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1 form multi-protein assemblies that span the cell envelope.
2 drugs and other harmful compounds across the cell envelope.
3 lasmic membrane and its transport across the cell envelope.
4 sis of major components of the mycobacterial cell envelope.
5 megadalton nanomachine that spans the entire cell envelope.
6 ntified are known or predicted to affect the cell envelope.
7 as well as the architecture of the bacterial cell envelope.
8 cts the speciation of U(VI) on the bacterial cell envelope.
9 I) surface complexes formed on the bacterial cell envelope.
10 panning the inner and outer membranes of the cell envelope.
11 nts, intercellular lipids, and the cornified cell envelope.
12 glycolipids/lipoglycans of the mycobacterial cell envelope.
13 d the transport of TbpB across the bacterial cell envelope.
14 ecific phenotypes consistent with an altered cell envelope.
15 the presence of key genes suggests a diderm cell envelope.
16 at affect immunomodulatory components of the cell envelope.
17 hat the gene products are distributed in the cell envelope.
18 as for the anchoring of the holdfast to the cell envelope.
19 ers and by changing the composition of their cell envelope.
20 hat mostly target elements in the parasite's cell envelope.
21 partite pumps thus span the entire bacterial cell envelope.
22 cted, restricted localization of TonB in the cell envelope.
23 e to host factors that disrupt the bacterial cell envelope.
24 proteins and coordinated constriction of the cell envelope.
25 esponse because it affects the mycobacterial cell envelope.
26 represents the hallmark of the mycobacterial cell envelope.
27 glycolipid composition of the mycobacterial cell envelope.
28 e study of the biosynthesis of the bacterial cell envelope.
29 c spindle controls cytokinetic events at the cell envelope.
30 ducing disulphide bonds into proteins in the cell envelope.
31 charide is the most exposed component of the cell envelope.
32 erentially to type I cohesins located at the cell envelope.
33 ence factors across their highly hydrophobic cell envelope.
34 ssemble a structure, the pseudopilus, in the cell envelope.
35 s, dramatically modified the M. tuberculosis cell envelope.
36 lationships of the various components of the cell envelope.
37 rowth rate and increased permeability of the cell envelope.
38 e crescentin structure to associate with the cell envelope.
39 rovides a route for LPS transport across the cell envelope.
40 maintenance of OM connections throughout the cell envelope.
41 urbing the assembly of the Corynebacterineae cell envelope.
42 teins serve essential roles in the bacterial cell envelope.
43 are independent of its association with the cell envelope.
44 hich are key components of the mycobacterial cell envelope.
45 the divisome, for proper constriction of the cell envelope.
46 e cytotoxic domain of the colicin across the cell envelope.
47 DNA transfer and that LpqM is located in the cell envelope.
48 tiple genes with putative association to the cell envelope.
49 otics because of its durable and impermeable cell envelope.
50 c levels by mediating drug efflux across the cell envelope.
51 f functional properties of the Gram-positive cell envelope.
52 he mechanical integrity of the Gram-negative cell envelope.
53 Escherichia coli are protected by a complex cell envelope.
54 ria to secrete proteins across their complex cell envelope.
55 glycolipids/lipoglycans of the mycobacterial cell envelope.
56 tiation and incorporation into the cornified cell envelope.
57 elope compared to those with a Gram-negative cell envelope.
58 septal peptidoglycan and constriction of the cell envelope.
59 ridazine, agents that impact M. tuberculosis cell-envelope.
60 the primary and outermost boundary of their cell envelopes.
61 cteria is critical for the assembly of their cell envelopes.
62 ricrin is a major component of the cornified cell envelope, a highly insoluble structure composed of
63 ses that it is important to define bacterial cell envelopes according to whether they have one (monod
70 isolate--had changes in the structure of the cell envelope and alterations in membrane permeability a
71 ntly have an uneven distribution through the cell envelope and are only found at one or both poles in
72 l to the integrity of the highly impermeable cell envelope and are potential therapeutic targets.
78 , transcribes genes required to maintain the cell envelope and is activated by conditions that destab
79 promises the integrity of the S. Typhimurium cell envelope and temporarily renders the bacterium avir
80 undergoes energized motion in the bacterial cell envelope and that ExbBD couples this activity to th
81 ese proteins form a complex that bridges the cell envelope and that has been proposed to cause fusion
84 rgy metabolism genes; and an upregulation of cell envelope and transport and binding genes in the mut
87 the degraded compound, (2) properties of the cell envelope, and (3) the localization of the reacting
88 n filaments, as a component of the cornified cell envelope, and as a source of natural moisturizing f
89 through several morphological stages of the cell envelope, and FtsZ is required but not sufficient t
90 re essential components of the mycobacterial cell envelope, and their biosynthetic pathway is a well
91 re essential components of the mycobacterial cell envelope, and their biosynthetic pathway is one of
92 escentin structure is stably anchored to the cell envelope, and this cellular organization requires M
93 that it plays a critical role in maintaining cell envelope architecture during growth and division.
96 nsaturated fatty acids in the B. burgdorferi cell envelope are potential targets for oxidative damage
97 inery needed for NB translocation across the cell envelope are widespread in Gram-negative bacteria,
98 termediary metabolism, and remodeling of the cell envelope as a means of defending spirochetes agains
99 se proteins, final destination is within the cell envelope as either membrane-anchored or cell wall-a
100 rence for necromass in the form of microbial cell envelopes as well as plankton and algal detritus.
101 ate filament-like protein crescentin forms a cell envelope-associated cytoskeletal structure that con
103 ampens proinflammatory responses through the cell envelope-associated serine hydrolase Hip1 (hydrolas
105 baumannii are far-reaching - from impacts on cell envelope biogenesis and maintenance, bacterial phys
106 e set of genes encoding products involved in cell envelope biogenesis, peptidoglycan remodeling, upta
107 contrast, BsrG strongly interferes with the cell envelope biosynthesis, causes membrane invagination
108 (PAT) not only play a structural role in the cell envelope but also contribute to the ability of M. t
109 ains enhance the structural integrity of the cell envelope by anchoring the beta-barrels within the p
110 monstrate that disruption of the V. cholerae cell envelope by chemical treatment with polymyxin B sim
111 at ultimately undermine the integrity of the cell envelope by depleting the inner membrane of phospho
114 aptic cleft, and would depolarize other hair cells enveloped by the same neuritic process increasing
117 modifications of components of the bacterial cell envelope can enhance resistance to antimicrobial ag
118 or upregulation of genes associated with the cell envelope can modulate vesicle production or remodel
119 tty-acid availability limits growth rate and cell envelope capacity, revealing that fatty-acid synthe
120 model in which fatty acid availability sets cell envelope capacity, which in turn dictates cell size
121 possible role for these genes in regulating cell envelope characteristics in the intracellular envir
122 nger for microorganisms with a Gram-positive cell envelope compared to those with a Gram-negative cel
126 stem alters the expression of genes encoding cell envelope components and proteins involved in centra
130 fication of specific Gram-negative bacterial cell envelope components, such as capsule, O-antigen and
131 erminant of Mycobacterium tuberculosis (Mtb) cell envelope composition and virulence, but the substra
133 first time, that despite the discrepancy in cell envelope concentrations of FepA and TonB ( approxim
134 tensive and tightly integrated modulation of cell envelope constituents, chemotaxis/motility machiner
136 entration of U(VI) adsorbed on the bacterial cell envelope control the kinetics of U(VI) bioreduction
137 n physiology are likely general responses to cell envelope damage and are not unique to T2S mutants.
139 antituberculosis drugs which also target the cell envelope, did not induce the expression of MT2816/R
140 f phage Sf6 is ejected through the bacterial cell envelope during infection and its C-terminal knob i
141 lectron microscopy images of the Caulobacter cell envelope exhibited outer membrane disruption, and c
142 ts that a mechanism for transport across the cell envelope exists, yet knowledge of proteins that may
144 K-rich clay occurs within and exterior to cell envelopes, forming where the supply of Fe had been
146 to be equally divided between cytosolic and cell envelope fractions, whereas SecA2 was predominantly
152 (a member of the LiaFSR system that controls cell envelope homeostasis), from daptomycin-resistant En
155 e and mechanical properties of the S. aureus cell envelope in both types of clinically relevant strai
156 between the inner and outer membranes of the cell envelope in Gram-negative bacteria, maintains cell
157 nvolved in the formation of the mycolic acid cell envelope in mycobacteria and provides a previously
158 sponse system modulates the integrity of the cell envelope in part by controlling peptidoglycan amida
160 also incorporated into the loricrin knockout cell envelope, in addition to the small proline rich pro
161 pumps in the overall flux process across the cell envelope, in this study we have carefully studied b
163 amnose polysaccharides that are critical for cell envelope integrity and cell shape and also represen
166 antagonistically to IreK and interfere with cell envelope integrity, antimicrobial resistance, and G
167 llar motility, exopolysaccharide production, cell envelope integrity, cell division and normal morpho
168 ies have indicated the importance of ECA for cell envelope integrity, flagellum expression, and resis
169 may function in establishing and maintaining cell envelope integrity, polysaccharide biosynthesis, ir
172 ow the molecular components of the bacterial cell envelope interact with each other to mediate cell w
178 rning activity, and that localization to the cell envelope is not required for patterning of heterocy
179 stinctive component of the Corynebacterineae cell envelope is the mycolyl-arabinogalactan (mAG) compl
181 s, the stalk, a cylindrical extension of all cell envelope layers, is the least well characterized at
184 CR signaling is inhibited by M. tuberculosis cell envelope lipoglycans, such as lipoarabinomannan and
185 The LytR-CpsA-Psr proteins are important for cell envelope maintenance in many Gram-positive species.
188 ns observed in the DeltaMceG mutant, such as cell envelope modifications, suggest a pleiotropic funct
194 enzymatic release of free mycolic acids from cell envelope mycolates during mycobacterial growth.
196 enome sequencing and characterization of the cell envelope of a clinical pair of vancomycin-resistant
198 e present the first detailed analysis of the cell envelope of an important but neglected member of th
200 ffective longitudinal Young's modulus of the cell envelope of Escherichia coli (50-150 MPa), Bacillus
201 he physical and mechanical properties of the cell envelope of Escherichia coli are poorly understood.
202 members of the Tol-Pal system that spans the cell envelope of Gram-negative bacteria and contributes
203 em is a multi-protein complex that spans the cell envelope of Gram-negative bacteria and promotes the
206 complex, a widely conserved component of the cell envelope of Gram-negative bacteria, is required to
209 ully coordinate constriction of a tripartite cell envelope of inner membrane, peptidoglycan (PG), and
210 to determine the thiol concentration on the cell envelope of intact Bacillus subtilis to nanomolar c
211 predominant stress-bearing structure in the cell envelope of most bacteria, and also a potent stimul
212 ein (MOSP) is a prominent constituent of the cell envelope of Treponema denticola (TDE) and one of it
214 revealed a structural difference between the cell envelopes of L. interrogans and Leptospira biflexa
215 nt properties on the bacterium in respect of cell envelope organisation and interaction with the envi
217 n as the "tail needle" likely functions as a cell envelope-penetrating device to promote ejection of
222 e Psp system in persister cell formation and cell envelope protection in bacterial pathogens and prov
224 the streptococcal CXC protease, S. pyogenes cell envelope proteinase, we developed a combination vac
225 functional groups are hypothetical proteins, cell envelope proteins and proteins involved in DNA meta
226 mitigation of stress generated by misfolded cell envelope proteins but promotes expression of genes
227 enzymatic subunits, bind cohesin modules of cell envelope proteins, thereby anchoring the cellulosom
229 n interesting research material for defining cell-envelope proteins without experimental cell disrupt
232 cts of the hydrolases on the M. tuberculosis cell envelope resulted in a significant decrease (60-80%
233 ing reveals a novel role for FtsK in linking cell envelope septation events and yields further eviden
235 functionality.IMPORTANCECaudovirales encode cell envelope-spanning proteins called spanins, which ar
236 B7 lipoprotein, and VirB9 interact to form a cell envelope-spanning structural scaffold termed the "c
237 a dynamic bacteriophage-like structure and a cell-envelope-spanning membrane-associated assembly.
239 125 DNA bar-coded mutants to identify novel cell envelope stress and acute acid shock phenotypes ass
240 is important for survival under a number of cell envelope stress conditions and in gastrointestinal
241 t in addition to anaerobiosis, adaptation to cell envelope stress is a critical requirement for E. co
244 MprAB as part of an evolutionarily conserved cell envelope stress response network and demonstrate th
245 l, these data demonstrate that RpoE is a key cell envelope stress response regulator and, similar to
246 k protein (Psp) system is a highly conserved cell envelope stress response required for virulence in
247 gnal transduction system, which controls the cell envelope stress response system in other gram-negat
248 encodes a homolog of the CpxRA two-component cell envelope stress response system originally characte
255 ECF factors typically respond to cell wall/cell envelope stress, iron levels, and the oxidation sta
258 at Amj is expressed under the control of the cell envelope stress-response transcription factor sigma
263 cated in resistance to antibiotics and other cell envelope stressors mediated, in part, by regulation
264 s enhance our understanding of mycobacterial cell envelope structure and dynamics and have implicatio
265 r Tol-Pal complex is thus a key component of cell envelope structure and function, mediating OM const
266 Together, sigma(M) and sigma(X) regulate cell envelope structure to decrease access of nisin to i
269 the system indicate adaptation to different cell envelope structures, bacterial lifestyles, and/or b
270 t into the evolutionary origins of primitive cell envelope structures, of which the S-layer is consid
271 encing, transcriptomics, ultrastructure, and cell envelope studies were carried out on two relatively
273 coordinated invagination of the multilayered cell envelope such that each daughter receives an intact
276 rease the resistance of B. subtilis to other cell envelope-targeted antimicrobial agents, indicating
277 centration causes a pressure drop across the cell envelope that drives changes in cell shape, such as
278 otected by an unusual and highly impermeable cell envelope that is critically important for the succe
279 comprises the S-layer (surface layer of the cell envelope that is external to the outer membrane), h
280 of proteins located in the Escherichia coli cell envelope that transduces energy and contributes to
281 aerobiales are remnants of an ancient diderm cell envelope that was present in the ancestor of the Fi
282 r adaptive success is linked to their unique cell envelopes that are extremely resistant to chemical
283 urface-associated carbohydrates of the Ldb17 cell envelope: the Gro3P decoration of the major surface
284 encoding proteins that are localized to the cell envelope; these include metallopeptidases, multidru
287 TolB to facilitate translocation through the cell envelope to reach their cytotoxic site of action.
290 s help to clarify fundamental differences in cell envelope ultrastructure between spirochetes and gra
293 rs of the Firmicutes with Gram-negative-type cell envelopes, was recently moved from Clostridia to a
294 recognized the peptide-target complex in the cell envelope, we next analyzed substrate binding by the
296 e to SDS and had unique protrusions from its cell envelope when examined by cryo-electron tomography,
297 at HtaA is associated predominantly with the cell envelope when the organism is grown in minimal medi
298 247 may exert some of its effects within the cell envelope whereas other activities occur in the cyto
299 of periplasmic flagella and the surrounding cell envelope, which enable this enigmatic bacterium to
300 ructions provided detailed structures of the cell envelope, which is significantly different from tha
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