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1 EE788 prevents entry into the S-phase of the cell division cycle.
2 htly coupled to a single complete eukaryotic cell division cycle.
3 both centrioles and centrosomes in a single cell division cycle.
4 ant volume fluctuations that occur over each cell division cycle.
5 maker is not influenced by the status of the cell division cycle.
6 restricting DNA replication to once in every cell division cycle.
7 romosome replication and does not follow the cell division cycle.
8 tion of cell division starting at the second cell division cycle.
9 plays important roles in the control of the cell division cycle.
10 tion of the S phase program in the mammalian cell division cycle.
11 rescentus that controls an early step in the cell division cycle.
12 that is responsible for re-initiation of the cell division cycle.
13 complex on chromatin during G1 phase of the cell division cycle.
14 ribosome synthesis and specific steps in the cell division cycle.
15 e p27 IDR controls entry into S phase of the cell division cycle.
16 at did not affect the G1/S transition of the cell division cycle.
17 ses (Cdks) are the central regulators of the cell division cycle.
18 genesis is likely to be under control of the cell division cycle.
19 at the core of the machinery that drives the cell division cycle.
20 try into, continuation of, and exit from the cell division cycle.
21 radation of p21, a negative regulator of the cell division cycle.
22 having progressed beyond mid-S phase in the cell division cycle.
23 origin to one round of DNA replication each cell division cycle.
24 tures of cells in the committed phase of the cell division cycle.
25 to chromatin isolated from each phase of the cell division cycle.
26 that would have normally withdrawn from the cell division cycle.
27 novel mechanism that exerts control over the cell division cycle.
28 he effects of oncogenic Ras on arrest of the cell division cycle.
29 e critical for the proper progression of the cell division cycle.
30 ntry into the DNA synthetic phase (S) of the cell division cycle.
31 block pRb(+) cells in the G(1)-phase of the cell division cycle.
32 ial for cell viability and regulation of the cell division cycle.
33 s either continue to proliferate or exit the cell division cycle.
34 nges characteristic of cells re-entering the cell division cycle.
35 lication machinery during the S phase of the cell division cycle.
36 ck entry into the DNA-synthetic phase of the cell division cycle.
37 incompletely understood events, arrests the cell division cycle.
38 he G1 to S phase transition of the mammalian cell division cycle.
39 not enter the DNA synthetic (S) phase of the cell division cycle.
40 inhibitor p27 is a negative regulator of the cell division cycle.
41 metabolites change significantly through the cell division cycle.
42 -oncogenes are thought to have a role in the cell division cycle.
43 types involves permanent withdrawal from the cell division cycle.
44 in the understanding of the machinery of the cell division cycle.
45 le in coordinating pre-mRNA splicing and the cell division cycle.
46 icting inappropriate entry of cells into the cell division cycle.
47 s important players in the regulation of the cell division cycle.
48 l role of RB in cell differentiation and the cell division cycle.
49 aggregation required progression through the cell division cycle.
50 ation involves permanent withdrawal from the cell division cycle.
51 ow metabolic fluxes are coordinated with the cell division cycle.
52 ptional activity per DNA copy throughout the cell division cycle.
53 tion of CDK1 expression had an impact on the cell division cycle.
54 sight into the regulation of CDK1 during the cell division cycle.
55 erwent reversible phosphorylation during the cell division cycle.
56 emble of interacting cells in a model of the cell division cycle.
57 olling periodic gene expression in the human cell division cycle.
58 unction in cytoskeletal organization and the cell division cycle.
59 ying microtubule cytoskeleton throughout the cell division cycle.
60 chromatin need to be duplicated during each cell division cycle.
61 other growth genes to promote entry into the cell division cycle.
62 y which cell growth is controlled during the cell division cycle.
63 ade and controlled through the course of the cell-division cycle.
64 s have transient interactions throughout the cell-division cycle.
65 tosis is the last critical decision during a cell-division cycle.
66 veloped that assumes expanding volumes and a cell-division cycle.
67 ecular link between the cilia life cycle and cell-division cycle.
68 tion of the centrosome is a key event in the cell-division cycle.
69 mutations that affect various aspects of the cell-division cycle.
70 th each other and with progression through a cell-division cycle.
71 iescent and, therefore, able to re-enter the cell-division cycle.
72 fore the morula stage after only two to four cell division cycles.
73 ycle times that emerge as 12-hr synchronized cell division cycles.
74 rochromatin is gradual and requires multiple cell division cycles.
75 sis of the aneuploid cells during subsequent cell division cycles.
76 rning circadian clock-dependent synchronized cell division cycles.
77 ith each other, resulting in circadian-gated cell division cycles.
78 protect chromosome ends from erosion during cell division cycles.
79 ion complex component, double-parked protein/cell division cycle 10-dependent transcript 1, is not on
80 conserved dual-specificity phosphatase human cell-division cycle 14A (hCDC14A) associates with the ac
82 roliferating cell nuclear antigen (PCNA) and cell division cycle 2 (cdc2) kinase elaborated significa
83 predicted a possible phosphorylation site by cell division cycle 2 (Cdc2), which directly phosphoryla
85 antigen, cyclin-dependent protein kinase 4, cell division cycle 2 kinase, DNA synthesis, and apoptos
86 g three genes, two nuclear (beta-tubulin and cell division cycle 2) and a gene from the plastid genom
88 k4, and cdk6 were induced during G1, whereas cell division cycle-2 (cdc2) was induced concomitantly w
92 s showed that induction of expression of the cell division cycle 20 gene (Cdc20), a key regulator of
95 e uncover the E3 ubiquitin ligase Cdc20-APC (cell division cycle 20-anaphase promoting complex) as a
97 G(2)/M phase, partially through induction of cell division cycle 25 (Cdc25) isoform C (Cdc25C) degrad
99 henotype that is linked to overexpression of cell division cycle 25 (Cdc25)A phosphatase and cell-cyc
101 phorylation of p27 at Thr187 was mediated by cell division cycle 25A (Cdc25A), confirmed using Cdc25A
105 ents also decreased the levels of Cdc25B and cell division cycle 25C (Cdc25C) phosphatases with an in
107 on of the let-7 target cell cycle regulators cell division cycle 34 (Cdc34) and E2F transcription fac
111 at shock protein 90) and its cofactor Cdc37 (cell division cycle 37 protein) are crucial to prevent t
113 s regulated by SRC-like adaptor 2 (Sla2) and cell division cycle 42 (Cdc42) independently of Sla2's r
118 GTPases have suggested that reduced Duo and cell division cycle 42 (Cdc42) transcript expression is
119 his idea, altered expression of genes in the cell division cycle 42 (CDC42)-CDC42 effector protein (C
121 the nascent axon, and the Rho GTPase Cdc42 (cell division cycle 42) activates the mPar6alpha/Par3 (P
125 s of the Rho family, including Rho, Rac, and cell division cycle 42, regulate the actin cytoskeleton.
127 demonstrate that T cell-specific deletion of cell-division cycle 42 (Cdc42) GTPase causes a profound
128 ) during S phase in yeast, and Sld3 recruits cell division cycle 45 (Cdc45) to minichromosome mainten
129 and the replicative helicase containing the cell division cycle 45 (Cdc45)/minichromosome maintenanc
132 we explore the effect of phosphorylation of cell division cycle 6 (Cdc6), a DNA replication initiati
133 hat the origin-recognition complex (ORC) and cell-division cycle 6 (Cdc6) proteins recognize and enci
138 of Ste7, thereby favoring the resumption of cell division cycling after pheromone-induced growth arr
139 cells that have normally withdrawn from the cell division cycle and are committed to terminal differ
140 gulate progression through each stage of the cell division cycle and as such are major targets for de
141 ciates with Cul4A/DDB1 during an unperturbed cell division cycle and both Chk1 phosphorylation and re
143 ells, it leads to a reversible exit from the cell division cycle and entry into G0, a cell cycle stat
144 abscission of the midbody at the end of the cell division cycle and for phosphorylation and activati
145 amined Scc2 chromatin association during the cell division cycle and found that the affinity of Scc2
146 on by RNase enzymes, different phases of the cell division cycle and growth rates, and the existence
147 ENP-A is deposited at centromeres during the cell division cycle and identify an evolutionally conser
148 hat the genome is completely duplicated each cell division cycle and in how the division of cells is
149 dilute aquatic environment, coordinates the cell division cycle and multiple cell differentiation ev
151 alysis of 53 conditional lethal mutations in cell division cycle and RNA synthesis related genes, rev
153 budding yeast Saccharomyces cerevisiae, the cell division cycle and sporulation are mutually exclusi
154 to specific subcellular addresses during the cell division cycle and sporulation, and proper localiza
155 ated destruction of Dup is necessary for the cell division cycle and suggest that Geminin inhibition
156 eting of Epac during different stages of the cell division cycle and the structural features that are
157 d us to explore the relationship between the cell division cycle and the yeast respiratory oscillatio
159 ntous cells blocked at various stages of the cell division cycle and two unusual classes of mutants w
161 ate coordination with other processes during cell division cycles and response to environmental cues.
162 ates that metabolic cycling does not require cell division cycling and that metabolic synchrony does
163 ic clues about cooperative regulation of the cell-division cycle and apoptosis by these oncogenes.
164 e abnormal centriole numbers within a single cell-division cycle and provide insights into the regula
165 central role in coordinating the eukaryotic cell division cycle, and also serve to integrate diverse
166 may exist between pre-mRNA splicing and the cell division cycle, and fission yeast Dsk1 appears to p
167 s a key cellular system coordinated with the cell division cycle, and major efforts in systems biolog
168 gest subunit of ORC, is regulated during the cell division cycle, and thus ORC is a dynamic complex.
169 and its subcellular localization during the cell division cycle are linked through the action of GSK
172 heckpoint controls ensure that events of the cell-division cycle are completed with fidelity and in t
173 xcept kinesin family member 18B (KIF18B) and cell division cycle associated 3 (CDCA3) were of confirm
175 olic cycling occurs during the phases of the cell division cycle associated with mass accumulation in
176 mutants are exposed to iron, they arrest the cell division cycle at the G1 regulatory point Start.
178 n a low-activity state during an unperturbed cell division cycle but at the same time keeps Chk1 prim
179 ordinates the serial events required for the cell division cycle, but no Cdk1 substrate has been iden
180 protein phosphatases positively regulate the cell division cycle by activating cyclin-dependent prote
181 s, p21 and p27, which regulate the mammalian cell division cycle by inhibiting cyclin-dependent kinas
183 mosome replication are triggered during each cell division cycle by the initiator protein, DnaA.
184 size primarily modulates the duration of the cell-division cycle by controlling the G1/S transition k
185 induce DNA damage, but also during aberrant cell-division cycles caused by activated oncogenes and i
186 ant decrease in protein levels of cyclin B1, cell division cycle (Cdc) 25B, and Cdc25C, leading to ac
188 died the interaction of two oscillators, the cell division cycle (CDC) and the yeast metabolic cycle
189 monstrate, by using mathematical modeling of cell division cycle (CDC) dynamics, a potential mechanis
190 We report a detailed characterization of cell division cycle (cdc) genes in the differentiating g
192 bution of immunohistochemical markers of the cell division cycle (CDC) in 5 of the 16 neurogenic nich
193 ng the actin cytoskeleton, and with selected cell division cycle (cdc) mutations affecting processes
194 ved in the cell cycle, notably cyclin, E2F1, cell division cycle (CDC), and minichromosome maintenanc
196 n the other hand, cyclins A1, A2, B1 and B2, cell division cycle (CDC)2 and its kinase, CDC25 A and B
197 in that it segregated at the very end of the cell division cycle: cells showed a single fluorescent f
198 lthough Cdc7p levels are constant during the cell division cycle, Dbf4p and Cdc7p-Dbf4p kinase activi
202 h types of oscillations are coupled with the cell division cycle, exhibit period determination by CK1
203 ylation by DDK to form an active CMG [Cdc45 (cell division cycle gene 45), Mcm2-7, GINS (Go, Ichi, Ni
205 s involved in cellular processes such as the cell division cycle, gene transcription, the DNA damage
206 e female germ-line clones of these potential cell division cycle genes and to identify those which ca
207 e-wide program of gene expression during the cell division cycle in a human cancer cell line (HeLa) w
210 of eukaryotic chromosomes occurs once every cell division cycle in normal cells and is a tightly con
211 evels remain relatively unchanged during the cell division cycle in primary human T lymphocytes and i
212 ly uncouples centrosome duplication from the cell division cycle in prostate cancer cells through CEP
213 ssion and failed to enter the S phase of the cell division cycle in response to CSF-1 stimulation.
215 ive agents of malaria, have evolved a unique cell division cycle in the clinically relevant asexual b
217 the developmental program to the asymmetric cell-division cycle in C. crescentus, studies of flagell
218 pond to mating pheromones by arresting their cell-division cycle in G1 and differentiating into a cel
219 /DDB1 ubiquitin ligase during an unperturbed cell division cycle, in response to replicative stress a
220 number of fundamental processes comprise the cell division cycle, including spindle formation, chromo
221 enome is transcribed periodically during the cell division cycle, indicating that properly timed gene
222 ogenic transformation, such as arrest of the cell division cycle, induction of cell differentiation,
226 of apoptosis (programmed cell death) to the cell division cycle is essential for homeostasis and gen
229 ing organisms, divergence from the canonical cell division cycle is often necessary to ensure the pro
232 hat in such a state, progression through the cell division cycle is reversibly arrested in an orderly
236 ve cell fates; glucose, which stimulates the cell division cycle, is a potent inhibitor of sporulatio
237 hat drive photosynthetic cell growth and the cell division cycle; it also exhibits a highly choreogra
239 IIIalpha is phosphorylated on Ser123 by the cell division cycle kinase Cdk2 beginning early in S pha
240 ase 5 (Cdk5), a member of the Cdc2 family of cell division cycle kinases that has recently been copur
242 t regulatory nodes that govern both the host cell division cycle machinery and the initiation of cent
244 cdc123, homolog of mammalian D123, is a new cell division cycle mutant with a G2 delay at permissive
245 ne was identified previously in a screen for cell division cycle mutants and the cdc24(+) gene was de
250 ng is associated with the G0/G1 phase of the cell division cycle of slowly growing budding yeast, tra
251 fection of cells by many viruses affects the cell division cycle of the host cell to favor viral repl
253 d, taking advantage of the rapid synchronous cell division cycles of Xenopus eggs and cell-free syste
255 cell production depends on the length of the cell-division cycle of progenitor cells during neurogene
257 h its Large T stabilization domain region to cell division cycle protein 20 (Cdc20) and, possibly, cd
265 ogen-activated protein kinase (MAPK) and the cell-division cycle protein kinase Cdc2, are known to be
267 matin remodeling and progression through the cell division cycle, providing genetic clues to possible
268 rom Cdc42, a Rho family member important for cell-division cycle regulation, of lower eukaryotes, yet
269 22q11.2 was identical to the hCDCrel (human cell division cycle related) gene that maps to the regio
271 Progression through G2/M of the mammalian cell division cycle requires the coordinated expression
274 analysis, we found that MYC even within one cell division cycle resulted in a several-magnitude incr
276 ve BrdU labeling in vivo that the progenitor cell division cycle slows down markedly as their numbers
277 ypical periods of hours, are slower than the cell-division cycle, so the state of the oscillator has
278 transduction pathways that regulate an early cell division cycle step and the gain of motility later
279 can coincide exactly with the S phase of the cell division cycle, suggesting that oxidative metabolis
280 have found that, during the G2 phase of the cell division cycle, TFAP4 is targeted for proteasome-de
281 ar organisms and in meiosis, the specialized cell division cycle that gives rise to haploid gametes.
283 To understand the genetic control of algal cell division cycle that pertains to phytoplankton bloom
284 these cleavage cycles are replaced by longer cell division cycles that include gap phases and checkpo
285 rapid, early cleavage cycles are replaced by cell-division cycles that lengthen and acquire G (gap) p
286 rawal of nutrients triggers an exit from the cell division cycle, the induction of autophagy, and eve
287 n of one and only one daughter centriole per cell division cycle, the prevailing view is that centrio
288 ed and stimulated to synchronously enter the cell division cycle, they entered S phase 2-3 h earlier
290 ing a DNA checkpoint kinase that couples the cell division cycle to the circadian cycle abolishes syn
291 subfamily A) and the AAA-type ATPase Cdc48 (cell division cycle), Ubr1 directs the substrate to prot
292 Gap 1/DNA synthesis)-phase transition of the cell division cycle, ultimately resulting in decreased c
293 at 42 or 50 degrees C and during synchronous cell division cycles under normal growth conditions show
294 ribution of membranous structures during the cell division cycle underlie the cell division defects i
297 onse that uncouples DNA replication from the cell division cycle, which is reinforced in metazoans by
299 e poles during prophase and metaphase of the cell division cycle, with diminished staining of anaphas
300 ulation of cells in the mitosis phase of the cell division cycle, with some cells in the G(0)/G(1) ph
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