ライフサイエンスコーパス: cell disruption

1 ive rise to the UPB precursor membranes upon cell disruption.

2 to virtual homogeneity within a few hours of cell disruption.

3 ility disorder associated with enteric glial-cell disruption.

4 s in combination with high pressure nitrogen cell disruption.

5 h bacterial invasion, colonization, and host cell disruption.

6 ciliary beat frequency and caused ependymal cell disruption.

7 cell-envelope proteins without experimental cell disruption.

8 5AC mucin transcription only after bacterial cell disruption.

9 a carrier protein and is released only upon cell disruption.

10 epresentative strains that respond poorly to cell disruption.

11 exes were sensitive to acid washing prior to cell disruption and caveolae enrichment.

12 onist BK or the antagonist NPC17731 prior to cell disruption and caveolae enrichment.

13 n contrast, the Zaire GP induced endothelial cell disruption and cytotoxicity in both nonhuman primat

14 tionally, the new extraction method combines cell disruption and extraction in one step, and the appr

15 PKD activation was maintained during cell disruption and immunopurification and was associate

16 mproved, and in particular the procedures of cell disruption and storage were optimized.

17 lassical autolysis, ultrasound led to a high cell disruption, and after 20h of ultrasonic treatment,

18 en by thorough optimization of cell sorting, cell disruption, and PCR conditions.

19 e state after stroke and identify systemic B-cell disruption as a target for therapeutic manipulation

20 was determined to be a lytic virus, causing cell disruption beginning at 30 hpi.

21 After fermentation and cell disruption, BoNT/A(Hc) was purified by using a thre

22 The initial step in this technique involves cell disruption by homogenization, followed by clearance

23 ic disruption reagents and note that genetic cell disruption can be used for functional analyses of s

24 cterized by lipid accumulation that leads to cell disruption, can be found in hair follicle-associate

25 activity of a second gene, mec-6, rendering cell disruption conditionally dependent on genetic backg

26 o tissue type due to varying efficiencies of cell disruption during organelle isolation procedures or

27 Here we report on genetically mediated cell disruption effected by the toxic Caenorhabditis ele

28 ting with small CM indentations, which after cell disruption give rise to a membrane fraction that ca

29 rapid DNA isolation method using high-speed cell disruption (HSCD) incorporating chaotropic reagents

30 ure change just below that which would cause cell disruption leading to death.

31 After cell disruption, M-phase Golgi was recovered in two size

32 st that the changes in selectivity seen with cell disruption may be due to a loss of cellular factors

33 Ultra Turrax was the best cell disruption method, and refrigeration was the best p

34 en cavitation) with those obtained using the cell-disruption method employed by others (homogenizatio

35 hat seem to leak from acidic organelles upon cell disruption, microspheres are retained within these

36 Furthermore, acid washing of cells prior to cell disruption minimally affected the CLR-associated B2

37 treated human lung microvascular endothelial cells, disruption of B1R-CPM heterodimers inhibited B1R-

38 Compared to control cells, disruption of PRC2 in HCMV-infected THP1 or NT2D1

39 In normal NIH 3T3 cells, disruption of Stat3 signaling with dominant-negat

40 d organization of actin filaments in Sertoli cells, disruption of the blood-testis barrier and sperma

41 In native cells, disruption of the C terminus-syntrophin gamma 2 P

42 In intact yeast cells, disruption of the TRM9 gene results in the comple

43 replication in tumor-associated endothelial cells, disruption of tumor blood flow, and hypoxia withi

44 ed and membrane-bound proteins in eukaryotic cells, disruption of which is the basis of the congenita

45 h a novel mechanism mitigated in transformed cells: disruption of proteasome-targeted degradation of

46 as hyperphosphorylated in PTP-PEST-deficient cells; disruption of the c-Abl-PSTPIP1-PEST-type PTP ter

47 han other stimuli that decrypt TF, including cell disruption, TF pathway inhibitor inhibition, and ca

48 tection of microorganisms requires microbial cell disruption to release nucleic acids.

49 iscs for cancer treatment through mechanical cell disruption under an applied rotating magnetic field

50 Cell disruptions using ultrasonic energy transmitted thr

51 Host cell disruption was measured quantitatively by a trypan

52 s noted around the iStent inject head and SC cell disruption was observed at the iStent inject insert

53 displacement close to but below the point of cell disruption yielded calibrated signal amplitudes as