ライフサイエンスコーパス: cell antigen

1 g (Jedi) T cells enable visualization of a T-cell antigen.

2 esponse to HtrABb, indicating that it is a B-cell antigen.

3 gainst Her2/neu or CD24, a putative PAC stem cell antigen.

4 rin (PE), is a murine and human gammadelta T cell antigen.

5 bacteria is both a virulence factor and a B-cell antigen.

6 litis), and autoantibodies specific for beta-cell antigens.

7 eby activating immune responses against dead-cell antigens.

8 lenging to identify relevant peptide-based T cell antigens.

9 factor receptor alpha (PDGFR-alpha) and stem cell antigen 1 (Sca-1) have recently been identified as

10 mmat (ROR-gammat)(+)CD3(+)CD4(-)CD8(-), stem cell antigen 1 (Sca1)(+) entheseal resident T cells.

11 omparative gene expression profiling of stem cell antigen 1(-) (Sca1(-)) and Sca1(+) neonatal fibrobl

12 on, Stat5 activation, and expression of stem cell antigen 1.

13 l cell population that was positive for stem cell antigen 1.

14 the role of Toll-like receptor 4 (TLR4)/stem cell antigen-1 (Sca-1) signaling in this process, bacter

15 s in adult mouse myocardium all sharing stem cell antigen-1 (Sca1), based on side population (SP) phe

16 be documented by the down-regulation of stem cell antigen-1 and acquisition of responsiveness to gran

17 ny-forming unit progenitors coexpressed stem cell antigen-1 and CD45 and were adventitially located,

18 distinct from neighboring cells lacking stem cell antigen-1 or CD45 and contained a proliferative (Ki

19 ariable models revealed more blood dendritic cell antigen-1(+) (BDCA-1(+)) myeloid dendritic cells (m

20 expressed either SSEA-1(+) or Sca-1(+) (stem cell antigen-1).

21 ontained abundant stem cells expressing stem cell antigen-1, Wilms' tumor 1 (WT-1), and CD34, suggest

22 oE(-/-) mice and localized primarily to stem cell antigen-1-positive cells in the adventitia.

23 cells and is selectively enriched with stem cell antigen-1-positive monocyte/macrophage precursors.

24 LSK (lineage-negative, stem cell antigen-1-positive, c-Kit-positive) cells were quan

25 cell (MSC) markers, including CD105 and stem-cell antigen-1.

26 (SPC)(+) alveolar type 2 cells and in Clara cell antigen 10 (CC10)(+) Clara cells by use of cell-typ

27 Blood dendritic cell antigen 2 (BDCA-2; also designated CLEC4C or CD303)

28 We tested bone marrow stromal cell antigen 2 (BST-2) gene variants rs3217318, a 19-bas

29 sm of the host restriction factor BM stromal cell antigen 2 (BST-2).

30 tif 5alpha (TRIM5alpha), bone marrow stromal cell antigen 2 (BST-2)/tetherin, and certain apolipoprot

31 ypeptide 3 (APOBEC3) and bone marrow stromal cell antigen 2 (BST-2/tetherin/CD317) retroviral restric

32 Bone marrow stromal cell antigen 2 (BST2) is a cellular restriction factor w

33 Sca2 (surface cell antigen 2) is the only bacterial protein known to p

34 ransposon mutagenesis, we identified surface cell antigen 4 (Sca4) as a secreted effector of spread t

35 n (Ig) molecules that efficiently capture ss-cell antigens allows autoreactive B-lymphocytes bypassin

36 lex of the prototypical invariant NKT (iNKT) cell antigen alphaGalCer (KRN7000) bound to mouse CD1d h

37 of the NAb targets gD and gB and the novel T cell antigen and tegument protein UL40, and we compared

38 f inflammatory T-cell responses to apoptotic cell antigens and failure of long-tolerance induction.

39 These data suggest that stem-cell antigens and PD-L1 may be targeted for immunopreven

40 eated a novel category of tumor-associated T-cell antigens based on their exclusive and frequent repr

41 ch is expressed on immature B cells as the B-cell antigen-binding receptor (BCR).

42 e protein encoded by the Bone Marrow Stromal Cell Antigen (BST2) known to play an inhibitory role in

43 importance of specific immunity to malignant cell antigens by previous authors.

44 f self-reactive CD8(+) T cells, suggesting B-cell antigen capture and presentation are critical in vi

45 oid cell or with dedifferentiated Langerhans cell antigens, carry the BRAFV600E mutation.

46 xpression correlated with expression of stem cell antigen CD123.

47 meric antigen receptor (CAR) targeting the B-cell antigen CD19.

48 meric antigen receptor (CAR) targeting the B-cell antigen CD19.

49 ial cell adhesion molecule (EpCAM) and the T-cell antigen CD3, is approved as intraperitoneal therapy

50 tion of proteins mucin-5AC and prostate stem-cell antigen could identify high-grade dysplasia/cancer

51 unctions empowered by specific delivery of T cell antigens could be harnessed for tolerance induction

52 beta-Cell antigen delivered to DCIR2(+) DCs delayed diabetes

53 we describe the first report of a human mast cell antigen-dependent passive systemic anaphylaxis resp

54 alization-traits associated with increased B cell antigen drive.

55 d that pDCs accumulated at sites of CD8(+) T cell antigen-driven activation in a CCR5-dependent fashi

56 n epitope that is an immunodominant CD8(+) T cell antigen during primary infection of C57BL/6 mice wi

57 be broadly applicable to the selection of T cell antigens for inclusion in candidate vaccines for ma

58 face receptors (CD19-22) to immunodominant T-cell antigens from EBV proteins, including EBNA1, EBNA3B

59 e to Sphingomonas bacteria, which carry iNKT cell antigens, fully established phenotypic maturity of

60 Nevertheless, current T cell antigen identification approaches do not discern wh

61 rived 3beta, 14-3-3 sigma, and prostate stem cell antigen immunochemistry.

62 ient to initiate expression of smooth muscle cell antigens in ASC, only activin A IgG blocked the eff

63 physics, and functional assays to identify T-cell antigens in long-term survivors of pancreatic cance

64 ired by DCs to cross-prime CTLs against dead cell antigens in mice.

65 The overall role of modification of beta-cell antigens in type 1 diabetes has not been elucidated

66 The immunodominant CD4 T-cell antigens included both long proteins abundantly pre

67 The most powerful iNKT cell antigen is alpha-galactosylceramide (alpha-GalCer),

68 Autoimmune responses to meiotic germ cell antigens (MGCA) that are expressed on sperm and tes

69 d with (124)I-labeled A11 anti-prostate stem cell antigen minibody.

70 g the expression of the neuroectodermal stem cell antigen, nestin, and up-regulating the glial matura

71 ns from 1:100 to 1:102,400 using crude whole-cell antigens of the Karp, Kato, and Gilliam strains of

72 entation of established myeloma-associated T-cell antigens on the HLA ligandome level, we found a sub

73 ET data of mice implanted with prostate stem cell antigen-overexpressing tumors and injected with (12

74 emerged as a critical regulator of dendritic cell antigen presentation, but its role in T-cell immuni

75 echanism of protection may involve lack of B cell antigen presentation, which impairs T-cell activati

76 cyte to dendritic cell differentiation and T-cell antigen presentation.

77 e formation, subsequently causing unstable T cell-antigen-presenting cell (APC) conjugates.

78 antigen is relatively low, the avidity of T cell-antigen-presenting cell interactions is greatly enh

79 en shown to induce activation of endothelial cells, antigen-presenting cells, and platelets, resultin

80 s2294008, on 8q24.3 within the prostate stem cell antigen (PSCA) gene, as a risk factor for bladder c

81 studies have demonstrated that prostate stem cell antigen (PSCA) is an attractive target for immunoth

82 Prostate stem cell antigen (PSCA) is expressed on the cell surface in

83 We report that the radial glial cell antigen RC2 identifies the majority of proliferatin

84 Examination of lymph node cell antigen recall responses identified elevated immune

85 Nfkb1(SSAA) mutation decreased B cell antigen receptor (BCR) activation of NF-kappaB in F

86 RK activity and CLL proliferation required B cell antigen receptor (BCR) activation, as inhibition of

87 respond to antigens by engagement of their B-cell antigen receptor (BCR) and of coreceptors through w

88 es encoding the variable (V) region of the B-cell antigen receptor (BCR) are assembled from V, D (div

89 obial molecules, enhance signalling by the B-cell antigen receptor (BCR) by activating the actin-seve

90 Mature B cells coexpress both IgM and IgD B-cell antigen receptor (BCR) classes, which are organized

91 milar to resting mature B cells, where the B-cell antigen receptor (BCR) controls cellular survival,

92 B-cell antigen receptor (BCR) expression is a key feature

93 Binding of antigen to the B cell antigen receptor (BCR) initiates a multitude of eve

94 cytosolic Ca(2+) following ligation of the B cell antigen receptor (BCR) led to the assumption that t

95 Kinases downstream of B-cell antigen receptor (BCR) represent attractive targets

96 B cell activation is regulated by B cell antigen receptor (BCR) signaling and antigen intern

97 kinase (Erk) activation mediated by tonic B-cell antigen receptor (BCR) signaling and that this sign

98 Notch2 and B cell antigen receptor (BCR) signaling determine whether

99 The strength of B cell antigen receptor (BCR) signaling in response to ant

100 ism of Lin28b action nor the importance of B cell antigen receptor (BCR) signaling in this process wa

101 B cell antigen receptor (BCR) signaling is critical for B

102 In B-lymphocytes, the down-regulation of B-cell antigen receptor (BCR) signaling is critical for su

103 egulating tonic, but not antigen-mediated, B-cell antigen receptor (BCR) signaling through modulation

104 are selected for an intermediate level of B-cell antigen receptor (BCR) signalling strength: attenua

105 Nck bound directly to the B cell antigen receptor (BCR) via the non-immunoreceptor t

106 kinase is essential for signaling from the B cell antigen receptor (BCR), and thus for antibody respo

107 IRF8 dampened signaling via the B cell antigen receptor (BCR), facilitated antigen-specifi

108 ell development requires expression of the B cell antigen receptor (BCR), it remains unclear whether

109 into cytoplasmic signaling events through B-cell antigen receptor (BCR)-based signalosomes at the B-

110 B-cell antigen receptor (BCR)-mediated signaling plays a c

111 on compromises the activity of the pivotal B-cell antigen receptor (BCR)-proximal effector spleen tyr

112 ignaling pathways, some emanating from the B-cell antigen receptor (BCR).

113 ng from BAFFR, a receptor for BAFF and the B cell antigen receptor (BCR).

114 ok-3) attenuates signals transduced by the B cell antigen receptor (BCR).

115 receptor (IL-7R) and the precursor to the B cell antigen receptor (pre-BCR) in B lymphopoiesis has n

116 gnaling components of the precursor to the B cell antigen receptor (pre-BCR), including defects in BL

117 rough the pre-B stage triggered by the pre-B-cell antigen receptor (pre-BCR).

118 Signaling via the pre-T cell antigen receptor (pre-TCR) and the receptor Notch1

119 ) following suboptimal stimulation via the T cell antigen receptor (TCR) (induced T(reg) cells (iT(re

120 The locus encoding the T cell antigen receptor (TCR) alpha-chain and delta-chain

121 tions further differed in their use of the T cell antigen receptor (TCR) alpha-chain variable region

122 The interaction between the T cell antigen receptor (TCR) and antigenic peptide in com

123 acted in synergy with stimulation via the T cell antigen receptor (TCR) and coreceptor CD28 to accel

124 We found that signaling via the T cell antigen receptor (TCR) and cytokine stimulation pro

125 Signals through the T-cell antigen receptor (TCR) and interleukin-2 (IL-2) pro

126 olecules associated with activation of the T cell antigen receptor (TCR) and of immunological-checkpo

127 The expression of Myc is regulated by the T cell antigen receptor (TCR) and pro-inflammatory cytokin

128 d directly with the signal strength of the T cell antigen receptor (TCR) and required the coreceptor

129 ganization of CD4, and its relationship to T-cell antigen receptor (TCR) and the active form of Src k

130 h ITAM multiplicity) in the complex of the T cell antigen receptor (TCR) and the invariant signaling

131 We found that clonotypes expressing the T cell antigen receptor (TCR) beta-chain variable region 1

132 osphatase PTPN22 limited signaling via the T cell antigen receptor (TCR) by weak agonists and self an

133 Variable strengths of signaling via the T cell antigen receptor (TCR) can produce divergent outcom

134 nction correlates with genomically encoded T cell antigen receptor (TCR) chains, which suggests that

135 This was associated with distinct T cell antigen receptor (TCR) clonotypes, characterized by

136 The T-cell antigen receptor (TCR) complex contains 10 copies o

137 Interactions driven by the T cell antigen receptor (TCR) determine the lineage fate o

138 mplex (MHC) proteins are recognized by the T cell antigen receptor (TCR) dictates the homeostasis of

139 In the thymus, low-affinity T cell antigen receptor (TCR) engagement facilitates posit

140 T cell antigen receptor (TCR) engagement has been shown to

141 The interaction between the T cell antigen receptor (TCR) expressed by natural killer

142 ypes generally show simplified patterns of T cell antigen receptor (TCR) expression, rapid effector r

143 Crystal structures of a T cell antigen receptor (TCR) from a MAIT cell in complex

144 was strongly induced by activation of the T cell antigen receptor (TCR) in a pathway involving calci

145 nterleukin 17 (IL-17), but the role of the T cell antigen receptor (TCR) in this developmental proces

146 The T cell antigen receptor (TCR) is a multisubunit receptor o

147 The T-cell antigen receptor (TCR) is an assembly of eight type

148 Although heightened signaling via the T cell antigen receptor (TCR) is critical for the differen

149 Signaling via the T cell antigen receptor (TCR) is initiated by Src-family k

150 The self-reactivity of their T-cell antigen receptor (TCR) is thought to contribute to

151 The T cell antigen receptor (TCR) is unique in that its affini

152 requires interleukin 2 (IL-2) and agonist T cell antigen receptor (TCR) ligands and is controlled by

153 ) microscopy showed that signaling via the T cell antigen receptor (TCR) occurred during synapse tran

154 lection, which requires recognition by the T cell antigen receptor (TCR) of complexes of self peptide

155 The T cell antigen receptor (TCR) on the surface of T cells is

156 The semi-invariant MAIT T-cell antigen receptor (TCR) recognises riboflavin and fo

157 How the T cell antigen receptor (TCR) recognizes these cations bou

158 ocesses, but its role in signaling via the T cell antigen receptor (TCR) remains unknown.

159 ) characterized by either a semi-invariant T cell antigen receptor (TCR) repertoire (type I NKT cells

160 Here we show that the T-cell antigen receptor (TCR) repertoire of intestinal TH1

161 RNA-seq for assessing T-cell clonality and T-cell antigen receptor (TCR) repertoire of the neoplastic

162 tors and cell-intrinsic factors related to T cell antigen receptor (TCR) signal quantity and quality.

163 ng the nature of molecules involved in the T cell antigen receptor (TCR) signal transduction network,

164 deficiencies in thymic development and in T cell antigen receptor (TCR) signal transduction, in PTKs

165 ound that calcineurin was recruited to the T cell antigen receptor (TCR) signaling complex, where it

166 T cell antigen receptor (TCR) signaling drives distinct re

167 CD8(+) T cells in the thymus requires that T cell antigen receptor (TCR) signaling end in time for cy

168 immunity and requires a limited degree of T cell antigen receptor (TCR) signaling in response to sel

169 T cell antigen receptor (TCR) signaling in the thymus init

170 T-cell antigen receptor (TCR) signaling is essential for a

171 -reactivity, with a particular emphasis on T cell antigen receptor (TCR) signaling thresholds.

172 catalytic activity of Zap70 is crucial for T cell antigen receptor (TCR) signaling, but the quantitat

173 To investigate how Csk activity regulates T cell antigen receptor (TCR) signaling, we utilized a mou

174 ctivator of the RAS-MAPK pathway following T cell antigen receptor (TCR) signaling.

175 Mutations in T-cell antigen receptor (TCR) subunit genes cause rare imm

176 sensor of thresholds for signaling via the T cell antigen receptor (TCR) that was essential for T cel

177 The ability of a single T cell antigen receptor (TCR) to cross-react with multiple

178 Classically, signal transduction from the T cell antigen receptor (TCR) to ERK is thought to be regu

179 ever, the mechanisms that allow the clonal T cell antigen receptor (TCR) to functionally engage multi

180 A and Foxp1D induced by stimulation of the T cell antigen receptor (TCR) to inhibit the generation of

181 nd the tyrosine phosphatase CD45 underpins T cell antigen receptor (TCR) triggering, but how such seg

182 T cell proliferation is initiated by T cell antigen receptor (TCR) triggering, soluble growth f

183 played enhanced proximal signaling via the T cell antigen receptor (TCR) without an effect on the act

184 ting THEMIS in signaling downstream of the T cell antigen receptor (TCR), but the mechanistic underpi

185 Upon recognition of antigen by the T cell antigen receptor (TCR), roquin and regnase-1 protei

186 We demonstrate that upon ligation of the T-cell antigen receptor (TCR), the TCR associates with and

187 n T cells in response to engagement of the T cell antigen receptor (TCR), which induced expression of

188 activation of specific T cells through the T cell antigen receptor (TCR)-CD3 complex.

189 Thus, BATF amplifies T cell antigen receptor (TCR)-dependent expression of tran

190 We found that T cell antigen receptor (TCR)-driven signaling initially d

191 anistically, we demonstrate a link between T cell antigen receptor (TCR)-induced asymmetric expressio

192 operated Ca(2+) channel and contributed to T cell antigen receptor (TCR)-induced Ca(2+) influx, TCR s

193 The T cell antigen receptor (TCR)-peptide-major histocompatibi

194 igen-presenting molecules by the alphabeta T cell antigen receptor (TCR).

195 ative activation pathway downstream of the T cell antigen receptor (TCR).

196 T cells was regulated by pathogens and the T cell antigen receptor (TCR).

197 ling via both interleukin 7 (IL-7) and the T cell antigen receptor (TCR).

198 ing aberrant proliferation mediated by the T cell antigen receptor (TCR).

199 T cell effector functions require the T cell antigen receptor (TCR).

200 cell proliferation and stimulation via the T cell antigen receptor (TCR).

201 nducible interaction between ZAP70 and the T cell antigen receptor (TCR)/CD3 complex.

202 ouble-negative thymocytes positive for the T cell antigen receptor alphabeta subtype (TCRalphabeta) t

203 We identified Notch, the T cell antigen receptor and c-Myc as key controllers of T

204 Akt is known to be activated by the T-cell antigen receptor and the cytokine IL-2, but its rol

205 is a critical mediator of signaling by the T cell antigen receptor and the principal costimulatory re

206 eta was significantly lower than with anti-T-cell antigen receptor antibodies (Abs).

207 odissection, transcriptional profiling and T-cell antigen receptor beta-chain (TCRbeta) genotyping on

208 s to tyrosine kinases and may regulate the T-cell antigen receptor biological activities for this cel

209 f immature B cells after engagement of the B cell antigen receptor by suppressing the expression of t

210 th human T-lymphotropic virus type 1; (ii) T cell antigen receptor clonotype repertoires; and (iii) m

211 ns affecting Toll-like receptor signaling, B-cell antigen receptor coreceptors (eg, CD19), or enzymes

212 T cell antigen receptor delta (Tcrd) variable region exons

213 findings indicate that the affinity of the T cell antigen receptor for self antigen drives the differ

214 The T-cell antigen receptor is an assembly of eight single-pas

215 ed pMHC-II reveals the organization of the T cell antigen receptor ligand on the DC surface.

216 A diverse T cell antigen receptor repertoire in progenitor-deprived

217 TEC) is essential for generating a diverse T cell antigen receptor repertoire tolerant to self-antige

218 munocompetent T cells with a self-tolerant T cell antigen receptor repertoire.

219 We find that components of T cell antigen receptor signal machinery and several key t

220 The initiation of T cell antigen receptor signaling is a key step that can r

221 mocyte positive selection by enhancing the T cell antigen receptor signaling response to low-affinity

222 ical role in the initiation of B1 but not B2 cell antigen receptor signaling.

223 Previous work using B cell antigen receptor transgenic animals suggested that

224 AT) is a critical signaling hub connecting T cell antigen receptor triggering to downstream T cell re

225 Herein, we report that ablation of B cell antigen receptor ubiquitination in vivo uncouples t

226 ors display altered alpha3135-145-specific T-cell antigen receptor usage, HLA-DR15-alpha3135-145 tetr

227 ll malignancies depend on signals from the B-cell antigen receptor, and Bruton tyrosine kinase (BTK)

228 anced responses to weak stimulation of the T cell antigen receptor, when transferred into lymphopenic

229 ESCRTs) at the plasma membrane to generate T-cell antigen receptor-enriched microvesicles.

230 T cell antigen receptor-proximal signaling components, Rho

231 that are activated after engagement of the T cell antigen receptor.

232 led by signaling pathways initiated by the T cell antigen receptor.

233 s from the cancer microenvironment and the B cell antigen receptor.

234 reg cells separable from signaling via the T cell antigen receptor.

235 in signalling complexes downstream of the T-cell antigen receptor.

236 or gene loci and not by the specificity of T-cell antigen receptor/coreceptor signalling.

237 ting MR1-Ag complexes are recognized by MAIT cell antigen receptors (alphabeta T cell receptors (TCRs

238 s by selection of somatically hypermutated B cell antigen receptors (BCR) on immune complexes (ICs).

239 sors of these antibodies act as functional B-cell antigen receptors (BCRs) that initiate subsequent a

240 ival and transcription of IgG2a-containing B cell antigen receptors (BCRs).

241 l survival relies on signals transduced by B-cell antigen receptors (BCRs).

242 ed glycolipid antigens (Ags), with their NKT cell antigen receptors (NKT TCRs) exhibiting reciprocal

243 k to generate T cells expressing alphabeta T cell antigen receptors (TCR).

244 sponses are driven by interactions between T cell antigen receptors (TCRs) and complexes of peptide a

245 In mice, iNKT cells express T cell antigen receptors (TCRs) comprising a unique TCRalp

246 d molecules are subsequently recognized by T cell antigen receptors (TCRs) expressed on the surface o

247 T cells bearing gammadelta T cell antigen receptors (TCRs) function in lymphoid stres

248 Studies of individual T cell antigen receptors (TCRs) have shed some light on st

249 estion is whether an altered repertoire of T cell antigen receptors (TCRs) is associated with such ex

250 I-restricted self antigens by autoreactive T cell antigen receptors (TCRs) is established, but how au

251 Human T cell antigen receptors (TCRs) pair in millions of combin

252 The binding of T cell antigen receptors (TCRs) to specific complexes of p

253 Interactions of T cell antigen receptors (TCRs) with complexes of self pep

254 The interaction of alphabeta T-cell antigen receptors (TCRs) with peptides bound to MHC

255 spond rapidly to stress despite expressing T cell antigen receptors (TCRs), a hallmark of adaptive im

256 ate response is dependent on IL-33 but not T cell antigen receptors (TCRs).

257 scence, and next-generation sequencing for T-cell antigen receptors (TCRs).

258 elease for Zap70 kinases at phosphorylated T cell antigen receptors (TCRs).

259 s is controlled by tonic signaling through T cell antigen receptors and common gamma chain cytokine r

260 lymphocyte-like cells that lack T cell or B cell antigen receptors and mediate protective and repair

261 g the balance of persistent stimulation of T-cell antigen receptors and specific CD2-induced co-stimu

262 Gnathostomes use T- and B-cell antigen receptors belonging to the immunoglobulin s

263 Activation of T cell antigen receptors induced expression of pro-IL-1bet

264 known to provide a confined space in which B-cell antigen receptors undergo selection.

265 olvent-exposed and therefore accessible to T-cell antigen receptors were predicted to be immunogenic.

266 important class of immune receptors (e.g., T-cell antigen receptors) whose ligands are anchored to th

267 heckpoint inhibition, mAbs, and engineered T-cell antigen receptors, the incidence and pattern of mal

268 and involving synergistic stimulation via B-cell antigen receptors, toll-like receptor 7 (TLR7), and

269 cells, enriched for self antigen-specific T cell antigen receptors, was also present in healthy host

270 l now was thought to be mediated mainly by B cell antigen receptors.

271 s, peptides presented by HLA molecules and T-cell antigen receptors.

272 HC) restriction is the cardinal feature of T cell antigen recognition and is thought to be intrinsic

273 eptor (TCR) represents the central step in T cell antigen recognition.

274 x (MHC) restriction is a unique feature of T cell antigen recognition.

275 of the most noted haptens, are gammadelta T cell antigens, recognized directly by specific gammadelt

276 eralizable strategy for identifying the Treg cell antigens relevant to human autoimmunity.

277 n type 1 diabetes, loss of tolerance to beta-cell antigens results in T-cell-dependent autoimmune des

278 for the highly conserved rickettsial surface cell antigen, rOmpB, and suggest that the ability to eva

279 Microfold (M) cells, antigen-sampling cells in the intestine, were pre

280 Stem cell antigen Sca-1 is implicated in murine cancer stem c

281 Stem cell antigen (Sca) -1 is a member of the Ly6 gene family

282 2B adenosine receptors on mouse cardiac stem cell antigen (Sca)-1(+)CD31(-) mesenchymal stem-like cel

283 Dynamic tuning of CD8(+) T cell antigen sensitivity by inflammatory cytokines most

284 T-cell immunity against stem-cell antigen SOX2 and preserved humoral responses at stu

285 ntigen-specific effector CD8(+) and CD4(+) T cells, antigen-specific cytokine release, inhibition of

286 tion of antigen-specific follicular helper T cells, antigen-specific GC B cells, and high-affinity cl

287 e on development and function of human CD4 T cells, antigen-specific human CD8 T cells, and immunoglo

288 ) lymphocyte-activation gene 3 (LAG3)(+) TR1 cells, antigen-specific proliferative responses, and cyt

289 Herein, we discuss different forms of NK cell antigen specificity and how these responses may be

290 le in animal models of UL40, a novel HSV-2 T cell antigen that has been correlated with asymptomatic

291 Here, we report that a noted B cell antigen, the algae protein phycoerythrin (PE), is a

292 CD8(+) T-cell responses against islet cell antigens, thought to play a central role in diabete

293 Our data describe a novel aspect of memory T cell antigen threshold sensitivity that may critically r

294 DCs present cancer cell antigens to CD4(+) T cells through Jak-STAT signal

295 the identification of sufficient discrete T-cell antigens to develop subunit vaccines that produce s

296 e virus (RHDV) for presentation of foreign B-cell antigens to the immune system.

297 ive, as expected, while few novel dominant T cell antigens were IgE reactive.

298 ies specific to CD24, a putative cancer stem cell antigen, were effective against PAC xenografts that

299 cells (EBV(+) in >90% of cells) expressed B-cell antigens, were often CD30 and PD-L1 positive, and s

300 GD3 can serve as a natural killer T (NKT) cell antigen when presented on CD1d molecules expressed