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1 SBL phenotype (based on nonsusceptibility to ceftriaxone).
2 lation (120 assigned to gatifloxacin, 119 to ceftriaxone).
3 gs: cefixime, chloramphenicol, ofloxacin, or ceftriaxone.
4 r bactericidal effect on S. epidermidis than ceftriaxone.
5  ceftriaxone, and Haemophilus influenzae and ceftriaxone.
6 influenzae in combination with cefotaxime or ceftriaxone.
7 ter one day of treatment with the antibiotic ceftriaxone.
8 fully subcultured by day 3 after exposure to ceftriaxone.
9  the antibiotics imipenem, penicillin G, and ceftriaxone.
10 cortex (mPFC) were quantified in response to ceftriaxone.
11 d decreased susceptibility to penicillin and ceftriaxone.
12 chloramphinicol, amoxicillin-clavulanate and ceftriaxone.
13 with single doses of ofloxacin, cefixime, or ceftriaxone.
14 rom cattle, all of which were susceptible to ceftriaxone.
15 tes from cattle, which was also resistant to ceftriaxone.
16  cefotaxime and 114 of 138 as susceptible to ceftriaxone.
17 ephalopathy can be treated successfully with ceftriaxone.
18  were reversed by the beta-lactam antibiotic ceftriaxone.
19 lin-resistant isolate, increased the MICs of ceftriaxone (0.4 mug/mL) and cefixime (1.2 mug/mL) to le
20 day for 14 days (with or without intravenous ceftriaxone, 1 g) (n = 127 included in the analysis).
21                                              Ceftriaxone 100 mg/kg per injection twice per day (200 m
22  no difference in mortality with intravenous ceftriaxone (121 of 230 patients) as compared with intra
23 230 patients) as compared with intramuscular ceftriaxone (128 of 229 patients) (odds ratio, 0.88; 95%
24 amil 600 mg given every 12 h was superior to ceftriaxone 2 g given every 24 h for the treatment of As
25 s ceftaroline fosamil (600 mg every 12 h) or ceftriaxone (2 g every 24 h) for 5-7 days.
26 articipants were randomly allocated (2:1) to ceftriaxone (2 g or 4 g per day) or placebo.
27 el trial of intramuscular versus intravenous ceftriaxone (2 g twice daily for 10 days) in adults with
28     The patients received either intravenous ceftriaxone, 2 g daily for 30 days, followed by oral dox
29                  The efficacy of intravenous ceftriaxone, 2 g per day for 30 days, was evaluated in a
30                               Treatment with ceftriaxone (200 mg/kg, i.p.) upregulated core GLT1 expr
31                                              Ceftriaxone (200 mg/kg, IP) was administered (vs. saline
32 or placebo (232 patients) plus intramuscular ceftriaxone (230 patients) or intravenous ceftriaxone (2
33 ar ceftriaxone (230 patients) or intravenous ceftriaxone (235 patients).
34  amoxicillin-clavulanate and 3 injections of ceftriaxone; 3 had recurrent AOM; and for 2 others, the
35 ion) increased survival in mice treated with ceftriaxone (5 days) from 40% to 100%.
36 nol for 48 h during which time they received ceftriaxone (50 or 100 mg/kg, IP) or saline twice a day
37 ycin, 96% were susceptible to penicillin and ceftriaxone, 54% were susceptible to ciprofloxacin, and
38 tifloxacin (10 mg/kg per day) or intravenous ceftriaxone (60 mg/kg up to 2 g per day for patients age
39 f 9,863 nonmeningeal isolates tested against ceftriaxone, 82.7% were susceptible, 13.2% were intermed
40 ncomycin (1103, 14.4%; 95% CI, 13.7%-15.2%), ceftriaxone (825, 10.8%; 95% CI, 10.1%-11.5%), piperacil
41 ts provided evidence that the combination of ceftriaxone (a cephalosporin antibiotic) and lansoprazol
42 del of ethanol withdrawal, we tested whether ceftriaxone, a beta-lactam antibiotic known to increase
43 lace preference (CPP), we determined whether ceftriaxone, a beta-lactam antibiotic known to increase
44 ne self-administration, while treatment with ceftriaxone, a beta-lactam antibiotic previously shown t
45                    Other studies report that ceftriaxone, a glutamate transporter subtype-1 activator
46 nt of CDI in hospitalized patients receiving ceftriaxone, a high-risk antibiotic for CDI.
47 are the effectiveness of the ampicillin plus ceftriaxone (AC) and ampicillin plus gentamicin (AG) com
48 y; those with the highest rate had increased ceftriaxone administration and culturable Aspergillus, a
49 orin and azithromycin and those treated with ceftriaxone alone (9.1%; RR, 0.81; 95% CI, .18-3.60) or
50 liary side-effects, participants assigned to ceftriaxone also received 300 mg ursodeoxycholic acid tw
51                                              Ceftriaxone also reversed the cocaine-induced synaptic p
52 ipenem, filamentous cells with cefoxitin and ceftriaxone), amikacin and phages did not modify cell sh
53                                              Ceftriaxone, an antibiotic compound known to increase EA
54                                              Ceftriaxone, an expanded-spectrum cephalosporin, is an a
55  340 participants were randomly allocated to ceftriaxone and 173 to placebo.
56 it 90% of isolates increased by 1.5-fold for ceftriaxone and 2-fold for levofloxacin and remained the
57         Of those patients, 74 (60%) received ceftriaxone and 50 (40%) received oxacillin.
58 ct roles for xCT and GLT-1 in the actions of ceftriaxone and add to a body of literature finding evid
59 to defervescence in 17 patients treated with ceftriaxone and azithromycin combination was 3.2 days (+
60 (CDC) currently recommends dual therapy with ceftriaxone and azithromycin for gonorrhea to ensure eff
61 at day 14; among the 190 isolates with lower ceftriaxone and azithromycin MICs, only 13 (7%) had pers
62     Persistence was associated with elevated ceftriaxone and azithromycin MICs.
63 ntly more likely to exhibit elevated MICs of ceftriaxone and azithromycin than isolates from MSW (P <
64 as the undefined mechanisms of resistance to ceftriaxone and azithromycin underscore the importance o
65 ssed include whether to change the dosage of ceftriaxone and azithromycin used in the recommended dua
66 taphylococcus aureus strains were exposed to ceftriaxone and cefazolin at concentrations from 0 to 10
67  (and ESBL nonproducers) were susceptible to ceftriaxone and cefepime at the standard inoculum as wer
68 mutants to extended spectrum cephalosporins (ceftriaxone and cefepime) identified either PbpF or PonA
69 ance to the expanded-spectrum cephalosporins ceftriaxone and cefixime in Neisseria gonorrhoeae.
70  penA alleles on decreased susceptibility to ceftriaxone and cefixime, with the expectation that this
71 parate S. pneumoniae isolates susceptible to ceftriaxone and cefotaxime from those that are not susce
72                    Overall, the new M100-S12 ceftriaxone and cefotaxime interpretative breakpoints fo
73 M100-S12 document in January 2002 introduced ceftriaxone and cefotaxime MIC interpretative breakpoint
74 usceptibility of Streptococcus pneumoniae to ceftriaxone and cefotaxime.
75 sceptible to failing cephalosporins, such as Ceftriaxone and Cefotaxime.
76                                              Ceftriaxone and cefotetan, which lack antienterococcal a
77 lates were susceptible by routine testing to ceftriaxone and ceftazidime, respectively.
78 g resistance to tetracycline, norfloxacylin, ceftriaxone and ciprofloxacin were observed among Gram n
79 nt recommend dual therapy with intramuscular ceftriaxone and either azithromycin or doxycycline.
80  the EHR, we found that patients taking both ceftriaxone and lansoprazole had significantly longer QT
81                       Combination therapy of ceftriaxone and lansoprazole is associated with increase
82 agnosis, surgical admission, and duration of ceftriaxone and other antibiotics, for each day of doxyc
83                                  We compared ceftriaxone and piperacillin-tazobactam at doses ranging
84 and reduced susceptibility to penicillin and ceftriaxone and still predominated among declining serot
85 eriod at week 14, after the 2-week course of ceftriaxone and the 12-week course of the randomized stu
86                                Resistance to ceftriaxone and the fluoroquinolones, which are used to
87 kpoints (2 each for aztreonam, cefepime, and ceftriaxone, and 1 for cefazolin and ceftazidime).
88 ross species, particularly to penicillin and ceftriaxone, and across geographical regions.
89 ad negative skin test results to cefuroxime, ceftriaxone, and aztreonam and tolerated challenges.
90 activity between penicillins and cefuroxime, ceftriaxone, and aztreonam in all subjects with T cell-m
91 e subjects could be treated with cefuroxime, ceftriaxone, and aztreonam.
92 ephalexin, cefaclor, cefadroxil, cefuroxime, ceftriaxone, and aztreonam.
93 ibitory concentrations (MICs) of penicillin, ceftriaxone, and cefixime for a wild-type strain (FA19)
94 icillin-clavulanic acid, cefdinir, cefixime, ceftriaxone, and clarithromycin were the most reproducib
95 r-Kelly medium in the presence or absence of ceftriaxone, and cultures were monitored for up to 56 da
96 , and gentamicin, Neisseria meningitidis and ceftriaxone, and Haemophilus influenzae and ceftriaxone.
97 of various antibiotics (i.e., ciprofloxacin, ceftriaxone, and tetracycline) against Escherichia coli
98 8 patients received prophylactic intravenous ceftriaxone (antibiotic group) and 147 patients did not
99      Our data suggest that ciprofloxacin and ceftriaxone are appropriate empirical therapy for suspec
100                      Generic formulations of ceftriaxone are becoming more affordable and available,
101                     Long-term treatment with ceftriaxone attenuates the reinstatement of cocaine seek
102 expression in the NAc.SIGNIFICANCE STATEMENT Ceftriaxone attenuates the reinstatement of cocaine, alc
103                                  Combination Ceftriaxone-Azithromycin therapy may provide a therapeut
104 ast 12 months (96%), and were treated with a ceftriaxone-based regimen (95%).
105 alosporin and azithromycin was comparable to ceftriaxone-based regimens in the treatment of pharyngea
106                                              Ceftriaxone blocked methamphetamine-triggered reinstatem
107 ehicle, whereas 100 or 200, but not 50 mg/kg ceftriaxone blocked this response.
108 ll) inputs or treatment with the antibiotic, ceftriaxone, blocked reinstatement of morphine-evoked co
109 n was specific to lansoprazole combined with ceftriaxone but not with other cephalosporins.
110 es (sensitivity 98% for cefixime and 91% for ceftriaxone), but alternative resistance mechanisms have
111  were intermediate, and 21 were resistant to ceftriaxone by MIC testing.
112 losporins, such as cefepime, cefotaxime, and ceftriaxone, by 9.1 to 13.0%, bringing their in vitro ra
113  of ciprofloxacin (A), azithromycin (B), and ceftriaxone (C).
114                                              Ceftriaxone can be dosed once daily and is less expensiv
115 mate transporter GLT-1 (N-acetylcysteine and ceftriaxone) can decrease measures of drug seeking.
116 underwent challenges with cefuroxime axetil, ceftriaxone, cefazolin, and ceftibuten; and group C and
117 B was studied in combination with cefazolin, ceftriaxone, cefepime, imipenem, gentamicin, tigecycline
118 to effectively remove vancomycin, cefoxitin, ceftriaxone, cefepime, piperacillin-tazobactam, ampicill
119  determine antimicrobial susceptibilities of ceftriaxone, cefixime, and cefpodoxime in Neisseria gono
120 mine the susceptibility of N. gonorrhoeae to ceftriaxone, cefixime, and cefpodoxime, although we reco
121 % within 1 log2 dilution from each other for ceftriaxone, cefixime, and cefpodoxime, respectively.
122 lation coefficients of 92%, 91%, and 92% for ceftriaxone, cefixime, and cefpodoxime, respectively.
123  agreements were 99.1%, 98.3%, and 94.8% for ceftriaxone, cefixime, and cefpodoxime, respectively.
124    Isolates with decreased susceptibility to ceftriaxone, cefixime, azithromycin, and spectinomycin r
125 of 264 isolates were subjected to cefazolin, ceftriaxone, cefotaxime, ceftazidime, cefepime, and aztr
126 hoxazole and had reduced susceptibilities to ceftriaxone, cefotaxime, minocycline, and ciprofloxacin.
127  which enhances the bactericidal activity of Ceftriaxone/Cefotaxime against highly pathogenic MRSA in
128 g an oxyimino group (cefuroxime, cefotaxime, ceftriaxone, ceftazidime, or aztreonam) was associated w
129  group A, positive responses to 1 or more of ceftriaxone, cefuroxime, cefotaxime, cefepime, cefodizim
130 or more of the following agents: ampicillin, ceftriaxone, ciprofloxacin, gentamicin, and/or trimethop
131  alone (9.1%; RR, 0.81; 95% CI, .18-3.60) or ceftriaxone combined with azithromycin or doxycycline (1
132 e tested against cefepime, cefotaxime (CTX), ceftriaxone (CTR), clindamycin (CLI), erythromycin (ERY)
133                                     However, ceftriaxone did not reverse stress-induced synaptic pote
134 d immunoprecipitation of GLT-1 revealed that ceftriaxone does not upregulate GLT-1 and xCT through a
135         In this cohort of patients receiving ceftriaxone, doxycycline was associated with lower risk
136 tes had low MICs to amoxicillin, cefotaxime, ceftriaxone, doxycycline, linezolid, meropenem, penicill
137 ice treated with the beta-lactam antibiotic, ceftriaxone, during cocaine withdrawal.
138                                              Ceftriaxone elevated EAAT2 transcription in PHFA through
139              Salmonella group B resistant to ceftriaxone emerged in 2009 and 2010 (6.2% [2/32 isolate
140       We investigated the mechanism by which ceftriaxone enhances EAAT2 expression in primary human f
141        In separate groups of rats, 200 mg/kg ceftriaxone failed to block cue-induced food seeking, ar
142                                              Ceftriaxone failed to promote colonization at doses up t
143 istration were treated with the same dose of ceftriaxone followed by intracore or intrashell infusion
144 study groups received open-label intravenous ceftriaxone for 2 weeks before initiating the randomized
145 e aimed to assess the safety and efficacy of ceftriaxone for amyotrophic lateral sclerosis in a combi
146 ot inferior to intravenous administration of ceftriaxone for bacterial meningitis.
147  to be noninferior to the comparator agents (ceftriaxone for CABP and vancomycin plus aztreonam for A
148            Intra-NAc xCT knockdown prevented ceftriaxone from attenuating reinstatement and from upre
149     Intra-NAc GLT-1 knockdown also prevented ceftriaxone from attenuating reinstatement and from upre
150  were isolated on selective media containing ceftriaxone from the wetland compared to feces, suggesti
151 16% to 27%, and there was a decrease in both ceftriaxone (from 39% to 33%) and azithromycin (change f
152  significantly more often in the doxycycline-ceftriaxone group (35%) than in either of the other two
153 p were numerically higher than those for the ceftriaxone group (for the clinically evaluable populati
154 ycycline group, and 86.5% in the doxycycline-ceftriaxone group (P > 0.2).
155 liary adverse events were more common in the ceftriaxone group than in the placebo group (gastrointes
156 l group and 178 (74%) of 240 patients in the ceftriaxone group were clinically cured at the test-of-c
157 Significantly more participants who received ceftriaxone had serious hepatobiliary serious adverse ev
158 this consideration and its lack of toxicity, ceftriaxone has potential to manipulate glutamate transm
159                            The resistance to ceftriaxone has remained low, suggesting it is likely to
160 tended-spectrum cephalosporins--cefixime and ceftriaxone--have been recommended in the UK for treatme
161 failure, compared with 19 (16%) who received ceftriaxone (hazard ratio [HR] 1.04 [95% CI 0.55-1.98];
162 ns with high-level resistance to cefixime or ceftriaxone heralds the possible demise of beta-lactam a
163 , compared with four (7%) of 54 who received ceftriaxone (HR 0.24 [95% CI 0.08-0.73]; p=0.01).
164 treatment versus 15 (23%) of 65 who received ceftriaxone (HR 7.50 [95% CI 1.71-32.80]; p=0.01).
165 6, cefaclor in 82, and cefuroxime axetil and ceftriaxone in 22 subjects) were well tolerated.
166                 Our data support the role of ceftriaxone in alleviating alcohol withdrawal and open a
167 ising stage 2 data, stage 3 of this trial of ceftriaxone in amyotrophic lateral sclerosis did not sho
168 amil should be regarded as an alternative to ceftriaxone in empirical treatment regimens for this pat
169 stage 3 (efficacy), participants assigned to ceftriaxone in stage 2 received 4 g ceftriaxone, partici
170  safety of ceftaroline fosamil compared with ceftriaxone in the treatment of Asian patients admitted
171 xacin would be superior to the cephalosporin ceftriaxone in treating enteric fever.
172   When delivered to animals, the beta-lactam ceftriaxone increased both brain expression of GLT1 and
173                                 In addition, ceftriaxone increased glutamate uptake, a primary functi
174 ays disease onset and prolongs survival, and ceftriaxone increases EAAT2 activity.
175            Immunoblotting confirmed that the ceftriaxone-induced blockade of cocaine relapse was asso
176  cue-induced food seeking, arguing against a ceftriaxone-induced effect unique to extinction training
177  small interference RNA completely inhibited ceftriaxone-induced glutamate uptake activity in PHFA.
178 argeting antibiotics, such as vancomycin and ceftriaxone, inhibit the kinase activity of Stk1 and lea
179             Both piperacillin-tazobactam and ceftriaxone inhibited colonization by an enterococcal st
180 after each extinction session, rats received ceftriaxone (intraperitoneally), a beta-lactam antibioti
181       Taken together, our data indicate that ceftriaxone is a potent modulator of glutamate transport
182 ptional mechanism, and their coregulation by ceftriaxone is not mediated by physical interaction.
183                                              Ceftriaxone is recommended when parenteral antibiotic th
184 us pneumoniae with reduced susceptibility to ceftriaxone is reported increasingly, and alternatives a
185                                              Ceftriaxone is the foundation of currently recommended g
186  error, 13.0%), cefotaxime (m error, 21.2%), ceftriaxone (m error, 23.3%), tetracycline (M error, 11.
187  0.008 to 2 mug/ml); all were susceptible to ceftriaxone (median MIC, 0.015 mug/ml; range, 0.008 to 1
188                  Our results reveal that the ceftriaxone-mediated attenuation of cue-induced cocaine
189 on of the EAAT2 promoter was responsible for ceftriaxone-mediated EAAT2 induction.
190 n NAc core, but not shell, would reverse the ceftriaxone-mediated effect.
191                                 Furthermore, ceftriaxone-mediated upregulation of surface GLT1 expres
192 etive criteria were proposed for cefotaxime, ceftriaxone, meropenem, azithromycin, and minocycline.
193  from 1,386 clinical isolates suggest that a ceftriaxone MIC cutoff of 8 mug/ml is an excellent predi
194 oline was generally 16-fold more active than ceftriaxone (MIC(50), 4 mug/mL; MIC(90), 4 mug/mL; 97.9%
195  microg/mL) and was 16-fold more active than ceftriaxone (MIC(90), 2 microg/mL).
196 spectively) and was 16-fold more active than ceftriaxone (MIC(90), 2 microg/mL).
197 azithromycin (MIC90s, < or =0.12 microg/ml), ceftriaxone (MIC90s, 0.5 microg/ml), and levofloxacin (M
198 ncentrations (MICs) >256 mug/mL and elevated ceftriaxone MICs (>/=0.125 mug/mL).
199 ad azithromycin MICs >16 microg/mL and 5 had ceftriaxone MICs = 0.125 microg/mL by agar dilution.
200 er of clinical microbiology laboratories use ceftriaxone MICs as a proxy means of identifying bacteri
201 he isolates showed reduced susceptibility to ceftriaxone (MICs, >or=16 microg/ml).
202                                Isolates with ceftriaxone minimum inhibitory concentration >/= 1 micro
203 geal and rectal isolates, there were 10 with ceftriaxone minimum inhibitory concentration (MIC) >/=0.
204                                Isolates with ceftriaxone minimum inhibitory concentrations >/=2 micro
205    Twelve of the 15 Salmonella isolates with ceftriaxone minimum inhibitory concentrations of 16 micr
206                               In conclusion, Ceftriaxone modulated the expression of the glutamate tr
207       These findings suggest a mechanism for ceftriaxone modulation of glutamate transport and for it
208                         After treatment with ceftriaxone mono- or dual therapy (with azithromycin or
209  (+/- 1.7), whereas in 13 cases treated with ceftriaxone monotherapy, the time to defervescence was 6
210                           In the presence of ceftriaxone, neither system was able to recover Streptoc
211                     These data indicate that ceftriaxone normalizes multiple aspects of glutamate hom
212                                              Ceftriaxone, one of the beta-lactam antibiotics, is a st
213 at at high concentration neither Inh2-B1 nor Ceftriaxone or Cefotaxime alone was able to inhibit the
214 as observed at a micromolar concentration of Ceftriaxone or Cefotaxime in the presence of Inh2-B1.
215                    Participants received 2 g ceftriaxone or placebo twice daily through a central ven
216 icipants were randomly assigned (2:1) to 4 g ceftriaxone or placebo.
217 with or without a single intravenous dose of ceftriaxone, or 20 days of oral doxycycline.
218 or which the MIC of ceftazidime, cefotaxime, ceftriaxone, or aztreonam was >or=2 microg/ml or the MIC
219                           For a ceftazidime, ceftriaxone, or cefotaxime MIC of > or =2 microg/ml, a d
220 l E. coli) tested susceptible to cefotaxime, ceftriaxone, or ceftazidime.
221 strain 536 and received 536_P1 intranasally, ceftriaxone, or control.
222 were treated with antibiotics (azithromycin, ceftriaxone, or doxycycline) for 30 consecutive days.
223 illin (P<0.0001), ampicillin (P = 0.01), and ceftriaxone (P = 0.006).
224 ity (9 of 50 [18%] oxacillin vs 3 of 74 [4%] ceftriaxone; P = .01).
225 igned to ceftriaxone in stage 2 received 4 g ceftriaxone, participants assigned to placebo in stage 2
226 ug use occurred during the 2-week open-label ceftriaxone phase, and no serious drug-related adverse e
227 rescribing practice in 2010 from cefixime to ceftriaxone plus azithromycin.
228                                              Ceftriaxone promoted colonization at doses at least 0.5
229 ation of GLT-1 expression in astrocytes with ceftriaxone protected CA1 neurons from forebrain ischemi
230  43 patients developed CDI within 30 days of ceftriaxone receipt, an incidence of 5.60 cases per 10 0
231                                 Furthermore, ceftriaxone reduced ethanol drinking, suggesting that EN
232          Rapidly emerging resistance against ceftriaxone requires urgent reevaluation of antibiotic r
233                                              Ceftriaxone resistance among S. pneumoniae was 2.1% (10.
234                                              Ceftriaxone resistance increased in blood isolates from
235 esistance and DCS; rates of azithromycin and ceftriaxone resistance were 0.6% and absent, respectivel
236 pyema; the emergence of multidrug, including ceftriaxone, resistant serotype 19A strains; and the nee
237            Ten (77%) of the 13 patients with ceftriaxone-resistant infections were aged 18 years or y
238                                          The ceftriaxone-resistant isolate from the child was indisti
239                                         Both ceftriaxone-resistant isolates were closely related to t
240                                         Both ceftriaxone-resistant isolates were resistant to 13 anti
241                                     Among 70 ceftriaxone-resistant pneumococcal isolates, all were in
242  plasmids and beta-lactamases to compare the ceftriaxone-resistant S. enterica serotype typhimurium f
243                        Domestically acquired ceftriaxone-resistant Salmonella has emerged in the Unit
244                Nationally, the prevalence of ceftriaxone-resistant Salmonella increased from 0.5% in
245                                Patients with ceftriaxone-resistant Salmonella infections between 1996
246                                              Ceftriaxone-resistant Salmonella infections have recentl
247                                         Most ceftriaxone-resistant Salmonella isolates had similar Am
248                            The prevalence of ceftriaxone-resistant Salmonella was 0.1% (1 of 1326) in
249  analyzed the molecular characteristics of a ceftriaxone-resistant strain of Salmonella enterica sero
250  From 2012, we have observed an emergence of ceftriaxone-resistant strains also showing reduced susce
251                           We also found that ceftriaxone restores glutamate reuptake and attenuates t
252 nd microdialysis to test the hypothesis that ceftriaxone restores the function of both GLT-1 and xCT
253 n that chronic treatment with the antibiotic ceftriaxone restores xCT and GLT-1 expression following
254 ecreased susceptibility to both cefixime and ceftriaxone rose between 2007 and 2010 but was more noti
255                               In particular, ceftriaxone significantly decreases the growth rate of a
256  LOLA (as an add-on therapy to lactulose and ceftriaxone) significantly improves the grade of OHE ove
257 n, Streptococcus pneumoniae, vancomycin, and ceftriaxone, Streptococcus agalactiae, ampicillin, and c
258 y related gonococcal isolates with decreased ceftriaxone susceptibility and high-level azithromycin r
259 were interviewed and isolates with decreased ceftriaxone susceptibility were further characterized.
260 several of which also demonstrated decreased ceftriaxone susceptibility.
261 more slowly in participants who received 4 g ceftriaxone than in those on placebo (difference 0.51 un
262 utamate transport in the accumbens core with ceftriaxone the capacity of acute stress to augment the
263                                Resistance to ceftriaxone, the drug of choice for invasive salmonella
264 en percent of the isolates were resistant to ceftriaxone, the drug of choice for treating salmonellos
265               Except for the last trial with ceftriaxone, the fluoroquinolone gatifloxacin was associ
266                     In cultures treated with ceftriaxone, the pH of the culture medium did not change
267  47 (98%) of 48 isolates were susceptible to ceftriaxone (third-generation cephalosporin), and 57 (93
268   Notably, all organisms were susceptible to ceftriaxone throughout the study period (2005-2014), and
269 CT and GLT-1 are essential to the ability of ceftriaxone to attenuate cue-induced reinstatement of co
270  June 2005 and 31 December 2010 who received ceftriaxone to determine whether the additional receipt
271 ctrophysiology to investigate the ability of ceftriaxone to normalize measures of synaptic plasticity
272 s were followed from first administration of ceftriaxone to occurrence of CDI or administrative closu
273 of both proteins in mediating the ability of ceftriaxone to prevent cue-induced reinstatement of coca
274 ine from 10 to 20 days or adding one dose of ceftriaxone to the beginning of a 10-day course of doxyc
275          By adding antibodies to antibiotic (ceftriaxone)-treated mice, we further reduce the bacteri
276  PCR for up to 56 days in aliquots from both ceftriaxone-treated and untreated cultures.
277                         We found that 5 d of ceftriaxone treatment following cocaine self-administrat
278  the ceftaroline treatment group than in the ceftriaxone treatment group (59 of 69 [85.5%] vs 48 of 7
279                                   Similarly, ceftriaxone treatment prevented stress-induced potentiat
280                        In addition, although ceftriaxone treatment resulted in a reduction in the qua
281                                     Finally, ceftriaxone treatment was associated with lasting upregu
282 nt, noncultivable Borrelia burgdorferi after ceftriaxone treatment was examined.
283                 In the absence of cocaine or ceftriaxone treatment, xCT knockdown in the NAc increase
284          We found a significant reduction in ceftriaxone use with a concomitant increase in targeted
285                        In this comparison of ceftriaxone versus oxacillin for MSSA osteoarticular inf
286  MSSA osteoarticular infections treated with ceftriaxone versus oxacillin.
287 ession with oral antibiotics (31 of 74 [42%] ceftriaxone vs 25 of 50 [50%] oxacillin; P = .4).
288 illin; P = .7) and >6 months (43 of 56 [77%] ceftriaxone vs 26 of 32 [81%] oxacillin; P = .6).
289 ss was similar at 3-6 months (50 of 60 [83%] ceftriaxone vs 32 of 37 [86%] oxacillin; P = .7) and >6
290 bo group (gastrointestinal, 245 of 340 [72%] ceftriaxone vs 97 of 173 [56%] placebo, p=0.0004; hepato
291      The outcome after 14-day treatment with ceftriaxone was favorable in 87.8% of patients.
292 rapy of intravenous daptomycin, rifampin and ceftriaxone was initiated.
293                                              Ceftriaxone was neuroprotective in vitro when used in mo
294   Although no resistance to ciprofloxacin or ceftriaxone was observed, 20 isolates (7%) were nalidixi
295    Additionally, under our study conditions, ceftriaxone was suboptimum in a high proportion of patie
296 d of care treatment (including lactulose and ceftriaxone) was given in both groups.
297 administration of the beta-lactam antibiotic ceftriaxone, which promotes synaptic glutamate clearance
298           Multidrug resistance, including to ceftriaxone, will pose further difficulty in management
299 ducibility of broth microdilution testing of ceftriaxone with N. cyriacigeorgica and N. wallacei, tig
300                                              Ceftriaxone with or without a macrolide antibiotic is a

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