ライフサイエンスコーパス: cell group

1 eleration of wave speed (p < 0.05; n = 10-16 cells/group).

2 of left ventricular ejection fraction in the cell group.

3 cular density in the BZ was increased in the cell group.

4 essing neurons are concentrated in the A1/C1 cell group.

5 gned" to synapses on dendrites from a target cell group.

6 ls with only two parameters that differ with cell group.

7 gray matter corresponding to the dorsomedial cell group.

8 ted within the medial half of the hypocretin cell group.

9 rresponding to the mammalian A8 dopaminergic cell group.

10 ily within the dorsal half of the hypocretin cell group.

11 e and behavioral functions subserved by this cell group.

12 egion, nucleus of the solitary tract, and C3 cell group.

13 ninergic/gamma-aminobutyric acid (GABA)ergic cell group.

14 tral ventrolateral medulla (rVLM) and the A5 cell group.

15 a major functional division within the POMC cell group.

16 axonal projections of at least some labeled cell groups.

17 t with or are influenced by the serotonergic cell groups.

18 rons in the A5, locus coeruleus (LC), and A7 cell groups.

19 ormation is funneled through these important cell groups.

20 to investigate the relation between the two cell groups.

21 in the hiMNC group than in the PBS and CD34+ cell groups.

22 r Purkinje cells as well as in several other cell groups.

23 -shaped and similar to that of I(Ca) in both cell groups.

24 thin all the midbrain dopaminergic (DAergic) cell groups.

25 vian telencephalon and some allied brainstem cell groups.

26 A cells in the locus coeruleus and A5 and A7 cell groups.

27 cluding the initial formation of cooperative cell groups.

28 ars compacta and other dopamine-synthesizing cell groups.

29 s, and in the vicinity of the A1, A2, and A5 cell groups.

30 in the hypothalamus, amygdala, and A1 and A2 cell groups.

31 lateral lemniscus and in ventral periolivary cell groups.

32 tyrosine hydroxylase-immunoreactive (TH-ir) cell groups.

33 form of compensation and competition within cell groups.

34 avian brain focusing on pallidal and nigral cell groups.

35 ters of development or rules of formation of cell groups.

36 imarily the periventricular portion), and A6 cell groups.

37 ent retinal innervation of auditory thalamic cell groups.

38 eans of projections to distinct motoneuronal cell groups.

39 and KA2 were expressed in hypophysiotrophic cell groups.

40 ing pattern of dendrite distribution of both cell groups.

41 ogenesis within both slow- and fast-specific cell groups.

42 homotypic trans interactions between the two cell groups.

43 fos expression in select 5-HTergic brainstem cell groups.

44 ruleus and adjoining A5 and A7 noradrenaline cell groups.

45 re actually in monoaminergic and cholinergic cell groups.

46 and average HER2 gene copy number per tumor cell: group 1 (in situ hybridization [ISH]-positive): HE

47 For in vivo studies, 70 muL of DMEM without cells (group 1) or containing 1.5x10(6) (Null)MSCs (grou

48 ections of 70 microL of basal medium without cells (group 1) or containing 3x10(6) nontransduced MSCs

49 croL Dulbecco modified Eagles medium without cells (group 1) or containing male 1 x 10(6) nonprecondi

50 results from 4-week studies in DMEM without cells (group 1), SMs (group-2), SiPS (group-3), and SiPS

51 rosis area on day 28 was less in the CD34(+) cell group (15.6+/-0.9%) than in the PBS, loMNC, and hiM

52 The activating receptor NK cell group 2 member D (NKG2D) mediates antitumor immunit

53 ning male 1 x 10(6) nonpreconditioned Sca-1+ cells (group 2) or preconditioned Sca-1+ (group 3) cells

54 of immune cells, including eosinophils, mast cells, group 2 innate lymphoid cells and lymphocytes, wh

55 ells were observed in both the A4 and the A5 cell groups, 2% and 0.4%, respectively, of the total.

56 tion score was better preserved in the CD34+ cell group (21.8+/-0.5) than in the PBS, loMNC, and hiMN

57 gated whether the CTL activation receptor NK cell group 2D (NKG2D) contributes to the development of

58 duced TSLP, HLA class II, and natural killer cell group 2D (NKG2D) ligand expression in the lesional

59 RMA-S-RAE-1beta, which overexpresses the NK cell group 2D (NKG2D) ligand RAE-1beta, or when inoculat

60 cible-1 (RAE-1), a ligand for natural killer cell group 2D (NKG2D) receptor.

61 day 28 was significantly higher in the CD34+ cell group (30.3+/-0.9%) than in the PBS, loMNC, and hiM

62 rived endothelial progenitor cells [2.0 x 10 cells]), group 4 (critical limb ischemia treated with ex

63 ay 28 was significantly greater in the CD34+ cell group (721.1+/-19.9 per 1 mm2) than in the PBS, loM

64 Multistratified cells (Group a/b) were common and occurred with either O

65 in the LH project near the A7 catecholamine cell group, a group of noradrenergic neurons in the pons

66 The chick OMC comprises two distinct cell groups: a dorsal Edinger-Westphal nucleus of viscer

67 in micropunches containing catecholaminergic cell groups A1 through the middle region of C1 (A1-C1m).

68 ir was reduced or abolished in catecholamine cell groups A1, A1/C1, C1, C3, and A6 and in the paraven

69 led for TH were found in the dorsocaudal A10 cell group (A10dc) located in the periaqueductal gray ar

70 AP caused a nearly complete loss of TH-ir in cell groups A5, A7, subcoeruleus, and retrofacial C1 and

71 as undetectable in the pontine noradrenergic cell groups (A5 and A6/locus coeruleus).

72 The pontine noradrenergic cell groups, A5, A6 (locus coeruleus), and A7, provide t

73 a conceptual barrier to recognizing related cell groups across its border, thereby confounding our u

74 t of the hypothalamic and hindbrain neuronal cell groups activated by restraint suggesting a possible

75 in areas contain high and low levels of FMRP cell groups adjacent to each other or between layers of

76 Using rats, we first localized cell groups afferent to the paraventricular hypothalamic

77 Most of the human tau-immunoreactive cell groups also showed tau hyperphosphorylation at the

78 ve from 9 to 18 mum/s (p < 0.0005; n = 18-22 cells/group); an increase in Ca(2+) wave speed was also

79 er in areas adjacent to the A5 noradrenergic cell group and overlapping the facial nucleus lateral su

80 The shaping of the cone cell group and packing of its components precisely imita

81 nce between the multipotent adult progenitor cell group and placebo groups in global stroke recovery

82 he relationship between the A5 noradrenergic cell group and superior salivatory nucleus, or for PRV-B

83 ce included most of the ipsilateral auditory cell groups and pathways at these levels.

84 racterized by rapid tumor growth, with small cell groups and single cells invading into the surroundi

85 demonstrated by wake-promoting monoaminergic cell groups and was previously found in cells localized

86 aseline of approximately 50% to 31.3+/-3.9% (cell group) and 33.3+/-3.1% (control).

87 rostral ventrolateral medulla (C1 adrenergic cell group), and the A5 noradrenergic cell group in the

88 ld (A8 cell group), followed by the VTA (A10 cell group), and very few fibers within substantia nigra

89 A ventral pallidum, a basal cholinergic cell group, and medial and lateral bed nuclei of the str

90 ons from PL (but not ACd) to implicated aBST cell groups, and from these to PVH.

91 These projection cell groups, and hence the higher-order visual areas to

92 ne on noradrenergic neurons of the A7 and A5 cell groups, and serotonergic neurons of the nucleus rap

93 rcuate), major cholinergic and monoaminergic cell groups, and specific sensory relay and association

94 on of the modules revealed common as well as cell group- and condition-specific pathways, GO-Terms an

95 Within this region several catecholaminergic cell groups appear to be glutamatergic, including but no

96 lopmental strategies by which axons from one cell group are "assigned" to synapses on dendrites from

97 hroughout much of the brain, whereas stained cell groups are distributed in well-defined regions.

98 their locations, 3 neuronal complexes and 17 cell groups are identified on the bases of their morphol

99 5-HT2C-IR labeling included the dorsomedial cell group as well as the dorsolateral, ventromedial and

100 ides were blindly re-reviewed and epithelial cells grouped as either benign or high-grade atypia (HGA

101 nobutyric acid (GABA)ergic and glutamatergic cell groups, as identified by the expression of glutamic

102 ory bulb), including limbic and hypothalamic cell groups associated with sex-typical behavior.

103 mplex axons, in turn, influence similar size cell groups at different VL locations.

104 When members of this cell group become highly activated, they elaborate a wid

105 activation of the dorsal medial cell column, cell group beta, or caudal medial accessory olive produc

106 2 weeks and 9 of 9 aortas at 6 weeks in the cell group but in none of the aortas in the medium group

107 Among subtypes of amacrine cells grouped by neurotransmitter phenotypes, the glycin

108 up B), or alphabeta- and gammadelta-TCR(+) T cells (group C) engrafted and had an average chimerism l

109 e zones were co-distributed with epinephrine cell groups C1-C3, suggesting that epinephrine neurons m

110 including but not limited to the adrenergic cell groups C1-C3.

111 he nucleus was seen as a diffusely organized cell group closely related to the brain stem reticular f

112 neurons as well as neurons in certain brain cell groups compared to control animals without conditio

113 to a somewhat different set of motoneuronal cell groups compared with other species fits the concept

114 rgic neurons in both dorsal and caudal raphe cell groups contain TASK channel transcripts (approximat

115 hat the mesopontine descending noradrenergic cell groups contribute to the analgesic effects of both

116 hat a specific subset of brainstem 5-HTergic cell groups contributes to the regulation of intrusion-e

117 These premotor cell groups coordinate sympathetic control with ongoing

118 that activity fluctuations in each of these cell groups correlate with choice variability, and the f

119 d from neurons in the ventrolateral VMN, the cell group crucial for estrogen induction of lordosis.

120 n the VH gene repertoire of the two memory B cell groups derived from broader usage of VH gene segmen

121 The two PYR cell groups did not differ significantly in electrophysi

122 at the BSTal and BSTsc are parts of the same cell group (dorsal and ventral to the anterior commissur

123 infusion of autologous peripheral blood stem cells; group E).

124 a terminalis (BSTalg) contains four distinct cell groups embedded within an undifferentiated anterola

125 dense immunostaining, and neurons in all AMe cell groups except the anterior neurons were labeled.

126 Core SEB-responsive cell groups exclude a medullary-PVH circuit implicated i

127 Both pontine cell groups express the transcription factor FoxP2 and b

128 major forebrain and diencephalic TH-positive cell groups expressed zDJ-1.

129 ance and magnitude of functional activity in cell groups expressing either the MOR-1 or DOR-1 genes,

130 s, we identified a large number of prominent cell groups expressing high levels of FMRP at the subcor

131 highest density in the retrorubral field (A8 cell group), followed by the VTA (A10 cell group), and v

132 t) as well as in the activation of 5-HTergic cell groups following aggressive encounter.

133 Despite the well known importance of the VTA cell group for incentive motivation functions, relations

134 ventrolateral preoptic nucleus (VLPO), a key cell group for producing behavioral sleep.

135 We suggest that this organization of cell groups forms by expansion of contiguous development

136 ue combinations of these genes mark specific cell groups from the time they are generated to their la

137 capture microdissection of specific cells or cell groups from tissues were developed to solve this pr

138 However, at week 4, the cell group had a significant recovery in ejection fracti

139 While the adrenergic C1 cell group has been extensively characterized both physi

140 However, cell-specific lesions of these cell groups have never been able to reproduce the deep c

141 trast to these other monoaminergic "REM-off" cell groups, histamine neurons are active in cataplexy a

142 The most common type of cell (group I) had relatively small somata and one to th

143 oprevented as compared with immortalized HBE cells (group I) and those augmented in chemoprevented as

144 Killing by Ag-specific cytotoxic T cells (group I) was completely inhibited by treatment wi

145 e response rate for each prespecified immune cell group (IC2/3: 27% [95% CI 19-37], p<0.0001; IC1/2/3

146 line categories: epithelial (group I), clear cell (group II) and mesenchymal (group III).

147 moprevented as compared with transformed HBE cells (group II) included known RA-target genes as well

148 killing by the broadly specific T cytotoxic cells (group II) was only partially inhibited by either

149 Cell groups immunoreactive for ChAT were observed in the

150 a set of interconnected limbic and autonomic cell groups implicated as primary sites of stress-relate

151 tivate a stereotyped set of limbic forebrain cell groups implicated in constraining stress-induced hy

152 e IP has reciprocal connections with several cell groups implicated in sleep/wakefulness regulation.

153 retin) neurons of the perifornical area, two cell groups implicated in the regulation of wakefulness.

154 M) and non-PDF evening (E) cells are coupled cell groups important for morning and evening behavior,

155 The midbrain dopaminergic cell group in birds known as the nucleus tegmenti pedunc

156 The nucleus incertus (NI) is a distinct cell group in caudoventral regions of the pontine perive

157 sly undescribed, seasonally variable AVP-lir cell group in the anterior tuberal hypothalamus, a vocal

158 nergic cell group), and the A5 noradrenergic cell group in the brainstem.

159 We identify a DA cell group in the diencephalon as a common source for in

160 otonin neurons, the densest HDAC6-expressing cell group in the mouse brain, dramatically reduced acut

161 In addition, we identified a CGRP cell group in the premamillary nuclei and showed that it

162 mesopontine tegmental cholinergic neurons, a cell group in which CHT expression has yet to be charact

163 oncentrations of labeled neurons to specific cell groups in hand VPL.

164 y in and around midbrain dopamine-containing cell groups in hormonally intact adult male and female r

165 red by adhesive interactions among different cell groups in multiple stages.

166 icity towards dopaminergic and noradrenergic cell groups in non-human primates.

167 holinergic, glutamatergic, and GABAergic PPT cell groups in regulating cortical activity and behavior

168 remotor neurons that project to motoneuronal cell groups in the brainstem and spinal cord.

169 ral pallidal subdivision as well as specific cell groups in the brainstem, including the substantia n

170 f the MLd receive inputs from three distinct cell groups in the caudodorsal brainstem.

171 medial medulla or in the A2-C2 catecholamine cell groups in the dorsal hindbrain.

172 orsolaterally in nTTD, terminate in specific cell groups in the dorsolateral nTTDo and in PrV.

173 ral nervous system, including stress-related cell groups in the hypothalamus (paraventricular and arc

174 Feeding relies on distinct cell groups in the hypothalamus, the activity of which a

175 solitary tract, magnocellular neurosecretory cell groups in the hypothalamus, ventrolateral medulla,

176 iour by dynamic reorganization of functional cell groups in the hypothalamus.

177 o-expression with markers for wake-promoting cell groups in the lateral hypothalamus (Hcrt), tuberoma

178 SEB also activated cell groups in the limbic forebrain (lateral septal n, m

179 ions of dorsal thalamic nuclei, dopaminergic cell groups in the mesencephalon and pons, the principal

180 In contrast, many cell groups in the midbrain and hypothalamus exhibit low

181 Deficiencies in neurotransmitter-specific cell groups in the midbrain result in prominent neural d

182 cataplexy suggests different roles for these cell groups in the normal regulation of environmental aw

183 Catecholaminergic cell groups in the pons (LC) and medulla (VLM, NTS) were

184 AT-1 alpha (p < 0.001) after culture of both cell groups in the presence of 100 units/ml IFN-gamma pl

185 ucleus of the trapezoid body and periolivary cell groups in the superior olivary complex.

186 ecies, originates from a number of different cell groups in the telencephalon and diencephalon.

187 We show that X-cells group in two highly divergent clades, robustly sis

188 d neurons were observed in all noradrenergic cell groups, in both the dorsolateral and the ventrolate

189 They also innervate pontine noradrenergic cell groups, including the locus coeruleus (LC) and A5.

190 A network of interconnected limbic forebrain cell groups, including the medial prefrontal cortex (mPF

191 lved in vocalization, i.e., the motoneuronal cell groups innervating soft palate, pharynx, and larynx

192 ppears to be specific to individual neuronal cell groups instead of being associated with all neurons

193 fitness tradeoffs drive the transition of a cell group into a multicellular individual through the e

194 d many other multicellular phenomena, motile cells group into a collective and migrate persistently i

195 l PRV injection to identify central neuronal cell groups involved in parasympathetic regulation of Ch

196 induction throughout the cortical mantle, in cell groups involved in sensory information processing,

197 In addition, central noradrenergic cell groups involved in splenic innervation were charact

198 e and caudal anterolateral area of the BST - cell groups involved in visceromotor responses.

199 This cell group is also heterogeneous-subsets of neurons can

200 Since this newly discovered cell group is predicted to play a role in regulating car

201 ain at the resolution of individual neuronal cell groups is not known.

202 r function are clustered together in a dense cell group known as area X that sits within the surround

203 urons in the ventral tegmental area (VTA), a cell group known to promote reinforcement and aspects of

204 urons modulate the activity of other central cell groups known to participate in the regulation of ca

205 nucleus y, the external cuneate nucleus, and cell group l.

206 This novel cell group likely serves as a relay or integration point

207 y suggest rather that the net output of this cell group may serve normally to restrain cytokine-induc

208 e restraint stress-induced activation of PVH cell groups mediating autonomic and neuroendocrine respo

209 e coronary artery, and either 50x10(6) MPCs (cell group, n=6) or saline (control, n=6) was injected i

210 d electrophysiologic (n = 5-6 animals, 21-25 cells/group), neuroanatomic (n = 6-8/group), and behavio

211 ricular nucleus (pPVN) via noradrenergic (A2 cell group) neurones in the nucleus tractus solitarii (N

212 tral paraflocculus which target a particular cell group, nodulus/ventral uvula inhibition targets a l

213 neurons, in the A5, A6, and A7 noradrenergic cell groups nor within the main cardiorespiratory center

214 tivated by glucoprivation, but unlike the C1 cell group, not by hypotension.

215 eral medulla, including the C1 catecholamine cell group; nucleus of the solitary tract; and dorsal mo

216 tern of axonal projections from one distinct cell group of the bed nuclei of the stria terminalis, th

217 ng factor (CRF) neurons in the anterolateral cell group of the bed nucleus of the stria terminalis (B

218 nomic centers including the C1 catecholamine cell group of the rostral ventrolateral medulla (RVLM) a

219 rodorsal medial amygdala (MeApd), the medial cell group of the sexually dimorphic preoptic area (medi

220 rsal preoptic nucleus (PdPN), and the medial cell group of the sexually dimorphic preoptic area (mSDA

221 Small cell groups of 50-100 cells were prepared from establish

222 enic plants have been regenerated from small cell groups of rice using a simpler, faster, and more ef

223 at during song perception, catecholaminergic cell groups of the brainstem actively participate in aud

224 jecting cells in the A1 and A2 noradrenergic cell groups of the caudal medulla were activated during

225 ) and PB external lateral-inner (PBel-inner) cell groups of the dorsolateral pons.

226 PV) and paratenial (PT) nuclei are prominent cell groups of the midline thalamus.

227 d on the present evidence, the noradrenergic cell groups of the pons (A5 and A6) do not contain eithe

228 vated most brain areas, and nearly all TH-ir cell groups of the postembryonic brain were already esta

229 trated that alpha-synuclein levels in the DA cell groups of the substantia nigra/ventral tegmental co

230 ian SCN is composed of functionally distinct cell groups, of which some are light induced and others

231 osynaptically connected either to some other cell group or, especially, to a single cell.

232 cterized by their parcellation into distinct cell groups, or nuclei, that can be histologically defin

233 brain or periphery (e.g., HVC, dopaminergic cell groups, or the syrinx) is required to enhance the q

234 ockade were intermediate between the two PYR cell groups (P < 0.05).

235 importance of parkin in maintaining specific cell groups, perhaps those with a high-energy demand and

236 ne expression differences occurred in the B9 cell group, pontomesencephalic reticular formation, medi

237 ocated in raphe pallidus (RP), parapyramidal cell group (PP), and the B3 region.

238 y associated with TASK-1 activation in these cell groups probably accounts for specific CNS effects o

239 ms of OT, but also vasopressin (VP), and >10 cell groups produce each peptide in any given species.

240 Thus, the pontine noradrenergic cell groups project in a roughly topographic and complem

241 d/or gradual loss of the identified neuronal cell group provides a neurophysiological basis for the p

242 thin catecholaminergic neurons in any of the cell groups quantified.

243 To identify motoneuronal cell groups receiving input from the NRA, the same seven

244 st signaling through a midbrain dopaminergic cell group, reminiscent of recurrent mesocortical loops

245 f primates and demonstrate that the isolated cell groups represent digits 1-5 in a mediolateral seque

246 (S)-motility (coordinated movement of large cell groups) requires both type IV pili and fibrils (ext

247 al neurochemical distribution of three major cell groups, serotonin (5-hydroxytryptamine; 5-HT), gamm

248 harmonic, involves selective hippocampal CA1 cell groups showing frequency doubling of firing periodi

249 Each Tv neuron resides within a neuronal cell group specified by the LIM-homeodomain gene apterou

250 atic conditions, and find that the different cell groups (SS2: young-deformable SS-RBCs, ISCs: rigid-

251 of neural inputs (the most varied of all BST cell groups), suggests that the BSTam is part of a stria

252 ber of remaining DMH neurons, and lesions in cell groups surrounding the DMH did not block entrainmen

253 ds, a transformation achieved by a hindbrain cell group termed the velocity-to-position neural integr

254 movements, including those of interconnected cell groups, termed collective cell movements.

255 ucleus (STN) is a glutamatergic diencephalic cell group that develops in the caudal hypothalamus and

256 The NRA is the only cell group that has direct access to the motoneurons inv

257 gnocellular part of the red nucleus (RMC), a cell group that participates in both the tonic and phasi

258 al factors that work together to establish a cell group that regulates neuroendocrine functions and b

259 The latter depends on a network of cell groups that activate the thalamus and the cerebral

260 raphe nucleus (DRN), adjoining mesencephalic cell groups that are strategically positioned to influen

261 ke and sleep emerges from the interaction of cell groups that cause arousal with other nuclei that in

262 ge of noradrenergic neurons in the A7 and A5 cell groups that contained Fos was significantly increas

263 time points, PrP(Sc) was localized to brain cell groups that directly project to the hypoglossal nuc

264 l cortex (V1) harbor morphologically diverse cell groups that have corticocortical and corticosubcort

265 duces changes in brainstem catecholaminergic cell groups that may play a neuromodulatory role in beha

266 f neuronal cell death and identify transient cell groups that may undergo wholesale elimination perin

267 eying information to downstream hypothalamic cell groups that modulate neuroendocrine function.

268 d nomenclature for the perioculomotor (pIII) cells groups that have distinctive projections and neuro

269 he nucleus raphe pallidus, the parapyramidal cell group, the raphe obscurus (RO), and the B3 region.

270 re several catecholaminergic and cholinergic cell groups, the periaqueductal gray, several brainstem

271 e-I) neurons in the pre-Botzinger complex, a cell group thought to be important in generating respira

272 thus performs coordinated social motility of cell groups through the extension and retraction of type

273 minal nerve, nucleus laminaris and scattered cell groups throughout the isthmus and pontine reticular

274 odel fits the responses of each of the three cell groups to the three different stimulus protocols wi

275 The contributions of specific peptide cell groups to these processes remain unknown, however.

276 entricular myocytes, randomized (minimum: 30 cells/group) to normothermia: (cell media for 2 hours/37

277 cells expressing OSCAR were found in the RAW cell group treated with either RANKL alone or RANKL and

278 Individual cells and small cell groups undergo constant cell rearrangements and app

279 rgence of the sequentially-timed activity of cell-groups underlying the learned behavior.

280 PLA2s were mixed with non-transfected HEK293 cells, group V and X PLA2s showed strong transcellular l

281 patients in the multipotent adult progenitor cell group vs 59 [97%] of 61 in the placebo group).

282 ptic area (DLPO) and in the A7 noradrenergic cell group were retrogradely labeled but lacked Fos expr

283 ibutions from the A1/C1 and A2/C2 adrenergic cell groups were also observed, particularly in the case

284 MT/OT-like immunoreactive (MT/OT-lir) cell groups were found in the anterior parvocellular, po

285 PACAP-containing cell groups were found to be retrogradely labeled from t

286 Small neuronal cell groups were infiltrated with a dense network of var

287 tivity compared with the C-F group, and both cell groups were positively immunostained for STRO-1.

288 l clones and hybridomas from both of these T cell groups were responsive to APC pulsed with GAD65(524

289 n the A1, C1, A2/C2, C3, or A6 catecholamine cell groups were similar in newborn and adult rats, as w

290 found in locus coeruleus/A5/A7 noradrenaline cell groups, whereas the extent of neuronal loss was low

291 merge in a wave as a patterned array of 6-10-cell groups, which are recognizable by expression of Ato

292 ated the role of brainstem catecholaminergic cells groups, which project to the forebrain, in estradi

293 ithin untreated, stressed, and drug-tolerant cell groups while generating high heterogeneity between

294 itory GABAergic and excitatory glutamatergic cell groups whose exact neurobiological roles are unclea

295 The vlBNST is a heterogeneous cell group with multiple efferent projections.

296 served at rostral and mid levels of the POMC cell group with VGLUT2-POMC neurons dominating in latera

297 Transformation of small cell groups with intact cell walls was carried out with

298 Functionally heterogeneous cell groups with sleep-related discharge patterns are lo

299 To explore the organization of both cell groups within the C group, we performed small injec

300 Septa also separated other cell groups within VPM and VPL, specifically in the medi