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1 o showed a reduced ability to colonize chick ceca.
2 erial killing via an ion channel activity of CecA.
3 intestine and longer-term clearance from the ceca.
4 were increased approximately 2.5-fold in the ceca and colons of H. bilis-inoculated mice.
5  quantitatively detect H. hepaticus in mouse ceca and feces using the ABI Prism 7700 sequence detecti
6 gly expressed around ENCCs as they enter the ceca and hindgut.
7 cytoplasm of epithelial cells of the gastric ceca and posterior stomach.
8 ow decreased commensal colonization of chick ceca and reduced colonization of BALB/cByJ mice compared
9 centration of the enzyme was observed in the ceca, and a 7.5-fold concentration occurred in the excre
10 sults support a mechanism of action in which CecA binds to and covers the membrane surface, thereby c
11 eads to reduction in C. jeuni numbers in the ceca by nine weeks post infection.
12 of the insect antibiotic peptide cecropin A (CecA) in the phospholipid bilayer membrane was determine
13 lly, ET3, which is strongly expressed in the ceca, inhibits the chemoattraction of NCC to glial-deriv
14 -infected mice were colonized at high rates: ceca of 50/50 wild-type mice and 168/170 IL-10(-/-) mice
15     Helicobacter spp. were cultured from the ceca of 62 of 79 mice.
16 been isolated from the inflamed stomachs and ceca of adult Syrian hamsters.
17            Salmonella more rapidly clear the ceca of birds administered the modified probiotic than o
18                                              Ceca of C. jejuni 11168-infected mice were colonized at
19                               At 16 wpi, the ceca of H. bilis-infected Rag2(-/-) mice treated with co
20           Analysis of the gene expression in ceca of H. hepaticus infected mice revealed 25 up-regula
21 e gamma-H2AX was significantly higher in the ceca of H. saguini-infected gnotobiotic mice than in the
22 , but IFN-gamma expression was normal in the ceca of IL-23p19-deficient mice during serotype Typhimur
23 nalysis of C. jejuni localization within the ceca of infected mice determined that the primary differ
24 athology scores was observed in the ilea and ceca of mice infected with the ibeA mutant.
25 tion of gamma interferon was observed in the ceca of mice infected with the ibeA mutant.
26      While H2 levels were greatly reduced in ceca of mice treated with antibiotics, both the Deltahyc
27 s Bacteroidetes populations long term in the ceca of mice, but the presence of C. albicans during cef
28 ells against microbial Ags isolated from the ceca of normal animals was observed.
29                   At 18 h postchallenge, the ceca of resistant C57BL/6 mice were histologically unrem
30 causes an acute inflammatory reaction in the ceca of streptomycin-pretreated mice that involves T-cel
31 causes an acute inflammatory reaction in the ceca of streptomycin-pretreated mice.
32 cing microbial richness and diversity in the ceca of stressed mice.
33  in the commensal E. coli harvested from the ceca of the stressed mice.
34 thetaiotaomicron was then harvested from the ceca of these hosts during the suckling period (postnata
35                Furthermore, histology of the ceca revealed that mice administered IL-12 antisera fail
36 tion of the beta-glucanase isolated from the ceca testified to its origin from the transgenic barley.
37 as little effect on bacterial numbers in the ceca, the main site of colonisation, where C. jejuni per
38 ortantly, increased fibrin deposition in the ceca was not associated with increased plasma fibrin whe
39 s ineffective in clearing C. jejuni from the ceca within the production lifetime of chickens, althoug

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