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1 lular distribution of (64)Cu was measured by cell fractionation.
2 ion, as determined by immunofluorescence and cell fractionation.
3 sociated with microsomal membranes following cell fractionation.
4 sed form of gB could be coisolated following cell fractionation.
5 ablished both through immunofluorescence and cell fractionation.
6 by indirect immunofluorescence as well as by cell fractionation.
7  also confirmed by coimmunoprecipitation and cell fractionation.
8 eins in Escherichia coli was used along with cell fractionation.
9  live cell imaging, immunocytochemistry, and cell fractionation.
10 labelling, domain-specific biotinylation and cell fractionation.
11                                              Cell fractionation according to lineage depletion and AL
12 -cell imaging, deconvolution microscopy, and cell fractionation, all concurred that exogenous rSPARC
13  found predominantly as a soluble protein by cell fractionation, all three proteins were found to loc
14                              We show that by cell fractionation, almost all MPP10 was found in isolat
15                              Microscopic and cell fractionation analyses of parasites expressing one
16              Immunocytochemical staining and cell fractionation analysis demonstrated that sentrin mo
17 pitope tag into NIH 3T3 cells and subsequent cell fractionation analysis shows that the PATE protein
18 A11 in cells by using immunofluorescence and cell fractionation analysis.
19 2 in the nucleolar fraction as determined by cell fractionation and by immnocytochemical analysis.
20                                              Cell fractionation and confocal immunofluorescence micro
21                                              Cell fractionation and confocal localization of formin I
22                                              Cell fractionation and confocal microscope studies revea
23                                         Both cell fractionation and confocal microscopic analyses of
24                         In the present work, cell fractionation and confocal microscopy studies demon
25 n null mice (Peri-/- MEF), we demonstrate by cell fractionation and confocal microscopy that up to 50
26                                     Based on cell fractionation and confocal microscopy, we find SH2-
27 gating enzyme 9 (Ubc9) in HCC cell nuclei by cell fractionation and confocal microscopy; physical int
28                                              Cell fractionation and cytological experiments suggest t
29 degraded with a t1/2 of 2.9 h, were found by cell fractionation and density-shift experiments to resi
30                                              Cell fractionation and electron-microscopic immunolabeli
31                                              Cell fractionation and extractions with MgCl(2) indicate
32                                        Using cell fractionation and fluorescence methods, we demonstr
33 h most of the CAD in the cell was cytosolic, cell fractionation and fluorescence microscopy showed th
34  underlying Tor-dependent signaling, we used cell fractionation and immunoaffinity chromatography to
35                                              Cell fractionation and immunoblot analyses using polyclo
36                                              Cell fractionation and immunoblot analysis confirmed thi
37 A::TnphoA) mutants do not elicit the HR, but cell fractionation and immunoblot analysis revealed that
38                                              Cell fractionation and immunoblotting showed mrnp 41 in
39                                              Cell fractionation and immunoblotting showed that PEB1a
40  their secretion phenotypes were assessed by cell fractionation and immunochemistry.
41                                        Using cell fractionation and immunocytochemical experimental a
42                                              Cell fractionation and immunocytochemistry indicated tha
43                                              Cell fractionation and immunocytochemistry show caveolae
44                                              Cell fractionation and immunocytochemistry studies demon
45                         In this work we used cell fractionation and immunocytochemistry to determine
46 taining RNA are all detected on membranes by cell fractionation and immunoelectron microscopy.
47                                              Cell fractionation and immunofluorescence analyses indic
48 n of ERalpha to the nucleus as shown by both cell fractionation and immunofluorescence microscopic st
49 ght into this process, we applied mechanical cell fractionation and immunofluorescence microscopy to
50 venous etoposide administration, followed by cell fractionation and immunofluorescence studies of var
51                                           By cell fractionation and immunogold electron microscopy, w
52                                              Cell fractionation and immunogold labeling show that in
53 as predominantly localized in the nucleus by cell fractionation and immunostaining.
54                                              Cell fractionation and in situ hybridization analysis of
55                                 Furthermore, cell fractionation and in vitro methylation assays demon
56                                              Cell fractionation and labeling studies with [(3)H]palmi
57 nt Ras localization to the PM as measured by cell fractionation and photobleaching.
58                                              Cell fractionation and PP1 catalytic subunit isolation s
59                                              Cell fractionation and protease accessibility experiment
60                                              Cell fractionation and qRT-PCR analysis indicated that L
61                                              Cell fractionation and quantitative RT-PCR revealed that
62                             We have utilized cell fractionation and ribosome profiling to obtain a ge
63 of FMR1 and the FXR proteins, we carried out cell fractionation and sedimentation experiments with mo
64 l subcellular localization were evaluated by cell fractionation and Western blot analysis.
65                                              Cell fractionation and Western blotting demonstrated tha
66                      We determined by use of cell fractionation and Western blotting that, during in
67                                              Cell fractionation and western-blot analysis localized t
68                         Protease protection, cell fractionation, and affinity purification experiment
69   In this report, using immunocytochemistry, cell fractionation, and chromatin immunoprecipitation an
70 were assessed by arteriovenous measurements, cell fractionation, and immunofluorescence.
71 ing immunogold labeling/electron microscopy, cell fractionation, and protein analysis techniques, we
72 vidence using immunofluorescence microscopy, cell fractionation, and SNARE protein interaction studie
73 ked Golgi fraction was isolated by classical cell fractionation, and the protein complement (the Golg
74 alphas, using immunofluorescence microscopy, cell fractionation, and total internal reflection fluore
75 nce localization, sucrose density gradients, cell fractionation, and yeast two-hybrid experiments.
76                                           By cell fractionation, ANKRA is found both in the cytosol a
77                     We previously reported a cell fractionation approach that includes the selection
78                                              Cell fractionation as well as indirect immunofluorescenc
79            In the present study, we found by cell fractionation assays and confocal microscopy that p
80           NMD substrates classified based on cell fractionation assays as "nucleus associated" or "cy
81                                              Cell fractionation assays localized p32 to the P100 frac
82              Developmental 2D Western blots, cell fractionation assays, and analysis of a missense pr
83 d the associated nascent RNA are prepared by cell fractionation, avoiding immunoprecipitation or RNA
84 r intracellular calreticulin released during cell fractionation because it was expressed on circulati
85                                              Cell fractionation by sucrose gradient centrifugation in
86                        These results suggest cell fractionation can be used to study the uptake of mo
87      Using transmission electron microscopy, cell fractionation, cell wall sorting signal domain swap
88 ng immunohistochemistry/confocal imaging and cell fractionation/co-immunoprecipitation, we found that
89             This hypothesis was supported by cell fractionation, coimmunoprecipitation, and coimmunol
90                                              Cell fractionation, coimmunoprecipitation, and immunogol
91                                              Cell fractionation combined with quantitative assays was
92                                              Cell fractionation confirmed the lysosomal localization
93                                 In addition, cell fractionation data indicate that Cdc37 is found in
94                                              Cell fractionation demonstrated a predominant decrease i
95                            OptiPrep gradient cell fractionation demonstrated that BK channels were co
96 lular localization of the kinase activity by cell fractionation demonstrated that it is enriched in c
97 immunofluorescence, immunogold labeling, and cell fractionation demonstrated that rat slit diaphragms
98                                            A cell fractionation experiment using cisplatin-treated He
99                                              Cell fractionation experiments also revealed that phosph
100                                  Strikingly, cell fractionation experiments and confocal immunofluore
101                                              Cell fractionation experiments and cryoimmunoelectron mi
102                                              Cell fractionation experiments confirmed that macroH2A1
103 tron microscopy, using anti-MompA serum, and cell fractionation experiments confirmed the localizatio
104 N-terminus and site-directed mutagenesis and cell fractionation experiments confirmed this protein is
105                                              Cell fractionation experiments demonstrated that LdK39 w
106           Subsequent immunocytochemistry and cell fractionation experiments fully supported the concl
107                                              Cell fractionation experiments indicated that the change
108                      Mutational analysis and cell fractionation experiments indicated that the inv(16
109                                              Cell fractionation experiments on freshly isolated CD4+
110 TAF1 colocalizes with UBF in Hela cells, and cell fractionation experiments provided further evidence
111                                 Furthermore, cell fractionation experiments revealed that significant
112                      Confocal microscopy and cell fractionation experiments revealed that upon LPS st
113          GFP fusion protein localization and cell fractionation experiments show that this E3 ligase
114                                              Cell fractionation experiments showed that caspase-3 act
115                                  Pulse-chase cell fractionation experiments showed that chitosome pro
116                                              Cell fractionation experiments showed that ExoT is trans
117                                              Cell fractionation experiments showed that it is localiz
118                                              Cell fractionation experiments showed that meningococcal
119                                              Cell fractionation experiments suggested that Inp53p ass
120                                              Cell fractionation experiments surprisingly revealed two
121 immunolocalization, immunoprecipitation, and cell fractionation experiments, here we show association
122       AKT2 was localized to the cytoplasm by cell fractionation experiments, immunocytochemistry, and
123             In agreement with the results of cell fractionation experiments, immunofluorescence micro
124                                           In cell fractionation experiments, more signal recognition
125 ntermediate was isolated in the periplasm in cell fractionation experiments.
126 experiments to calculate the effective whole-cell fractionation factors between water and organic mat
127                                              Cell fractionation, fluorescence imaging and immunoelect
128               Subcellular localization using cell fractionation followed by immunoblot detection, as
129                                              Cell fractionation followed by Western blot analysis ind
130 ncluding several attempts to isolate them by cell fractionation from different cell types.
131  fluorescent protein-fused PIMT proteins and cell fractionation-immunoblot analysis revealed that apa
132 bes catalyzed by transglutaminase 2 prior to cell fractionation, immunoprecipitation, and detection w
133 and function of beta-catenin was analysed by cell fractionation, immunoprecipitation, immunoblots, QR
134  test this, we measured V-ATPase assembly by cell fractionation in HEK293T cells treated with and wit
135  in EAAT4 and GluRdelta2 by protein blot and cell fractionation in SCA5 autopsy tissue.
136  trafficking in control and BDL livers using cell fractionation in the context of in vivo pulse-chase
137                       Immunofluorescence and cell fractionation indicate that Bud1p remains associate
138                                              Cell fractionation indicated specific loss of acidic lip
139                                              Cell fractionation indicated that Frmpd1 stabilizes AGS3
140                                              Cell fractionation into a cytoskeleton-unbound and a cyt
141                                              Cell fractionation localized both raf1CAAX and raf1(257L
142                                        Using cell-fractionation methods, we observed that approximate
143                                              Cell fractionation of Arabidopsis leaves revealed that A
144                                        Crude cell fractionation of infected cells using detergent lys
145   Using quantitative confocal microscopy and cell fractionation of Nef-expressing cells and HIV-1-inf
146                                              Cell fractionation of SCP-2 overexpressing L-cells and W
147 mY RNAs are largely cytoplasmic in wild-type cells, fractionation of knockout cells revealed that the
148                However, extraction of viable cells, fractionation of lineages, and in vitro analysis
149       beta-Catenin signaling was assessed by cell fractionation or immunoconfocal microscopy to detec
150                            Refinement of the cell fractionation procedure indicated that the completi
151 zed with double-label immunofluorescence and cell fractionation procedures.
152 ial patterning, we developed a novel ex vivo cell fractionation/reconstitution assay.
153                Immunoelectron microscopy and cell fractionation reveal that both proteins travel thro
154 ccessibility, TX-114 phase partitioning, and cell fractionation revealed that Msp exists as distinct
155                                              Cell fractionation revealed that SecADelta11 and SecADel
156                                              Cell fractionation revealed that variation in quantity o
157 scence assay, surface proteolysis, and novel cell fractionation schemes revealed that MOSP in TDE exi
158                                              Cell fractionation showed that adenosine or an ADORA2A o
159                                              Cell fractionation showed that approximately 80% of the
160               Consistent with these results, cell fractionation showed that both the GFP-2a fusion an
161 Furthermore, immunofluorescence staining and cell fractionation showed that erlotinib treatment led t
162                                        Liver cell fractionation showed that macrophages and activated
163                                              Cell fractionation showed that Omega-3 fatty acids induc
164                                              Cell fractionation showed that the phosphorylated pool o
165                                              Cell fractionation showed that the viral preintegration
166                                              Cell fractionation shows that both EGF-Rs and cAbl are f
167                                              Cell fractionation shows that cyclins A, E, and Cdk2 are
168 on by both immunofluorescence microscopy and cell fractionation shows that the export of Pma1p from t
169 ows little or no endo H resistance, although cell fractionation still needs to be optimized for these
170                              In addition, in cell fractionation studies an estimated 25% of Mr 34000
171                                              Cell fractionation studies and confocal microscopy showe
172                                              Cell fractionation studies and green fluorescent protein
173 ce its plasma membrane localization based on cell fractionation studies and immunoelectron microscopy
174                                  Biochemical cell fractionation studies as well as confocal images of
175                                              Cell fractionation studies demonstrate that exogenous Ap
176                                              Cell fractionation studies demonstrate that PANCR is pri
177                                     Previous cell fractionation studies demonstrated that chitin synt
178                                              Cell fractionation studies demonstrated that expression
179                                              Cell fractionation studies determined that IL-12 and TNF
180              However, immunofluorescence and cell fractionation studies identified Bax activation and
181                                              Cell fractionation studies indicated that NO donors caus
182                                              Cell fractionation studies localize the 47- and 49-kDa p
183                                              Cell fractionation studies localize the three additional
184                              Furthermore, in cell fractionation studies of mitotic cells, phospho-PP-
185                                              Cell fractionation studies of primary bovine articular c
186                                              Cell fractionation studies of the t(2;5) translocation-c
187                                              Cell fractionation studies revealed that approximately 9
188                                              Cell fractionation studies revealed that cytosolic SNAP-
189                                              Cell fractionation studies revealed that KSR formed a co
190                                              Cell fractionation studies revealed that membrane-associ
191                                              Cell fractionation studies revealed that the N terminus
192 rog ECA(CYC) per milligram (dry weight), and cell fractionation studies revealed that these molecules
193                       Immunocytochemical and cell fractionation studies revealed that TM6SF2 was pres
194                                              Cell fractionation studies showed that active p35/CDK5 w
195                              Biochemical and cell fractionation studies showed that BfpB is a 58-kDa
196            Triton X-114 phase separation and cell fractionation studies showed that dominant negative
197                                     Infected-cell fractionation studies showed that insoluble fractio
198                                       Direct cell fractionation studies showed that latent virus is p
199 erinuclear and cytosolic localization, while cell fractionation studies showed that most of the p38(J
200                                              Cell fractionation studies showed that NF90 and its hete
201                                              Cell fractionation studies showed that only a small amou
202                                              Cell fractionation studies showed that SPN binds prefere
203                                              Cell fractionation studies showed that the fluorescent M
204                       Immunofluorescence and cell fractionation studies showed that the major MV memb
205                    In vivo cell labeling and cell fractionation studies showed that the majority of W
206                                              Cell fractionation studies showed that the NAE amidohydr
207                              Biochemical and cell fractionation studies suggest caveolae contain func
208 sus coimmunoprecipitated (Shc-antibody), and cell fractionation studies that suggested that the Shc.P
209                        Immunocytological and cell fractionation studies with a specific antibody reve
210                                              Cell fractionation studies with S. gordonii further corr
211 assessed by chemical cross-linking following cell fractionation studies with VP40 transfected cells.
212 surprisingly, immunofluorescence microscopy, cell fractionation studies, and studies with enhanced gr
213                Using confocal microscopy and cell fractionation studies, butyrate pretreatment of a h
214                             Data from recent cell fractionation studies, however, predominantly those
215 ivo, together with coimmunoprecipitation and cell fractionation studies, provide compelling evidence
216                                           In cell fractionation studies, the 46-kDa protein cofractio
217 udged by two-hybrid, immunofluorescence, and cell fractionation studies.
218 d supported by early electron microscopy and cell fractionation studies.
219 in antibody staining, which was confirmed by cell fractionation studies.
220  nucleoids and/or mitochondrial ribosomes in cell fractionation studies.
221 eolin-1 by immunofluorescence microscopy and cell fractionation studies; and (iii) a caveolin-3-deriv
222                                              Cell-fractionation studies showed that it is the monocyt
223                     Immunohistochemistry and cell-fractionation studies suggested that mutant beta-ca
224 itor treatments either prior to or following cell fractionation suggested the presence of a cell enve
225                                              Cell fractionation suggests that the overexpressed prote
226 to the plasma membrane as assessed by either cell fractionation, surface biotinylation, or the plasma
227                                           By cell fractionation, surface rTPO fractionated distinctly
228  fraction was substantiated by two different cell fractionation techniques.
229  microsurgery as well as isolation by common cell fractionation techniques.
230                               We now show by cell fractionation that folate receptors also must be cl
231          Herein we show, with morphology and cell fractionation, that all the components of a mitogen
232                    By immunofluorescence and cell fractionation, the exocyst subunits were found to s
233 hIP-chip), ChIP-quantitative PCR (qPCR), and cell fractionation, the stable association of RBF1 with
234                                           By cell fractionation, these signaling molecules cosediment
235 o microscopy, immunoelectron microscopy, and cell fractionation to enter transferrin-positive REs wit
236 nd immunogold electron microscopy as well as cell fractionation to identify the intracellular localiz
237                                              Cell fractionation, two-phase partitioning, and detergen
238  tagging (LOPIT), which combines biochemical cell fractionation using density gradient ultracentrifug
239                                              Cell fractionation was done to analyze this further.
240                Using confocal microscopy and cell fractionation, we demonstrated that up to 40% of en
241 g co-localization with organelle markers and cell fractionation, we determined that COMMD1 is located
242               Using ChIP-sequencing data and cell fractionation, we have compared the genomic distrib
243 ting the subcellular distribution of Chk1 by cell fractionation, we observed that around 20% of it lo
244                                        Using cell fractionation, we show that PLCgamma2-IP(3)-Ca(2+)
245                                        Also, cell fractionation/Western blot studies revealed that a
246 cytochemical findings have been supported by cell fractionation, which demonstrated that full-length
247                                   We coupled cell fractionation with stable isotope labeling with ami

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