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1 ence (G0) allows cycling cells to reversibly cease proliferation.
2     Unlike antigen-stimulated T cells, which cease proliferation after a finite number of cell divisi
3                           Normal human cells cease proliferation after a finite number of population
4 ype 1 (HTLV-1)-infected HeLa and SupT1 cells cease proliferation and become senescent immediately aft
5 tum (dpc), the stage in mice when germ cells cease proliferation and begin differentiation into prosp
6  cells in the external granule cell layer to cease proliferation and begin migration to the internal
7 essed at progressively higher levels as NPCs cease proliferation and differentiate into postmitotic n
8 e number of population doublings before they cease proliferation and enter a growth-arrest state term
9 sors only induce SPARC expression after they cease proliferation and migration, and transform into ma
10 n of HPV gene expression causes the cells to cease proliferation and undergo senescence.
11                       The T-cell line Jurkat ceased proliferation and died shortly after virus infect
12                                        Cells ceased proliferation and differentiated when FGF4, hepar
13 keratinocytes increase intracellular Ca(2+), cease proliferation, and differentiate passively in resp
14 n the absence of nos-1 and nos-2, germ cells cease proliferation at the end of the second larval stag
15             Third, cells in the cement gland cease proliferation completely as they begin to accumula
16 RNA is initially detectable when osteoblasts cease proliferation, increasing during differentiation a
17  granulosa cells in p21(Cip1)-deficient mice ceased proliferation similarly to those in wild-type mic
18 tured RB1(-/-) calvarial osteoblasts fail to cease proliferation upon reaching confluence or followin
19 of reporter systems, we show that HeLa cells cease proliferation within one or two division cycles af
20 r transplantation, whereas adult hepatocytes ceased proliferation within the first month; 2) both the

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