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1 the regulation of root growth by controlling cell elongation.
2 BL1), that also antagonized BR responses and cell elongation.
3 gen peroxide and a corresponding decrease in cell elongation.
4 ittle is known about the regulation of fibre cell elongation.
5  lacking both enzymes have a lethal block in cell elongation.
6 ns-Golgi network (TGN) is required for plant cell elongation.
7 owth inhibition, and this was due to reduced cell elongation.
8  of MapZ and FtsZ positioning and subsequent cell elongation.
9  is to repress JA signaling and allow normal cell elongation.
10 d allow the expression of genes required for cell elongation.
11  of periaxin, a protein required for Schwann cell elongation.
12  type III carboxypeptidase in the control of cell elongation.
13     Binding of BR to BRI1 primarily promotes cell elongation.
14 icating that HBI1 is a positive regulator of cell elongation.
15 cal wall of the cell, supporting its role in cell elongation.
16 further demonstrate roles in BR promotion of cell elongation.
17 ith actin cytoskeletal assembly during fiber cell elongation.
18 pporting a role of HERK1/THE1 in BR-mediated cell elongation.
19 ly independent of the BR pathway to regulate cell elongation.
20 f the mechanism that brings about controlled cell elongation.
21 , extensin arabinosylation--is important for cell elongation.
22 nam) and compound A22, both of which inhibit cell elongation.
23 otype with defects in both cell division and cell elongation.
24 n of both interdivision time and the rate of cell elongation.
25 xpansin (EXP) that are involved in promoting cell elongation.
26 arrays of microtubules that drive apicobasal cell elongation.
27  plants were dwarf, due primarily to reduced cell elongation.
28 a scaffolding protein thought to orchestrate cell elongation.
29  that are likely to regulate the response of cell elongation.
30 nd might be a general mechanism in polarized cell elongation.
31 l before cell constriction and contribute to cell elongation.
32  a common transcriptional network to promote cell elongation.
33  that a distinct regulatory pathway promotes cell elongation.
34 und that these molecules are dispensable for cell elongation.
35 e Rho-kinase (Rok) and myosin II in anaphase cell elongation.
36         Depletion of CgtAV did not result in cell elongation.
37 agA to gastric epithelial cells and initiate cell elongation.
38 stalk synthesis may be a specialized form of cell elongation.
39 ortant roles in various processes, including cell elongation.
40 n to be involved in cell wall metabolism and cell elongation.
41 n-lipid interactions, and perhaps slow fiber cell elongation.
42 alk elongation is a more constrained form of cell elongation.
43 mbryos by patterning coordinated fast muscle cell elongation.
44 ctivity in terms of degree and extent of AGS cell elongation.
45  growth and vascular differentiation but not cell elongation.
46 ificial proton pump activation inhibits root cell elongation.
47 -lacZ marker, and form the floor by directed cell elongation.
48 but not by fast muscle cells for fast muscle cell elongation.
49 d migration of oriC and 50 times faster than cell elongation.
50 1) and hormone applications that reduce root cell elongation.
51  expression of fliC, early flagellation, and cell elongation.
52 not inhibit the initial, rapid phase of lens cell elongation.
53 ut also decreased primary wall thickness and cell elongation.
54 ganized cortical microtubules and suppressed cell elongation.
55 ificial proton pump activation inhibits root cell elongation.
56 re dependent on actomyosin contractility and cell elongation.
57 eral root cap associated with the control of cell elongation.
58 dependent (non-genomic) activity of auxin in cell elongation.
59 th through effects on cell proliferation and cell elongation.
60 ment, oriented cell division, and apicobasal cell elongation.
61 ell wall softening at normal stages of rapid cell elongation.
62 he cleavage site by undergoing two phases of cell elongation.
63 ing dependent on both cell proliferation and cell elongation.
64 arily known for its role in controlling root cell elongation.
65 rotein transduces GA signal to promote fibre cell elongation.
66                 These included lack of fiber cell elongation, abnormal proliferation in prospective l
67 ittle consideration has been given as to how cell elongation affects the distribution of the key regu
68 ing both a transient decrease in the rate of cell elongation after 1.5 h but also a longer term gradu
69 ral root initiation in roots showing reduced cell elongation after auxin treatment.
70 utation that results in constitutive swarmer cell elongation also increased wosA transcription.
71               Overexpression of srk1 induces cell elongation, an indication of cell cycle G(2) delay,
72 appear to correlate with differences between cell elongation (anaerobic conditions) versus cell divis
73 e set of genes including those implicated in cell elongation and 16% of the genes affected in herk1 t
74 abidopsis thaliana, resulting in anisotropic cell elongation and a weak cell wall.
75  remodeling the basement membrane, promoting cell elongation and actin cytoskeletal reorganization, a
76 ibutes more strongly than the PKA pathway to cell elongation and adhesion, whereas nutrient limitatio
77 7, a novel cell polarity protein, the normal cell elongation and alignment upon leaving the primitive
78 d shade by the cotyledons triggers hypocotyl cell elongation and auxin target gene expression.
79 ex triggers extensive and unexpected Schwann cell elongation and branching to form long, parallel pro
80      This accumulation promotes asymmetrical cell elongation and causes differential growth between t
81 ls show aberrant morphology characterized as cell elongation and cell body rounding, loss of lamellip
82  well established roles of BRs in regulating cell elongation and cell division events, BRs also gover
83 ct modes of cell wall synthesis, involved in cell elongation and cell division, which are believed to
84                   Depletion of oMAP4 impairs cell elongation and cell-cell fusion.
85  processing in MarP-deficient cells leads to cell elongation and chain formation, a hallmark of proge
86  stomatal responses require reversible guard cell elongation and contraction.
87 , whereas ROCK1 depletion selectively led to cell elongation and defective tail retraction.
88 omatids from the cleavage site in a two-step cell elongation and demonstrate the role of myosin efflu
89 n is not as severe but also results in polar cell elongation and differentiation defects.
90 hape of the ocular lens depend on epithelial cell elongation and differentiation into fiber cells, fo
91 proteins are core cell wall synthases of the cell elongation and division machinery, and represent at
92          Auxin is a phytohormone involved in cell elongation and division.
93 ivities of cytoskeleton complexes that drive cell elongation and division.
94 revealed that Galpha(12/13) are required for cell elongation and efficient dorsalward migration durin
95     Instead, mutation of hob1+ led to slight cell elongation and faulty cell cycle arrest upon nutrie
96 stress for 24 h resulted in flow adaptation: cell elongation and formation of actin stress fibers ali
97 on to discover kinesins that are involved in cell elongation and found Gh KINESIN-4A expressed abunda
98 lant growth regulators, yet both can promote cell elongation and growth.
99  both the transcription of genes involved in cell elongation and hypocotyl growth.
100 oteolytic activity correlated with increased cell elongation and increased single-cell migration.
101 e retraction, but can support some germ band cell elongation and is thus not a full phenocopy of ush
102 r cell polarity (PCP) proteins that regulate cell elongation and mediolateral alignment.
103                During development, epidermal cell elongation and microtubule alignment occur simultan
104 to the midcell plane where it promotes zonal cell elongation and normal morphology.
105  of pcDNA3.1-pet in HEp-2 cells, we observed cell elongation and other morphological changes similar
106 l aluminum responses, others are involved in cell elongation and plant development.
107 l responses, whereas others are required for cell elongation and plant development.
108 n due to GA3-stimulation or TE-inhibition of cell elongation and production rate in leaves for both c
109 r endogenous GA4 content in leaf and greater cell elongation and production rates under the untreated
110 the individual contributions of differential cell elongation and proliferation to defining the apical
111 endent actin remodeling promotes endothelial cell elongation and proper organization of VE-cadherin i
112 to BR-related phenotypes, including impaired cell elongation and reduced expression of BR target gene
113 gulates shootward auxin transport to control cell elongation and root growth.
114 in and ethylene in control of root epidermal cell elongation and root hair elongation.
115 ial cells in culture, purified toxin induces cell elongation and rounding, followed by exfoliation of
116 cking ampD, to analyze PG degradation during cell elongation and septation.
117 ed microtubules during polarized endothelial cell elongation and that depletion of FMNL3 retards elon
118 ransition zone at or near the onset of rapid cell elongation and the epitope is similarly restricted
119          Here, we examine the role of PME in cell elongation and the regulation of its secretion and
120 urther emphasizes the importance of RG-II in cell elongation and the utility of glycosyltransferase i
121 n of cell wall pectin is a key to regulating cell elongation and ultimately the shape of the plant bo
122 -EXPL2 genes showed limited correlation with cell elongation and was not induced by GA.
123 icity and porosity, important parameters for cell elongation and water uptake.
124 s, rearrange the actin cytoskeleton to cause cell elongation, and alter junctional zona occludens 1 a
125  equatorial myosin II recruitment, prevented cell elongation, and caused a remarkable spindle defect
126 ctivity is required for cell adhesion, polar cell elongation, and cell differentiation.
127  pathways were involved in lignan synthesis, cell elongation, and fatty acid biosynthesis, all of whi
128 as necessary for persistent cell protrusion, cell elongation, and stable cell orientation in 3D colla
129 for the maintenance of junctional integrity, cell elongation, and suppression of proliferation, pheno
130 play redundant roles in cell wall mechanics, cell elongation, and the axial growth of various vegetat
131 shortening of XyGs that normally accompanies cell elongation appears to be slightly reduced, galactos
132 diameter) nanotubes elicited a dramatic stem cell elongation (approximately 10-fold increased), which
133     We show that cortical changes underlying cell elongation are more sensitive to depletion of Rok a
134 ating growth to predawn, when conditions for cell elongation are optimal.
135 re short and thick, have reduced anisotropic cell elongation, are suppressed in a root-waving phenoty
136 domain oriented to the cortical cells during cell elongation as well as subsequent polar nuclear move
137 egulators of apical constriction-independent cell elongation, as alpha-Spectrin and integrin mutant c
138 o muscle detachment and failure in epidermal cell elongation at the 2-fold stage.
139 ults from differential growth (i.e. enhanced cell elongation at the proximal abaxial side of the peti
140 3D) array that is oriented transverse to the cell elongation axis in wild-type plants and is oblique
141 lied either parallel or perpendicular to the cell elongation axis.
142 here bacterial cell division is arrested and cell elongation begins.
143 ysis showed that decreased cell division and cell elongation both contributed to the shortened leaves
144 ve regulator in light-mediated inhibition of cell elongation but a positive regulator in light-regula
145  affects the ability of cytokinin to inhibit cell elongation but not cell proliferation.
146 e complementation of fliL restores wild-type cell elongation but not motility.
147 lates Arabidopsis root growth by controlling cell elongation, but it is currently unknown whether GA
148 ponsive MAP kinases, Fus3 and Kss1, promotes cell elongation, but only Fus3 promotes chemotropic grow
149 's abilities to regulate gene expression and cell elongation, but these defects are rescued by TPL fu
150 role of these phosphatases in the control of cell elongation by BL.
151 Consistent with a model in which CagA causes cell elongation by inhibiting the disassembly of adhesiv
152 elix-loop-helix (HLH) factors, which promote cell elongation by interacting antagonistically with ano
153  crescentus, FtsZ also plays a major role in cell elongation by spatially regulating the location of
154 t Cell Elongation posits that auxin promotes cell elongation by stimulating cell wall acidification a
155 t cell elongation posits that auxin promotes cell elongation by stimulating cell wall acidification a
156 esulting in differential gene expression and cell elongation causing the organ to bend.
157 gical changes in susceptible GNBs, including cell elongation, cell swelling, or lysis, at 90 min.
158 ve1 and kobito1, previously shown to disrupt cell elongation, cellulose synthesis, vascular different
159                                The degree of cell elongation correlated with the length of the traili
160 ally expressed, which is consistent with the cell elongation defect phenotype and the changes in the
161 y, and the yip4a yip4b double mutants have a cell elongation defect.
162 II activation, only partially suppressed the cell-elongation defect and the furrowing delay, but prev
163 ygalacturonase gene NIMNA (NMA) that lead to cell elongation defects in the early embryo and markedly
164 S-A causes apical surface irregularities and cell elongation defects.
165 emergence of lateral root primordia and root cell elongation depended on the rootward auxin stream an
166 hain of antagonistic switches that regulates cell elongation downstream of multiple external and endo
167 m distachyon with dramatically enhanced root cell elongation due to increased cellular auxin levels h
168                    We show that differential cell elongation during apical hook development is defect
169  bands in Periaxin-null mice impairs Schwann cell elongation during nerve growth.
170                    This is caused by reduced cell elongation during the cold photoperiod.
171 int to a major role for HPATs in influencing cell elongation during tip growth in plants.
172 dies demonstrated that they are required for cell elongation during vegetative growth as herk1 the1 d
173 lization of apical junctions, which promotes cell elongation, during epithelial morphogenesis.
174 SMCs, suggesting that NOR-1 is a mediator of cell elongation effects.
175            Phototropism, or the differential cell elongation exhibited by a plant organ in response t
176 s from the surface ectoderm, as evidenced by cell elongation, exit from the cell cycle, and expressio
177 xin and brassinosteroid signaling as well as cell elongation/expansion, and increased expression of A
178     One of the most abundant proteins in the cell, elongation factor Tu, was found to be more oxidati
179 root elongation pom2 mutants are impaired in cell elongation, fertility, and microtubule-related func
180       Pulling on the ECM resulted in initial cell elongation, followed by disengagement and retractio
181 ion of the XyGs is not strictly required for cell elongation, for lengthening the polymers that occur
182 t responds to exogenous strain by undergoing cell elongation, forming polarized apical microtubules,
183                                           AP cell elongation forms a gradient culminating at the post
184                                       The AP cell elongation gradient remains when mesoderm invaginat
185  of cell cycle, cell wall, organ initiation, cell elongation, hormone homeostasis, and meristem activ
186 cytoskeleton is understood to participate in cell elongation; however, a detailed description and mol
187 EPIYA-D motif impacted early changes in host cell elongation; however, the degree of elongation was c
188 to be non-responsive to gibberellin (GA) for cell elongation, hypersensitive to the GA synthesis inhi
189 al abiotic stress conditions could stimulate cell elongation in an ethylene-dependent manner.
190 arriers mediates the control of differential cell elongation in apical hook development.
191 environmental, and developmental controls of cell elongation in Arabidopsis hypocotyl.
192 id-dependent molecular circuit that promotes cell elongation in Arabidopsis hypocotyls.
193 ng lipid changes alter cellulose content and cell elongation in Arabidopsis.
194 ory network by which cytokinin inhibits root cell elongation in concert with the hormones auxin and e
195        For genetic characterization of rapid cell elongation in cotton fibers, approximately 14,000 u
196 putative transcription factor that regulates cell elongation in developing styles.
197 K6 suppresses ectopic outgrowth and promotes cell elongation in different regions of the hypocotyl.
198 ion in dividing spermatocytes and failure of cell elongation in differentiating spermatids and disrup
199 cal microtubules and cell wall mechanics and cell elongation in flowering plants.
200            Mutations in CSI1 cause defective cell elongation in hypocotyls and roots and reduce cellu
201 opsis Galpha subunit, resulting in increased cell elongation in hypocotyls in darkness and increased
202 capable of intrinsically promoting hypocotyl cell elongation in light-grown plants, independently of
203 in biosynthesis pathway and thereby enhances cell elongation in low R:FR.
204 uxin, which regulates both cell division and cell elongation in plant development, are controlled by
205                                              Cell elongation in plants is controlled by environmental
206 se, the trehalose-degrading enzyme, inhibits cell elongation in response to heat and geldanamycin.
207 f genes involved in suppression of hypocotyl cell elongation in response to Rc signals.
208 nsporter is required for ROS homeostasis and cell elongation in roots and that this balance is pertur
209 tore is required for (and sets the rates of) cell elongation in roots.
210                                              Cell elongation in the basal part of the hypocotyl under
211 ators that act downstream of HLS1 to control cell elongation in the hypocotyl.
212 a1, but not bif2, also function in promoting cell elongation in the inflorescence.
213 t slow muscle migration triggers fast muscle cell elongation in zebrafish, we hypothesize that migrat
214 xin, showing that periaxin regulates Schwann cell elongation independent of its role in the dystrogly
215 yB-interacting capacity to control hypocotyl cell elongation, independently of its ability to bind DN
216              After DF3gamma34.5 infection of cells, elongation initiation factor 2alpha phosphatase a
217 tip involve cellular mechanisms that include cell elongation, intercalation, convergent extension, pr
218                                    Growth by cell elongation is a morphological process that transcen
219                                    Bacterial cell elongation is controlled by actin-MreB while cell d
220 mechanism of plant growth and morphogenesis, cell elongation is controlled by many hormonal and envir
221 n Hedgehog signaling is blocked, fast muscle cell elongation is disrupted.
222 fiber cells, suggesting that secondary fiber cell elongation is incomplete.
223 ) primary roots to water deficit showed that cell elongation is maintained preferentially toward the
224                                  The rate of cell elongation is maximal near the apical meristem and
225                                              Cell elongation is promoted by different environmental a
226 o a highly mobile ring-like structure during cell elongation is quickly followed by the recruitment o
227 he epigenomic state and cell division versus cell elongation is suggested, as no differences in DNA m
228                                    Polarized cell elongation is triggered by small molecule cues duri
229      Moreover, similar to Brachypodium, root cell elongation is, in general, robustly buffered agains
230 er, similar to Brachypodium distachyon, root cell elongation is, in general, robustly buffered agains
231 steroid (BR), and gibberellin (GA), regulate cell elongation largely by influencing the expression of
232 1 cKO lenses displayed delayed primary fiber cell elongation, lenses from both Rac1 cKO strains were
233 onal laccase enzymes play important roles in cell elongation, lignification and pigmentation in plant
234 t necessary for glycan polymerization by the cell elongation machinery, as is commonly believed.
235 e dorsal convergence movements with impaired cell elongation, mediolateral orientation, and consequen
236 ant suggests that cell wall insertion during cell elongation normally occurs along two helices of opp
237 t tip where transition from cell division to cell elongation occurs.
238 n African rice, which regulates longitudinal cell elongation of the outer and inner glumes.
239 LF (rapid alkalinization factor), suppresses cell elongation of the primary root by activating the ce
240 gs is required for the specific processes of cell elongation or division, while the cell wall synthes
241 th, possibly repressing cell division and/or cell elongation or the length of time that cells elongat
242         Here we further characterized the AP cell elongation patterns during GBE, by tracking cells a
243 ways were important in the H. pylori-induced cell elongation phenotype.
244  proposed to cause cell crowding, leading to cell elongation (placode formation).
245 he long-standing Acid Growth Theory of Plant Cell Elongation posits that auxin promotes cell elongati
246 he long-standing Acid Growth Theory of plant cell elongation posits that auxin promotes cell elongati
247       Protrusion occurs through differential cell elongation, probably mediated by Islet, as we find
248 Z, MurG fails to accumulate near midcell and cell elongation proceeds unperturbed in appearance by in
249  In this study we have used the differential cell elongation process during apical hook development t
250  It was hypothesized that AQPs contribute to cell elongation processes by allowing water influx acros
251 fied ethylene pathway genes that may control cell-elongation processes functioning at the intersectio
252 aterial precedes and predicts the process of cell elongation provide support for the idea that the in
253  show that the GA-induced boost of hypocotyl cell elongation rate is not dependent upon the maintenan
254 the doubling time, rather than in the single-cell elongation rate, (iii) the division rate increases
255                      During compression, the cell elongation rate, proliferation rate, DNA replicatio
256 ggested that the observed changes in abaxial cell elongation rates during ethylene treatment should r
257 nversion to a physiological phenotype namely cell elongation, reduced proliferation, lowered angiogen
258 pose that activation of UNC-82 kinase during cell elongation regulates thick filament attachment or g
259 3 genes preferentially expressed in fiber, a cell elongation regulator, PRE1, is strikingly At biased
260  regulating expression of photosynthetic and cell elongation-related genes in etiolated seedlings.
261                                              Cell elongation required Rac1 and Cdc42 but not phosphat
262                          The coordination of cell elongation requires E-cadherin-mediated cell-cell a
263 systematic, parallel analysis of endothelial cell elongation response to different fluidic shearing p
264   Spirogyra produces ethylene and exhibits a cell elongation response to ethylene.
265 cosyl Yariv reagent (betaGlcY) that disrupts cell elongation results in the persistence of (1-->4)-be
266 he roles of two other proteins important for cell elongation, RodA and MreB.
267 l processes, including cell differentiation, cell elongation, seed germination, and response to abiot
268 idoglycan hydrolysis, a process required for cell elongation, separation of progeny cells, and cell w
269 n pollen tubes, both the Ca(2+) gradient and cell elongation show oscillatory behavior, reinforcing t
270 rase-transpeptidase PBP1A interacts with the cell elongation-specific transpeptidase PBP2 in vitro an
271 2, which is commonly branded as an essential cell elongation-specific transpeptidase, switches its lo
272  Treatment of HeLa cells with D-e-Cer caused cell elongation, spreading inhibition, rounding, and det
273 tage, a stomata differentiation stage, and a cell elongation stage.
274  rapidly separate in a manner independent of cell elongation, suggesting the existence of a mitotic a
275 BH1 causes a severe dwarf phenotype, but the cell elongation suppression mechanism is still not well
276 lobacter crescentus is a specialized form of cell elongation that confers to the cell substantial adv
277 rated apical constriction and Rac1-dependent cell elongation that controls cell shape and thus curvat
278 ck revealed that although plasmolysis slowed cell elongation, the cells nevertheless "stored" growth
279 trient limitation the MAPK pathway regulates cell elongation, the PKA pathway regulates unipolar budd
280                     Cytokinin regulates root cell elongation through ethylene-dependent and -independ
281 these conditions, by concomitantly promoting cell elongation through intrinsic transcriptional-regula
282 teract to consolidate the phase of hypocotyl cell elongation to peak at dawn under diurnal cycles in
283                    The marked sensitivity of cell elongation to Rok depletion was highlighted by RNAi
284 n antibiotic which specifically inhibits the cell elongation transpeptidase penicillin binding protei
285                                              Cell elongation triggered by human body temperature invo
286 verexpression of PUN1 results in exaggerated cell elongation under conditions of nitrogen stress.
287 ng-term, phy-imposed inhibition of hypocotyl cell elongation under prolonged, continuous irradiation.
288 on cell-autonomously to mediate mediolateral cell elongation underlying intercalation during notochor
289 PCP) signaling is essential for mediolateral cell elongation underlying these movements, but how this
290 BC transporter, FtsEX, which is required for cell elongation, unlike cell division as in Escherichia
291 ds (BRs), play essential roles in modulating cell elongation, vascular differentiation, senescence an
292 or instance, brassinosteroids (BRs) regulate cell elongation, vascular differentiation, senescence an
293 w that bacterial cell division is coupled to cell elongation via a direct and essential interaction b
294                                              Cell elongation was measured optically and protein accum
295 shion to Vpr; however, no additive effect of cell elongation was observed when srk1 and vpr were coex
296 y prior to the colonization of land and that cell elongation was possibly an ancestral ethylene respo
297  zones of cell proliferation and the zone of cell elongation where differentiation begins.
298      Phosphorylated CagA leads to epithelial cell elongation, which is dependent on the number of var
299 e focused on the earliest cell shape change, cell elongation, which occurs during anaphase B and prio
300                         Parallel flow caused cell elongation with enhanced stress fibers and p-FAK, a

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