ライフサイエンスコーパス: cell debris

1 This causes a buildup of unengulfed cell debris.

2 capable of phagocytizing extracellular lens cell debris.

3 ced by low doses of cytotoxic agents, clears cell debris.

4 in response to self-DNA, self-RNA, and dead cell debris.

5 d CCL5, by human macrophages stimulated with cell debris.

6 tal rippling is a result of scavenging lysed cell debris.

7 ddition to congestion of the swim bladder by cell debris.

8 enveloped viruses from microvesicles and/or cell debris.

9 d and engulfment of unicellular organisms or cell debris.

10 ced between the co-precipitated proteins and cell debris.

11 After removal of cells and cell debris, a 0.50 mL sample (e.g., cell culture medium

12 diated by dendritic cell uptake of apoptotic cell debris and associated nucleic acids, whereas the su

13 n, buffer exchange, and selective removal of cell debris and by-products of the reactions.

14 end products (RAGE), inhibited responses to cell debris and conditioned medium and reduced the numbe

15 of whole, intact apoptotic cells, as well as cell debris and foreign organisms to which these molecul

16 Instead, DCs capture B cell debris and generate T cells specific for EBV latenc

17 loss of collagen and matrix, accumulation of cell debris and intense intimal inflammation.

18 nt on oxidized LDL (OxLDL), apoptotic cells, cell debris and modified proteins in the vessel wall, ac

19 taminants, the particle size distribution of cell debris and the physical properties of the resultant

20 Moreover, the removal of necrotic cell debris and the regeneration of endothelial cell str

21 lmonary vascular congestion, edema, necrotic cell debris, and gross inflammatory infiltration when co

22 chemokines, injury-conditioned medium, dead cell debris, and high mobility group box chromosomal pro

23 tissues, where they ingest and degrade dead cells, debris, and foreign material and orchestrate infl

24 tides were overproduced and solubilized from cell debris by denaturation and refolding.

25 show that ischemic cell death and uptake of cell debris by macrophages in the heart fuel a fatal res

26 led fibrocytes, and promotes phagocytosis of cell debris by macrophages.

27 antly enhanced the phagocytosis of apoptotic cell debris by monocyte-derived cells.

28 of cancer cells followed by phagocytosis of cell debris by MPhi.

29 Antigens derived from apoptotic cell debris can drive clonal T-cell deletion or anergy,

30 Cell debris can elevate IOP during the initial culture p

31 Significant levels of cell degeneration and cell debris, characteristic of necrosis, were observed i

32 d phagocytosis pathway and that internalized cell debris co-localizes with alphaVbeta5 and with RAB7

33 al cell debris persisted for months, granule cell debris disappeared rapidly.

34 Abnormally liberated iris pigment and cell debris enter the ocular drainage structures, leadin

35 ignificantly more silver was present in the "cell debris" fraction (known to contain the cell wall an

36 a peptide deposits from the normal brain and cell debris from injured brain tissue.

37 Dendritic cells exposed to cell debris from tumors expressing mEGP are similarly co

38 containing mucin goblets together with other cell debris, further indicating apoptosis of the goblet

39 th AIM in facilitating the clearance of dead cell debris in injured kidney, which is a key response i

40 junctival or subretinal haemorrhage and mild cell debris in the anterior vitreous) were generally mil

41 that enhancing endogenous clearance of tumor cell debris is a new therapeutic target that may complem

42 s intact even after multiple wounding, while cell debris is simultaneously removed using laminar flow

43 Whereas pyramidal cell debris persisted for months, granule cell debris di

44 ng permanent magnet, Further, removal of the cell debris, proteins, and carbohydrates was done using

45 by chemotherapy or targeted therapy ("tumor cell debris") stimulate primary tumor growth when coinje

46 e lens could result in accumulation of toxic cell debris that could contribute to UV light-induced ca

47 r cells, yet it simultaneously creates tumor cell debris that may stimulate inflammation and tumor gr

48 which results in the build up of a plaque of cell debris that severely impairs feeding.

49 ly high-pressure cultures revealed scattered cell debris throughout the meshwork in greater amounts t

50 Given the importance of clearing apoptotic cell debris to prevent inappropriate exposure of TLRs to

51 ium and delivers it along with the apoptotic cell debris to the lysosomal compartment.

52 examined host cell types and accumulation of cell debris were observed in infections with the human i

53 have posterior nuclear cataracts composed of cell debris, whereas the remaining fiber cells appear ge

54 d mainly concentrated around broken cells or cell debris with floating open ends, eDNAs produced via