pericardial cavity formation.
12 es where metal coordination is necessary for cavity formation.
13 ely for the central hydrophobic phenomena of cavity formation and association of inert gas solutes.
14  post-implantation embryos triggers amniotic cavity formation and developmental progression.
15                Hydrophilic surfaces suppress cavity formation and reduce the water-water correlation
16 seous tuberculous lesions and the subsequent cavity formation are probably the most dangerous process
17 al and show simulation results demonstrating cavity formation by iron starvation.
18 f the origin of hydrophobicity, derived from cavity formations by Pratt and Pohorille and Madan and L
19 e carefully designed to evaluate the role of cavity formation/dispersive interaction on the chromatog
20  and Cl(-) in aqueous solutions increase the cavity formation energy cost and the polar interaction e
21 ical model of mushroom-like architecture and cavity formation in Pseudomonas aeruginosa biofilms.
22 vere WMD including rarefaction, necrosis and cavity formation in the corpus callosum, external and in
23                                              Cavity formation in zonal occludens-1-depleted cells is
24 of CPEB or zonal occludens-1 impairs central cavity formation, indicating a loss of cell polarity.
25             Specifically, the probability of cavity formation is enhanced in the vicinity of hydropho
26  kcal/mol, the unfavorable thermodynamics of cavity formation may be the major factor in destabilizin
27              In contrast, the probability of cavity formation or the free energy of binding of hydrop
28 t relative to the energy of the mobile-phase cavity formation process.
29 d RH4 indicates that both steric overlap and cavity formation provide strong driving forces for oligo
30 ts "fatness." In solution, the extra work of cavity formation to accommodate a fluorocarbon, compared
31 t destabilizing, allowing the energy cost of cavity formation to be estimated.
32 halpy of solution and calculated enthalpy of cavity formation using the scaled particle theory.
33 ccessible surface area term, as an index for cavity formation, were correlated with the free energies

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