ライフサイエンスコーパス: cecal

1 trations, prebiotic administration increased cecal acetate and propionate and reduced isobutyrate con

2 fection, have persistent, albeit ameliorated cecal and colonic inflammation.

3 iated with ileal microbiota alterations, the cecal and colonic microbiota of MMP7(-/-) and wild-type

4 similar baseline symptoms, bowel habits, oro-cecal and colonic transit, and fasting concentrations of

5 rRNA gene sequencing and quantitative PCR of cecal and fecal samples demonstrated that GP dramaticall

6 Cecal arrival of the infused nutrient was determined by

7 on transcripts, and differentially modulated cecal bacterial populations.

8 Correlations among metabolic parameters and cecal bacterial taxa identified in this study suggest th

9 to as isolate microfluidicus 1, from a human cecal biopsy.

10 xpression, which in turn leads to epithelial cecal bud formation.

11 Cecal, but not colonic, pathology in C57BL/6 mice inocul

12 ations increases continuously from rectal to cecal cancers, supporting the 'colorectal continuum' par

13 is negatively correlated Actinobacteria with cecal cholesterol, intestinal and plasma LPC18:1, and Fi

14 e activity was detected in the distal ileal, cecal, colonic, and rectal epithelium, with selected cry

15 eukin-10 (IL-10)(-/-) mice revealed that the cecal colonizing ability of the ahpC mutant was signific

16 The cecal completion rate was 86% after erythromycin versus

17 dy, the largest to date on this subject, the cecal completion rate was higher with erythromycin than

18 Cecal completion rates, gastric and small bowel transit

19 Germfree mice that received cecal content transplants from "resistant" or "susceptib

20 We found that exposure of mice to sterilized cecal contents also resulted in a high mortality rate.

21 al early window of ontogeny, and transfer of cecal contents from the latter to the former suppressed

22 cal material of germfree mice colonized with cecal contents of conventionally raised animals.

23 rial strains, including those extracted from cecal contents of mice, bound poliovirus, with each bact

24 By colonizing adult germ-free mice with the cecal contents of neonatal and adult mice, we show that

25 l slurry, used intraperitoneal injections of cecal contents to induce polymicrobial peritonitis witho

26 TCDF-treated mouse cecal contents were enriched with Butyrivibrio spp. but

27 d concentrations of saturated fatty acids in cecal contents.

28 e challenged with sterilized or unsterilized cecal contents.

29 NSG mice given PGE2 had increased numbers of cecal CSCs and liver metastases compared with controls a

30 nto how C. jejuni responds and adapts to the cecal environment and reveals new functions involved in

31 In addition, it delays the initiation of cecal filling and T ICVt.

32 were delayed, with postoperative increase in cecal filling initiation and T ICVt, respectively.

33 filling, duodenal to terminal ileum transit, cecal filling initiation, and ileocecal valve transit (T

34 mmonia, intestinal glutaminase activity, and cecal glutamine content were compared between groups.

35 t mice led to increased mucosal destruction, cecal hemorrhage, and increased production of IFN-gamma

36 owever, NCR(+) ILC3 cells were essential for cecal homeostasis.

37 with FOLFOX was tested in a mouse model with cecal implantation of green fluorescent protein-labeled

38 Lyn(-/-) mice exhibited profound cecal inflammation, bacterial dissemination, and morbidi

39 revented the development of colonic, but not cecal, inflammation in H. hepaticus-infected anti-IL-10R

40 ive was to determine the association between cecal insertion time and colorectal neoplasm detection r

41 Cecal insertion time may be clinically relevant as misse

42 Information on the impact of cecal insertion time on colorectal neoplasm detection is

43 The mean cecal insertion time was 5.9 (SD, 4.4 minutes).

44 olorectal neoplasm detection by quartiles of cecal insertion time were 36.8%, 33.4%, 32.7%, and 31.0%

45 We observed a significantly higher rate of cecal intubation in patients with fair or better bowel p

46 In patients with incomplete colonoscopy, cecal intubation is sometimes unsuccessful due to a redu

47 advances in five prominent quality measures: cecal intubation rate, adherence to recommended screenin

48 Cecal intubation was successful in 96 of 100 repeat colo

49 Following cecal intubation, the colonic mucosa was carefully inspe

50 . difficile induces tryptophan catabolism in cecal lamina propria cells, which restricts C. difficile

51 elow the cecal valve at 25%, 50%, and 75% of cecal length, defined as severe, moderate, and mild seps

52 cells were isolated from rats 24 hours after cecal ligation and double puncture (2CLP) or sham surger

53 Cecal ligation and excision were performed on presymptom

54 assay was performed before and 5 hours after cecal ligation and incision (rats only; n = 5 per group)

55 Animals underwent cecal ligation and incision and remained anesthetized fo

56 Sepsis was induced by cecal ligation and incision followed by closure of the a

57 d interleukin-10 were observed 5 hours after cecal ligation and incision in rats (p < 0.05 vs baselin

58 Sham controls underwent laparotomy but not cecal ligation and incision.

59 Adult Wistar rats underwent cecal ligation and perforation (CLP), and serum and brai

60 Anesthetized mice were randomized to cecal ligation and perforation (vs. sham) with or withou

61 o death 5 hrs 11 mins +/- 0 hr 36 mins after cecal ligation and perforation at 38 degrees C.

62 In the cecal ligation and perforation group, increased luminal

63 plasma mediators; high tidal volume but not cecal ligation and perforation impaired lung function.

64 Sepsis was induced by cecal ligation and perforation in Sprague-Dawley rats.

65 High tidal volume and cecal ligation and perforation increased individual bron

66 Priming of high tidal volume by cecal ligation and perforation intensified plasma and br

67 The model used was cecal ligation and perforation modified so that alone it

68 roups: Sham, cecal ligation and perforation, cecal ligation and perforation plus immunoglobulin G (25

69 munoglobulin G (250 mg/kg, intravenous), and cecal ligation and perforation plus immunoglobulins enri

70 The sepsis group was subjected to a cecal ligation and perforation technique, whereas the co

71 ere divided into the following groups: Sham, cecal ligation and perforation, cecal ligation and perfo

72 erforation, high tidal volume, saline; or 7) cecal ligation and perforation, high tidal volume, dexam

73 ) sham, high tidal volume, dexamethasone; 6) cecal ligation and perforation, high tidal volume, salin

74 n and perforation, nonventilated, saline; 3) cecal ligation and perforation, nonventilated, dexametha

75 mized to: 1) sham, nonventilated, saline; 2) cecal ligation and perforation, nonventilated, saline; 3

76 p was subjected to abdominal surgery without cecal ligation and perforation.

77 the temperature goal, sepsis was induced by cecal ligation and perforation.

78 osphorylation by 31 +/- 11% following murine cecal ligation and puncture (CLP) at 8 h and 34 +/- 9% f

79 Sepsis was induced by cecal ligation and puncture (CLP) in 77 male rats.

80 eneficial in polymicrobial sepsis induced by cecal ligation and puncture (CLP) in mice.

81 urvival after experimental sepsis induced by cecal ligation and puncture (CLP) in mice.

82 Here, we show that cecal ligation and puncture (CLP) in rats impairs the os

83 Akt in CMs exposed to C5a in vitro and after cecal ligation and puncture (CLP) in vivo In both cases,

84 To address this, our model of cecal ligation and puncture (CLP) induced sepsis stratif

85 Using the mouse cecal ligation and puncture (CLP) model of sepsis, the a

86 eritubular microenvironment using the murine cecal ligation and puncture (CLP) model of sepsis.

87 ild-type (WT) and SP-D knockout (KO) mice in cecal ligation and puncture (CLP) model.

88 r2 in inflammation following endotoxemia and cecal ligation and puncture (CLP) models of sepsis.

89 saccharide-induced endotoxemia model and the cecal ligation and puncture (CLP) peritonitis model.

90 We developed a biotelemetry-based model of cecal ligation and puncture (CLP) that standardizes the

91 Cecal ligation and puncture (CLP) was used as a polymicr

92 rosis factor (TNF)-induced lethal shock, and cecal ligation and puncture (CLP) were performed in gene

93 In this study, we employed cecal ligation and puncture (CLP), a clinically relevant

94 ee RNA was significantly increased following cecal ligation and puncture (CLP), an animal model of po

95 llowed by treatment with AB103; or underwent cecal ligation and puncture (CLP), followed by treatment

96 the model of polymicrobial sepsis induced by cecal ligation and puncture (CLP), we investigated the r

97 ry responses, and organ injury in a model of cecal ligation and puncture (CLP), with or without antib

98 e report that hepatic SR-BI protects against cecal ligation and puncture (CLP)-induced sepsis as show

99 segmented filamentous bacterium (SFB) after cecal ligation and puncture (CLP)-induced sepsis using m

100 Mice undergoing cecal ligation and puncture (CLP)-induced sepsis were tr

101 nhibitory CpG-ODN (iCpG-ODN) 1 hour prior to cecal ligation and puncture (CLP)-induced sepsis.

102 ole of TLR3 in cardiac dysfunction following cecal ligation and puncture (CLP)-induced sepsis.

103 psis, severe sepsis, and septic shock and in cecal ligation and puncture (CLP)-induced septic mice.

104 bitor, improves survival in a mouse model of cecal ligation and puncture (CLP)-induced septic shock.

105 were subjected to LPS (2 mg/kg) or sepsis by cecal ligation and puncture (CLP).

106 oach was utilized, and sepsis was induced by cecal ligation and puncture (CLP).

107 ained from rats subjected to sham surgery or cecal ligation and puncture (CLP).

108 Lethal sepsis was induced in rats by cecal ligation and puncture (CLP).

109 54) and Fas in response to sepsis induced by cecal ligation and puncture (CLP).

110 of NLRP3 in polymicrobial sepsis induced by cecal ligation and puncture (CLP).

111 using flow cytometry during sepsis caused by cecal ligation and puncture (CLP).

112 O) mice at 20 h after induction of sepsis by cecal ligation and puncture (CLP).

113 ance in an animal model of sepsis induced by cecal ligation and puncture (CLP).

114 IP); 3) cecal ligation and puncture; and 4) cecal ligation and puncture + trametinib.

115 Cecal ligation and puncture also led to the alternative

116 after intraperitoneal lipopolysaccharide and cecal ligation and puncture and decreased vascular leak,

117 Murine models of cecal ligation and puncture and intratracheal lipopolysa

118 Procedures included induction of sepsis by cecal ligation and puncture and measurement of heart fun

119 ate groups of nontransplanted mice underwent cecal ligation and puncture and were euthanized 24 hours

120 When compared with sham-operated animals, cecal ligation and puncture animals developed hypotensio

121 creased greatly in mice after endotoxemia or cecal ligation and puncture as compared with sham mice.

122 applied 2 stringent animal models of sepsis: cecal ligation and puncture as well as intraperitoneal i

123 nal-regulated kinase signaling 6 hours after cecal ligation and puncture attenuated increases in circ

124 spleen were significantly elevated following cecal ligation and puncture but were reduced by the abse

125 d pituitary hormones increased 24 hours post-cecal ligation and puncture but were significantly lower

126 Sepsis, induced by cecal ligation and puncture for 24 and 48 hours.

127 as used to induce traumatic brain injury and cecal ligation and puncture for sepsis.

128 Serial blood specimens were obtained after cecal ligation and puncture for serum creatinine, interl

129 Wild-type mice made septic via cecal ligation and puncture had decreased crypt prolifer

130 Sepsis was induced by cecal ligation and puncture in 52 rats and was treated w

131 Polymicrobial sepsis, created by cecal ligation and puncture in a mouse model, augmented

132 g host defense to polymicrobial sepsis after cecal ligation and puncture in gene-deficient mice.

133 alloproteinase-8 improves survival following cecal ligation and puncture in mice, making it a potenti

134 pothesis, we induced polymicrobial sepsis by cecal ligation and puncture in wild-type (WT) mice and t

135 Model of polymicrobial sepsis induced by cecal ligation and puncture in wild-type and toll-like r

136 er intraperitoneal lipopolysaccharide and/or cecal ligation and puncture included mortality, vascular

137 Female Wistar rats submitted to cecal ligation and puncture method.

138 .002) and reduced by a similar proportion in cecal ligation and puncture mice (p = 0.004).

139 d 85% (p = 0.007), whereas in the jejunum of cecal ligation and puncture mice sodium-dependent glucos

140 cterial titers compared with vehicle-treated cecal ligation and puncture mice that succumbed at 48 h.

141 These changes are paralleled in cecal ligation and puncture mice.

142 Using the cecal ligation and puncture model of polymicrobial sepsi

143 st (CJ-12,255) would improve survival in the cecal ligation and puncture model of sepsis in adult fem

144 ation and generate a physiology-based murine cecal ligation and puncture model that is more similar t

145 The adhesive was also effective in a cecal ligation and puncture model, preventing sepsis and

146 Using the cecal ligation and puncture model, we demonstrated that

147 significantly increased survival in a murine cecal ligation and puncture model.

148 hages in DUSP3(-/-) mice in both the LPS and cecal ligation and puncture models.

149 of vehicle, endotoxin preconditioning in the cecal ligation and puncture mouse model of sepsis led to

150 for 2 weeks and then subjected to sepsis by cecal ligation and puncture or endotoxin injection.

151 ine production during septic shock caused by cecal ligation and puncture or endotoxin-induced shock.

152 was induced in Mye-K/O and wild-type mice by cecal ligation and puncture or intraperitoneal injection

153 ered both before and after sepsis induction (cecal ligation and puncture or lipopolysaccharides) intr

154 Forty Sprague-Dawley rats were randomized to cecal ligation and puncture or sham surgery.

155 Mice underwent cecal ligation and puncture polymicrobial sepsis and wer

156 r diet demonstrated increased survival after cecal ligation and puncture relative to mice receiving l

157 In wild-type mice, cecal ligation and puncture resulted in splenocyte apopt

158 DIE) and surviving (SUR) mice suffering from cecal ligation and puncture sepsis (days 6-28).

159 he initiation of sepsis improved survival in cecal ligation and puncture sepsis (neurokinin-1 recepto

160 Wild-type mice were subjected to the cecal ligation and puncture sepsis model.

161 ofol on LTB4 production in vivo and in vitro Cecal ligation and puncture surgery was performed in mic

162 semination, after K. pneumoniae infection or cecal ligation and puncture surgery.

163 ld for acute physiologic deterioration after cecal ligation and puncture that has adequate face and c

164 Mice underwent cecal ligation and puncture to induce peritonitis.

165 Mice were subjected to cecal ligation and puncture to induce sepsis or underwen

166 Mice underwent cecal ligation and puncture to induce sepsis.

167 Mice were randomized to cecal ligation and puncture to model critical illness (n

168 optive transfer experiments were followed by cecal ligation and puncture to test the hypothesis that

169 n inflammatory gene expression at 20 h after cecal ligation and puncture was not due to resolution of

170 Cecal ligation and puncture was performed 1 week later.

171 Mice that underwent cecal ligation and puncture were randomly assigned to re

172 ment also exacerbated septic shock caused by cecal ligation and puncture when given after the onset o

173 Sepsis was induced by cecal ligation and puncture with the cecum ligated below

174 ally relevant model of polymicrobial sepsis (cecal ligation and puncture), DJ-1(-/-) mice had improve

175 of sepsis-induced indirect pulmonary injury (cecal ligation and puncture).

176 induced ALI) and indirect ALI (systemic LPS, cecal ligation and puncture).

177 Twenty rodents (10 cecal ligation and puncture, 10 sham) were killed at 24

178 after intraperitoneal lipopolysaccharide and cecal ligation and puncture, also resulted in decreased

179 Mice underwent cecal ligation and puncture, and an HD-X11 wireless tele

180 e bone marrow during late sepsis, induced by cecal ligation and puncture, and demonstrated that they

181 cytokines were increased in serum following cecal ligation and puncture, and interleukin 10 and mono

182 l signs were repeated at 25 or 49 hours post-cecal ligation and puncture, blood was collected, animal

183 severity of illness and improved survival in cecal ligation and puncture, both as a pretreatment and

184 Forty-eight hrs after cecal ligation and puncture, cardiac performance was est

185 Sepsis was induced in C57Bl/6J mice by cecal ligation and puncture, followed 6 hours later by a

186 Six hours after cecal ligation and puncture, mice were randomized to fou

187 4) After 24 hours of cecal ligation and puncture, norepinephrine was infused

188 In polymicrobial sepsis initiated by cecal ligation and puncture, RvD2 ( approximately 2.7 nm

189 3) Six, 12, and 24 hours after cecal ligation and puncture, thoracic aorta segments wer

190 1) Six, 12, and 24 hours after cecal ligation and puncture, vascular reactivity to phen

191 At 24 or 48 hours post-cecal ligation and puncture, vital signs were measured,

192 In a murine model of infection using cecal ligation and puncture, we demonstrated microvascul

193 Four hours after cecal ligation and puncture, Wistar rats were randomly a

194 nt mice exhibited enhanced susceptibility to cecal ligation and puncture- and LPS-induced sepsis, whi

195 scopy, collagen elastic modulus was lower in cecal ligation and puncture-exposed rats at 24 hours (1.

196 femoral neck were significantly reduced for cecal ligation and puncture-exposed rodents at 24 hours

197 dulus was similar at 24 hours but reduced in cecal ligation and puncture-exposed rodents at 96 hours

198 ononuclear cells rescued wild-type mice from cecal ligation and puncture-induced mortality.

199 Cecal ligation and puncture-induced murine sepsis induce

200 The salutary effects of resveratrol on cecal ligation and puncture-induced myocardial dysfuncti

201 Upon lipopolysaccharide or cecal ligation and puncture-induced peritonitis, these m

202 bacterial dissemination, and inflammation in cecal ligation and puncture-induced polymicrobial sepsis

203 ous Ag-specific CD4 T cell populations after cecal ligation and puncture-induced sepsis and analyzed

204 jury develops in mice predicted to die after cecal ligation and puncture-induced sepsis compared with

205 ation of iCRT3 to C57BL/6 mice, subjected to cecal ligation and puncture-induced sepsis, decreases th

206 om toll-like receptor 9--deficient mice with cecal ligation and puncture-induced severe sepsis expres

207 signal-regulated kinase blockade attenuated cecal ligation and puncture-mediated up-regulation of cy

208 erapy significantly improved the survival in cecal ligation and puncture-treated scavenger receptor B

209 Sepsis was induced by cecal ligation and puncture.

210 and severe polymicrobial sepsis produced by cecal ligation and puncture.

211 hock and to polymicrobial septic shock after cecal ligation and puncture.

212 of mice with polymicrobial sepsis induced by cecal ligation and puncture.

213 psis-intra-peritoneal lipopolysaccharide and cecal ligation and puncture.

214 as induced in 10-wk-old male C57BL/6 mice by cecal ligation and puncture.

215 xic shock and polymicrobial sepsis following cecal ligation and puncture.

216 or water for 12 weeks and then subjected to cecal ligation and puncture.

217 nergic activity decreased six-fold following cecal ligation and puncture.

218 ect on cytokine/chemokine serum levels after cecal ligation and puncture.

219 Male C57Bl6 mice were made septic using cecal ligation and puncture.

220 pe mice were subjected to sham laparotomy or cecal ligation and puncture.

221 d much higher mortality after endotoxemia or cecal ligation and puncture.

222 ls of intraperitoneal lipopolysaccharide and cecal ligation and puncture.

223 and mortality after intraperitoneal LPS and cecal ligation and puncture.

224 and mortality after intraperitoneal LPS and cecal ligation and puncture.

225 e production and a lower survival rate after cecal ligation and puncture.

226 ansia species and the survival benefit after cecal ligation and puncture.

227 ls are a critical factor in sepsis following cecal ligation and puncture.

228 Polymicrobial sepsis was induced by cecal ligation and puncture.

229 re not a critical factor in sepsis following cecal ligation and puncture.

230 inflammation in septic mice challenged with cecal ligation and puncture.

231 mice, ranging from 339 to 529 minutes after cecal ligation and puncture.

232 re euthanized at 18 hours after induction of cecal ligation and puncture.

233 himeric mice were generated and subjected to cecal ligation and puncture.

234 n wild-type mice when subjected to sepsis by cecal ligation and puncture.

235 sham control + trametinib (1 mg/kg, IP); 3) cecal ligation and puncture; and 4) cecal ligation and p

236 provides effective protection 18 hours post cecal ligation and puncture; using adrenal-specific scav

237 ELA-treated cecal ligation puncture rats were the only group to 1) d

238 -induced cardiac dysfunction was produced by cecal ligation puncture to assess hemodynamic efficacy,

239 Apelinergic agonist infusion counteracted cecal ligation puncture-induced myocardial dysfunction b

240 Ultrasound treatment 24 hours before cecal ligation puncture-induced sepsis was effective in

241 s cardiovascular function and survival after cecal ligation puncture-induced sepsis.

242 ride injection and is extended by 1 wk after cecal ligation.

243 Recently, using a cecal-ligation and puncture (CLP) model of sepsis, we sh

244 In this study, using the cecal-ligation and puncture mouse model of sepsis, we sh

245 reduced mortality in lipopolysaccharide and cecal-ligation-and-puncture models of sepsis, but not in

246 o increase survival rates of mice undergoing cecal-ligation-and-puncture-(CLP).

247 Furthermore, polymicrobial sepsis induced by cecal ligature and puncture resulted in early death of t

248 series included older patient age, rectal or cecal location, surface coating with oral contrast mater

249 ls of short chain fatty acids (SCFAs) in the cecal material and enhancement of cell density and crypt

250 Mice receiving C57BL/6J cecal microbes demonstrated choline diet-dependent enhan

251 in recipients on choline diet that received cecal microbes from C57BL/6J inbred mice; however, durab

252 control diet, soy significantly shifted the cecal microbial community of LCR rats, resulting in a lo

253 L-carnitine supplementation in mice altered cecal microbial composition, markedly enhanced synthesis

254 train, NZW/LacJ, were selected as donors for cecal microbial transplantation into apolipoprotein e nu

255 Following dog-associated dust exposure, the cecal microbiome of protected animals was extensively re

256 significant shift in the composition of the cecal microbiome.

257 health, adipose tissue inflammation, and the cecal microbiota in ovariectomized (OVX) rats bred for l

258 Ribotyping of IgA-coated cecal microbiota showed Proteobacteria evading antibody

259 ce harboring these xenomicrobiota or a mouse cecal microbiota, along with germ-free "bystanders," rev

260 ate the cellular sources of IFN-gamma in the cecal mucosa during the acute phase of an S. Typhimurium

261 c cells and macrophages were observed in the cecal mucosa of mice infected with the curli mutant.

262 majority of IFN-gamma-positive cells in the cecal mucosa were neutrophils.

263 tants that grew 15% faster in vitro in mouse cecal mucus and motile E. coli MG1655 envZ missense muta

264 D and grows ca. 15% slower in vitro in mouse cecal mucus and on several sugars present in mucus compa

265 e mutants that grew slower in vitro in mouse cecal mucus yet were able to cocolonize with the faster-

266 Administration of cecal mycobiomes from rats that had been separated from

267 Cecal nitrate measurements suggested that the murine cec

268 ying effects of alpha-defensin deficiency in cecal or colonic infection or disease.

269 OD-scidIL-2Rgamma(-/-) (NSG) mice were given cecal or subcutaneous injections of LS-174T or human pri

270 deep sequencing of C. jejuni mutants in the cecal outputs, several novel factors required for effici

271 We used the model of cecal pole ligation and puncture for induction of polymi

272 -155, and miR-193b* were found in mice after cecal pole ligation and puncture-induced sepsis.

273 w hours of challenging mice with adjuvant or cecal puncture, B cells accumulate in the bone marrow re

274 It is absent from the cecal region just prior to ENCC arrival, but becomes str

275 SSP) of >/=10 mm that were in the right-side/cecal regions of the colon.

276 F from the membrane) rapidly developed large cecal serrated polyps.

277 stines of mice promoted development of small cecal serrated polyps.

278 Furthermore, KGM increased cecal short-chain fatty acid contents, and both KGM and

279 corticosterone, microbiota composition, and cecal short-chain fatty acids were measured.

280 wk), or elderly (20-24 mo) mice underwent a cecal slurry model of intra-abdominal sepsis.

281 In our cecal slurry model, no survival advantage was seen among

282 ndotoxemia by intraperitoneal injection with cecal slurry or lipopolysaccharide, respectively.

283 Our second model, cecal slurry, used intraperitoneal injections of cecal c

284 , including four ileal, two ileocecal, three cecal, three appendicular, and 14 sigmoid colon lesions.

285 d expression of proinflammatory cytokines in cecal tissue.

286 (iNOS) were significantly upregulated in the cecal tissues of infected mice.

287 usceptibility to amebiasis was assessed, and cecal tissues were analyzed for changes in gene expressi

288 bursa of Fabricius, gastrointestinal tract, cecal tonsil, and trachea.

289 Droppings, cecal tonsils and 5 internal organs were sampled and cul

290 astric emptying, antral contractions and oro-cecal transit after ingestion of a solid meal were inves

291 Antral contractions and oro-cecal transit were not different.

292 el-related symptoms, rectal sensitivity, oro-cecal transit, and colonic (whole intestine) transit.

293 Mice on semipurified I3C-diet had increased cecal Tregs, ILC3s, and gammadelta T cells and an increa

294 Apc(Min/+) mice and NSG mice with orthotopic cecal tumors were given vehicle (controls), PGE2, celeco

295 nd puncture with the cecum ligated below the cecal valve at 25%, 50%, and 75% of cecal length, define

296 ted the role of IL-17A, IL-17F, and IL-22 in cecal versus colonic inflammation.

297 l for mortality in cases of sigmoid (SV) and cecal volvulus (CV) taking into account preoperative and

298 Intestinal transit and cecal water content were also significantly higher in Ta

299 e acids was observed along with an increased cecal weight.

300 ter losses of stool consistency and body and cecal weights than those that were not exposed to PPI.