ライフサイエンスコーパス: celiac sprue

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1 e therapeutic potential for the treatment of celiac sprue.

2 gy for oral peptidase supplement therapy for Celiac Sprue.

3 f a highly restricted diet for patients with celiac sprue.

4 t gastrointestinal bleeding in patients with celiac sprue.

5 ocking agents for the potential treatment of Celiac Sprue.

6 Seventy-eight patients with celiac sprue (59 women and 19 men) treated with a gluten

7 Celiac Sprue, a widely prevalent autoimmune disease of t

8 ore, diagnostic investigation of diarrhea in celiac sprue after treatment seems warranted.

9 ility of the gastrointestinal tract, such as celiac sprue and scleroderma, and their relationship wit

10 n be detected in about half of patients with celiac sprue and should be added to the list of factors

11 plays a critical role in the pathogenesis of Celiac Sprue by binding to epitopes derived from dietary

12 HLA-DQ2 predisposes an individual to celiac sprue by presenting peptides from dietary gluten

13 genesis of various human disorders including celiac sprue, certain neurological diseases, and some ty

14 An analysis of 15 untreated celiac sprue (CS) patients demonstrated that approximate

15 bowel syndrome (IBS-D) may have undiagnosed celiac sprue (CS).

16 The majority of patients with celiac sprue experience diarrhea before diagnosis.

17 Endomysial antibodies indicate celiac sprue in asymptomatic patients, and multiple endo

18 Celiac sprue is a chronic disease, which usually occurs

19 Celiac sprue is a multifactorial disease characterized b

20 Celiac Sprue is an HLA DQ2 (or DQ8)-associated autoimmun

21 Celiac sprue is important to recognize and treat by glut

22 Iron deficiency complicating celiac sprue is usually attributed to the malabsorption

23 Celiac disease, also known as celiac sprue, is a gluten-induced autoimmune-like disord

24 olitis has been reported in association with celiac sprue, lymphoma, and hypogammaglobulinemia.

25 Celiac Sprue, or gluten-sensitive enteropathy, is an inh

26 work for understanding the role of tTGase in Celiac Sprue, our results lay the groundwork for the des

27 P as an oral therapeutic enzyme for treating celiac sprue, our results provide a strong foundation fo

28 e found in all food grains that are toxic to Celiac Sprue patients but are absent from all nontoxic f

29 esentation in the small intestinal mucosa of Celiac Sprue patients therefore represents a potentially

30 ary gluten in the small intestinal mucosa of celiac sprue patients, our findings reveal a new strateg

31 recognized by gluten-responsive T cells from celiac sprue patients.

32 +) T cells from small intestinal biopsies of Celiac Sprue patients.

33 or of the inflammatory response to gluten in Celiac Sprue patients.

34 gut-derived human T cell lines from 14 of 14 Celiac Sprue patients.

35 rial glutenases (proposed oral therapies for celiac sprue) were proteolyzed in the absence or presenc

36 tic steatorrhea; and 7 patients with treated celiac sprue who had normal intestinal histologic featur

37 After treatment of celiac sprue with a gluten-free diet, chronic diarrhea p

38 e transglutaminase, the major autoantigen in Celiac Sprue, with substantially greater selectivity tha

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