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1 o showed a reduced ability to colonize chick ceca.
2 erial killing via an ion channel activity of CecA.
3 intestine and longer-term clearance from the ceca.
5 quantitatively detect H. hepaticus in mouse ceca and feces using the ABI Prism 7700 sequence detecti
8 ow decreased commensal colonization of chick ceca and reduced colonization of BALB/cByJ mice compared
9 centration of the enzyme was observed in the ceca, and a 7.5-fold concentration occurred in the excre
10 sults support a mechanism of action in which CecA binds to and covers the membrane surface, thereby c
12 of the insect antibiotic peptide cecropin A (CecA) in the phospholipid bilayer membrane was determine
13 lly, ET3, which is strongly expressed in the ceca, inhibits the chemoattraction of NCC to glial-deriv
14 -infected mice were colonized at high rates: ceca of 50/50 wild-type mice and 168/170 IL-10(-/-) mice
21 e gamma-H2AX was significantly higher in the ceca of H. saguini-infected gnotobiotic mice than in the
22 , but IFN-gamma expression was normal in the ceca of IL-23p19-deficient mice during serotype Typhimur
23 nalysis of C. jejuni localization within the ceca of infected mice determined that the primary differ
27 s Bacteroidetes populations long term in the ceca of mice, but the presence of C. albicans during cef
30 causes an acute inflammatory reaction in the ceca of streptomycin-pretreated mice that involves T-cel
34 thetaiotaomicron was then harvested from the ceca of these hosts during the suckling period (postnata
36 tion of the beta-glucanase isolated from the ceca testified to its origin from the transgenic barley.
37 as little effect on bacterial numbers in the ceca, the main site of colonisation, where C. jejuni per
38 ortantly, increased fibrin deposition in the ceca was not associated with increased plasma fibrin whe
39 s ineffective in clearing C. jejuni from the ceca within the production lifetime of chickens, althoug
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